Acquired brain injuries and CTE Flashcards

1
Q

what are the impacts of ABI on biological functioning

A

seizures, movement impairment and smeel/oflaction impairment

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2
Q

what causes seizures

A

occur as a result of scars left by brain injury that produce a sudden abnormal electrical disturbance in the brain

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3
Q

what causes movement impairment

A

paralysis occurs because the brain is unable to send adequate motor neural messages to the bodys skeletal muscles (can occur as a result of injury to any part of tthe brain)

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4
Q

why does olfactory/smell impairment occur

A

an obstructed nasal airway or harmed neural pathway

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5
Q

smell/olfactory impairment may be…

A

temporary or permanent loss of smell, loss of some smells, increased sensitivity to some smells

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6
Q

impact of ABI om psychological functioning

A

memory loss, personality changes, increased susceptibility

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7
Q

how does memory loss occur

A

if certain areas of the brain which control memory are damaged (e.g. hippocampus)

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8
Q

memory loss can be..

A

short term, long term, complete amnesia

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9
Q

how do personality changes occur

A

following a brain injury, particuraly to the frontal lobe, can cause alterations in an individuals emotional and behavioural regulations

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10
Q

increased susceptibility to mental health disorders occur why

A

dysfunction in certain areas of the brain, brain injury can induce or exacerbate mental health

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11
Q

impact of ABI on social functioning

A

job productivity, social support, antisocial behavioural

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12
Q

CTE stands for

A

chronic traumatic encephalopathy

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13
Q

chronic traumatic encephalopathy

A

a progressive and fatal brain disease associated with repeated head injuries and concussion

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14
Q

neurodegenerative disease

A

disease characterised by the progressive loss of neurons in the brain

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15
Q

How is CTE a neurodegenerative disease

A

because in CTE, damage to neurons occurs overtime and brain functioning progressively worsens

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16
Q

how can CTE cause death

A

CTE can become fatal as the brain reaches its point of [degeneration in which it is difficult to function and sustain life

17
Q

who is CTE most prevelent in

A

older athletes, war veterans and other people who have had repeated concussions, typically appears 8-10 yrs after concussions

18
Q

symptoms of CTE

A

memory loss, behavioural changes, problems with attention and concentration

19
Q

how are the four stages of cte described

A

very mild, mild, moderate and severe

20
Q

what occurs during stage 1 (very mild) of CTE

A

headaches, loss of attention and concentration

21
Q

what occurs during stage 2 (mild) of CTE

A

mood swings, depression

22
Q

what occurs during stage 3 (moderate) of CTE

A

memory loss, executive dysfunction

23
Q

what occurs during stage 4 (severe) of CTE

A

dementia, language difficulties and paranoia

24
Q

how can CTE be comppletely confirmed/diagnosed

A

through a post-mortem autopsy, not possible to diagnose CTE through neuroimaging

25
what indicates that CTE is present in an individual
the build-up of p-tau (a protein) in regions of the brain - this can be determined through a structural neuroimaging technique
26
27
neurofibrillary tangles
stops the neuron from transporting essential substances, impairs communication and results in neural death
28
why cant CTE be diagnosed easily
symptoms are very simsilar to parkinsons and alzheimers
29
what are the treatments of CTE
CTE is incurable and irreversable but medications can be used to manage symptoms and therapies to stabilise mood, cognition and behaviour, prevention is key
30
how is CTE prevented
timely treatment of concusion, adhering to rest rules, wearing helmets