Arrhythmias Flashcards

(34 cards)

1
Q

Treatment of VT - Haemodynamically Compromised

A

Emergency DC Cardioversion

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2
Q

Treatment of VT - Haemodynamically Stable

A

IV lidocaine or IV amiodarone

May need DC cardioversion if unsuccessful

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3
Q

Treatment of Bradycardia - Rate >40bpm and Asymptomatic

A

No treatment needed

Look for a cause and stop any drugs which could be contributing

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4
Q

Presentation of Sick Sinus Syndrome

A
Sinus node dysfunction 
Bradycardia +/- arrest 
Sinoatrial block 
SVT alternating with bradycardia/asystole 
AF and thromboembolism may occur
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5
Q

Treatment of Symptomatic Sick Sinus Syndrome

A

Pacing

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6
Q

Acute Management of SVT

A

Vagotonic manoeuvres
IV adenosine or verapamil
DC shock if compromised

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7
Q

Maintenance Therapy of SVT

A

Beta blockers or verapamil

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8
Q

Maintenance Therapy of AF/flutter

A

Beta blocker or verapamil
Alternatively, digoxin or amiodarone
Flecanide for pre exited AF

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9
Q

Causes of First and Second Degree Heart Block

A
Normal variant 
Athletes
Sick sinus syndrome 
Ischaemic heart disease 
Acute myocarditis 
Drugs (beta blockers, digoxin)
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10
Q

Treatment of First Degree Heart Block

A

None needed

Follow up recommended to monitor development to ore advanced block

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11
Q

Causes of Mobitz Type I Block

A

Vagal in origin

Usually block in AV node

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12
Q

Treatment of Mobitz Type I Block

A

Generally only requires monitoring

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13
Q

Causes of Mobitz Type II Block

A

Block at infranodal level (e.g. Bundle of His)

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14
Q

Treatment of Mobitz Type II Block

A

Permanent pacing due to higher risk of progression to complete heart block

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15
Q

Causes of Third Degree Heart Block

A
Ischaemic heart disease 
Idiopathic (fibrosis)
Congenital 
Digoxin toxicity 
Aortic valve calcification 
Cardiac surgery/trauma 
Infiltration
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16
Q

Treatment of Third Degree Heart Block

A

Dependant on cause, but ventricular pacing generally needed

17
Q

Treatment for Bradycardia - Symptomatic or Rate below 40bpm

A

IV Atropine
No response = temporary pacing wire
If needed = Isoprenaline infusion, or external cardiac pacing

18
Q

What is the most common childhood arrhythmia?

A

Supraventricular re-entry tachycardia

19
Q

How does IV Adenosine slow the heart rate?

A

Block the secondary circuit at the AV node to return the heart to sinus rhythm

20
Q

What are the two main mechanisms of arrhythmia production?

A

Defects in impulse formation

Defects in impulse conduction

21
Q

How does pathological altered automaticity lead to arrhythmias?

A

A latent pacemaker subverts the SA node’s function as the heart’s normal pacemaker

22
Q

How does triggered activity lead to arrhythmias?

A

Afterpolarisations are triggered by a normal AP

Can get early after depolarisation or late depolarisation

23
Q

When do Early Afterdepolarisations occur?

A

During the inciting AP within Phase 2 and Phase 3

Associated with prolongation of AP and drugs prolonging QT interval

24
Q

When do Late Afterdepolarisations occur?

A

After complete depolarisation

Associated with Ca2+ overload provoked by catecholamines, digoxin, heart failure

25
What defects in impulse formation lead to arrhythmias?
Altered automaticity | Triggered activity
26
What defects in impulse conduction lead to arrhythmias?
Re-entry Conduction block Accessory tracts
27
How does re-entry lead to arrhythmias?
Re-entry occurs when a self sustaining current occurs which stimulates and area of myocardium repeatedly/rapidly Re-entrant circuits require: Unidirectional block Slowed retrograde conduction velocity
28
How do accessory tract pathways lead to arrhythmias?
Electrical pathways that bypass AV node (e.g. Bundle of Kent) Ventricles receive impulses from both normal and accessory pathways
29
Sudden onset tachycardia with recovery of normal sinus rhythm after carotid sinus massage or adenosine is characteristic of which arrhythmia?
Paroxysmal Supraventricular Tachycardia
30
What is the most common cause of Paroxysmal Supraventricular Tachycardia?
Atrioventricular nodal reentrant tachycardia
31
What are the three kinds of atrial fibrillation?
Paroxysmal Persistent Permanent
32
What is paroxysmal atrial fibrillation?
Episodes of AF terminate spontaneously
33
What is persistent atrial fibrillation?
Episodes are non self terminating
34
What is permanent atrial fibrillation?
Continuous AF which cannot be cardioverted | Treatment centres on rate control and anticoagulation