Anomolies Flashcards

(58 cards)

1
Q

what tissues are interacting in week 6 of tooth development?

A

epithelium and ectomesenchyme

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2
Q

when is bud stage?

A

week 8

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3
Q

what stage of tooth development results in disruption that is seen on clinic?

A

bud stage at week 8

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4
Q

when is cap stage?

A

week 11

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5
Q

when do layers form in tooth development?

A

cap stage at week 11

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6
Q

when is bell stage?

A

week 14

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7
Q

what are the 5 classifications of anomalies?

A

quantity
size and form
formation disturbances
hereditary disturbances of structure
other

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8
Q

how do you differentiate developmental and environmental anomalies?

A

Developmental – affect all the teeth as they require the same gene expression

Environmental – will only affect teeth that were developing at the time when the environmental factor was an issue

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9
Q

what are the 3 quantitative developmental disorders?

A

anodontia/ hypodontia
hyperdontia

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10
Q

what is hypodontia?

A

missing teeth due to failing to develop

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11
Q

what are the 2 types of hypodontia?

A

anodontia
oligodontia

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12
Q

what is anodontia?

A

total lack of teeth in one or both dentitions

  • no alveolar bone
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13
Q

what is oligodontia?

A

more than 6 primary or permanent teeth are absent

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14
Q

aetiology of hypodontia?

A

genetic, environmental or both

sometimes single-gene disorders

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15
Q

what syndromes is hypodontia associated with?

A

trisomy 21 (down syndrome)
ectodermal dysplasia

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16
Q

what do people with ectodermal dysplasia not have?

A

sweat glands

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17
Q

what are supernumerary teeth?

A

additional teeth

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18
Q

where do you find mesiodens?

A

midline
peg shaped teeth

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19
Q

what do supplemental teeth look like?

A

normal teeth

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20
Q

what is a risk associated with conical supernumeraries?

A

if they dont erupt they could migrate towards the nose

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21
Q

what skeletal dysplasia disorder may lead to supernumerary teeth?

A

cleidocranial dysostosis

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22
Q

what gene switches on and allows teeth to develop?

A

MSX1

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23
Q

what are the size and shape anomalies?

A

microdontia
macrodontia
dens en dente
talon cusp

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24
Q

what is microdontia?

A

one or more teeth appear smaller than normal, including peg shaped teeth

25
what is macrodontia?
teeth are larger than normal
26
what are forms of macrodontia?
double teeth dens en dente (dens invaginatus) talon cusps (dens envagintus)
27
what are the 2 causes of macrodontia?
fusion gemination
28
what is fusion?
union of 2 tooth buds
29
what is gemination?
single root 2 crowns
30
what is dens en dente?
localised area where the crown is folded inwards
31
what is clinically important about dens en dente?
caries can lead to pulpitis and periapical infection
32
what are talon cusps?
adjunct cusp arises from the cingulum or CEJ in anterior teeth
33
what are types of structural developmental anomalies?
hereditary: amelogenesis imperfecta, dentinal dysplasia, dentinogenesis imperfecta non hereditary: MIH, fluorosis, turners tooth
34
what is amelogenesis imperfecta?
defect in function of ameloblast and mineralisation of enamel matrix
35
what are the 2 types of amelogenesis imperfecta?
hypoplastic hypomineralised
36
what does hypoplastic amelogenesis imperfecta present as?
thin but hard enamel; normal bond strength
37
aetiology of hypoplastic amelogenesis imperfecta?
teeth different shape before eruption
38
what does hypomineralised amelogenesis imperfecta present as?
full thickness enamel but soft; impaired bond strength
39
aetiology of hypomineralised amelogenesis imperfecta?
breakdown occurs after tooth erupts
40
how is amelogenesis imperfecta inherited?
autosomal dominance
41
what are dental defects associated with amelogenesis imperfecta?
pulp calcification taurodontism delayed eruption gingival overgrowth skeletal anterior open bite
42
what are the types of hypomineralised amelogenesis imperfecta?
hypocalcified hypomature
43
what does hypomature AI present as?
chalky orange/brown/white
44
what does hypocalcified AI present as?
enamel only persists on cervical part
45
what is dentinogenesis imperfecta?
autosomal dominant inherited condition
46
what is type I DI?
associated with osteosis imperfecta - less severe
47
what is type II DI?
DI on its own - more significant
48
what is type III DI?
"Brandywine isolate"
49
how is DI normally managed?
bisphosphonates
50
what does DI present as in the primary dentition?
amber attrition pulp obliteration spontaneous abscesses
51
what does DI present as in the permanent dentition?
grey/translucent short root pulp obliteration spontaneous abscesses
52
what is MIH?
developmental defect in enamel structure with less mineral content due to disruption to the maturation/ late transitional stage of amelogenesis
53
what teeth are most affected by MIH?
1st permanent molars and central incisors
54
what can MIH appear as?
demarcated enamel opacities ranging in colour hypoplastic breakdown of teeth
55
what does fluorosis arise from?
ingestion of high fluoride concentrations during amelogenesis
56
what is turners tooth?
area of hypoplastic enamel following infection around the interradicular area of a primary tooth
57
what teeth are usually affected by turners tooth?
successional tooth - often 2nd premolars
58
what is it called when a root has an irregular bend?
dilacerated root