10 - Arrhythmias P. 1 Flashcards

1
Q

Native latent pacemakers

A

Sinoatrial node - 60-100 bpm
Atrioventricular junction - 40-60 bpm
Purkinje system - 20-40 bpm

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2
Q

Action potential phases

A

Phase 0 - resting state (-80mV)
—cell repol —
Phase 1 - na channels close = NA flux ceases
Phase 2 - k channel opens Ca++ flux continues - plateau phase
Phase 3 - Ca++ closes, K+ fluc out continues
—gradual and spontaneous depol—
Phase 4 - K+ channel closed, active transport of Na+ and K+ return to resting state concentration

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3
Q

Arrhythmias are generated by?

A
  1. Altered impulse formation

2. Altered impulse conduction

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4
Q

Altered impulse formation examples

A
  • altered normal automaticity
  • increased sinus node automaticity (sympathetic)
  • increased sinus node automaticity (parasympathetic)
  • escape rhythms
  • ectopic beats & rhythms
  • abnormal automaticity
  • triggered activity
  • early after depol
  • delayed after depol
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5
Q

Altered impulse conduction examples

A
  • Conduction block
  • transient vs permanent
  • unidirectional/bidirectional
  • functional/fixed
  • reentry
  • primarily unidirectional
  • includes accessory pathways (WPW)
  • multidirectional (unstable pathway_
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6
Q

Supraventricular arrhythmias

A
PAC
Ectopic Atrial Rhythm
MAT
Atrial Flutter
Atrial Fibrillation
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7
Q

Junctional arrhythmias

A
(Nodal)
PJC
Junctional Rhythm
AV node re-entry tachy (AVNRT)
AV re-entry tachycardia (AVRT)
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8
Q

Ventricular arrhythmia

A
PVC
Idioventricular rhythms
V tach
Polymorphic ventricular tachycardia (torsades)
V Fib
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9
Q

If the monitor looks terrible but the pt looks fine?

A

Treat the pt no the monitor

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10
Q

Assess stability of pt

A
HOTN
Alter mental
Shock
Ischemic chest discomfort
Acute HF

Follow ACLS alogrithms

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11
Q

Tachycardia ACLS protocol

A

O2, EKG
Assess stability
Unstable - cardiovert
Stable - fix causes

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12
Q

Bradycardia ACLS protocol

A

O2 ekg
Assess stability

Stable - look for the why
Unstable - ATROPINE, dopamine, EPI

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13
Q

Shock, shock, everybody shock, & another shock

A

V fib pulseless V tac ACLS

Shock 
Shock 
Epi
Shock 
Amnioterone 
Shock
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14
Q

Reversable causes of MI

H’s and T’s

A
Hypovelemia
Hypoxia
Hydrogen ion (acidosis)
Hypo-hyperkalemia
Hypothermia
Tension pneumothorax
Tamponade, cardiac
Toxins
Thrombosis, pulmonary
Thrombosis, Coronary
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15
Q

Cardiac arrest drugs

A

Epinephrine - 1mg q 3-5 min
Amiodarone -
- 1st dose = 300 mg bolus
- 2nd dose = 150 mg

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16
Q

Cardiac arrest shock therapy

A

1st
- Biphasic 120-200J
2nd and subsequent
- Monophasic 360J

17
Q

Approach for stable arrhythmias

A
ID arrhythmia
Mechanism 
Consequences
Treatment (best for this one)
Patient HX 
PE
Diagnostics
18
Q

Stable arrhythmias diagnostic tests

A

Lab - CBC, TSH, Electroyltyes, Drugs, HCG
Electrical - ECG, Holter/event/implantable
CXR
TTE
Treadmill
Tilt-table
Electrophysiologic testing

19
Q

Implantable pacemaker indications

A

Heart block
Sinus node dysfunction
Syncope
Dilated cardiomyopathy

20
Q

Cardioverter-defibrillator indications

A

Non sustained VT
Spontaneous VT
Cardiac arrest survivors
Syncope of undetermined etiology (when pharm fails)

21
Q

Types of pacemakers

A
Temporary
-Transcutaneous
-Transvenous
Permanent
-ventricular
-dual chamber
-biventricular

ICD/Cardioverter

22
Q

Temp pacemakers

A

Reversible bradycardia
Acute rheumatic fever
Pending surgery

*dont leave the hospital

23
Q

Permanent pacemaker types

A

Ventricular

  • single chamber
  • dual chamber
  • atria and ventricle

Biventricular
-both ventricles

24
Q

AICD

A

Automated implantable cardioverter defribillator

Pts at risk for SCD

  • CAD
  • depressed LV function
  • runs of VT
  • LVEF <30%
  • long QT, brugada syndrome
25
Q

Pacemaker spike before QRS

A

Ventricular pacing

26
Q

Pacemaker spike before p and before QRS

A

Dual chamber

27
Q

Pacemaker spike in 2,3,AVF?

A

L ventricle

28
Q

Pacemaker spike before p

A

Atrial pacing

29
Q

2 pacemaker spikes before QRS, none before p

A

Dual chamber pace