Acute Myocardial Infarction Flashcards

1
Q

What is chronic stable angina?

A

When there is fexied stenosis of the coronary artery whcih leads to demand led ischaemia.

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2
Q

Angina can radiate through to arms and up to jaw. True or False?

A

True, it has the possibility of radiation.

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3
Q

What is acute coronary syndome and what is included in it?

A
Any acute presentation of coronary artery disease.
It includes:
Unstable angina
Acute NSTEMI
STEMI
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4
Q

Acute MIs can come in 2 forms, what are they?

A

ST elevation MI

Non ST elevation MI.

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5
Q

What are the causes for acute coronary syndromes?

A

They are on a spectrum from the build up of an atherosclerotic plaque. When the plaque ruptures, a thrombus can form and at this moment this is when acute coornary syndomes can occur.

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6
Q

Acute coronary syndromes are unstable and safe. True or False?

A

Partially true, they are unstable, HOWEVER they are dangerous, due to the unpredictability.

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7
Q

What is the main cause of occlusion of an artery to cause acute coronary syndrome?

A

The plaque rupturing and a thrombus forming due to the coagulation cascade.

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8
Q

How would an MI present?

A

Severe crushing central chest pain
Radiation to jaw and arms (particularly left)
Similar to angina, more severe and not relieved by GTN.
Associated with sweating, nausea and vomiting.

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9
Q

What the main differences between angina and an MI?

A

Angina is a much shorter period of time and occurs on exertion not at rest like an MI.

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10
Q

How would you diagnose an MI and what would you look for?

A

ECG:
ST elevation
T wave inversion
Q waves.

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11
Q

What classifies as a STEMI?

A

> /=1mm ST elevation in 2 adjacent limb leads
/=2mm ST elevation in at least 2 continuous precordial leads
New onset bundle branch block.

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12
Q

What are the evolving ECG changes of an acute MI during and after an MI?

A

First few hrs- ST elevation
First day- Q wave formation and T wave inversion
‘Old’ MI- Q waves and inverted T waves.

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13
Q

What is the anatomical site of an MI is there is ST elevation in leads II, III and aVF?

A

Inferior MI

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14
Q

What is the anatomical site of an MI is there is ST elevation in leads V1-V4

A

Anteroseptal.

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15
Q

What is left bundle branch block?

A

QRS is much broader

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16
Q

What test can be done to diagnose an MI but what has to be considered when using this test?

A

Troponin levels I and T as they are specific to cardiac muscle and C can be found in skeletal muscle.
The test is very sensitive and a diagnosis should not be made solely on a troponin test (“Troponinitis”).

17
Q

What is the mode of early treatment for an STEMI?

A
Analgesia
Anti-emetic 
Aspirin + clopidogrel/ticagrelor
GTN if high BP
Oxygen if hypoxic
Primary angioplasty
Thrombolysis if angioplasty not available.
18
Q

Why and when is thrombolysis used in MIs and what are the risks?

A

If the clot still hasnt been destroyed thrombolysis can be administered which clears the artery.
If the patient cannot recieve an angioplasty in 2hrs then thrombolysis is given.
However has a major risk of haemorrhage and failure to re-perfuse.

19
Q

What are complications nof acute MIs?

A
Death
Arrhythmic complications
-VF
Structual complications
-Cardiac rupture
-Ventricular septal defect
-Mitral valve regurge.
-LV aneuryms
-Inflammation
-Acute pericarditis
Functional complications
-Acute V failure
-Chronic cardiac failure
-Cardiogenic shock.
20
Q

What is the Killip Classification and what is it used for?

A

It is used to determine in-hospital mortality
I - No signs of heart failure (6%)
II - Crepitations <50% lung fields (17%)
III - Crepitations >50% lung fields (38%)
IV - Cardiogenic shock (81%)

21
Q

What is the most important thing to remember about ECG in acute non ST elevation MI?

A

The ECG may appear normal.

22
Q

What is troponin testing?

A

Embolisation
Microvascular circulationa
Myonecrosis

23
Q

What can TnT also be elevated in?

A
CCF
Renal Failure
PE
Sepsis
Stroke/TIA
Pericarditis/myocarditis
Post arrhythmia
24
Q

What is a class II MI?

A

When there is an MI that occurs secondary to ischaemia due to inbalance of O2 with demand from coronary spasm, embolism, anemia, hyper/hypotension, arrhythmias.

25
Q

What is MINOCA?

A

Myocardial Infarction with Non Obstructed Coronary Arteries.

26
Q

ECG of NSTEMI can have T wave depressions. True or False

A

True.

27
Q

Once a patient has been treated post MI, as a doctor what should you ensure?

A

That the patient realises their life isn’t going to be majorly hindered.
That they understand their drug therapy and what to expect.

28
Q

What are the 4 pahses of cardiac rehabilitation?

A

Phase 1- In patient
Phase 2- Early post discharge
Phase 3- structured exercise programme
Phase 4- Longer term maintenance of physical activity and lifestyle change.

29
Q

Who are the cardiac rehabilitation team?

A
Cardiac rehab nurses
Physiotherapy exercise classes
Smoking cessation specialists
Pharmacists
Cardiologists.