Arrythmias Flashcards
(26 cards)
Sinus bradycardia- causes
Meds: beta blockers, CCB, amiodarone, Li, dig
Increased vagal tone: athletes, sleep, IMI
Metabolic: hypoxia, sepsis, mexedema, hypothermia, hypoglycemia
OSA
Increased ICP
Sinus brady tx
if no sx: atropine, b1 agonists (short term), pacing
Sinus brady- MOST COMMON CAUSE
Dr. Flitter: The most common cause for a pause is a blocked PAC
Sick Sinus Syndrome- causes
periods of unprovoked SB
SA arrest
tachy-brady syndrom
chronotropic incompetence with endo trach tube
Sick sinus syndrome- tx
often need combo
BB, CCB, dig for tachy
PPM for brady
AV block- Type I
prolonged PR (>200ms); 1:1 conduction
AV block Type II Mobitz I (Wenckebach)
progressively prolonged PR
worsens with carotid sinus massage, improves with atropine
AV T II Mobitz I (Wenckebach)- causes
abnml AV node- IMI, inflammation, myocarditis, high vagal tone, drugs
AV T II Mobitz I (Wenckebach)- tx
often paroxysmal, nocturnal, asx
Septal MI
V1-V2 +/- avR; proximal LAD
Anterior MI
V3-V4; LAD
Apical MI
V5-V6; Distal LAD, LCx, RCA
Lateral MI
I, avL; LCx
Inferior MI
II, III, avF; RCA (85%), LCx (15%)
RV MI
V1-V2 & V4R (most sensitive); proximal RCA
Posterior MI
ST depression V1-V3, ST elevation in V7-V9 (posterior leads); RCA, LCx
SVT- atrial
Sinus tach (pain, fever, hypovolemia, hypoxia, PE, anemia) Atrial tach (catchols, EtOH, dig, CAD, COPD) Multifocal atrial tach (increased automaticity at multiple sites; seen with underlying pulm dz) Atrial flutter (macroreentry- usually in RA) A fib (irregular AVN bombardment, often from pulm veins)
AV Jxn
AVNRT (reentrant circuit)
AVRT: (reentrant circuit and accessory path; may show pre-excitation (WPW))
NPJT: increased jxnal automaticity
Unstable SVT tx
cardioversion per ACLS
ST tx
treat underlying stressor
AT tx
BB, CCB, adenosine
Amiodarone?
Radiofrequency ablation, class IC/II antiarrhythmics
AVNRT or AVRT tx
Vagal maneuvers
Adenosine
CCB or BB, DCCV
Ablation
NPJT
CCB, BB, amiodarone
AF
BB, CCB, dig, AAD