6. Periodontal disease and conditions Flashcards
(46 cards)
When does plaque induced gingivitis occur?
During puberty
What are two conditions that commonly result in plaque induced gingival enlargement?
mouth breathers
active ortho treatment
What drugs commonly cause gingival enlargement?
Anti-seizure (Dilantin)
Cyclosporine (immunosuppressant)
Calcium Channel Blockers (diltiazem, nifedipine, amoldipine)
Does drug induced gingival enlargement occur over edentulous areas?
No
Where are gingival abscesses found?
marginal gingiva or interdental papilla
Where is it most common to see pericornitis?
3rd molars
What are the signs and symptoms associated with Vitamin C deficiency gingivitis?
edematous, spongy non-specific gingiva
spontaneous bleeding
delayed wound healing
What is the primary bacteria in chronic periodontitis?
P. gingivalis
What are the signs and symptoms of ANUG?
rapid painful onset of inter proximal and marginal necrosis and ulceration
At what age is ANUG typically seen?
late teens/early 20s in the US
younger in less developed
T/F: Patients with diabetes are at increased risk of earlier onset of periodontitis even if they have similar plaque/calculus levels as healthy controls
True
Where is localized aggressive periodontitis in the primary dentition most commonly seen
primary molars
What is the etiology of prepubertal periodontitis?
A.a.
Leukocyte chemotaxis defect
Cementum defect
What is definition of localized aggressive periodontitis?
bone loss around incisors, first molar and no more than 2 other teeth
pt otherwise systemically healthy, age of onset 10-15
What is etiology of localized aggressive periodontitis?
A.a
neutrophil chemotaxis and phagocytosis
over reactive monocyte response
genetic defect in gene encoding IgG2
What is the dental sequale of hypophosphatasia?
early loss of primary teeth due to abnormal cementum formation
Exfoliated with intact roots, before complete root formation, in order of eruption
permanet teeth may be unaffected
What are the 5 levels of severity of hypophosphatasia?
perinatal (lethal) infantile childhood adult odontohypophosphatasia
What is the etiology of hypophosphatasia
defect or deficient in tissue nonspecific alkaline phosphatase (TNSALP)
How do you diagnose hypophosphatasia
low AKP increased phosphoethanolamine (urine) or phosphate (blood)
What is the enzyme therapy for treatment of hypophosphatasia
Asfotase Alpha
What is the definition of mucogingival defect
pocket depth > width of attached keratinized gingiva (KG
Where is it most common to have a mucogingival defect
lower incisors due to labial position
How do you calculate attached keratinized gingiva
Attached KG = MGJ to FGM (note sites < 1 mm)
What is pseudo-recession?
recession like appearance without root exposure