Airway & Breathing Flashcards

1
Q

What makes people breathless?

A

Pain, exercise, obesity, exercise, genetics, smoking

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2
Q

Name 5 common respiratory conditions?

A
Chest Infection
Pneumonia
Emphysema
Dyspnoea
Respiratory failure
Asthma
COPD
PE
TB
Bhronchiectasis
Bhronchitis
CF
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3
Q

What is Kussmaul breathing?

A

Kussmaul breathing is a deep and labored breathing pattern often associated with severe metabolic acidosis, particularly diabetic ketoacidosis (DKA) but also kidney failure

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4
Q

What is dyspnoea

A

Difficulty in breathing

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5
Q

What is orthopnoea

A

Difficulty in breathing when lying down

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6
Q

Signs of dyspnoea

A

Orthopnoea – DIB Lying down
Use of accessory muscles
Nasal Flaring
Head bobbing (Children)

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7
Q

What is cheyne-stokes breathing?

A

an abnormal pattern of breathing characterized by progressively deeper, and sometimes faster, breathing followed by a gradual decrease that results in a temporary stop in breathing called an apnea. The pattern repeats, with each cycle usually taking 30 seconds to 2 minutes

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8
Q

What is Stridor?

A

High pitched noise on inspiration or expiration, indicates a disturbance to the airflow in the upper respiratory tract

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9
Q

What is stertor?

A

Snoring during sleep or altered consciousness

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10
Q

What is a wheeze?

A

Whistling heard on expiration, indicates resistance to airflow in lower respiratory tract

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11
Q

What is a rattle?

A

Heard on inspiration and expiration, associated with secretions in the lower respiratory tract (death rattle!)

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12
Q

How would you position the breathless patient?

A

Upright position chair/bed

Leaning over pillow placed on hospital table (orthopneic position)

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13
Q

What is asthma?

A

Chronic inflammatory disease of the airways
Causes hyper-responsiveness, mucosal oedema and mucous production
Allergy is the strongest predisposing factor
Inflammation leads to cough, chest tightness, wheezing and dyspnoea

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14
Q

What is Status Asthmaticus?

A

medical emergency where symptoms do not respond to bronchodilators (aggressive treatment/ventilation/ICU)

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15
Q

What is Type I respiratory failure?

A

Type I respiratory failure involves low O2, and normal or low CO2
Occurs because of damage to lung tissue. Lung damage prevents adequate oxygenation of the blood (hypoxaemia); however, the remaining normal lung is still sufficient to excrete the CO2 being produced by tissue metabolism. This is possible because less functioning lung tissue is required for CO2 excretion than is needed for oxygenation of the blood.

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16
Q

What is Type II respiratory failure?

A

Type II respiratory failure involves low O2, with high CO2
‘Ventilatory failure’ - occurs when alveolar ventilation is insufficient to excrete the CO2 being produced. Inadequate ventilation is due to reduced ventilatory effort, or inability to overcome increased resistance to ventilation – it affects the lung as a whole, and thus CO2 accumulates. Complications: damage to vital organs due to hypoxaemia, CNS depression due to increased CO2 levels, respiratory acidosis (CO2 retention). This is ultimately fatal unless treated.

17
Q

List causes of Type I respiratory failure?

A

disease that damage lung tissue, including pulmonary oedema, pneumonia, acute respiratory distress syndrome, and chronic pulmonary fibrosing alveoloitis

18
Q

List causes of Type II respiratory failure?

A

COPD, chest-wall deformities, respiratory muscle weakness (e.g. Guillain-Barre syndrome) and central depression of the respiratory centre (e.g. heroin overdose).

19
Q

What is subjective data in respiratory assessment?

A

Subjective is that the patient (the subject) tells us. So these are the things we cant see/verify with our own eyes.
Pain
Shortness of breath
Feelings
What the pt says “I can’t breathe” “I’m exhausted”
Past health Hx
Family health Hx

20
Q

What is objective data in respiratory assessments?

A
Objective = measurable, can see with our own eyes
 Age/gender
 RR  HR SpO2  IPPA  BP
Diaphoresis/Pale
ECG (e.g. showing sinus tachycardia)
Accessory muscle use, nasal flaring etc
Pursed lip breathing/tripod position
21
Q

What are the normal ABG values?

A
pH 7.35-7.45
paCO2 35-45mmHg
paO2 80-100mmHg
HCO3 22-26mmol/l
BE +/- 2mmol
22
Q

What causes respiratory acidosis?

A

Alveolar hypoventilation
Inadequate perfusion
Mechanical ventilation

23
Q

What diseases cause alveolar hypoventilation?

A
COPD
Obesity hypoventilation syndrome
Chest wall deformities
Neuromuscular disorders
Central alveolar hypotension
24
Q

What diseases cause inadequate perfusion?

A

Shock - cardiogenic, hypovoleamia

25
Q

What are the compensatory mechanisms to restore ABG to normal limits?

A

Initial response: increased RR and depth
Increase in minute ventilation
Increased HR
Possible vasoconstriction
Peripheral chemoreceptors detect hypoxia and initiate compensatory mechanisms

26
Q

What is Non Invasive Ventilation?

A

Non Invasive Positive Pressure Ventilation (NIPPV) delivers positive pressure breaths to a spontaneously breathing patient.
Delivered by a mask with an airtight seal
Reduces the occurrence of patients being intubated

27
Q

What does CPAP stand for?

A

Continuous Positive Airway Pressure

28
Q

What patients is CPAP used for?

A

Commonly used in patients with
Pulmonary oedema
COPD
Asthma

29
Q

Why use CPAP?

A

Support for spontaneously breathing patients and ventilated patients
The raised positive pressure assists in reducing the work of breathing on inspiration
Increases gas exchange and reduces hypoxia

30
Q

What does BiPAP stand for?

A

Bilevel Positive Airway Pressure

31
Q

How does BIPAP work?

A

Involves-
IPAP (Inspiratory positive airway pressure)
A higher pressure is delivered on inspiration
EPAP (Expiratory positive airway pressure)
Lower pressure (but still positive) on expiration

32
Q

What patients benefit from BiPAP?

A
Commonly used in-
high dependency patients
Neurological disorders (Guillain Barre syndrome)
OSA (Obstructive sleep apneoa)
COPD
Asthma
Post extubation weaning issues
33
Q

What do we call mechanical ventilation?

A

Invasive Positive Pressure Ventilation (IPPV)

34
Q

Indications for IPPV?

A

Inability to protect own airway (gag response diminshed (GCS reduced)
Inadequate breathing pattern rate and/or depth
inability to sustain O2 demands of the body
hypercapnia