Acute emergencies and pre-hospital care Flashcards

1
Q

What are the criteria for refering a patient from primary care to nephrology?

A

HTN after a trial of 4 different anithypertensive agents
eGFR <30 decreased by >5 in a year
Albumin:creatinine ratio > 70 not known to be associated with diabetes
ACR > 30 with persitence haematuria (UTI excluded)
Suspected or rare genetic causes of CKD
Suspected renal artery stenosis
Supsected complications of CKD e.g. anaemia, gout, secondary hyperparathyroidsim

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2
Q

How does a subdural haematoma prsent?

A

Fluctuating GCS on a background of head injury
CT finidngs of a cresent shaped haemorrhage
Delayed presentation due to lower pressure (venous system)
Intracranial bleed between the meningeal layers of the dura matter and arachnoid matter

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3
Q

How does a subarachnoid haemorrhage present?

A

Sudden onset severe thunderclap headache associated with vomiting and a rapid drop in GCS
Occurs between the arachnoid membrane and the pia matter
The normally black subarachnoid cisterns and sulci may appear white in acute hemorrhage on CT
On CT scans, subarachnoid hemorrhage (SAH) appears as a high-attenuating, amorphous substance that fills the normally dark, CSF-filled subarachnoid spaces around the brain

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4
Q

What triad does rhabdomyolysis inculde?

A

Dark urine
Generalised muscle weakness
Myalgia

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5
Q

What CK level is diagnostic of rhabdomyolysis?

A

> 5x normal range

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6
Q

What is Horner’s syndrome?

A

Anhidrosis
Miosis (small pupils)
Pstosis

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7
Q

Why might there be arm pain and weakness in Horners syndrome?

A

INvasion of the brachial plexus

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8
Q

When should you be suspicious of necrotising fasciitis?

A

An important consideration in all patients with skin infection is necrotising fasciitis, also known as Eron class IV cellulitis. Highly suggestive clinical features include pain out of proportion to the exam, crepitus and symptoms of systemic toxicity (e.g. pyrexia, hypotension).

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9
Q

When might trigeminal neuralgia warrant MRI?

A

Red flag symptoms and signs that may suggest a serious underlying cause (e.g. deafness/aural involvement, optic neuritis, family history of multiple sclerosis, age of onset under 40 years).

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10
Q

Treating GCA with visual compromise?

A

Treatment with 60mg oral prednisolone stat and urgent referral to rheumatology and ophthalmology to be seen same working day

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11
Q

Initial management of an anyphlactic reaction?

A

Give 0.5ml of 1:1000 adrenaline IM

Consider half dose if taking amitriptyline, beta blockers, or imipramine

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12
Q

Initial management of choking adult?

A

. Stand behind them and slightly to one side & support their chest with one hand & lean
them forward so the object blocking their airway will come out of their mouth.

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13
Q

Management of acute angle glucoma in community?

A

Oral acetazolamide and contact opthalmology immediately for urgent admission
Lie supine

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14
Q

How does acute angle gluacoma present?

A

Hyperacute visual changes
Painful red eye - unilateral
Pupil fixed and dilated

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15
Q

If a patient presents in primary care with a single TIA (resolved) in the last week what action should be taken?

A

300mg loading dose aspirin (if not already on oral anticoagulation)
Refer for specialist review within 24hrs

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16
Q

If a patient presents in primary care with a single TIA (resolved) 7-10 days ago what action should be taken?

A

300mg loading dose aspirin (if not already on oral anticoagulation)
Refer for specialist review within 7 days

17
Q

If a patient presents with an un-resolved TIA or multiple TIAs in 7 days what action should be taken by GP?

A

Urgent admission to hospital

300mg aspirin loading dose if not already on anticoagulation