ARDS Flashcards

(35 cards)

1
Q

ARDS definition

A

Rapid onset of noncardiac pulmonary edema

Progressive refractory hypoxemia

Extensive lung tissue inflammation

Small blood vessel injury

Multisystem organ failure

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2
Q

Pathophysiology of ARDS

A

Acute lung injury

Unregulated systemic inflammatory response

Damaged capillary membranes leak

Damage alveolar membrane

Fluid enters alveoli

Significant tissue hypoxia results

Metabolic acidosis

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3
Q

Risk Factors for ARDS

Direct Insults

A

Direct insults

Aspiration of gastric contents
Inhalation injuries
Smoke inhalation
Salt water inhalation

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4
Q

Risk Factors for ARDS

Indirect Insults

A

Indirect insults

Overall body sepsis
Trauma
Gastrointestinal infections

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5
Q

The greatest risk factor for ARDS is

A

Aspiration

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6
Q

Initial manifestations of ARDS begin

A

24–48 hours post insult

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7
Q

Early signs of ARDS

A

Dyspnea and tachypnea are early signs

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8
Q

S/S of ARDS

A

Progressive respiratory distress
Hypoxia
Hypercapnia
Agitation, confusion, and lethargy

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9
Q

Diagnostic Tests for ARDS

A

ABG analysis to determine oxygen levels

Chest x-ray to determine fluid in lungs

Blood tests such as CBC, blood chemistry, and blood cultures

Sputum culture to determine cause of infection

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10
Q

Pharmacological Therapy

A

No definitive drug therapy

Nitric oxide

  • Reduces intrapulmonary shunting
  • Improves oxygenation

Surfactant therapy

NSAIDS and corticosteroids are being studied

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11
Q

Nonpharmacologic Therapy

A
Mechanical ventilation
Artificial airways
Proper nutrition
Adequate amounts of fluids
Other clinical therapies
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12
Q

The mainstay of ARDS management is

A

Mechanical Ventilation

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13
Q

Types of ventilators
Negative vs Positive pressure
Noninvasive

A

Negative-pressure ventilators
*Create negative pressure externally to draw chest outward and air into lungs

Positive-pressure ventilators
*Push air into lungs

Noninvasive ventilation (NIV)

  • Uses face mask
  • Sleep apnea
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14
Q

ARDS

What is happening?

A

A: Atelectasis
R: Refractory hypoxemia (hallmark sign)
D: Decreased lung compliance
S: Decreased surfactant

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15
Q
Complications of Mechanical Ventilation
Why?
HAP
Barotrauma, pneumothorax
Cardiovascular effects
GI effects
A

Hospital-acquired pneumonia (HAP): loss of humidity and filtering from upper airway
Barotrauma: PEEP
Pneumothorax: PEEP
Cardiovascular effects: Increased pressure in the chest decreases cardiac output
Gastrointestinal effects: Misplacement of tube (monitor for abdominal distention)

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16
Q

Artificial Airways
What do they do?
Oropharangeal vs Naropharangeal

A

Inserted to maintain patent air passage

Oropharyngeal airways
*Stimulate gag reflex

Nasopharyngeal airways

  • Better tolerated by alert clients
  • Frequent oral care important
17
Q

Artificial Airways

Endotracheal tubes

A

General anesthesia, emergency situations
Specialized education to insert
Guided by laryngoscope
Client unable to speak while tube in place

18
Q

Artificial Airways

Tracheostomies

A

Long-term airway support
Opening to trachea through neck
Percutaneous or surgical insertion

19
Q

Nursing Considerations

Fluids and Nutrition

A

Monitor I & O
Renal perfusion
Catheter
Arterial line

20
Q

What effect does Mechanical Ventilation have on Cardiac Output?

A

increased intrathoracic pressure can decrease CO sec return of blood to the heart. Urine Output is an early sign.

21
Q
Other Clinical Therapies for ARDS
Position?
Medications?
I/O?
Swan Ganz?
A

Prone positioning

Antibiotics
Low-molecular-weight heparin

Careful fluid replacement
Attention to nutrition

Swan-Ganz line to monitor

  • Pulmonary artery pressures
  • Cardiac output
22
Q

Weaning from ventilator support
When to start?
Why wean?

A

When: Begins after underlying process corrected

T-piece, CPAP
SIMV, PSV

Why: Reconditioning respiratory muscles
Terminal weaning

23
Q

Modes with positive pressure ventilators

A

Continuous positive airway pressure (CPAP)
Bilevel ventilator (BiPAP)
Assist-control mode ventilation (ACMV)
Synchronized intermittent mandatory ventilation (SIMV)
Positive end-expiratory pressure (PEEP)
Pressure-support ventilation (PSV)
Pressure-control ventilation (PCV)

24
Q

Ventilator settings

A

Rate, tidal volume, oxygen concentration

12–15 ventilator breaths per minute initially

25
Symptoms of Barotrauma | *Pneumothorax
Tracheal Deviation Asymmetrical Chest Rise Absent Lung Sounds Rice Crispies from air trapped
26
Aspirating secretions through a catheter When to suction? Effects on patient?
Sterile technique Open-tipped, whistle-tipped Yankauer device for oral suctioning Nursing decision to suction * Based on clinical need, not fixed schedule * Suctioning irritates mucous membranes Uncomfortable for patient Patient may feel panicked by loss of oxygen
27
Nursing diagnoses may include: ``` Risk for _____ Ineffective _____ Ineffective _____ Impaired _____ Decreased _____ Dysfunctional _____ Imbalanced _____ Acute _____ ```
Nursing diagnoses may include: ``` Risk for Acute Confusion Ineffective Airway Clearance Ineffective Breathing Pattern Impaired Gas Exchange Decreased Cardiac output Dysfunctional Ventilator Weaning Response Risk for Imbalanced Fluid Volume Imbalanced Nutrition: Less Than Body Requirements Risk for Infection Acute Pain Anxiety ```
28
Client Goals with Planning ``` Be oriented _____ Receive adequate _____ Be free of ______ Maintain _____ Maintain _____ Receive adequate _____ Be free of _____ Have no development of _____ Manage _____ Cope with or be free from _____ ```
Goals may include that client will: ``` Be oriented with each interaction Receive adequate ventilatory support Be free of pulmonary tissue damage Maintain patent airways Maintain adequate cardiac output Receive adequate nutrition Be free of signs, symptoms of infection Have no development of thrombosis Manage pain successfully Cope with or be free from anxiety ```
29
Common Interventions Monitor-
Lab work and specimens Suction airways as needed Monitor * Vital signs hourly * Oxygenation status * Neurological status * Lung and heart sounds
30
Position Interventions
Maintain HOB at 30° or higher Prone position as tolerated 3–4x/day
31
Common Medication Interventions
Provide analgesia, anxiolytics, sedation Beta-agonist to maintain patent airways
32
Common Interventions Fluid balance? Glucose levels? Pulses?
Monitor hemodynamic status Monitor renal function Place Foley catheter IV fluids as needed Monitor glucose levels Assess peripheral pulses
33
Interventions | Maintain patent airway
Maintain patent airway * Suction as needed * Obtain sputum for culture * Chest physiotherapy as ordered * Secure endotracheal or tracheostomy tube * Maintain adequate hydration
34
Interventions | Promote spontaneous ventilation
Promote spontaneous ventilation Assess, document Respiratory rate, VS, O2 saturation every 15–30 minutes Promptly report worsening data Administer O2 as ordered, monitor response Place in Fowler or high-Fowler Minimize activity, energy expenditures
35
ARDS Pneumonic
A- atelectasis R- refractory hypoxemia D- decreased lung compliance S- surfactant (damaged)