ARDS + respiratory Flashcards

(66 cards)

1
Q

bronchial circulation

A

does not participate in gas exchange
left bronchiole circulation is rooted from aorta
right sided bronchioles are supplied by arteries (intercostal, subclavian, mammary)
veins empty into vena cava

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2
Q

ventilation

A

mechanical act of moving air into and out of respiratory tree
involves musculoskeletal and nervous sustems

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3
Q

respiration

A

process of transporting O2 and CO2 across alveolar capillary membranes by diffusion across concentration gradient

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4
Q

physiology of ventilation

A

diaphragm contracts and flattens
increases volume of thoracic cavity
creates a relative negative intrapulmonary pressure

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5
Q

compliance

A

ability of lungs and thorax to stretch and expand given a change in pressure
decreased in pulmonary fibrosis
increased in emphysema and COPD

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6
Q

age related changes

A

decreased cough/laryngeal reflexes
decrease in alveoli, respiratory muscle strength
increased VQ mismatches, AP diameter, residual volume

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7
Q

autonomic respiration

A

controlled by brainstem

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8
Q

voluntary ventilatory effort

A

controlled by cerebral cortex

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9
Q

central chemoreceptors

A

detect level of H ion in blood

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10
Q

peripheral chemoreceptors

A

sensitive to oxygen, CO2, and H levels

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11
Q

dead space units

A

ventilation exceeds perfusion

pulmonary embolism or pulmonary infarct

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12
Q

shunt unit

A

perfusion exceeds ventilation

pneumonia or atelectasis

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13
Q

silent unit

A

ventilation and perfusion are impaired

severe ARDS or pneumothorax

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14
Q

oxyhemoglobin dissociation curve

A

97% of O2 is bound to Hgb (SaO2)

3% dissolved in serum (PaO2)

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15
Q

shift right

A

fever
acidosis
rise in CO2 (hypercapnia)
rise in 2, 3 diphosphoglycerate

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16
Q

shift left

A

hypothermia
rise in pH (alkalosis)
low CO2

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17
Q

PaO2

A

80-100 mm Hg

reflects the partial pressure of O2 in arterial blood

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18
Q

SaO2

A

93-100%

represents saturation of Hgb with O2

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19
Q

type 1 respiratory failure

A

hypoxemic failure

stems from a disruption of O2 transport from alveolus to arterial flow

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20
Q

type 2 respiratory failure

A

hypoxemic hypercapnic failure
originates in musculoskeletal or anatomical lung dysfunction/suppression
high concentration of CO2 in alveolus = hypercapnia
inability to replace CO2 with O2 = hypoxemia

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21
Q

type 1 respiratory failure nursing dx

A

impaired gas exchange

r/t: alterations in alveolar capillary membrane, excessive secretions, VQ mismatch

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22
Q

type 1 respiratory failure interventions

A

provide supplemental O2
positioning
maintain airway patency
treat underlying causes of ACM alterations, VQ mismatches

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23
Q

type 2 respiratory failure nursing dx

A

ineffective breathing pattern

r/t: alveolar hypoventilation, musculoskeletal dysfunction, neurological trauma

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24
Q

type 2 respiratory failure interventions

A

provide mechanical ventilation as needed
treat causes of alveolar hypoventilation
optimize musculoskeletal dysfunctions
optimize neurological defects

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25
s/s of hypoxia
``` tachypnea, dyspnea tachycardia, dysrhythmias HTN confusion, restlessness lethargy ```
26
low flow O2 therapy
``` nasal cannula (up to 6 LPM) simple mask (8-12 LPM) nonrebreather mask (prevent room air from being inhaled) Venturi mask (adjustable dial with desired FiO2 and LPM flow) ```
27
high flow O2 therapy
delivers O2 at higher rate than pt's inspiratory flow rate may have some positive pressure effect wide nasal cannula, nasal pillow, face mask
28
mechanical ventilation
NIV or invasive
29
mechanical ventilation desired outcomes
``` relief of sx of respiratory distress rest fatigued muscles of respiration improved oxygenation, ventilation pH balance stabilization of chest wall provision of sedation/anesthesia ```
30
noninvasive ventilation (NIV) indications
COPD exacerbation obesity hypoventilation syndrome cardiogenic pulmonary edema lung contusions
31
noninvasive ventilation contraindications
apnea | recent airway or GI surgery
32
noninvasive ventilation
supports ventilation without insertion of artificial airway ventilation through upper airway using an interface with a tight seal most common: CPAP, BIPAP
33
CPAP
continuous positive airway pressure allows more lung units to be available for gas exchange helps open alveoli and prevent atelectasis during expiration increases partial pressure of O2 in the alveoli
34
BiPAP
bilevel posiive airway pressure allows clinician to set higher inspiratory pressure and lower expiratory pressure reduces work of breathing
35
noninvasive ventilation concerns
skin protection anxiolytics tissue ischemia 2/2 tight fit of mask
36
invasive mechanical ventilation
provided by positive pressure ventilator | positive pressure is applied at patient's airway by means of naso/endotracheal tube or tracheostomy
37
endotracheal intubation
larygnoscope stethoscope to auscultate for placement suction with orotracheal device bag valve mask
38
nursing actions during endotracheal intubation
alert physician to decreases in SaO2 below 90% auscultate for correct tube placement note marking on tube at lip monitor for patient to wake because of choking sensation
39
tracheostomy indications
pts requiring long term mechanical ventilation (?21 days) | pts with airway obstruction
40
tracheostomy
incision in the neck at cricothyroid membrane to access trachea and create a stoma to insert tube
41
tracheostomy complications
``` bleeding infection ulceration dysphonia tube obstruction fistula ```
42
controlled mandatory ventilation
tidal volume delivered at a set rate independent of patient effort
43
assist-control ventilation (AC)
patient regains some control of rate of breathing
44
synchronized intermittent mandatory ventilation (SIMV)
patient receives a minimum number of breaths with set Vt | may take additional breaths as determined by the pt
45
CPAP ventilator setting
used frequently as pts become ready for extubation | often used in conjunction with pressure support ventilation to offer bilevel vent pressures
46
pressure control ventilation
less frequently used | provides a set pressure instead of tidal volume during respiratory cycle
47
apnea alarm low minute volume alarm
check if pt is connected to ventilator ensure pt is not overly sedated pt may require higher level of ventilatory support
48
high pressure alarm
ensure tubing is not kinked or compressed assess need for suctioning assess for ventilator dyssynchrony report alarm to responsible clinician
49
high minute volume alarm
assess for anxiety assess for s/s hypoxia (tachycardia, HTN, restlessness, cyanosis) pt may require higher level of vent support or sedation
50
endotracheal suction
every 2-4 hours | facilitates airway patency
51
ventilators and restraints
restraints prevent accidental extubation
52
ventilators and nutrition
early enteral nutrition reduces risk of nutritional deficiencies routine GI assessment aspiration risk
53
weaning protocols
``` is pt: hemodynamically stable? SaO2 > 92% ? able to follow simple commands? FiO2/ABGs ```
54
acute respiratory distress syndrome (ARDS)
inflammatory disorder that damages the alveolar capillary membrane and interferes with gas exchange 2/2 sepsis usually
55
ARDS criteria
1. acute onset hypoxemia 2. diffuse b/l opacities on chest radiography that cannot be explained by HF or nodules 3. moderate to severe impairment of oxygenation
56
exudative phase of ARDS patho
capillary membranes leak and protein rich fluid fills alveoli type 1 alveolar cells destroyed hyaline membranes are formed
57
exudative phase of ARDS assessment
chest x-ray may be normal or show dependent infiltrates tachypnea, dyspnea change in LOC lung sounds may be clear
58
fibroproliferative phase of ARDS patho
``` type II alveolar cells are damaged surfactant production declines peak inspiratory pressure increases compliance declines V/P mismatch ```
59
fibroproliferative phase of ARDS assessment
``` chest x ray shoes diffuse b/l infiltrates and elevated diaphragm refractory hypoxemia with hypercarbia crackles on auscultation right HF develops agitation ```
60
recovery/fibrotic phase of ARDS patho
development of fibrotic tissue in the ACM resulting in alveolar disfigurement decreased compliance pulmonary HTN dead space ventilation increased
61
recovery/fibrotic phase of ARDS assessment
``` leukocytosis, fever worsening infiltrates on CXR decreased tissue perfusion tachycardia + hypotension lactic acidosis end-organ dysfunction ```
62
early s/s of ARDS
``` dyspnea, tachypnea cyanosis abnormal breath sounds change in LOC confusion retrosternal discomfort fever ```
63
vent management of ARDS
``` tidal volume 4-8mL/kg plateau pressure <30 cm H2O FiO2 > 60% judicious use of PEEP permissive hypercapnia maintain SaO2 88-95% ```
64
extracorporeal life support (ECMO) indications
ARDS with: severe hypoxemia uncompensated hypercapnia, pH <7.15 end expiratory plateau pressures >35-45 cm H2O who fail prone positioning trial
65
ARDS medications
``` neuromuscular blocking agents statins corticosteroids vasodilators surfactant beta agonists ```
66
contraindications for prone positioning
spinal injuries elevated ICP abdominal compartment syndrome hemodynamic instability