Session 8 Flashcards

1
Q

Why are most CXR from a PA view?

A

Accurate assessment of cardiac size and the scapula are rotated out of the way

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2
Q

Outline DR of the DR ABCDE approach for interpreting CXR’s

A

Details/Demographics/projection
RIPE - rotation (spinous process between clavicles), inspiration (5-7 ant. Ribs at mid clav. Line), penetration (vertebrae just visible through heart), everything visible that should be (1st rib, costophrenic angle)

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3
Q

Outline ABCDE of the DR ABCDE approach for interpreting CXR’s

A

Airway - check trachea central and hila (left higher than right)
Breathing - lungs, pleural spaces, lung interfaces
Circulation - aortic arch, pulmonary vessels
Diaphragm - free gas/mass
Everything else - bones (fracture/mass) and soft tissues

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4
Q

What is a pneumothorax, what is the most common cause, how does it look on a CXR and what are the clinical features?

A

Air in pleural space. Trauma most common cause. Large if >2cm from inner chest wall at level of hilum. Under tension if mediastinum pushed away and the hemidiaphragm is flattened.
CXR - visible pleural edge and loss of lung markings beyond edge.
Clinical features - tachycardia,dyspnoea, acute pleuritic chest pain, hyper resonant on percussion

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5
Q

What is a pleural effusion what are the clinical features and what is seen on a CXR?

A

Collection of fluid in pleural space. There is a uniform white area, loss of costophrenic angle, obscured hemidiaphragm and obscured upper border.
Clinical features - pleuritic chest pain, dyspnoea, stoney dullness on percussion

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6
Q

What is a lobar lung collapse and what can cause it?

A

Volume loss within lung lobe (loss of air in alveoli). Causes include bronchial obstruction (tumour, mucus, foreign body, iatrogenic) or external pulmonary compression (effusion/mass)

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7
Q

What is seen on a CXR in lobar lung collapse?

A

Elevation of hemidiaphragm, mediastinum pulled towards lung, increase in density (less air) and possible compensatory overinflation of opposite lung

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8
Q

What is consolidation?

A

Replacement of alveolar air by fluid, cells, pus or other material

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9
Q

How are space occupying lesions classified and what are their causes?

A

Nodule if <3cm, mass if >3cm. Single vs multiple.

Causes - malignancy (primary/secondary), benign mass, congenital

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10
Q

What is seen on a CXR of pulmonary oedema due to cardiac failure?

A

Perihilar shadowing and cardiac enlargement

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11
Q

What is the interstitial space?

A

A potential space between the alveolar cells and the capillary basement membrane only apparent in disease states

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12
Q

What are the clinical features of interstitial lung disease?

A

Breathlessness, reduced exercise tolerance, dry cough, tachypnoea, reduced chest movement and course crackers

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13
Q

What are the different types of interstitial lung disease?

A

Occupational - asbestos, coal workers pneumoconiosis
Treatment related - radiation, chemotherapy
Connective tissue disease - Rh. Arthritis
Immunological - sarcoidosis, extrinsic allergic alveolitis
Idiopathic - fibrosing alveolitis

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14
Q

What is fibrosing alveolitis?

A

Progressive inflammatory condition of the lungs of unknown cause. There are increased activated alveolar macrophages that attract neutrophils and eosinophils -> local lung damage. CXR shows small lungs with micro nodular shadowing, predominantly lower zone. Finger clubbing. Treatment - steroids.

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15
Q

What is extrinsic allergic alveolitis?

A

A number of conditions in which the inhalation of organic material triggers a diffuse allergic reaction in the interstitium. Can be acute or chronic.

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16
Q

What is an acute form of extrinsic allergic alveolitis?

A

Farmers lung - exposure to an antigen in mouldy hay produces influenza type illness with dry cough and breathlessness

17
Q

What is an chronic form of extrinsic allergic alveolitis?

A

Bird fanciers lung - long exposure to antigens in birds leads to malaise, dry cough and breathlessness over months/years

18
Q

What is asbestos exposure associated with?

A

Can develop long after exposure:
Benign pleural plaque
Asbestosis (alveolitis -> fibrosis)
Mesothelioma

19
Q

What are the features of sarcoidosis?

A

Affects many body systems - most commonly lungs, skin and eyes. There is a non caseating granuloma present. Fluid in the airways contains lots of inflammatory cells. It is staged I-IV from hilar adenopathy to irreversible lung fibrosis. Steroids for I to III

20
Q

Give some examples of occupational lung diseases

A

Asthma - lab worker. Rat urine
Diffuse fibrosis - demolition. Asbestos
Nodular fibrosis - miner/demolition. Coal dust/ silicon/ asbestos
Alveolitis - farmer/pigeon

21
Q

List the different types of pleural effusion

A

Blood - haemothorax
Chyle (lymph from intestines) - chylothorax
Pus - empyema
Serous fluid - simple fluid

22
Q

What can cause a transudate pleural effusion?

A

Increased hydrostatic pressure - cardiac failure
Decreased capillary oncotic pressure - hypoalbuminaemia
Increased capillary permeability - sepsis

23
Q

What can cause a exudate pleural effusion?

A

Neoplasms - mesothelioma/secondary
Infections - pneumonia/TB
Immune disease - RA, connective tissue disease
Abdominal disease - pancreatitis, sub phrenic abscess

24
Q

What can result from an unabsorbed pleural effusion?

A

Pleural fibrosis -> reduced lung volume and compliance

25
Q

What congenital wall abnormalities produce a restrictive pattern on spirometry?

A

Scoliosis/kyphosis

Rarely pectus carinatum/excavatum