Arrhythmias Flashcards

(37 cards)

1
Q

definition of

  • bradycardia
  • tachycardia
A

bradycardia = < 60 bpm tachycardia = > 100 bpm

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2
Q

causes of sinus bradycardia

A

physiological

beta blockers

ischaemia

sick sinus syndrome

hypothermia

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3
Q

acute management of bradycardia

A

1st line: 500 micrograms atropine - can repeat doses up to 3g

2nd line options: isoprenaline / transcutaneous pacing if unstable

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4
Q

a narrow complex tachycardia (QRS <0.12s) originates where?

A

above the AV node - an ‘SVT’

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5
Q

what causes an SVT

A

an electrical signal re-entering the atria from the ventricles - creates a self perpetuating electrical loop

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6
Q

what are the main types of SVT

A
  1. Atrioventricular nodal re-entrant tachycardia: re-entry point is back through AV node
  2. Atrioventricular re-entrant tachycardia: re-entry point is an accessory pathway (e.g. Wolf Parkinson White)
  3. Atrial tachycardia: electrical activity originates somewhere other than SA node
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7
Q

1st line management for an SVT

A

vagal manœuvres - valsalva - carotid sinus massage

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8
Q

2nd line management for SVT

A

if vagal manoeuvre unsuccessful:

  • IV adenosine
  • IV verapamil in asthmatics

DC cardioversion if above unsuccessful

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9
Q

what is the accessory pathway called in Wolf Parkinson white

A

Bundle of Kent

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10
Q

ECG changes in Wolf Parkinson White

A

slurred upstroke of QRS - delta wave

short PR

left axis deviation (right sided pathway)

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11
Q

Definitive treatment of Wolf Parkinson White

A

radio frequency ablation

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12
Q

characteristic appearance of atrial flutter on ECG

A

Sawtooth baseline

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13
Q

characteristic appearance of atrial fibrillation on ECG

A

irregularly irregular

absent P waves

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14
Q

1st line drug for controlling rate in atrial fibrillation + flutter

A

beta-blocker

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15
Q

what patients are offered immediate cardioversion for AF?

A

If AF present for < 48 hours or severely unstable

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16
Q

options for cardioversion in AF

A

pharmacological:

  • flecainide
  • amiodarone if structural heart disease

electrical:

  • DC cardioversion
17
Q

what patients are suitable for delayed cardioversion in AF?

what must be done beforehand?

A

AF present for > 48 hours + stable

anticoagulate for 3 weeks prior

18
Q

what is first degree heart block

A

fixed prolonged PR interval (>0.2s)

19
Q

what is second degree heart block: Mobitz Type 1

A

gradual lengthening of PR interval until a QRS complex is dropped

20
Q

what is second degree heart block: Mobitz Type 2

A

fixed prolonged PR interval with QRS complexes dropped in

  • 2: 1 (every other p wave followed by QRS)
  • 3:1 ( 2 P waves with no QRS followed by 1 P wave with a QRS)
21
Q

what is 2:1 heart block

A

2 P waves for each QRS complex

( 1 P wave not followed by QRS, next P wave followed by QRS)

22
Q

what is third degree heart block

A

complete heart block

  • no relationship between P waves + QRS
23
Q

what types of heart block require pacing

A

Mobitz type 1 if symptomatic

Mobitz type 2 + 3rd degree – permanent pacing

24
Q

what is bifasicular heart block

A

RBBB with left axis deviation

25
what is trifasicular block
RBBB + Left axis deviation + 1st degree heart block (fixed prolonged PR)
26
ECG appearance of ventricular tachycardia
regular broad QRS \>100 bpm no P or T waves
27
management of VT in - stable patient - unstable patient
stable = amiodarone unstable = DC cardioversion
28
what is Torsades de Pointes
polymorphic VT that occurs in patients with long QT precipitated by: - hypokalaemia / hypocalcaemia / hypomagnesmia - amiodarone, citalopram, macrolides
29
management of torsades de pointes
correct electrolyte disturbances / remove causative drugs magnesium sulphate infussion defibrillation if VT occurs
30
when does ventricular fibrillation occur
post MI
31
What rhythms can be shocked
pulseless VT VF
32
what are ventricular ectopics
premature ventricular beats - individual random broad QRS complexes on background of normal ECG
33
what is ventricular bigeminy
ventricular ectopic following every sinus beat
34
what is an escape beat
a beat that comes late
35
what is ventricular hypertrophy? most common cause?
increase in left ventricle muscle mass _not_ volume hypertension
36
voltage criteria for LVH
negative component in V2 and positive component in V5 must add to \> 7
37
affect of LVH on the heart
strain - shows as ST depression - blood supply cant match demand