After the Stroke - Brancamp Flashcards

(56 cards)

1
Q

What is cerebral plasticity?

A

The brain’s potential for reassigning to a different brain region functions that are lost when brain tissue is damaged. This diminishes with age.

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2
Q

Why is the severity of the persisting consequences of brain injury increased with age?

A

Because cerebral plasticity decreases with age.

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3
Q

Describe some characteristics of aphasia.

A
  1. is neurogenic
  2. is acquired
  3. involves language problems
  4. is not a problem of sensation, motor function or intellect
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4
Q

What is Aphasia?

A

An impairment of the ability to comprehend and formulate language; a multi modality disorder represented by a variety of impairments in auditory comprehension, visual and reading comprehension, oral-expressive language and writing.

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5
Q

Aphasia is not…….?

A
  1. a disturbance of articulation
  2. a product of mutism
  3. a product of aphonia
  4. language disorder experienced in altered states of awareness
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6
Q

What is aphonia?

A

Lack of ability to phonate sound at the level of the vocal cord or larynx.

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7
Q

What is dysarthria?

A

A collective name for a group of speech disorders resulting from disturbances in muscular control over the speech mechanism due to damage of the CNS or PNS. It designates problems in oral communication due to paralysis, weakness or incoordination of speech musculature.

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8
Q

What is apraxia?

A

A neurogenic speech disorder resulting from impairment of the capacity to program sensorimotor commands for positing and movement of muscles of the volitional production of speech. It can occur without significant weakness or neuromuscular slowness and in the absence of disturbances of conscious thought or language.

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9
Q

What is agnosia?

A

lack of knowing

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10
Q

What is dyscalculia?

A

Impaired ability to do math.

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11
Q

What is alexia/dyslexia?

A

Impairment in reading.

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12
Q

What is agraphia?

A

Impairment in writing.

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13
Q

Where is Broca’s area (area 44)?

A

Inferior frontal lobe.

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14
Q

Describe the pathway when someone hears something spoken and then formulates a reply.

A

Info comes into Wernickes area (mostly on the left side) and then it goes through the arcuate fascicles to broca’s.

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15
Q

Condiction aphasia is a problem with what structures?

A

The arcuate fasciculus.

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16
Q

What is the function of Broca’s area?

A

Motor programming for articualtion.

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17
Q

What is the function of the pre central gyrus for language?

A

Activation of muscles for articulation.

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18
Q

What is the function of the arcuate fasciculus in language?

A

Transmission of linguistic info to anterior areas from posterior areas.

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19
Q

What is the function of Wernickes area for language?

A

Comprehension of oral language.

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20
Q

What is the function of the angular gyrus in language?

A

Integrates visual, auditory, and tactile info and carries out symbolic integration for reading. Located in the left parietal lobe.

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21
Q

What is the function of the supra marginal gyrus in language?

A

Symbolic integration for writing.

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22
Q

What is the function of the corpus callosum in language?

A

Transmission of information between the hemispheres.

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23
Q

What is PWA and what are some characteristics of it?

A

Posterior Wernicke’s Aphasia.
A person with aphasia (PWA) produces some speech, or even abundant speech, which does not conform to the grammatical rules of the language being used
Errors include omission of words, erroneous choice of words, errors in word order.
During the first hours or days of onset, the aphasic person may not produce speech, but most will attempt to communicate by gesture or facial expression.

24
Q

A person with PWA has difficulty with what?

A

A PWA often has difficulty comprehending a purely verbal command.
Verbal messages through auditory or visual means without gestures, facial expressions or emotional intonation.
Errors range from almost complete inability to comprehend a simple command to mild deficits with complex sentences.

25
Does a person with PWA have awareness of their environment?
A PWA is aware of self and environment (they are not confused, demented or psychotic), has an appropriate level of awareness. He/she is intent on communicating thoughts regarding his/her condition and environment. Exceptions are possible during the first few hours after acute infarcts and late in the chronic stages for a few.
26
General characteristics of speech fall into what two general categories?
FLUENT – approximates normal speech in terms of the rate of word production, length of sentences, melodic intonation, and ease in speaking [9+ words/utterance] NONFLUENT-rate is slow, sentence length is short, melodic contour is lost, production is effortful, may be more pauses than actual words. [0-5 words/utterance].
27
What is an utterance?
A group of words that is said on one breath.
28
What are some signs of aphasia?
Failure to repeat words or sentences is a hallmark of aphasia; ability may be completely lost or characterized by phonemic paraphasias or omissions of sounds/words. Presence of impaired repetition has localization value – perisylvian region of the dominant hemisphere
29
Give an example of a phonemic paraphasia.
A phenome is a sound disruption. For example saying boy when you wanted to say toy.
30
Where is the zone of language in the brain?
In the perisylvian region.
31
Describe some disturbances of auditory comprehension that may occur with aphasia.
Ability to decode auditory input Auditory comprehension rarely preserved in aphasia Deficits can range from inability to understand: single words sentences multiple-step commands narrative discourse
32
What are some disturbances of grammatical processing that may occur with aphasia?
AGRAMMATISM refers to difficulty ordering words and putting them together in a sentence. Content words are relatively spared Agrammatic patients have difficulty producing function words and bound morphemes (problems with tense).
33
What are some disturbances of reading and writing that may occur with aphasia?
Patients with auditory comprehension deficits usually have some reading impairment Reading impairment can appear in a pure form without auditory comprehension or writing impairment. In most cases of aphasia, reading, writing, and auditory comprehension are impaired together, but not to the same degree.
34
What are some characteristics of Broca's aphasia?
``` Dysfluent Increased effort in speaking Speech is agrammatic, telegraphic, groping Often accompanied by dysarthria and/or apraxia of speech Comprehension better than expressive Repetition abnormal Naming is poor Writing reflects spoken output ```
35
Are patients with Broca's aphasia aware of their deficit?
Yes, they are: very aware of their communication and physical impairments easily upset by failed communication attempts cooperative and task oriented
36
What are some characteristics of Wernicke's aphasia?
Pt. usually appears normal with no dysarthria or limb weakness/paralysis. Speech is fluent but often presents with paraphasias and jargon Syntax is normal Comprehension is poor Reading is poor Repetition is poor. Word-salad
37
What are some characteristics of patient's with Wernicke's aphasia?
Most patients are alert, attentive and task-oriented Mild Wernicke’s: aware of errors and generally follow conversational turn taking Moderate: rarely notice errors or attempt repairs; attentive & cooperative but get off topic easily (verbal tangents) Severe: attentive, but severe comprehension deficits interfere with all communication tasks; unaware of errors and communication failure
38
Describe global aphasia.
Severe impairment in both comprehension and expression Concrete understanding of symbols; may not fully comprehend abstract or inferential information May not understand simple words, may only understand icons or pictures Performance usually enhanced by materials or activities that are automatic or emotionally laden
39
What are some characteristics of patients with global aphasia?
Speech is severely limited and often characterized by stereotypical utterances (whatawhy), overlearned phrases (how-de-do), or expletives. Most are attentive, task-oriented and socially appropriate (which helps to differentiate from the confused or demented patient). Occasionally comprehend personally relevant questions fairly well Some reliably answer yes/no questions
40
Describe conduction aphasia.
SOL is thought to be arcuate fasciculus or temporal lobe in the auditory association area Fluent speech; auditory comprehension deficits range from moderate to mild Hallmark: inability to repeat long sentences - grossly impaired repetition & relatively preserved language comprehension. Prosody may be affected because of pauses and hesitations Alert, attentive and task-oriented; aware of errors in speech and writing; attempts repairs (‘Why can’t I say that?’)
41
What is prosody?
The ability to have vocal inflection and affect in speech.
42
Describe transcortical aphasia.
Preserved repetition is defining characteristic of the transcortical aphasias (Wernicke’s, Broca’s and arcuate fasciculus are spared) Lesions are outside of the perisylvian zone Transcortical Motor Aphasia Transcortical Sensory Aphasia Mixed Transcortical Aphasia
43
Describe anomic aphasia.
Anomia – word finding difficulties; is common in aphasia and other neurologic disorders (dementia, encephalopathy). Often the residual impairment of a more severe aphasia Spontaneous speech is fluent and grammatically correct, but marred with frequent word-retrieval failures. Average phrase length WNL Auditory comprehension & repetition are relatively intact
44
Cardiovascular events are the leading cause of what?
Right hemisphere disorders.
45
Will all adults with right hemisphere disorders have cognitive/communication deficits?
No. 50% of all patients with RHD 80-90% of patients in rehab Diagnosis depends on who does the evaluation
46
What are some communication deficits associated with right hemisphere damage?
1. deficits in facial recognition -prosopagnosia 2. deficits in comprehending facial expressions and expressing using facial expressions 3. prosodic deficits 4. inferencing deficits 5. discourse deficits
47
What are some neuropsychiatric deficits associated with right hemisphere damage?
1. anosognosia - lack of self awareness 2. depression 3. capgras delusion - patient believes a loved one or pet has been replaced by an identical imposter 4. visual hallucinations 5. paranoid hallucinations
48
What are some other deficits associated with right hemisphere damage?
1. Visuoperceptual deficits include simultagnosia (cannot perceive more than one object at a time) and cerebral achromatopsia (color-blindness due to brain damage) 2. Attentional deficits include neglect and sustained and selective attention deficits
49
What are some things that patients with right hemisphere disorder do not have trouble with?
Few if any problems in comprehending or expressing language syntax (grammar), morphology (eg, word endings), and/or phonology (speech sounds) Do not have classical aphasia lexical retrieval (coming up with words they want to say) is like that of age-matched healthy individuals Trouble with naming pictures/objects may be due to visual-perceptual or visuospatial difficulties not comprehension
50
Describe neglect with respect to brain damage.
1. May be called -Hemispatial, left or visuospatial neglect, hemineglect 2. Inattention to the left side 3. Occurs in 13-80% of patients with RHD 4. Lesion localization: Inferior parietal lobe; parieto-temporal juction Inferior frontal, underlying white matter, thalamus, basal ganglia
51
What are the 3 types of neglect?
1. Motor Neglect Reduced use of left side of body (less than capable) 2. Tactile Neglect Reduced response/recognition of tactile stimulation (greater than sensory deficit) 3. Auditory Neglect Reduced processing of auditory stimuli from left – poorer localization of sounds
52
What is the course of neglect with relation to right hemisphere damage?
Variable performance – hour to hour, day to day; within and across neglect types Often resolves within first few weeks Usually accompanied by anosognosia Presence of neglect - more likely to have other cognitive/communicative deficits
53
What is neglect dyslexia?
When the person omits letters/words, substitutes letters, parts of words or words on a line.
54
What is neglect dysgraphia?
When the person writes only on the lefts side to the page but has preserved letters/words.
55
What is aprosodia?
1. Production: flat, monotone speech, rate sounds fast 2. Comprehension: problems interpreting prosody 3. RHD possibly effects emotional prosody more than linguistic prosody
56
Prosody and affective disorders are characterized by what?
1. Impaired emotion & nonverbal communication 2. Production: Use fewer emotionally-charged words Reduced facial expressions Reduced animation Flat affect – reduced animation, reduced prosody/intonation 3. Comprehension: Misinterpretation of emotional cues (e.g., facial expression, body language, intonation)