assessment of blood pressure Flashcards

(24 cards)

1
Q

what are the normal BP values in dogs and cats

listed as the same in both

A

Systolic: 90 - 140mmHg
Mean: 70 - 100mmHg
Diastolic: 50 - 80mmHg

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2
Q

How is MAP calculated

A

MAP = Diastolic + ((systolic - diasolic)/3)

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3
Q

how is doppler BP measured

A
  • cuff applied then a doppler transduced placed distally over peripheral artery (coupling gel)
  • cuff inflated until no blood flow then slowly inflated until audible signal = systolic BP
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4
Q

Advantages of doppler

A

Non invasive
- more accurate than other non invasive in dysrrhymias and small patients

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5
Q

how is oscillometric BP measured?

A
  • Cuff attached to machine which detects oscillations from arterial flow
  • these are greatest at the mean
  • the systolic and diastolic are then calculated from the mean.
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6
Q

what are the advantages and limitations of oscillometric

A
  • non invasive
  • less prone to human error than doppler from speed of cuff deflation.
  • less accurate in very small patients or those with dysrhymias, check that the machine is counting the HR correctly.
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7
Q

How can you help to reduce the limitations of indirect BP measurement

A
  • standardise process
  • appropriate cuff number 30-40% of limb circumference
  • select largery if between two
  • limit vertical distance from heart to cuff (sternal or lateral recumbancy)
  • want the level of the right atrium - below overestimates and above underestimates
  • can correct by 0.8mmhg/cm if more than 10com below/above
  • discrad 1st reading and average from 5 -7
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8
Q

What are the general limitations of indirect BP measurement

A

rely of peripheral flow so effected by hypovolaemia, hypothermia, vasoconstriction, vasopressors and hear failure
- tend to overestimate

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9
Q

how is direct arterial BP measured

A

arterial catheter in the dorsal pedal artery is attached to a monitor via a fluid line and a pressure transducer
- gives MAP, systolic, diastolic and generates a waveform.
- most accurate

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10
Q

What are some problems associated with arterial BP monitoring

A
  • need regularly line flushing as prone to occlusion by clots and bubbles (dampens waveform)
  • complications; medication given in error, accidendental removal + bleeding, thrombosis of the limb and technical ability to place.
  • can be hard to place in small patients or if have very poor perfusion
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11
Q

hypertension is a BP>150/95 (115). What are the risk categories for organ damage

A

i. <150mmHg, minimal

ii. 150 - 159mmHg mild

iii. 160 - 179mmHg moderate

iv. >180mmHg severe

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12
Q

What ocular changes may be seen with systemic hypertension

A

acute onset blindness, intraocular haemorrhage, retinal detachment
- on exam you may note torturous retinal vessels oedema or degeneration

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13
Q

What neurological changes can be seen with systemic hypertension?

A

altered mentation, disorientation, seizures, imbalance, head tilt and nystagmus.
should respond to BP reduction
- make sure TBI and cushing response ruled out

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14
Q

What renal changes can be seen with systemic hypertension?

A
  • usually more insidious onset
  • azoatemia, increased SDMA, proteinuria.
  • note hypertension can be both cause and effect of Renal issues
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15
Q

Hypertension is inappropriately high SVR either along or with changes in blood volume

discuss the pathophysiology

A
  • sympathetic stimulation increases catecholamies, angiotensin2, endothelian 1 and vasopressin. local vasodilators (NO and prostacyclin) are reduced.
    volume expansion due to inadequate renal excretion or sodium and water retension.
  • inflammation with oxadative stress leads to endothelial dysfuntion and reducing NO bioavailibility
  • causes vascular remodelling resulting in cytokine production, endothelial adhesion and WBC upreg
    -leads to target organ damage or SIRS
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16
Q

what may cause hypertension

A
  • situational
  • primary (rare) diagnosis of exculsion, usually proteinuria with low USG, other tests normal.
  • secondary (common), lost of disesaes eg renal, endocrine, hepatic, anaemia, polycytheamia, neoplasic.
  • each disease has variable mechanism but most cause inappropriate hormone secretion:
    RASS activation, adrenal cortex tumours acuse aldoserone production and adrenal medulla catecholamines.
17
Q

What are the treatment goals for hyperstension

A
  • want to reduce to 160mmHG as a minimum
  • aim for 10% reduction first hour the further 15% subsequent hrs.
  • if concurrent kidney disesae looking for UOP drop by 50%.
18
Q

What/how are arterial vasodilators used to treat hypertension

A
  • SNP or Hydralazine
  • Both increase NO concentrations leading to arterial dilation.
  • given as a CRI
  • ideally art line BP monitoring
19
Q

What/how are ACE inhibitors used to treat systemic hypertension

A
  • Enalapril and benazepril
  • Reduces production of angiotension 2 leading to reduced vasoconstriction and sodium and water retension at the kidney
  • used for more chronic hypertension associated with cardiac and renal disease. unlikely to lower BP enough in emergencies.
19
Q

What/how are calcium channel blockers used to treat hypertension

A
  • Amlodipine
  • reduce smooth muscle constriction by inhibiting calcium channels in vasculature
  • rapid onset oral amodip should help in an hour and the dose can be repeated.
20
Q

when are alpha adrenergic ANTAGONISTS useful

A
  • Prazosin and ACP
  • cause smooth muscle relaxation via effects on the alpha receptors
  • most useful in hypertension resulting from pheochromocytomas and aldosterone secreting tumours.
21
Q

Beta adrenergic antagonists - examples of and when could they treat hypertension

A

-atenolol and propanolol
- cause a reduction in renin secretion and therefore the effects of the RAAS ( vasoconstriction via angiotensisn 2, water rentention from ADH and NA and water retenstion from aldosterone.
- most useful in pheochromocytomat and aldo sterone secreting tumours

22
Q

What ARB are used in hypertension

A
  • angiotensin 2 receptor blockers
  • eg telimasartan