6/7-ECGs Flashcards

(40 cards)

1
Q

What is the normal conduction pattern in the heart?

A
  • electrical activity starts at Sino Atrial Node
  • impulse spreads across atria
  • impulse delayed at atrio ventricular node to allow complete atrial contraction
  • conduction spreads to bundle of His-right/left bundle branch-purkinje fibres to stimulate myocardial contraction
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2
Q

Name the colours and locations of the limb leads

A

Right upper limb lead - red - right shoulder
Left upper limb lead - yellow - left shoulder
Right lower limb lead - black - right ankle
Left lower limb lead - green - left ankle

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3
Q

Name the colours and locations of the chest leads

A

V1 - red - 4th right intercostal space
V2 - yellow - 4th left intercostal space
V3 - green - 5th left intercostal space (closest to sternum)
V4 - blue - 5th left intercostal space
V5 - orange - 5th left intercostal space
V6 - purple - 5th left intercostal space (closest to mid clavicular line)

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4
Q

Where can the QRS complex normally be seen?

A

aVL, I, II, aVF

Which is between -30 and +90 degrees

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5
Q

What is left axis deviation?

A

Where the QRS complex is present at less than -30 degrees

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6
Q

What causes left axis deviation?

A

Inferior wall MI
LV hypertrophy
Left anterior bundle block

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7
Q

What is right axis deviation?

A

Where the QRS complex is found above 90 degrees

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8
Q

What causes right axis deviation?

A

RV hypertrophy
Acute right heart strain (e.g. Pulmonary embolism)
Left posterior bundle block

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9
Q

What is the standard recording speed of ECGs?

A

25mm per second

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10
Q

How many seconds are equivalent to big and small squares?

A

Big square = 0.2 seconds

Small square = 0.04 seconds

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11
Q

Where is the P-R interval measured from and what is the normal range?

A

Measured from start of p to start of q

Normally 3-5 small squares

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12
Q

What does a long P-R interval indicate?

A

Indicates a slow atria ventricular conduction, 1st degree heart block

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13
Q

Where is QRS width measured from and what is the normal range?

A

Measured from start of q to end of s

Normally 2-3 small squares

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14
Q

What does a wide QRS complex indicate?

A

Indicates abnormal conduction for ventricular depolarisation

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15
Q

Where is Q-T interval measured from and what is the normal range?

A

Measured from start of q to end of t

Normally 0.35-0.43 seconds

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16
Q

What does a prolonged Q-T interval indicate?

A

Indicates prolonged repolarisation of ventricles leading to arrhythmias e.g. prolonged QT syndrome

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17
Q

Where is S-T segment measured from?

A

End of s to start of t

18
Q

What does a raised or depressed S-T segment indicate?

A

Should be equal to baseline (isoelectric), raised/depressed indicates ischaemia/MI

19
Q

What is the equation to calculate a regular heart rate?

A

No of small squares per minute / no of small squares between peeks
=1500/x

20
Q

What is needed to be classed as sinus rhythm?

A

Identical round P waves
Followed by QRS complex every time
Regular rhythm

21
Q

Why are the depolarisation/repolarisation waves orientated in the same direction?

A

Repolarisation occurs in opposite direction so usually inverted deflections orientated in same direction

22
Q

What differences are there between the depolarisation and repolarisation waves?

A

Repolarisation waves are more prolonged and lower amplitude

23
Q

What are the features of atrial fibrillation?

A

Wavy baseline
No p waves
Irregularly irregular rhythm

24
Q

What causes atrial fibrillation?

A
Hypoxia 
Hypertension
Sepsis 
Alcohol 
Ischaemic heart disease
25
What are the consequences of untreated atrial fibrillation?
Clot leading to stroke | Rapid ventricular rate leading to hypertension/angina/ heart failure
26
How is atrial fibrillation treated?
Rate control - digoxin/ beta blockers Anti-arrhythmics- amiodrone Anti-thrombotics - warfarin/ dabigatron
27
What causes ventricular fibrillation?
Ischaemic heart disease Hypoxia Post MI Prolonged QT arrhythmia
28
How is ventricular fibrillation treated?
Implanted cardiac defibrillator | Anti-arrhythmics - amiodorone
29
Describe the features of first degree heart block
Prolonged P-R interval greater than 0.2 seconds | Sinus rhythm
30
Describe the features of 2nd degree heart block, mobitz type 1
Progressive p-r elongation until QRS is not conducted
31
Describe the features of 2nd degree heart block, mobitz type 2
Fixed PR interval until sudden dropped QRS complex
32
Which mobitz type has a high risk of developing into third degree heart block?
Mobitz type 2
33
Describe the features of 3rd degree heart block
No relationship between atrial and ventricular activity Wide QRS complex (from escape rhythm) Bradycardic 30-40 bpm
34
How is heart block treated?
Anticholinergics - atropine Beta agonist - isoprenaline Pacemaker if severe
35
What causes heart block?
``` MI Rheumatic fever Calcium channel blockers Beta blockers Sarcoid ```
36
Describe the features of ventricular ectopic beats
Wide, abnormally shaped QRS complexes as impulse is spread slower by abnormal conduction system to ventricles
37
Describe the stages of a STEMI on an ECG over several weeks
Acute - ST elevation in leads facing injured area Hours later - ST elevation, depressed R wave, Q wave begins 1/2 days later - t wave inversion p, Q wave deepens Days later - ST normalises, T wave still inverted Weeks later - T wave normalises, Q wave persists
38
What effect does hyperkalaemia have on ECGs?
RMP less negative, so voltage gated Na channels inactivated making heart less excitable, causing conduction issues leading to ventricular fibrillation
39
What effect does hypokalaemia have on ECGs?
RMP more negative, leading to Low t waves High u wave (after t wave) Low ST segment
40
What are the differences between atrial and ventricular fibrillation?
Atrial has coordinated ventricular contraction, ventricular doesn't Atrial has cardiac output, ventricular doesn't Atrial has a pulse, ventricular doesn't Atrial has an irregularly irregular heart rate, ventricular has no heart beat