Shock Flashcards

1
Q

shock

A

failure of the circulatory system to maintain adequate perfusion of vital organs

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2
Q

what occurs when there is a buildup of wastes?

A

the body switches from aerobic to anaerobic metabolism

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3
Q

how do we treat shock in general?

A

it is cause specific

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4
Q

cardiovascular system parts

A
  1. pump (heart)
  2. set of pipes (blood vessels and arteries)
  3. contents (blood)
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5
Q

what are the types of shock and which part of the cardiovascular system do they correlate with?

A

pump (heart) = cardiogenic
pipes (blood vessels and arteries) = circulatory
contents (blood) = hypovolemic

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6
Q

what are the 3 subcategories of shock under circulatory shock?

A

septic, neurogenic, anaphylactic

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7
Q

what is the most common type of shock?

A

hypovolemic

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8
Q

what is the most common form of cardiogenic shock?

A

MI

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9
Q

what is normal MAP?

A

70-105 mmHG

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10
Q

what value of MAP must we obtain and why?

A

> = 65mm Hg for perfusion

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11
Q

how do we calculate MAP?

A
    3
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12
Q

what do we assess with pulse pressure?

A

narrowing

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13
Q

what are cellular changes that occur during shock?

A
  • anaerobic metabolism
  • inc cell perm = influx of Na + H20, efflux of K+
  • mitochondrial damage leading to death
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14
Q

what are assessments and interventions we perform when a pt is entering shock?

A
  • apply O2 d/t dec perfusion
  • check perfusion: cap refill, colour, LOC (is it altered)
  • RR (is it slowing down?)
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15
Q

what is the first change we see with our pt if they may be entering shock?

A

a change in LOC - may be irritated, annoyed, agitated, sharp

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16
Q

what are the systemic effects of shock in relation to the cardiovascular system?

A
  • myocardial deterioration
  • DIC
  • vasoconstriction
  • release of lysosomal enzymes
17
Q

lysosomal enzymes

A
  • vasoactive substances
  • catecholamine: epinephrine, dopamine
  • histamine
  • vasoactive polypeptides (bradykinin, angiotensin)
18
Q

MODS

A
  • phase of progression of shock by any cause

- altered organ fx that requires medical intervention to support continued organ fx

19
Q

how does MODS progress?

A

usually starts in lungs, followed by cardiac instability

then progresses to hepatic, GI, renal, immunologic and CNS

20
Q

mortality rate of MODS

A

high - one organ = 20% mortality rate, 4 or more organs = 70% mortality rate

21
Q

who is at increased risk for MODS?

A

elderly, people with comorbidities, malnutrition, surgical or trauma wounds

22
Q

hypovolemic shock

A
  • internal or external loss of circulating volume
  • blocked venous return
  • organ hypoperfusion
  • stages of compensation
23
Q

management of hypovolemic shock

A

early recognition, stop and replace losses if indicated

  • treat underlying cause
  • blood loss, fluid shifts, dehydration, ascites or edema
  • fluid and blood replacement
  • fluid redistribution
  • meds depending on underlying cause
24
Q

crystalloids

A
  • electrolyte solutions that move easily between intravascular and interstitial spaces
  • can be given without altering [ ] of plasma e-

ie) NS, LR 3%
- 3:1 crystalloid to blood ratio

25
Q

colloids

A
  • large molecule IV solutions
  • generally plasma proteins
  • expand intravascular volume by exerting oncotic pressure
    ie) albumin and synthetics
26
Q

positioning for a pt in shock

A

modified trendelenburg position