Lecture 7 Flashcards

1
Q

What is the mechanism of action behind specific positioning and postural drainage?

A
  • gravity opens airspaces by passively stretching the lung
  • increases stretch = increased surfactant production - reduced surface tension
  • increased compliance of lung portion that was placed upper most - i.e reversed atelectasis
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2
Q

What factors are CI/P’s to HDT?

A
  • MAP < 6-65
  • BP variability
  • arrythmias
  • tachy/bradycardia
  • hypovolaemia
  • inotrope requirement
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3
Q

What’s more effective for sputum clearance - side lying + MHI or HDT + MHI in CV stable pts?

A

HDT+MHI

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4
Q

What are CIs/P’s to positioning?

A
Spinal Cord injury
Skeletal traction
Acute brain injury (i.e change in ICP)
Craniotomy
Rib #
UWSD (can turn pt onto chest drain side - but monitor to see the drain amounts not affected)
Cardiac disease (arrhythmias)
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5
Q

Define manual hyperinflation and what do Berney et al (2004) recommend as a dosage for MHI/VHI

A

The delivery of a larger than normal tidal breath (1.5-4x normal Vt) with an anesthetic or resuscitation circuit

6 sets of 6 breaths

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6
Q

What are the different types of MHI circuits?

A

Laerdal circuit - 1.6L capactiy; resuscitation circuit; works with or without oxygen

  • Mapleson C - more secretions cleared with this circuit
  • Mapleson F - this is what we use - 2L capacity
  • PEFR greater with mapleson circuits
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7
Q

What is the rationale for gas/secretion movement in MHI?

A

Deep breath = increased volume - surfactant production- reduced surface tension and better compliance

Slow inspiration = reduces effect of airway resistance on ventilation distribution

Inspiratory hold = collateral ventilation

Rapid release for expiration = aids secretion clearance - but expiratory flow has to exceed inspiratory flow by 10% (velocity 1000 cm/sec)

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8
Q

What are the benefits of MHI?

A
  • pulmonary compliance (deep breath)
  • arterial oxygenation (hold)
  • clearance of airway secretions (fast expiratory flow)
  • prevents/treats atelectasis (deep breath/hold)
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9
Q

What is the benefit of VHI over MHI for decruitment?

A

Since VHI doesn’t require disconnection from the ventilator - PEEP can be maintained and this avoids potential de-recuitment of alveoli

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10
Q

What parameters can you monitor during MHI and WHEN do you monitor?

A
  • before, during, after
  • SpO2
  • HR/BP/MAP/ECG
  • Airway pressures (<40 for MHI; <20kpa for suction)
  • EtCO2
  • Ausc pre/post
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11
Q

MHI - what are the precautions and modifications for an unstable respiratory system?

A

Precautions:

  • PEEP > 10cmH2O
  • FiO2 > 0.6

Mods:
- Use PEEP valve

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12
Q

MHI - what are the precautions and modifications for an unstable cardiovascular system?

A

Precautions:

  • hypotension
  • brady/tachycardia

Mods:

  • smaller volume - slowly increasing size of breath
  • intersperse big/small breaths
  • encourage spontaneous effort
  • no hold
  • ensure complete expiration - no PEEP

all this reduces the positve pressure on the venous channels with improves venous return to the heart

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13
Q

MHI - what modifications would you make for:

  1. Stiff lungs (ARDS eg)
  2. Hyperinflated pts
  3. Raised ICP
A
  1. Slow inspiratory flow rate/pressure manometer
  2. Ensure complete expiration (no PEEP)
  3. Pressure manometer - keep <40 cmH20
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14
Q

When do you know to suction a patient?

A
  1. Visible, audible, palpable secretions
  2. Respiratory signs:
    - desat, increasing PIP, increased WOB, increased RR, decreasing Vt, coarse crackles
  3. Cardiovascular
    - increased HR/BP
  4. Individual
    - sweaty (diaphoretic), restless, agitated
  5. Ventilator graphics
    - saw tooth pattern
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15
Q

What does a normal ventilator graphic look like? What does a saw tooth pattern indicate?

A

Normal graphic - triangular in shape, 80% expired in 1 second

Saw tooth pattern indicated condensate build up or loose secretion build up

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16
Q

What are respiratory hazards of suctioning?

A
  1. Hypoxia
  2. Bronchospasm
  3. Damage to trachea/bronchial mucosa
  4. Atelectasis
  5. Reduction in lung compliance/FRC
17
Q

What are cardiac hazards of suctioning?

A

Increased and decreased BP

18
Q

What are neuro hazards of suctioning?

A

Increased ICP

Changes in cerebral blood flow

19
Q

Saline is not recommended for routine use prior to suctioning. What factors can you optimise prior to using saline?

A

HHMM

  1. Humidification
  2. Hydration
  3. Mobilisation
  4. Mucolytics
20
Q

What are adverse effects of hyperinflation?

A
  1. Increase/decrease:
  2. MAP
  3. Q
  4. PAP
  5. Paw
21
Q

When is above the cuff suction used and what are it’s benefits?

A

When ventilated > 72 hrs

Benefits:

  • reduced ventilatory assoc pneumonia (VAP)
  • reduced ICU LOS
  • reduced MV
22
Q

What is ICU acquired weakness?

A
  • seen in 25-60% of MV pts who are ventilated > 1 week
  • diffuse symmetric generalized mm weakness
  • unclear etiology
23
Q

What are the benefits of early mobilisation?

A
  • peripheral mm strength
  • resp mm strength
  • physical fxn indices
  • HRQOL
  • LOS in ICU