applied anatomy of the heart Flashcards

(62 cards)

1
Q

what are the borders of the heart?

A
  • Upper right: 3rd costal cartilage
  • Upper left: left 2nd costal cartilage
  • Lower right: 6th costal cartilage
  • Lower left: 5th intercostal space midclavicular line
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2
Q

what is the cardiac plexus made up of?

A

vagus nerve
sympathetic nerves
general visceral afferent nerves

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3
Q

where does the vagus feed into the heart and what effect does it have on the heart?

A

feeds into the SAN

parasympathetic - slows down HR

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4
Q

where do sympathetic nerves from the heart enter the spinal cord?

A

T1-T5

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5
Q

what effect do sympathetic nerves have on the heart?

A

increase the rate of firing

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6
Q

where do general visceral afferents feed into?

A

spinal levels T1-T5

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7
Q

where is referred heart pain felt and why?

A

dermatomes T1-T5

brain can’t differentiate between GVA and somatic nerves bc they enter the spinal cord at the same spinal level

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8
Q

what are the branches of the right coronary artery?

A

posterior interventricular/descending artery

marginal branch

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9
Q

what are the branches of the left coronary artery?

A

circumflex branch
left marginal branch of circumflex artery
left anterior descending

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10
Q

what artery supplies the SAN?

A

right coronary artery

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11
Q

what artery supplies the AVN?

A

right coronary artery

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12
Q

what arteries supply the bundle branches?

A

interventricular (descending) arteries

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13
Q

what is a right dominant heart?

A

posterior descending artery is supplied by the RCA which supplies the myocardium of the inferior 1/3 of the interventricular septum

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14
Q

what is a left dominant heart?

A

PDA supplied by the LCA. All of the IV septum supplied by the LCA

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15
Q

what is the risk in a left dominant heart?

A

LCA blockage means both bundle branches have their blood supply cut off

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16
Q

what happens as a result of bundle branch block?

A
  • bundle branch becomes ischaemic
  • doesnt conduct impulses properly
  • use altered pathways for depolarisation (cardiac myocytes conduct the impulses instead of using the bundle branches)
  • slows impulse speed
  • prolongs QRS
  • loss of ventricular synchrony
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17
Q

what are the 3 most common places for stenosis of coronary arteries?

A

left anterior descending
right coronary artery
circumflex artery

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18
Q

how long does it take for severe ischaemia to register on an ECG?

A

minutes

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19
Q

how many electrodes and perspectives are there on an ECG?

A

10 electrodes

12 perspectives

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20
Q

how many chest leads are there and how many perspectives do they provide?

A
6 chest leads
6 perspectives (horizontal plane)
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21
Q

how many limb leads are there and how many perspectives do they provide?

A

4x limb leads

6x perspectives

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22
Q

what leads give a lateral view of the heart?

A

I, aVL, V5 and V6

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23
Q

what leads give an anterior view of the heart?

A

V3, V4

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24
Q

what leads give a septal view of the heart?

A

V1, V2

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25
what leads give an inferior view of the heart?
II, III and aVF
26
what leads correspond with the right coronary artery?
main artery on the inferior surface | leads II, III, aVF
27
what leads correspond with the left anterior descending artery?
main artery on the anterior surface | V3, V4
28
what leads correspond with the circumflex?
main artery on the lateral surface | I, aVL, V5, V6
29
where are the heart valves located?
o Aortic – upper right 3rd costal cartilage o Pulmonary – upper left 2nd costal cartilage o Tricuspid – lower right 6th costal cartilage o Mitral – lower left 5th intercostal space midclavicular line
30
where do you auscultate the heart valves?
o Aortic – right 2nd intercostal space o Pulmonary – left 2nd intercostal space o Tricuspid – 6th intercostal space o Mitral – 5th intercostal space midclavicular line
31
what causes the characteristic lub dub noises of the heart?
``` o Lub (S1) when mitral valves and tricuspid close (systole) o Dub (S2) when aortic and pulmonary valves close (diastole) ```
32
what 2 things can go wrong with the heart valves?
stenosis | regurgitation
33
what is stenosis?
when the valves dont open properly
34
what is regurgitation?
when the valves dont close properly
35
what type of murmur is mitral regurgitation?
systolic murmur
36
what causes mitral regurgitation?
Mitral valve doesn’t close properly – can hear when blood passes back through valve into the atria
37
what does mitral regurgitation sound like?
pansystolic | often louder in late systole
38
where is mitral regurgitation heard?
heard at the apex
39
what is the most common form of valvular heart disease?
mitral regurgitation
40
what type of murmur is aortic stenosis?
systolic murmur
41
why do you hear turbulence in aortic stenosis?
as blood has to be pushed through stenotic aortic valve
42
describe the different severities of aortic stenosis throughout systole and why this occurs
less severe in early systole more severe in late systole bc time taken to generate pressure to pass through stenotic valves
43
where is aortic stenosis heard?
right second intercostal space
44
what does mitral stenosis sound like?
rarely produces a soft rumbling diastolic murmur
45
what does aortic regurgitation sound like?
complex and often absent
46
what are the further complications of aortic regurgitation?
decreased cardiac output bc of regurgitation elevates preload and afterload LVH - typically very dilated
47
what causes change in heart size
- changes based on workload | - workload can be physiological (athletes, pregnancy) or pathological (valvular disease, atrial fibrillation)
48
define preload
volume of blood in ventricles at the end of diastole
49
define afterload
resistance ventricle must overcome to circulate blood
50
what does preload increase in?
hypervolemia, valve regurgitation, heart failure
51
what does afterload increase in?
(chronic) hypertension, vasoconstriction, valve (aortic) stenosis, outflow stenosis
52
what is concentric hypertrophy?
hypertrophic growth of a hollow organ without overall enlargement – walls thicken, and capacity/volume are reduced
53
how does concentric hypertrophy occur?
increased afterload bc of aortic stenosis or chronic hypertension increased resistance wall thickness increases to overcome resistance - new sarcomeres
54
what are the disadvantages of wall thickness increasing?
compliance is reduced (stiff) | ventricular filling is compromised (diastolic dysfunction)
55
what is eccentric hypertrophy?
dilation of the left ventricular chamber (normal response to healthy exercise)
56
what causes eccentric hypertrophy?
o Aortic and mitral regurgitation o Systolic dysfunction (loss of cardiac inotrophy) o Volume overload (hypervolaemia due to ventricular or renal failure) o Others eg alcohol cocaine
57
what causes atrial enlargement?
in response to mitral or tricuspid valve pathology
58
what is atrial remodelling?
any persistent change in atrial structure
59
what is the link between atrium size and the risk of atrial fibrillation and why?
larger the atrium the higher the risk of AF bc you're disrupting the electrical conduction pathways
60
how is atrial fibrillation diagnosed?
absent P waves on an ECG reduced cardiac output thrombi and syncope
61
how is left atrium enlargement seen on an x-ray?
seen as a double density over the right atrium
62
how is left ventricular enlargement seen on an x-ray?
heart looks like a boot