Final: Ophthalmology Flashcards

1
Q

What is the most common cause of miosis?

A

Uveitits

(other possibiltiy= corneal disease, or drugs)

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2
Q

How are uveal cysts treated? Do they need to be treated?

A

Diode laser ablation

In horses they must be treated (impede vision)

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3
Q

What are the Stars of Winslow?

A

End on capillaries of the choriocapillaris

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4
Q

What does a steeper eyelash angle indicate?

A

Bleparospasm - PAIN

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5
Q

How do you open the eyelids?

A

Index finger engages supraciliary sulcus

Sedation: xylazine, detomidine, torb, romifidine

Nerve blocks

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6
Q

What are the 3 spots that you can block to obtain akinesia of the upper eyelid? What nerve are you blocking?

A

Caudal to posterior ramus of mandible

Dorsal to highest point of zygomatic arch

Zygomatic arch caudal to bony process of frontal bone

Auriculopalpebral nerve

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7
Q

Where do you block the frontal nerve?

A

Supraorbital foramen

Blue in pic

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8
Q

What can you do to overcome the challenge of having to use eyedrops in a horse? What is required to place this?

A

Place a Subpalpebral Lavage (SPL)

Sedation (detomidine +/- torb, topical AX (propara or teracane), nerve blocks (frontal and auriculopalp))

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9
Q

What is the most common cause of blindness in horses? Which breeds are at-risk?

A

Equine recurrent uveitis (Moon blindness, periodic ophthalmia)

Appaloosas, Drafts, European warmbloods

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10
Q

What is the most commonly implicated/suspected trigger for moon blindness?

A

Leptospirosis

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11
Q

What clinical signs are seen in recurrent uveitis when the infection is acute and active?

A

Aqueous flare

Hypopyon

Fibrin (yellow hue)

Hyphema

Miosis

Iris hyperpigmentation

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12
Q

What clinical signs are seen in recurrent uveitis when the infection is chronic and active?

A

Band keratopathy (mineral infiltrates into the cornea)

Glaucoma: episcleral injection, buphthalmos, corneal edema

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13
Q

What are some scars of ERU that interfere with vision?

A

Posterior synecia

Cataract formation (blinding)

Phthisis bulbi (blinding)

Retinal degeneration/detachment (blinding)

Optic nerve degeneration

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14
Q

What is the treatment plan for active ERU?

A

Systemic NSAIDs (Banamine, Phenybutazone, Aspirin)

Systemic corticosteroids (Prednisone, Dexamthasone - caution LAMINITIS )

Systemic antibiotics (Doxycyline- based on titer)

Topical steroids (Pred- C/O’d w/ CORNEAL DISEASE )

Topical NSAIDs (Flurbiprofen, Diclofenac)

Topical mydriatics (Atropine- caution COLIC)

Treat for at least 2 weeks post resulution

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15
Q

What is the referral treatment option for ERU?

A

Cyclosporin implants

Effective for over 2 years

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16
Q

What features in an examination suggest that a corneal ulcer is infected?

A

Deep ulcer

Diffuse corneal edema

Yellow-white corneal infiltrate

Keratomalacia/Melting

Deep corneal vascularixation (Crown of Thorns)

Severe reflex uveitis (miosis, aqueous flare hypopyon)

17
Q

What treatment is warrented for keratomalacia?

A

Agressive (q1-2 hrs) anticollagonase therapy

Autologous serum

EDTA

Acetylcysteine

18
Q

What are the most commonly identified organisms in infected equine ulcers?

A

Fungi:

  • Aspergillus*
  • Fusarium*
  • Candida*

Bacteria:

  • Pseudomonas aeruginosa*
  • B-hemolytic streptococcus*
19
Q

What are the 2 diagnostic tests you should run for a corneal ulcer?

A
  1. C&S (bacterial and fungal)
  2. Corneal cytology
20
Q

What is the treatment for acute superficial non-infected (uncomplicated) corneal ulcers?

A

Topical broad spectrum antibiotics TID

Atropine

Systemic NSAID

Recheck 5-7 days

21
Q

What is the treatment for complicated corneal ulcers?

A
  1. Topical broad-spec ABs: Triple antibiotic or Cefazolin (gr+) + Ofloxacin or Tobramycin (gr -)
  2. Antifungals (Voriconazole)
  3. Topical antiprotinase therapy (Serum)
  4. Atropine q6-24 hours
  5. Oral or IV NSAIDs (Banamine)
  6. Subpalpebral lavage
  7. Protective eye cup
22
Q

When should you consider referal for surgical repair of corneal ulcers? What is this surgeries?

A

>50% stromal depth

Rapidly progressive

Melting

No vascular response

Perforation

Sx: Conjunctival graft

23
Q

What clinical signs suggest the presence of a deep stomal abscess?

A

Fluorescein stain negative

Deep white corneal infiltrate

Corneal edema

Severe reflex uveitis

+/- vacularization

24
Q

How are deep stromal abscesses treated?

A

Aggresibe medical therapy!

Topical: Ofloxacin, Voriconazole (have good penetration), Atropine

Banamine

25
Q

When is surgery indicated for a deep stromal abscess? What are the surgical options?

A

If there is no clinical improvement after 24-48 hours of appropriate/aggressive medical therapy

Sx: Penetrating keratoplasty/ Corneal transplant, Intrastromal voriconazole inhection

26
Q

What are the 4 main predilection sites for SCC? What clinical signs indicate SCC in these locations?

A

Eyelid: hyperemic, erosive plaques, dark crusts, large fleshy mass

Conjunctiva/cornea/orbit: raised pink mass, Cobblestone appearance

Third eyelid/medial canthus: fleshy mass, erosions, crusts

Limbus (usually starts lateral) - UV exposure): fleshy mass

27
Q

What is the most important diagnostic recommendation with SCC?

A

Histopathology

28
Q

What are the treatment options for SCC?

A

Surgical excision (Enucleation, conjunctivectomy, third eyelid excision) + Adjunct therapy (cryo, CO2 laser…) + Lifelong monitoring

Communicate need for possible multiple treatments

29
Q

How is SCC prevented?

A

UV avoidance!

e.g. protective masks