Bariatric Flashcards
(46 cards)
Addison Disease Definition and etiology
primary adrenal insufficiency; insufficent cortisol and aldosterone due to destruction, partial or complete to adrenal glands
2ndary could be caused 2/2 decr ATCH release caused by a pituitary tumor or prolonged striod use. 2dary is more common but we will focus on primary
Etio:autoimmune reaction, infection, metastized cancer, CMV, tuberculosis, and hemmorage
Addison Disease Risk fxs, gender and age
Surgery, pregnancy accident injury or trauma, infection, sweating overexertion during physical activity or failure to take steroid therapy in persons who have chronic adrenal insufficiency Slightly higher in women 40-60 years
Addison Disease Clinical Presentation:
- Cortisol mobilizes nutrients, modifies the body’s response to inflammation, stimulates the liver to raise blood sugar and helps to control the amount of water in the body.
- Hypoglycemia can ensue
- weak, exhausted, hypotensive, anorexia, weight loss, nausea and vomiting, Emotional disturbance and poor stress mgmt and darkenign of the skin
- Aldosterone regulates salt and water levels which affects blood volume and blood pressure.
- decr can cause hypotension, decr CO, dehydration,
- Addisonian crisis, can progress quickly to hypovolemic shock (e.g., hypotension, tachycardia, and loss of consciousness) from rapid fluid loss
Addison Disease diagnostic dx
-blood and urine tests. Cortisone and alodsterone should incr with ATCH synthetic admin
Addison Disease med mgmt
- replacing fluids, electrolytes, glucose and cortisol
- synthetically manufactured corticosteroids and mineralocorticoids
Addison Disease Physical TherapyTests and Measures and Implications for evaluation
- check vitals, stress can increase the risk for Addisonian crisis
- get baseline for ROM, MMT and areobic state
- proper dose of replacement medication is taken every day
- focus on strength
- CONTRA- aqua therapy; hot and humid
Grave’s Disease Definition and Etiology
Autoimmune condition and a form of hyperthyroidism with an increase in t4 production; primary role to regulate metabolism
- makes up of 85% of hyperthyroiodism cases
- thyroid gland; stimulation of metabolism, therefore, causes hypermetabolism and increased sympathetic nervous system activity
Grave’s Disease risk fxs gender and age
- Immunologic and genetic factors
- more common in women with a family history of thyroid conditions
- psoriatic arthritis, rheumatoid arthritis, and Sjögren Syndrome
- 4 times as likely to affect women than it is to affect men-
- 20-50 yrs
Grave’s Disease Clinical presentation
- symmetric enlargement of the thyroid
- nervousness, heat intolerance, sweating, diarrhea, tremor, and palpitations, dramatic weight loss despite an increase in appetite
- exophthalmos, afrib
Grave’s Disease dx tests
Radioactive iodine uptake
- blood tests to test TSH
Grave’s Disease Med MGMT
Antithyroid medication, radioactive iodine (first-line therapy for those over 18 and not prego), and partial or subtotal thyroidectomy
Grave’s Disease Physical TherapyTests and Measures and Implications for evaluation and prax
exercise intolerance and reduced exercise capacity should be looked out for 2/2 weakness and myopathy
- watch vitals and cardio signs bc of unpredictable cardio signs
- watch for signs/ be observant of possible signs of hypoparathyroidism, which include tetany, muscle twitching, and numbness and tingling around mouth, fingers, or toes
- address bone mineral density and poor nutrition
- cardiopulmonary rehabilitation should be a focus
Gastric Bypass definition and description of procedure
- caloric restriction through a significant reduction in stomach capacity via medical procdure
- “Roux-en-Y” is the gold standard
- stomach is either stapled or transected into a smaller “gastric pouch” of anywhere from 15-30 mL
Gastric Bypass Characteristics for Qualification
- Weight 100 pounds or 100% above desirable weight
- BMI >40 kg/m2
- BMI >35 kg/m2with medical comorbidities
- Failure of nonsurgical attempts at weight reduction
- Absence of endocrine disorders that can cause massive obesity
- Psychological stability-Basic understanding of how obesity surgery causes weight loss
- Realization that surgery itself does not guarantee weight loss
- Absence of alcohol and drug abuse
- Commitment to post-op follow-up
Gastric Bypass Risk Fxs
- Nephrolithiasis (kidney stones)
- Hepatic failure
- Cholelithiasis (gall bladder stones)
- Malnutrition (vitamin and mineral deficiencies)
- Reflux
- Small bowel obstruction
- Hemorrhage
- Iron-deficiency anemia
- Gastric prolapse
- Postoperative bleeding
- Postoperative leaking
- Atelectasis; pneumonia
- Pulmonary embolism
- Significant decrease in bone health
- Death
Gastric Bypass Clinical Presentation: Signs/symptomsBefore and After Procedure
Before -Hypertension (BP above 120/80) -BMI >35 kg/m2 -Presence of diabetes mellitus (A1C of >6.5% or fasting blood sugar test of >126 mg/dL glucose on two separate occasions) -Presence of sleep apnea -Decreased activity tolerance -Decreased quality of life After -Malnutrition of protein -Severe/mild iron deficiency anemia -Nausea/vomiting -Hair loss from iron/folate/vitamin B12deficiencies -Diarrhea/bad fecal odor
Gastric Bypass Dx tests
- blood tests
- phych test
- BMI
- nutrition consult
- DxA scan for bone mineral density
Gastric Bypass med mgmt
- multi vitamin for mal nutrition
- constant lab work ups every 3 months
- re- do of sx if it was outeaten
Gastric Bypass Physical TherapyTests and Measures and Implications for evaluation and prax
- sense effects of sx dont happen over night help pt get back to activity goals and monitor activity tolerance
- tend to suffer from increased heat intolerance, so err on the side of caution to reports of excessive sweating and “over-heating” to avoid heat stroke/exhaustion
- high risk of bone fractures
- low intensity short duration
- moderate aerobic exercise of at least 150 min/week and strength training 2-3 times/week can have a positive effect on bone mineral density
- strengthen core and rpevent incision site adhesions
- With Ex Rx **
- Increased heat intolerance
- Need for equipment modifications to fit body size
- Reported pain as obesity tends to increase risk of LBP and joint pain
- Selection of appropriate footwear as obesity can lead to poor foot mechanics
Cushing’s Definition and Etiology
exposure of excessive glucocorticoids for extended periods of time due to injection or tumors causing endogenous overproductions
- Hyperfunction of the adrenal glands from an adenoma,also with carcinomas in rarity.
oExcessive corticosteroid medications, like those suffering from inflammation, chronic allergy or even MSK disorders involving pain.
- excess ATCH
-uncontrolled type 2 diabetes, hypertension, or early-onset osteoporosis.
Cushing’s Risk Fxs gender and age
- Chronic use of corticosteroids
- Genetics
- Females> men
18-25
Cushing’s clinical presentation
- “moon-face” in which the face enlarges and becomes rounded with washed out jaw/cheek lines
- increased truncal obesity and specific supraclavicular and cervicodorsal (buffalo hump) fatty enlargements.
- hyperglycemia, hypertension, proximal muscle wasting and bone mineral density (BMD) loss resulting in osteoporosis
- Mental changes like memory loss, poor concentration and “steroid psychosis”
Cushing’s Diagnostic tests
- 24-hour urinary cortisol test
- Lowdose dexamethasone suppression test –Measures response of ACTH by adrenal glandfollowing dexamethasone
- Late-night salivary cortisol test; nomral are low levels at night
- MRI of pituitary
Cushing’s Med MGMT
-Surgery, chemotherapy, radiation or otherdrugs aimed at inhibiting, or lessening cortisol in the body