Brainscape's Knowledge GenomeTM

Browse over 1 million classes created by top students, professors, publishers, and experts.


See full index

Yr1 - S2 - Formatives > Yet more cardio class stuff lol > Flashcards

Yet more cardio class stuff lol Flashcards

1
Q

The QT interval is too long if it is more than

A

0.42 seconds

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

preload

A

initial stretching of cardiomyocytes before contraction

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

CO =

A

HR x SV

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

after load

A

load against which the muscles tries to contract

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

increase in TPR then an increase in aortic pressure leads to ventricle having to work harder to open aortic valve so less energy so SV

A

decreases

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

Vessels/ Veins (capacitance vessels) effect

A

preload

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

resistance vessels (arterioles) effect

A

afterload

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

the fast depolarising stage of cardiac AP is due to

A

an increase in Na+

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

resting membrane potential is set buy

A

leaky potassium channels

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

depolarisation is due to the opening of

A

K+ leaky channels

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

plateau phase is due to

A

calcium

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

contractility is the effect of the sympathetic system

A

releasing noradrenaline which increase calcium available for EC coupling

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

Beta 1 adrenoreceptors

A

increase HR and increase Contractility

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

Valsalva manœuvre will

A

increase vagal tone

parasympathetic

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

MAP +

A

CO x TPR

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

Blood clot is

A

fibrin clot + platelet plug

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

prostacylcins and NO will

A

reduce platelet aggregation

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
18
Q

heparin

A

inactivates thrombin

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
19
Q

fibrinogen to fibrin transformed by

A

thrombin

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
20
Q

Noradrenaline acting on alpha 1 receptors will

A

reduce flow

increase TPR

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
21
Q

Angiotensin II released in response to

A

low blood volume

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
22
Q

Angiotensin II leads to

A

arteriolar constriction

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
23
Q

ADH is released in response to

A

low blood volume

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
24
Q

ADH release causes

A

arteriolar constriction

so increased TPR

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
25
Q

atrial niuretic peptide is released in response to

A

high blood volume

26
Q

atrial niuretic peptide release causes

A

arteriolar dilation

27
Q

an increase in hydrostatic pressure (standing up) will…

A

reduce

SV

EDV

VR

Preload

MAP

28
Q

Reflex response ill…

A

increase sympathetic tone

decrease vagal tone

29
Q

an increase in sympathetic tone will

A

increase HR, CO, SV, contractility

30
Q

Long term BP is controlled by what 4 things?

A

aldosterone

angiotensin II

atrial niuretic peptide

vasopressin (ADH)

31
Q

an increase in thoracic pressure will decrease

A

VR

EDV

SV

CO

MAP

32
Q

Baroreceptors are

A

sensors located on blood vessels that detect changes in BP

33
Q

Aortic arch 1

A

nothing

maxillary artery

34
Q

aortic arch 2

A

nothin

maxillary artery

35
Q

aortic arch 3

A

internal carotid

36
Q

aortic arch 4

A

aortic arch

37
Q

aortic arch 5

A

nothing

38
Q

aortic arch 6

A

pulmonary arteries

39
Q

angiotensin II stimulates the release of …… and …….

A

aldosterone

ADH

40
Q

ADH is produced in response to

A

low blood volume

41
Q

ADH is produced in the

A

hypothalamus

42
Q

ANP is released in response to …. and …..

A

increase blood volume

increased distension of atrium

43
Q

ANP inhibits….

A

renin release

44
Q

Anticoagulants 2 main drugs

A

heparin

warfarin

45
Q

low molecular weight heparin is often used in

A

venous thromboembolism

46
Q

Beta blockers work by

A

antagonising the sympathetic system

47
Q

beat blockers example

A

propranolol

48
Q

beta blockers are often used in

A

angina

hypertension

post MI

49
Q

be careful when using beta blockers in

A

asthma

heart block

50
Q

ACE inhibitors are often used in

A

hypertension

heart failure

post MI

51
Q

Loop diuretics are used to treat

A

heart failure

52
Q

Thiazide like diuretics are used to treat

A

IHD

hypertension

heart failure

53
Q

Vasodilators example

A

nitrates

54
Q

vasodilators (such as nitrates) are used in

A

IHD

hypertension

heart failure

55
Q

Statins work by reducing

A

LDL cholesterol

56
Q

Statins example

A

simvastatin

57
Q

Sodium channel blockers are also known as

A

anti-arrhythmias

58
Q

anti platelets are given following an

A

MI or stroke

59
Q

anti platelets examples (2)

A

aspirin

clopidogrel

60
Q

Calcium antagonists treat

A

hypertension and angina

Yr1 - S2 - Formatives (6 decks)

Brainscape helps you reach your goals faster, through stronger study habits.
© 2024 Bold Learning Solutions. Terms and Conditions