56: Digital Deformities - Yoho Flashcards

1
Q

define hammertoe

A

A deformity in which the proximal phalanx of one of the smaller toes is bent upward or is dorsiflexed at the metatarsal-phalangeal joint, and the middle and distal phalanges are sharply bent downward, so that they form a more acute angle with the proximal phalanx

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

most common etiology of digital deformities

A

biomechanical dysfunction

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

cavus foot and toe deformity

A

need to rule out a neuromuscular condition

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

anatomy review

A
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

Stabilization of the lesser toes against the ground at the MTPJ’s provided mainly by …

A

FDL and FDB

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

extensor sling vs. extensor wing

A
  • Dorsiflexion at the lesser MTPJ’s is accomplished by the EDL through the extensor sling which supports the proximal aspect of the proximal phalanx.
  • Dorsiflexion of the middle and distal phalanges is mediated through the extensor wing controlled primarily by the lumbricals.
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

what holds the extensor tendons in place

A
  • Held in place by 2 fibrous sheaths: Extensor hood
  • Proximal part: Extensor sling: fibers encircle and attach to the plantar plate
  • Interosseous muscles contribute to the sling
  • Distal part: Extensor wing: Formed by expansions of the lumbricals
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

long and short flexors are _____ phase muscles

A

stance

plantarflex and stabilize digits during stance phase of gait

flexors –> Plantarflexory force to the MTPJ (non-weight-bearing), but dorsiflex MTPJ in weight-bearing.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

lumbricales are a _____ phase muscle

interossei are a _______ phase muscle

A

swing

stance

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

anatomy review

A
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

how many interossei are there? what do they do?

A

seven

4 dorsal, 3 plantar

  • Pass above the deep transverse metatarsal ligament but below the transverse axis of the MTPJ and insert into the base of the proximal phalanx.
  • Stabilize in the transverse plane and plantarflex the MTPJ.
  • Balances the reverse buckling effect of the flexors during stance phase of gait.
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

how many lumbricals? what do they do?

A

four muscles arising from FDL tendons

  • Course beneath the deep transverse intermetatarsal ligament.
  • Insert medially into the base of the proximal phalanx and extensor hood
  • Plantarflex and adduct the MTPJs and dorsiflex the IPJs
  • Balance the effect of the long extensors and passive pull of the long flexors during swing phase of gait.
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

static pathologic anatomy assoc with biomechanics

A

associated with HAV

confining shoegear

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

3 dynamic pathologic anatomy digital deformities

A

flexor stabilization

flexor substitution

extensor substitution

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

describe flexor stabilization etiology

A
  • pronated foot
  • late stance phase of gait
  • FDL & FDB have gained mechanical advantage over the interossei muscles
  • Often a adducto-varus rotation of the 5th digit: due to weakness of the quadratus plantae
  • _Most common type*_
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

how does flexor stabilization dynamic deformity lead to toe deformity?

A
  • Flexibile pes valgus deformity with excessive STJ pronation
  • Causes an unlocked/hypermobile MTJ: forefoot hypermobility

•Flexors fire earlier and longer to try to stabilize osseous structures (overpower the interosseous m)

•Ineffective at controlling the forefoot structures

17
Q

how is quadratus plantae weakness associated with flexor stabilization etiology?

A
  • Excessive STJ pronation
  • Forefoot abduction
  • Changes the lateral vector force of the QP
  • More medial pull by the FDL creates a adducto varus rotation of the 5th digit (maybe 4th as well)
18
Q

flexor stabilization etiology and peripheral neuropathy

A

–> loss of intrinsic power

19
Q

describe flexor substitution dynamic etiology toe deformity

A
  • Supinated foot
  • Late stance phase of gait
  • Flexors have gained advantage over the interossei
  • Straight contracture of all lesser toes
  • Due to weak triceps surae
  • Deep posterior + lateral leg muscles try to substitute
  • Least common type
20
Q

how does the deep posterior muscle group contribute to flexor substitution dynamic etiology?

A
  • Tibialis posterior: primary decelerator of pronation & supinator of the rearfoot
  • Peroneus longus: plantarflexes 1st ray and causes STJ supination
  • FHL & FDL also have axis of supination around the STJ
  • Due to a weak tricep surae
  • These muscles fire earlier and longer
  • The deep flexors attempt to substitute for the weak triceps surae will not create heel lift
  • Flexors overpower the interossei and cause hammertoe deformities
  • High arched, late stance supinated foot with contracture of the toes results
21
Q

describe extensor substitution dynamic etiology toe deformity

A
  • EDL gained advantage over the lumbricals
  • Severe dorsal contracture at the lesser MTPJ is straight usually greater than 20 – 40 degrees
  • Severe dorsal contraction seen during propulsion, swing and heel contact
  • Excessive contracture at the MTPJ is also seen: Bowstringing
22
Q

bowstringing

A

excessive contracture at MTPJ

seen with extensor substitution etiology

23
Q

why have extensor substitution?

A
  • Anterior pes cavus
  • Ankle equinus
  • Weakness of the lumbricals
  • Biomechanical
  • Neurologic
  • Combination
24
Q

which etiology includes the hallux?

A

extensor substitution

25
Q

development timeline extensor substitution

A
  • Usually begins as a flexible deformity
  • Which may reduce completely during weightbearing
  • Deformity become more rigid as contractures develop
26
Q

what is an anterior cavus and how does it develop hammertoes?

A
  • Increase declination of the forefoot
  • EDL has longer to travel: tendon does not lengthen but a passive pull is then place on the digits at the joints
  • Increased retrograde force placed on the metatarsal heads increasing the anterior cavus deformity: vicious cycle
27
Q

how would ankle equinus contribute to hammertoes?

A
  • TA, EDL, EHL fire earlier and longer to attempt dorsiflexion at the ankle to clear the foot
  • Ineffective
  • EDL then gains advantage over lumbricals
28
Q

what is the role of the EDL in extensor substitution etiology hammertoes?

A
  • In order to dorsiflex the ankle: must move the MTPJ’s to their EROM before able to maximumly effect the ankle.
  • Loss of the lumbricals in neuromuscular conditions-excessive dorsiflexion will occur.
  • Severe digital deformity and subluxation.
  • Hammered hallux.
29
Q

hammer vs. claw vs. mallet toe

A
30
Q

what is a mallet toe?

A
  • Sagittal plane deformity
  • Distal phalanx is flexed on the middle
  • PIPJ and MTPJ not usually involved
  • Associated with a long digit
  • Presents as a flexion contracture at the DIPJ
  • Callus at distal tip
  • Limited extension at DIPJ
  • May or may not be reducible
  • Often seen with a long digit
31
Q

what is a clawtoe?

A
  • Middle & distal phalanges are flexed on a dorsiflexed proximal phalanx
  • Associated with cavus foot and neuromuscular disorders
  • Dorsal contracture of the MTPJ and plantar contracture of the interphalangeal joints
  • Plantar protrusion of the MTPJ is seen
  • Mechanical hyperkeratosis
  • Metatarsalgia
  • Flexible or rigid