Brainscape's Knowledge GenomeTM

Browse over 1 million classes created by top students, professors, publishers, and experts.


See full index

` > Arrhythmias 1 > Flashcards

Arrhythmias 1 Flashcards

1
Q

3 mechanisms of arrhythmia

A

Abnormal Automaticity
Triggered Activity
Reentry

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

3 reasons for abnormal automaticity

A

scar, ischemia, age

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

examples of abnormal automaticity

A

SSS, afib, PVCs, PACs

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

? + Torsades = Long QT

A

Bradycardia

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

Bradycardia + Early after depolarizations = ?

A

Torsades

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

Bidirectional VT + Dig = ?

A

Delayed after depolarizations

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

whereas Long QT is exacerbated by bradycardia, what exacerbate delayed afterdepolarizations?

A

catecholamines

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

3 things needed for reentry arrhythmia

A

2 pathways
slow/fast
unidirectional block in 1 of the pathways

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

In terms of Sn/Sp, what do NTG and Isoproterenol do in a tilt table test?

A

Increase Sn

Decrease Sp

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

HV Interval

A

normal = 35-55 msec
Autonomic factors little influence
if HV>100ms–> PPM needed (even if asymptomatic)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

Is infrahissian block serious?

A

yes! Can lead to asystole.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

Long A-H interval makes you think of?

A

slow/fast AVNRT

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

Late potentials on Signal Averaged ECG

A

Infarct, scar (slow conduction)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

Most common reason to get PPM in US?

A

Sinus node dysfunction

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

Class I indications for PPM in sinus node Dz

A

1) SND w/ symptomatic brady
2) SND w/ chronotropic incomp.
3) SND w/ symptomatic brady and required meds

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

Class III PPM for SND?

A

1) SND due to nonessential meds

2) asymptomatic

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

Class I for PPM w/ AVB

A

1) sxs brady
2) >3s pauses (>5s if a fib)
3) required meds that cause AVB
4) 2nd degree AVB w/ sxs brady
5) neuromuscular dz (muscular dystrophy, Kearns-Sayre)
6) AVB w/ exercise but no ischemia
7) awake, no sxs , escape rate 40 w/ either LV dysfxn or known to be infranodal block

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
18
Q

Class III for PPM w/ AVB

A

reversible AVB (lyme, drug tox, OSA)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
19
Q

Class I for PPM w/ bi/trifascicular block

A

1) intermittent 3rd degree AVB
2) 2nd degree type II AVB
3) alternating BBB

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
20
Q

Class I PPM for AVB post MI

A

1) persistent and symptomatic 2nd or 3rd deg AVB
2) persistent infrahissian block (R/LBBB)
3) transient blocks (incld. BBB)

21
Q

Class III PPM post MI

A

1) new BBB w/o AVB

2) transient AVB w/o BBB

22
Q

Class I for CRT

A
  • EF < 35%
  • QRS > 150ms
  • LBBB (not RBBB!)
  • Class II (NEW!), III, or ambulatory IV CHF
23
Q

what type of syncope is “ominous”

A

syncope w/ exertion

24
Q

what tests are low yield for syncope?

A

neuro

25
Q

Which phase of AP is If?

A

4

26
Q

What is phase 0 (upstroke) of SAN AP due to?

A

Ca

27
Q

Which AP phase and current does beta adrenergic stimulation affect?

A

Phase 4

If

28
Q

In what phase do early after depolarizations occur?

A

3

29
Q

What clinical scenario is associated with early after depolarizations?

A

Long QT

30
Q

What phase of AP are delayed after depolarizations seen in?

A

4

31
Q

Name 2 clinical scenarios associated with delayed after depolarizations

A

Calcium overload

Dig tox

32
Q

What mech of arrhythmia causes DAD’s?

A

Triggered activity

33
Q

If you see a wide complex tachycardia with beat to beat variation of axis, what is most likely diagnosis?

A

Bidirectional VT from dig toxicity

34
Q

What mech of arrhythmia is scar related VT (monomorphic)

A

Reentry

35
Q

Who usu gets primary vasodepressor response on tilt table test? Rx? (Not orthistatic b/c takes time)

A

LOL with BP drop in church

- hydration, salt, MIDODRINE

36
Q

What happens in cardio inhibitory response on tilt table test? Who gets it?

A

Pro drone of nausea then Asystole then BP drop then syncope

Young

37
Q

What to think of if HV< 35ms?

A

Pre-excitation

38
Q

What part of conduction system is most common area of irreversible AV block?

A

His bundle

39
Q

What is rate related BBB called?

A

Ashman phenomenon

40
Q

How do you program a ppm if there is AVB, you desire AV synchrony but atrial sensing only?

A

Dual chamber

VDD

41
Q

Why does high impedance (>3000) of a lead occur?

A

Lead fracture

42
Q

Why does low impedance (< 200) of a lead occur?

A

Insulation compromise

43
Q

Rx for Pacemaker Mediated Tachycardia

A

Inc PVARP to blind PPM to retrograde beat

44
Q

Pacemaker syndrome pt’s have vague complaints; what is Rx?

A

Programming changes

45
Q

What class indication is it to put PPM for infra-hissian block or HV > 100ms?

A

Class II (do it!)

46
Q

Where is the LV lead placed for CRT?

A

Lateral LV epicardium or CS if endocardial

47
Q

According to new guidelines, what class is PPM for QRS 120-149?

A

IIa

48
Q

What does beta adrenergic stimulation do to If channel?

A

Increases the current and makes threshold more negative

` (31 decks)

Brainscape helps you reach your goals faster, through stronger study habits.
© 2024 Bold Learning Solutions. Terms and Conditions