Patient's problems, PT management and outcome measures Flashcards

1
Q

Most common pt problems

A
> Impaired airway clearance
> Dyspnoea
> ↓ exercise tolerance
> Reduced lung vol
> Impaired gas exchange
> Airflow limitation
> Respiratory muscle dysfunction
> Dysfunctional breathing
> Pain
> MSK dysfunction (postural abnormalities, ↓ compliance or deformity of the chest wall)
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2
Q

Problem solving (Note)

A

Pt often present with more than one problem that is amenable to PT. In this situation, the intervention plan should focus on strategies that address as many pt problems as possible, using the best evidence and practice, and should be determined in collaboration with the individual and with a focus on self-management where appropriate.

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3
Q

Impaired Airway Clearance: problem

A

Normal airway clearance depends upon 2 mechanisms:

  • Mucociliary clearance
  • Effective cough

Normal volume of mucus in adult is up to 100 ml/day.
When the volume has increased so the individual becomes conscious of the presence of secretions on coughing or “clearing the throat” the mucus is defined as sputum. Its presence is abnormal.

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4
Q

Impaired Airway Clearance: clinical features

A

> Hx of usual daily sputum production
Altered breathing pattern due to ↑ work of breathing (WOB)
Infection (can produce fever and tachycardia)
Auscultatory findings: whezzing, diminished or absent breath sounds, bronchial breath sounds, crackles.
Chest Rx: sometimes show signs of lung collapse and/or consolidation
In the postoperative pt: ↑ volume of sputum; weak, ineffective moist cough; possible bacterial contamination of sputum; fever; and chest Rx changes consistent with atelectasis or pneumonia.

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5
Q

Impaired Airway Clearance: PT management

A

Factors to consider when choosing airway clearance TQ:

  • Evidence supporting TQ
  • pt age and ability to learn TQ
  • pt motivation
  • pt preference and confort
  • PT’s skill in teaching TQ
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6
Q

Impaired Airway Clearance: outcome measures

A

► Short-term outcomes can be monitored by:
> Change in sputum expectorated (weight, vol or rate of expectoration)
> VAS for ease of expectoration
> In acute conditions, chest Rx and auscultatory findings
> Radio-aerosol clearance may be used in studies of clearance TQs

► Long-term outcomes can be monitored by:
> Number of exacerbations, courses of antibiotics, hospitalizations, etc.
> Quality of life scales (St George’s Respiratory Disease Questionaire)
> Spirometry (pulmonary fx)

Improved cough or huff TQ may be associated with a ↓ in associated problems such as fatigue, dyspnoea, syncope, airflow limitation, arterial O2 desaturation or stress incontinence.

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7
Q

Dyspnoea: problem

A

Sensations experienced by individuals complaining of unpleasant or uncomfortable respiration, AKA breathlessness.

Some pt with moderate or severe disease, especially those with COPD, report marked dyspnoea when performing ADLs that involve the use of the ULs, especially when the ULs are unsupported.

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8
Q

Dyspnoea: clinical features

A

Some pt seek medical care when they become breathless playing sports, but many don’t seek help until breathlessness occurs during ADLs.

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9
Q

Dyspnoea: PT management

A

> Bronchodilators may ↓ dyspnoea and ↑ exercise tolerance, so inhaler TQ and timing should be optimized.
Positioning, breathing control and relaxation TQs.
Symptomatic relief may be achieved by ↑ mvmt of cold air onto pt’s face.
Pursed-lip breathing may be very effective in ↓ discomfort associated with dyspnoea.
Encouraging exhalation during effort may be helpful.
For selected pt with severe dyspnoea, education on energy conservation TQs during ADL is important.
Exercise training is effective relieving dyspnoea in pt with stable chronic lung disease and those with cardiac failure.
Walking aids that facilitate the forward lean position and arm support may ↓ dyspnoea, ↑ exercise tolerance and limit the extent of O2 desaturation in pt with COPD.
Ambulatory O2 only indicated if shows to produce benefit in terms of ↑ exercise tolerance and ↓ breathlessness.
For pt with COPD, high-intensity inspiratory muscle training (IMT) has been shown to ↓ dyspnoea.

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10
Q

Dyspnoea: outcome measures

A

Ax of dyspnoea at rest, during ADL and when exercising:
> Intensity: Borg RPE
> Exercise tolerance: walking test
> Fx limitation: scales such as Medical Research Council (MRC), New York Hearth Association (NYHA), University of California San Diego Shortness of Breath Questionnaire, etc

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11
Q

Decreased Exercise Tolerance: problem

A

Exercise tolerance in pt with respiratory or cardiovascular disease is invariably limited by dyspnoea, pain (chest or legs) or fatigue (general or local).

Depression and anxiety often accompany chronic respiratory or cardiovascular disease and may ↓ an individual’s confidence or motivation to exercise.

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12
Q

Decreased Exercise Tolerance: clinical features

A

> ↓ ability to exercise or perform ADLs → breathlessness, fatigue or pain.
↓ Fx exercise capacity (measured with field walking test)
When Exercise-Induced Asthma (EIA) ↓ in FEV1 and peak expiratory flow rate measured right after exercise.
In some pt respiratory muscle strength may be ↓ compared to normal.
For pt with acute cardiopulmonary dysfunction an exercise test in inappropriate.

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13
Q

Decreased Exercise Tolerance: PT management

A

> Pt with known or suspected EIA should use inhaled short-acting beta-agonist or NSAIDs before ex’s.

> Strong evidence supports benefits of exercise training for these pt. Also effective in prevention.

> Group training is recommended.

> Program should have warm-up, stretches, aerobic component, resistive training (when appropriate), and cool-down.

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14
Q

Decreased Exercise Tolerance: outcome measurements

A
> Ax of anthropometric variables
> Resting BP
> Peripheral muscle strength
> Fx ex capacity
> QoL (questionnaires)
> Ability to perform ADLs (questionnaires or self-report by pt)
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15
Q

Reduced Lung Volume: problem

A

Inability to expand the lung tissue. involves ↓ RV, Vt, ERV and/or IRV.

Main clinical consequences:

  • ↓ TLC and VC: pt unable to ↑ inhaled vol sufficiently to expand lung tissue
  • ↓ FRC: pt unable to sustain alveolar inflation
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16
Q

Reduced Lung Volume: main consequences

A

> Impaired oxygenation due to V/Q mismatch.
Ineffective cough
Increased WOB
Dyspnoea during physical activity

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17
Q

Reduced Lung Volume: PT management

A

When ↓ gas exchange or ↑ WOB → high sitting, sitting out of bed and ambulation encouraged.

Side-lying better than supine or slumped.

FRC may ↑ with Continuous Positive Airway Pressure (CPAP).

Breathing TQs can ↑ Vt and inspiratory capacity (in upright positions).

Ambulation increases Ve (pt should be encouraged to take frequent deep breaths to assist in lung re-expansion).

18
Q

Reduced Lung Volume: outcome measures

A

> Cough effectiveness.
Auscultation → changes in lung volume.
Chest Rx
Pulmonary fx tests

19
Q

Impaired Gas Exchange: problem

A

Type I respiratory failure is present when pt is awake and at rest when PaO2 is below 8 kPa (60 mmHg).

Type II respiratory failure is present when pt is awake and at rest when PaCO2 exceeds 6.7 kPa (50 mmHg) and meets the criteria for Type I.

20
Q

Impaired Gas Exchange: clinical features

A

Moderate to severe hypoxaemia: tachypnoea, restlessness, confusion, sweating, tachycardia, hypertension, skin pallor and cyanosis.

21
Q

Impaired Gas Exchange: PT management

A

In many cases O2 therapy is indicated. Has to be Rx by doctor. PT can administer.

Gas exchange may be optimized by positioning upright to ↑ FRC.

Physical activity may improve gas exchange by enhancing O2 transport.

22
Q

Impaired Gas Exchange: outcome measures

A

Arterial blood pressure, oximetry and measures of PaCO2 will reflect changes in gas exchange.

23
Q

Airflow Limitation: problem

A

Abnormal resistance or obstruction to airflow. Generally occurs together with other physiotherapy problems such as dyspnoea, ↓ ex tolerance or impaired airway clearance.

Airflow limitation may be reversible (asthma), partially reversible (chronic bronchitis) or irreversible (emphysema).

24
Q

Airflow Limitation: clinical features

A

> Chest tightness or wheeze, cough and breathlessness.
“Tight” cough and difficulty to clearing secretions.
↑ WOB
↑ respiratory rate
↑ I:E ratio
Recruitment of accessory muscles
Pursed-lip breathing in some cases
In long-standing disease, hyperinflated chest (barrel-shaped)

25
Q

Airflow Limitation: PT management

A

> Medication when limitation is reversible
Pt education (especially in asthma)
Airway clearance techniques
Force Expiration Technique (FET) useful to prevent dynamic airway collapse
Positive Expiratory Pressure (PEP) useful to splint open collapsible airways

26
Q

Airflow Limitation: outcome measures

A

Most important is QoL questionnaire:

  • Asthma Quality of Life Questionnaire (AQLQ)
  • Mini AQLQ
  • Marks Asthma Quality of Life Questionnaire (AQLQ-M)
  • Modified AQLQ-M
  • Asthma Control Questionnaire (ACQ)

Auscultation for short-term changes (wheeze)

Pulmonary fx tests

27
Q

Respiratory Muscle Dysfunction: problem

A

May be present in a range of clinical conditions:

  • Neuromuscular disorders
  • Connective tissue diseases
  • Chronic lung disease
  • Chest wall disorders
  • CHF (Chronic Heart Failure)
28
Q

Respiratory Muscle Dysfunction: clinical features

A

Weakness of the respiratory muscles is often advanced before clinical symptoms are present because the pressure required to initiate inspiratory flow represents only a small proportion of the max force-generating capacity of the inspiratory muscles.

Main clinical features:
> Unexplained reduction in VC
> Abnormal breathing pattern
> Nocturnal hypoxaemia and hypercapnia in the absence of chronic lung disease
> Dyspnoea
> ↓ ex tolerance
> Impaired airway clearance
29
Q

Respiratory Muscle Dysfunction: PT management

A

> Targeted respiratory muscle training (IMT, Inspiratory Muscle Training)
Whole body ex’s (as opposed to unsupported upper limbs ex’s)
Assistance with airway clearance
Non-invasive ventilation during night-sleep (sometimes also during daytime)

30
Q

Respiratory Muscle Dysfunction: outcome measures

A

> Pt perception of dyspnoea during ADL and ex → RPE or VAS
Questionnaires (e.g. Chronic Respiratory Disease Questionnaire)
PiMax and PeMax will determine whether changes in respiratory muscle strength have occured.
Observation of breathing pattern.
VC
Field walking test

31
Q

Dysfunctional Breathing Pattern: problem

A

Rarely occurs in isolation and is commonly associated to other problems.

Resolution of the problems may be accompanied by a return to a more normal breathing pattern.

Breathing problems resulting from neurological abnormalities or metabolic dysfunction can’t be treated with PT.

Dysfunctional breathing mainly occurs when the WOB is exaggerated such that pt overbreathe or have an abnormally high resp rate.

32
Q

Dysfunctional Breathing Pattern: clinical features

A

> Abnormalities in resp rate and depth, including excessive sighing or breath holding, and changes in the I:E ratio.
Observation and palpation: asymmetrical chest wall mvmt, paradoxical chest wall mvmt, asynchronous mvmt or respiratory alternans.
Accessory muscles overuse.
Arterial blood gas analysis: hypocapnia in hyperventilation or hypoxaemia and hypercapnia in spinal cord injury.
Diagnostic tests: voluntary hyperventilation provocation test, Nijmegen questionnaire and breath-holding time.

33
Q

Dysfunctional Breathing Pattern: PT management

A

Focus is on changing dysfunctional breathing patterns when they are not associated with strategies used by pt to ↓ other problems (dyspnoea or airflow limitation).

Relaxation and breathing control are encouraged.

Positive pressure vent indicated in cases of abnormalities to the chest wall or spinal cord injury.

34
Q

Dysfunctional Breathing Pattern: outcome measures

A

> Nijmegen Questionnaire
Breath-holding time
Diaries recording disability/distress/symptoms and QoL measures
Arterial blood gas analysis, transcutaneous CO2 and oximetry

35
Q

Pain: problem

A

> Chest pain of respiratory origin: often reported by pt who have chronic cough or dyspnoea as a result of the associated MSK pathology.

> Chest pain of cardiovascular origin

> Chest pain which may be unrelated to respiratory or cardiovascular disease:

  • Neural, muscular or skeletal pain: usually localized to the affected area (TOP). Usually reported as dull ache or sharp. Pain ↑ with resp mvmt, aggr by sh and trunk mvmt.
  • Oesophageal pain
  • Peptic ulceration and gallbladder disease: with peptic the pain is burning after meals, relieved by antacids. Biliary pain is colicky and felt on R side of abdomen, front and back of chest. May be related to ingestion of fatty food.
  • “Pseudoangine” due to hyperventilation syndrome: atypical chest pain, which mimics angina.
36
Q

Pain: clinical features

A

> VAS
On examination: signs of abnormal breathing pattern and systemic signs (sweating, pallor and tachycardia)
Chest expansion may be ↓ over painful area.
Associated ↓ in breath sounds on auscultation and pleural rub.

37
Q

Pain: PT management

A

> Dx and management of underlying cause are essential.
Anti-inflammatory agents or analgesics used for MSK, pleuritic and pericardial pain.
Heat modalities, interferential, TENS, acupuncture, manual therapy.

38
Q

Pain: outcome measures

A

> VAS

39
Q

MSK dysfunction: problem

A

Most common causes:
> Chest wall stiffness and abnormal posture due to neuromuscular disease.
> Sternotomy or thoracotomy.
> Age-related changes (e.g. ↓ in ROM due to costovertebral joint stiffness and ↓ in cartilage elasticity)
> Hyperkyphosis due to overused forward leaning posture.
> Neuromuscular disorders → muscle weakening →postural abnormalities.

40
Q

MSK dysfunction: clinical features

A

> Abnormal posture
↓ ROM of Cx, Tx and sh
Pain or stiffness resulting in decreased fx

41
Q

MSK dysfunction: PT management

A
> Postural correction
> Stretching techniques
> Mobs of Cx, Tx, ribs and sh
> Muscle strengthening ex's
> HEP / education
42
Q

MSK dysfunction: outcome measures

A

> ROM of Cx, Tx and sh
Muscle length and strength
VAS: for short-term changes
Generic QoL scale (i.e. SF-36): for long-term changes