Abnormal CXR Flashcards
objectives (31 cards)
Typical (lobar or segmental): MC type of pneumonia

- Pneumococcal pneumonia – Strep pneumoniae
- Infection contained within a single lobe or part of a lobe
- Affected tissue is more opaque aka infiltrate
- May contain air bronchograms
- Silhouette sign: helps localize the lobe
- Airspace dz, fluffy, cloudlike, indistinct borders
Bacterial Pneumonia
- Typically lobar, single consolidation

Bad Lobar Pneumonias: worse bacterial pathogens = bronchopneumoina
- Staph aureus …. Also gram negative bacteria (klebsiellae and pseudomonas)
- Patchy airspace dz involving several segments of lung, exudate may fill bronchi so may not have air bronchograms
- How to dx:
- Sputum gram stain
- Blood cultures
- PCR for respiratory viruses
- Legionella urinary antigen

Air Bronchogram
- normally you can’t see airways>> as they get more stuff on the outside (consolidation) you can see the air going through the airway

Atypical Pneumonia: “walking”
- MC mycoplasma pneumoniae
- No focal consolidation like bacterial, more diffuse and patchy
- Fine, reticular interstitial pattern in lungs

Atypical: PCP/PJP: always multi-lobar
- MC infection in pts with AIDS
- Fine, reticular interstitial pattern in lungs

Tuberculosis
- Parenchymal lung disease with Cavitary lesion
- CXR of active TB:
- Classic: focal infiltration of upper lobes (apical/posterior) or lower lobe (apical/superior)
- Can be unilateral or bilateral
- Cavitation may be present and inflammation/tissue destruction causing fibrosis
- Caseous granulomas
- Enlargement of hilar and mediastinal lymph nodes

Pleural effusion
- fluid trapped between visceral and parietal pleura
- Normally contains 5-20 mL of pleural fluid
- The rate of formation may be increased due to
- inc hydrostatic pressure- Left HF
- dec colloid osmotic pressure- hypoproteinemia
- inc capillary permeability- infection
- The rate of resorption can decrease by:
- Decreased absorption of fluid by lymphatics- tumor
- The rate of formation may be increased due to
- Normally contains 5-20 mL of pleural fluid

pleural effusion etiology
- Etiology: cbc, protein and LDH level inc (get serum sample), gram stain, cytology
- Transudates: bilateral, low protein low LDH
- CHF MCC
- Cirrhosis
- Nephrotic syndrome
- Exudates: unilateral, inc protein, inc LDH
- Malignancy and infection MCC
- Contains frank pus = empyema
- Rheumatic dz can cause bilateral or unilateral –older ppl
- Transudates: bilateral, low protein low LDH
free pleural effusion
- You can see a meniscal line with free pleural effusions
- Will have blurred costophrenic angles most of the time; will be white
- The fluid will move with gravity when a lateral decubitus is taken.
- You may need a lateral view to see the small effusion in the posterior mediastinum

loculated pleural effusion
- can mimic pneumonia/consolidation
- When you get a lateral decubitus and the fluid does not move with gravity.
- If fluid is in a position where it could not normally hold itself up – adhesions are holding it up

CXR view for pleural effusion
- Lateral decubitus with affected side down
- A pleural effusion can cause an atelectatic lung where the two layers are separated by fluid so the lung decreases in size but remains its shape.
Pneumothorax
Air trapped in pleural space

pneumothorax cause
- Trauma/iatrogenic- through a communication in the chest wall
- Spontaneous/COPD- thorugh lung parenchyma across visceral pleura
- Copd- blebs that burst
pneumothorax CXR
- look at the lining of the pleura at the periphery – they will be separated
- Absence of vessels outside the pleural line
- If it is a small pneumothorax, look at the apices first- up near clavicle!
- Which one?
- Expiratory Lateral decubitus with affected side up because air rises!
Tension pneumothorax
- “so much air outside that is pushes everything to other side of chest”
- Positive pressure that displaces the mediastinal structures (shifts them) to the opposite side of the pneumothorax
- Impair venous return and compromise cardiopulmonary function
- Can cause cardiac arrest if venous return sucks so BP will drop
- On CXR:
- Trachea deviated
- Flattening or inversion of diaphragm w/partial or complete collapse of lung
- You need to decompress this immediately if large enough….
- Mainstay of tx: chest tube if large enough; inserted at apices since air rises
- Body can absorb the air if it is a small one.

Hilar enlargement
- Hilar adenopathy: prominent hilar border that is lobulated or polycyclic in contour- GRAPES
- Aortopulmonary window is not clear
- Enlarged Lymph Nodes
- Lymph nodes have a lobular grape-cluster appearance on PA film
- Sarcoidosis most common cause: “bilateral hilar lymphadenopathy”
- Unilateral:
- Mets from bronchial CA
- Malignant Lymphoma-neoplasmas
- Infections - esp TB and Histoplasmosis

Bronchogenic Tumors
- Usually presents as solitary nodule
- If metastatic dz to lungs: multiple nodules
- Four types:
- Squamous cell carcinoma
- Central growing, in bronchi
- Adenocarcinoma
- Bronchoalveolar cell carcinoma
- Peripheral
- Small cell: centrally
- Large cell carcinoma
- Squamous cell carcinoma

Enlarged Pulmonary Arteries
- Pulmonary arteries have a branching pattern

Discuss the characteristics that help distinguish benign from malignant solitary pulmonary nodules
- Calcified nodule:
- The more calcium the more radiopaque that it is going to look
- Most likely benign
- Shape:
- Irregular = bad
- Speculated border- malignancy
- Nodules/masses: more sharply marginated than airspace dz and produce clear demarcation btwn nodule and lung tissue
- Nodule < 3 cm
- Mass > 3 cm
- Malignant mass: > 5 cm
- Change in size over time =malignant

Recognize the classic appearance of pulmonary metastases
- See more bilateral nodules – they can be large or small
- They can look like whatever they want no matter what type of cancer
- No size restriction/ not uniform- would be all over a lot of the time

Severe Emphysema / COPD
- COPD: lung disease characterized by persistent respiratory symptoms and airflow limitations that is due to airway or alveolar abnormalities (noxious particle exposure= cigarettes)
- On CXR: hyperinflation, flattening of diaphragm, inc retrosternal air space
- Long, narrow, vertical, hanging heart
- Hyperlucency of lungs (dec markings)
- On CXR: hyperinflation, flattening of diaphragm, inc retrosternal air space
- Emphysema is destruction of the alveoli à as they are destroyed there is a pocket of dead air
- Bullae/bleb formation w/emphysema
- Radiolucent CXR – ribs may look bigger, heart may look smaller, flattened diaphragm
- More barrel chested on lateral view

Pulmonary infarction (Hamptons Hump and Westermark sign)
- Pulmonary emboli
- CXR findings:
- Westermark Sign
- Truncated pulmonary artery
- Collapse of the distal vasculature creating the appearance of a sharp cut off on chest radiography.
- You are going to see this more centrally
- Hampton’s Hump
- Wedge shaped pleural based consolidation (mimics pneumonia)
- Will see a large obstruction further out into the lung
- Caused by Pulmonary Infarction associated with PE
- Wedge shaped pleural based consolidation (mimics pneumonia)
- Pleural effusion
- Elevated diaphragm
- CXR is usually normal - a normal film does NOT rule out a PE!!!
- Westermark Sign
- CXR findings:

Interstitial Lung Disease, e.g. idiopathic pulmonary fibrosis
- Has discrete reticular, nodular, or reticulonodular patterns
- Usually diffuse
- No air bronchograms present
- What causes ILD:
- Pulmonary fibrosis
- Drugs/ct dz, environmental
- Sarcoidosis
- Early edema from HF
- CT: “ground glass opacities” or “honeycombing” – starts in the periphery and have thick scarred vessels (scarring in the interstitium highlights vessels), more advanced can be seen more centrally
- CXR: Lacy, reticular pattern typically involving most lung fields but can be apical or basilar predominate






