all heart sounds and murmurs Flashcards

(44 cards)

1
Q

cause of heart sounds in general

A

turbulent blood flowtensing of cardiac structuresvalve closure

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2
Q

cause of S1 heart sound

A

AV (tricuspid and mitral) valve closure

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3
Q

what phase of cardiac cycle is S1

A

start of systole - isovolumic contraction

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4
Q

phases of the cardiac cycle

A

isovolumic contraction - period between which AV valves shut, SL valves open; ventricles contracting in a closed circuit
rapid ejection - opening of SL valves; c wave in JVP
reduced ejection - slowing of ejection ultimately causing closure of SL valves (aortic and pulmonary trunk pressures >ventricular pressure)isovolumic relaxation - relaxation of ventricles when both AV and SL valves are shut; V wave in JVPrapid ventricular filling - AV valves open causing rapid filling; y descent in jvpdiastasis - as filling of ventricles nears ~80%atrial systole - atria contract to top of the ventricles with last amount of blood; a wave in jvp

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5
Q

cause of S2 heart sound

A

SL (pulmonary and aortic) valve closure

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6
Q

what phase of cardiac cycle is S2

A

end of systole/beginning of diastole - isovolumic relaxation

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7
Q

cause of S3 heart sound

A

volume overload
tensioning of chordae tendinae at the end of rapid ventricular filling
sudden deceleration of blood against LV

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8
Q

cause of S4 heart sound

A

pressure overload

reflection of atrial wave against a poorly compliant ventricle (thickened or stiff)

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9
Q

pathologies where S4 is seen

A

LV hypertrophy secondary to HTN
aortic stenosis,acute MR
IHD, age +++
angina, MI sometimes - only thing seen acutely

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10
Q

pathologies where S3 is seen

A

normal in young people and athletes
in older people - congestive heart failure
physiological - thyrotoxicosis, pregnancy

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11
Q

symptoms of MS

A

dyspnea, orthopnea, PND - increased left atrial pressure
hemoptysis - ruptured bronchial veins
ascites, edema, fatigue - pulmonary HTN, decrease CO

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12
Q

signs of MS

A

mitral facies

peripheral cyanosis

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13
Q

pulse of MS

A

normal or reduce in volume - reduced CO

AF may be caused if left atrial enlargement

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14
Q

JVP in MS

A

normal height
prominent a wave if pulmonary HTN present
loss of a wave if AF

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15
Q

palpation in MS

A

tapping apex beat
RV heave
palpable P2

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16
Q

auscultation in MS

A

T/C - mid diastolic low pitched rumbling diastolic murmur
R - nil
loud S1 - valve cusps remain wide open at onset of systole
loud or palpable P2 if pulmonary HTN
opening snap - high LA pressure forces valve open

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17
Q

accentuation manoeuvres in MS

A

exercise

left lateral position

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18
Q

causes of MS

A

RHD

congenital parachute valve

19
Q

MR symptoms

A

dyspnea - increased LA pressure

fatigue - decreased CO

20
Q

MR signs

21
Q

MR pulse

A

normal or sharp upstroke due to rapid ventricular decompression
AF relatively common

22
Q

JVP in MR

23
Q

palpation in MR

A

displaced, diffuse and hyperdynamic apex beat
pansytolic thrill occasionally present at apex
parasternal heave due to LA enlargement behind the RV

24
Q

auscultation in MR

A

T/C - pansytolic murmur maximal at apex and usually radiating towards axilla
soft or absent S1-
LV S3 due to rapid LV filling in early diastole

25
MR accentuation manoeuvres
valsalva - longer and louder
26
causes of MR
mitral valve prolapse degeneration associated with age RHD papillary muscle dysfunction due to LV failure or ischemia cardiomyopathy - hypertrophic, dilated or restrictive CT diseases congenital
27
AR symptoms
occurs in late stage of disease exertional dyspnea, fatigue, exertional angina - decreased CO palpitations - hyperdynamic circulation
28
AR signs
none specifically but marfanoid features Ankolysing spondylitis or other seronegative arthropathies
29
AR pulse and BP
collapsing pulse 'water hammer' - most obvious if raising patient's arm while feeling radial pulse wide pulse pressure - DBP very low as leaking back constantly, SBP normal bisfiriens pulse - severe AR - venturi effect - rapid ejection and brief in drawing of aortic wall leading to a diminution of the pulse followed by rebound increase
30
AR JVP/neck
prominent carotid pulsations
31
AR palpation
displaced and dyskinetic apex beat | diastolic thrill may be felt at LSE if sitting up and exhaling
32
AR auscultation
T/C - early diastolic, decrescendo quality, high pitched. may extend for variable time into diastole loudest at 3/4 ICS soft A2 component systolic ejection murmur usually also present - aortic stenosis or turbulent flow across normal diameter aortic valve
33
AR accentuation manouvres
expiration and leaning forward
34
acute AR causes
valvular - IE | aortic root - marfan's syndrome - aortic dissection
35
chronic AR causes
``` valvular RHD (rarely the only murmur if so) congenital (bicuspid valve, VSD) seronegative arthropathy esp ankolysing spondylitis aoritc root dilation marfan's aortitis rheumatoid arthritis teritary syphilis dissecting aneurysm ```
36
differentiate acute AR to chronic AR
acute - no collapsing pulse (BP is low) and diastolic murmur is short
37
AS symptoms
exertional chest pain (50% no CAD) | exertional dyspnea and exertional syncope
38
AS signs
usually nothing remarkable
39
AS pulse
``` plateau pulse (anacrotic) OR late peaking and low volume (tardus et parvus) ```
40
AS JVP
n/a
41
AS palpation
hyperdynamic apex beat may be slightly displaced systolic thrill at base of heart (in aortic area)
42
AS auscultation
T/C - harsh (mid)systolic ejection murmur, crescendo - decrescendo R - maximal over aortic area and extending into carotids, may extend widely over precordium and extend to apex narrowly split or reversed S2 - delayed LV ejection
43
AS accentuation manouvres
sitting up and in full expiration | note - associated aortic regurg is common
44
AS causes
degenerative calcific aortic stenosis - elderly calcific in younger patients - congenital bicuspid valve rheumatic