Alveolar Bone and PDL Diseases Flashcards

(58 cards)

1
Q

Specialized mineralized connective tissue

A

Alveolar Bone

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2
Q

What is the Alveolar Bone composed of?

A

Intracellular substances and osteocytes

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3
Q

Structures in the Alveolar Bone ACT

A
  1. Alveolar Bone Proper
  2. Compact Bone
  3. Trabecular Bone
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4
Q

What forms the wall of the alveolar socket?

A

Alveolar Bone Proper

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5
Q

What is the Alveolar Bone Proper in a radograph?

A

Lamina Dura

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6
Q

What covers the bone and contains the alveolar process?

A

Compact Bone

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7
Q

Thickness of the Compact bone

A

0.1-0.4mm

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8
Q

What are the components of the Compact Bone? (7) PCCHIVE

A
  1. Periosteum
  2. Circumferential Lamellae
  3. Concentric Lamellae
  4. Interstitial Lamellae
  5. Haversian Canal (BV)
  6. Volkmann’s Canal
  7. Endosteum
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9
Q

A spongy or cancellous bone

A

Trabecular Bone

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10
Q

Where is the trabecular bone situated?

A

It occupies the space between the compact bone and alveolar bone proper

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11
Q

In the Maxillary Alveolar Process, bone is thicker on which side?

A

Palatal

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12
Q

In the Mandibular Alveolar Process, bone is thicker on which side?

A

Buccal

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13
Q

An area without bone coverage in the marginal portion

A

Dehiscense

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14
Q

Bone is present in the most coronal portion of the buccal but the defect is located more apically

A

Fenestration

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15
Q

What are the cells in the Alveolar Bone?

A
  1. Osteoblasts
  2. Osteoclasts
  3. Osteocytes
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16
Q

What is an Osteoid?

A

Bone lining cells that have been inactive; Unmineralized portion that forms prior to bone maturation

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17
Q

Mononucleated cells

A

Osteoblasts

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18
Q

Osteoblast is responsible for..?

A

Mineralization and formation of the bone

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19
Q

Osteoblasts synthesize the following: (2)

A
  1. Type I & V Collagen
  2. Cytokines & Growth factors
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20
Q

Entrapped osteoblasts in the lacunae

A

Osteocytes

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21
Q

What happens to osteocytes after bone formation?

A

It loses the ability to form matrix & becomes smaller

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22
Q

Bone resorbing cells

A

Osteoclasts

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23
Q

Osteoclasts releases what? AP

A

Acids & Proteolytic enzymes

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24
Q

Sequence of Resorptive Events (Osteoclast) ACRO

A
  1. Attachment of osteoclasts to bone
  2. Creates sealed environment
  3. Releases acid to demineralize the hard tissue
  4. Organic matrix is degraded by secretion of proteolytic enzymes
25
Coordinated Functions of the Periodontium TAHD
1. Turnover 2. Adaptation 3. Healing 4. Defense
26
Also known as “Tissue homeostasis”
Turnover
27
What is Adaptation?
Deposition and/or resorption occurs constantly in healthy tissue
28
Examples where a tooth is in hypofunction, afunction
No contact with opposite tooth
29
Adaptation in cases of reduced occlusal loading
Afunction, Hypofunction
30
Adaptation during increased occlusal loading
Hyperfunction, Parafunction
31
Refers primarily to the immune system and response of healthy tissues
Host Defense
32
Refers to the adaptability of the tissues; Ability to vary the rate of turnover in response to various mediators
Healing
33
Oral/Gingival Epithelium turnover rate
6 to 40 days (depends on the need of the epithelium)
34
PMN
Polymorphonuclear Neutrophils or Neutrophils
35
Coronoapical portion of JE
1-3mm
36
Where does cell division occur in JE
Basal Cell Layer
37
Turnover rate of JE
4-6 days
38
Does cementum undergo turnover?
No, only continuous deposition throughout life
39
Mechanism for protection against all mechanical challenges in the epithelium
Keratinization
40
GCF
Gingival Crevicular Fluid (tissue fluid flow)
41
What does the JE produce?
Epithelial attachment
42
Turnover of the GCT is mediated by..?
Cytokines & Growth factors (inflammatory mediators)
43
What is responsible for the synthesis and breakdown of collagen matrix?
Fibroblasts
44
Goal of Periodontium
Achieve homeostasis
45
Disease that is limited to the marginal, supracrestal soft tissues
Gingivitis
46
Characterized by plaque-induced inflammation of the papillary and marginal gingiva
Gingivitis
47
Clinical symptoms of Gingivitis (3)
1. Bleeding 2. Erythema 3. Swelling
48
In gingivitis, is the AB and PDL involved?
No
49
Treatment for Gingivitis (2) ApcM
1. Adequate plaque control 2. Maintenance
50
Inflammation of the gingiva extending into the deeper structures of the tooth-supporting apparatus
Periodontitis
51
Quality of Plaque
Type of bacteria present in the plaque
52
Anaerobic
Without oxygen
53
Aerobic
With oxygen
54
Risk factors that may progress a gingivitis case to periodontitis case:
1. Use of drugs 2. Smoking 3. Diabetes (systemic diseases) 4. Dry mouth 5. Syphilis 6. Poor oral hygiene 7. Bruxism
55
Inflammation-free apical migration of the gingival margin
Gingival Recession
56
Best way to prevent gingival recession
Gentle oral hygiene technique
57
Treatment options for Periodontitis CRR
1. Close or Open Root Planing 2. Regenerative Surgical Therapies 3. Resective Surgical Therapies
58
Rate of progression of Periodontitis may depend on: (5) QSPIR
1. Quality and quantity of plaque 2. Systemic health of the patient 3. Patient’s genetic constitution 4. Immune response 5. Risk factors