Arrhythmia Classification Flashcards
Types of arrhythmias
Sinus arrhythmias
Atrial arrhythmias
Nodal arrhythmias
Ventricular arrhythmias
Types of arrhythmias: Tachycardia
Sinus tachycardia Atrial tachycardia Atrial fibrillation/flutter AV Nodal Re-entry tachycardia Wolff Parkinson White Syndrome Ventricular tachycardia Ventricular fibrillation
Slow arrhythmias: Bradycardia
- Bradycardia
- AV Nodal block: 1st, 2nd, and 3rd degree
- Asystole
Heart Rates in BPM
500: Atrial or ventricular Fibrillation
350: Atrial flutter
150-250: Ventricular or supraventricular arrhthymias
60-100: Normal
40: Moderate bradycardia
20: Severe bradycardia
Etiology of arrhythmias: Metabolic
- Ectopic conduction/early afterdepolarization
- Ischemic tissue
- Electrolyte abnormalities
- Drug toxicity
- Genetic abnormalities (QT prolongation)
Etiology of arrhythmias: Structural
- Scarring/fibrotic areas of no conduction
- Cardiomyopathy and hypertrophy
- Accessory pathways/block/congenital malformations
Formation of arrhythmias: 2 main mechanisms
1. Disorder of impulse generation A: Abnormal automaticity B: Triggered activity, early or delayed afterdepolarizations 2. Abnormal conduction A. Block B: Re-entry
Disorder of impulse generation: Abnormal automaticity
Slope of phase 4:
- Increase in slope means increase in heart rate - Sympathetic activity increases heart rate - Cholinergic activity decreases heart rate - Antiarrhythmic drugs make changes on slope and change heart rate
Disorder of impulse generation: Afterdepolarizations
- EAD: Caused by opening of Na+, Ca+, and K+ channels
2. DAD: Caused by elevated systolic Ca+ ions
Afterdepolarizations
Early: Occuring during phases 2 or 3
Delayed: Occuring during phase 4
Caused by: Opening of L-type calcium channel, opening of Na+ channel, closing of K+ channel. These are caused by elevated systolic Ca+ ions (Digoxin), long QT syndrome, or K+ channel blockers
Abnormal Impulse Conduction: Block
- Area of tissue that does not conduct electrical impulse
- 3rd degree AV Nodal block: There is no association between Atria and Ventricles. They conduct impulses independently of each other.
Abnormal impulse conduction: Re-entry
There is a block in the tissue that causes the electrical impulse to jump back and move to other pathway
What are two ways to fix a re-entry arrhythmia?
- Increase refractoriness
2. Provide a two-way block