ASSCC 1 Flashcards
(35 cards)
6 CT findings in acute pancreatitis:
1) Fat stranding
2) Pseudocyst
3) Oedema
4) Collection
5) Abscess
6) Necrosis
Why does pancreatitis cause hyperglycaemia?
Pancreatic enzymes destroy β cells = increase in serum glucose
5 complications of acute pancreatitis:
1) Hypovolaemic shock
2) ARDS
3) Pseudocyst
4) Haemorrhagic pancreatitis
5) Sepsis
6) Multiorgan failure
6 causes acute pancreatitis:
1) Gallstones
2) Alcohol
3) Abdominal trauma
4) Post-ERCP
5) Thiazide diuretics
6) Steroids
7) Viral infections (mumps)
8) Hyperparathyroidism
3 exocrine secretions of pancreas:
1) Trypsinogen
2) Amylase
3) Lipase
Define pseudocyst:
Collection of amylase rich fluid enclosed in a wall of fibrous or granulation tissue. Takes > 4 weeks to form after onset of acute pancreatitis
4 symptoms of pseudocyst:
1) Vomiting
2) Epigastric swelling
3) Dyspepsia
4) Mild fever
5 complications pancreatitic pseudocyst:
1) Rupture
2) Bleeding from splenic vessels
3) Portal vein thrombosis
4) Infection
5) CBD obstruction
6) Cholangitis
4 line related complications of parenteral feeding?
1) Infection
2) Thrombosis
3) Pneumothorax
4) Haemothorax
2 feed related complications of parenteral feeding?
1) Fluid and electrolyte imbalances
2) Hypo/hyperglycaemia
3) Refeeding syndrome
4) Gut atrophy
6 TPN Indications:
1) Severe malnutrition >10% weight loss
2) Multiple trauma
3) Sepsis with MOF
4) Severe burns
5) Enterocutaneous fistula
6) IBD
7) Radiation enteritis
8) Short bowel
Routes of administration of TPN:
1) Central line
2) PICC
Type of electrolytes in TPN:
1) Sodium chloride
2) Calcium chloride
3) Potassium chloride
4) Magnesium chloride
What are the components of TPN?
Water Carbohydrates 50% Fats 30% Protein Vitamins Nitrogen Trace elements
kcal/g of energy of FAT:
9.3 kcal/g
kcal/g of energy of PROTEIN:
4.1 kcal/g
kcal/g of energy of CARBs:
4.1 kcal/g
4 Disadvantages of using glucose as sole energy source:
1) Critical ill pt unable to utilise due to stress response
2) Fatty liver
3) Deficiency in essential fatty acids
4) Respiratory failure (excess CO2 released on oxidation of glucose)
5 factors affecting epidural efficiency:
1) Level of injection
2) Drugs used
3) Dose of drug
4) Vasoconstrictors
5) Posture
6) Age, height, weight
Causes of gastric outlet obstruction:
Benign: pyloric stenosis due to chronic peptic ulceration
Malignant: Gastric or pancreatic carcinoma
ABG result gastric outlet obstruction:
Metabolic alkalosis
3 Biochemical abnormalities in gastric outlet obstruction:
1) Metabolic alkalosis
2) Hypochloraemia due to vomiting
3) Hypokalaemia due to ^aldosterone
Why is bicarbonate increased in gastric outlet obstruction?
- Increase in reabsorption of bicarb in renal tubules due to loss of Cl- due to vomiting
- Reduced pancreatic secretions = more bicarb rich secretions retained
Why paradoxical acuduria in gastric outlet obstruction?
- Hypovolaemia
- RAAS activation
- Na+ and H20 retained in exchange for H+ + K+
- Aciduria