Arrhythmias Flashcards

1
Q

Name the types of tachyarrhythmia.

A

Afib, SVPC, VT, VPC

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2
Q

Describe the pathogenesis of Afib

A

Atrial stretch (eg CHF, endocarditis, MVDV)

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3
Q

What ECG complexes are seen with ventricular disease?

A

Wide and bizarre QRS

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4
Q

What ECG complexes are seen with supraventricular disease?

A

Thin QRS with no P waves (superimposed on other complex)

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5
Q

What type of changes on an ECG may be seen with a lesion originating near the AV junction?

A

Negative P wave or no P wave, normal QRS complex

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6
Q

What changes would be seen on an ECG with a supraventricular tachycardia?

A

Superimposition of the P wave on the previous complex (t wave, st segment or QRS complex)

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7
Q

What drugs may be used in the treatment of Afib (minus those for primary disease)?

A

Dignoxin (negative chronotrope, vagomimetic - slows av conduction)
Diltiazem (negative inotrope)
Beta-blockers (negative inotrope)

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8
Q

In what situation would you NOT use beta blockers?

A

In cases of uncontrolled CHF

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9
Q

What changes on ECG may be seen with sinus arrhythmia?

A

Alternating periods of fast and slow rhythm (respiration), +/- irregular p wave confirguration

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10
Q

What is a sinus arrhythmia?

A

Irregular sinus rhythm originating in the SAN

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11
Q

What physiological factors may lead to increased vagal tone?

A

Vomiting, intubation, brachycephalic, elevated ICP, hypothermia, hypothyroidism

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12
Q

What events may lead to sinus tachycardia?

A

Stress, exercise, pain

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13
Q

What causes an SVPC?

A

Ectopic nodal tissue in the atria (other than SAN)

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14
Q

What is wolff-parkinson-white syndrome?

A

Accessory nervous pathway creation leading to supraventricular tachycardia

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15
Q

What treatment may be involved in an emergency SVT?

A

Vagal manoeuvres! Ca+ channel blockers - Diltiazem, Vermapil. Negative chronotropes - Digoxin. Beta-blockers - Esmolol

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16
Q

Why do junctional premature complexes generate negative P waves?

A

Retrograde spread of electrical signal to the atria

17
Q

What does a fusion complex suggest?

A

Paroxysmal ventricular tachycardia (none constant VT)

18
Q

Define ventricular tachycardia.

A

3 or more VPCs in a row

19
Q

What underlying causes lead to ventricular ectopic rhythms?

A

Catecholamine release, CHF, myocardial ischemia, acidosis, hypokalaemia, abdominal disease, thoracic trauma

20
Q

What is the treatment of choice for ventricular tachycardia?

A

IV Lidocaine. If persistent use beta blockers such as propranolol, amiodarone, soltalol etc

21
Q

Describe the emergency treatment of ventricular tachycardia.

A

IV access and lidocaine bolus, CRI amiodarone if Lido not affective. Within 24 hours oral mexiltine, propranolol, soltalol and amiodarone

22
Q

MOA amiodarone

A

Potassium channel blockers

23
Q

What mineral imbalance may lead to atrial standstill?

A

Hyperkalaemia

24
Q

Describe the changes associated with atrial standstill seen on ECG.

A

Absent P waves, spikey T waves, mildly prolonged QRS

25
Q

Describe the pathogenesis of atrial standstill

A

Continued SAN conduction but inability of the atrial myocardium to contract

26
Q

What causes an escape rhythm?

A

Slowing down or stopping of conduction through the SAN

27
Q

What is sinus sick syndrome?

A

SAN pathology leading to long pauses between QRS complexes, junctional escape complexes and atrial fibrillation (AV junction disease)

28
Q

What treatment is advised with sinus sick syndrome?

A

Pacemaker

29
Q

Name four examples of bradyarrhythmias.

A

Sinus brady, sinus arrest, sinoventricular rhythm, AV block

30
Q

Describe 1st degree AV block

A

Prolonged p-r interval

31
Q

Describe 2nd degree AV block

A

P without a QRS every few complexes (mobitz 1) or P without a QRS every other (mobitz 2)

32
Q

Describe 3rd degree AV block

A

No communication between P waves and QRS complex

33
Q

What does an atropine test of a bradyarrhythmia suggest?

A

That the murmur is vagally mediated.