basics Flashcards

(61 cards)

1
Q

which ventricle occupies most of the anterior surface

A

right ventricle

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2
Q

comes out of the right ventricle and
bifurcates into its left and right branches and heads into the lungs

A

pulmonary artery

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3
Q

comes out of the left ventricle, ascends to the level of the sternal angle, and then arches up and over the pulmonary arteries and then descends into the thorax

A

aorta

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4
Q

dump their blood collected deoxygenated blood into the right atrium

A

superior and inferior vena cava

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5
Q

dump oxygenated blood return from the lungs into the left atrium

A

pulmonary veins

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6
Q

sac around the heart

A

pericardium

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7
Q

inner lining

A

endocardium

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8
Q

heart muscle

A

myocardium

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9
Q

outer surface

A

epicardium

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10
Q

myocardium has what

A

intercalated disks with gap junctions = speed!

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11
Q

❖ Right atrium, near the
insertion of the SVC
❖ Inherent rhythm 60-100
bpm)
❖ Usually sets the heart
rate (sinus rhythm)
❖ Initiates Depolarization
Wave

A

SA node

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12
Q

❖ Anterior
❖ Middle (Wenckebach
branch)
❖ Posterior

A

internal pathways

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13
Q

Anterior Inter-Atrial band or Bachmann’s bundle

A

interatrial pathways

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14
Q

❖ Located at the base of the
right atria near the
interventricular septum
❖ Smaller cells, fewer gap
junctions = Slower
❖ Maximal rate is 40-60 bpm;
also maximal ventricular rate

A

AV node

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15
Q

❖ Only cellular connection between atria and ventricles
❖ Together AV node and AV bundle make up the AV junctional tissue
❖ If SA node fails, AV junctional tissue can control heart rate
❖ Intrinsic heart rate of 40-60 bpm
❖ Slows down the cardiac impulse
❖ Impulse is delayed before conducting down the bundle of His and its branches to depolarize the ventricles.

A

AV junction or bundle of HIS

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16
Q

left anterior bundle branch; thinner,
carries impulses to septum, left
anterior wall, and anterior papillary
muscle

A

left anterior fascicle

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17
Q

. post. bundle branch; thicker, carries impulses to posterior, inferior, left ventricular free wall and posterior papillary m. with dual blood supply, less likely to become
ischemic

A

left posterior fascicle

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18
Q

travel down towards the apex of the heart where they fan out into Purkinje fibers

A

both left and right BB

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19
Q

❖ Electrical Stimulus pass through
the ventricular myocardium
❖ If both the SA and AV node fail,
The purkinje fibers can generate and electoral signal
❖ the intrinsic rate for
the purkinje system is about
20 to 40 bpm
❖ Muscular Contraction starts at
the apex and circumferential
❖ Fast rate of action potential
generation, numerous sodium
ion channels and mitochondria
and fewer myofibrils

A

purkinje fibers

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20
Q

depolarization wave spreads upward from

A

apex

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21
Q

atrium squeezing

A

P wave

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22
Q

ventricle squeeze

A

QRS complex

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23
Q

heart restarts to do it all over again

A

T wave

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23
Q

normal conduction

A

sinus rhythm

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24
time for blood to leave atrium and fill ventricle
delay in AV node
25
abnormal conduction
atrial arrhythmias junctional rhythm ventricular rhythm
26
rate at which electricity is conducted
cardiac conductivity
27
rate at which electricity is conducted at internodal pathways
fast
27
rate at which electricity is conducted at SA node
slow
28
rate at which electricity is conducted at AV node
SLOW
29
rate at which electricity is conducted at right and left bundle branches
fast!
29
rate at which electricity is conducted at purkinje fibers
FASTER!!
30
ability to spontaneously generate an impulse (depolarize)
cardiac automaticity
31
primary pacemaker
SA node
31
outside of the sinus node pacemaker (non-sinus)
ectopic
32
blockage of stimuli through AV junction
AV heart block
32
disease of bundle branches
bundle branch block
33
damage to ventricular muscle
ST segment changes
34
#1 killer of all Americans, plaque and fat inside plumbing
coronary artery disease
35
Supply the anterior septum, the anterior wall, and in most cases apex. 9 o'clock to 1 o'clock
LAD
35
Supply the lateral wall 2 o'clock to 4 o'clock
LCX
35
Supply the posterior lateral segments, the inferior segments, and the posterior septum. 5 o'clock to 8 o'clock
RCA
36
what bproduces the sound of the heartbeat
As the heart valves close, the heart sounds of S1 and S2 arise from the vibrations emanating from the leaflets and the blood flow they “SHUT DOWN”. They SNAP CLOSED producing a noise
37
Closure of the Mitral valve and Tricuspid valve (end of diastole, beginning of systole)
S1
38
Closure of the Aortic valve and Pulmonic valve (end of systole)
S2
38
pathologic (BAD) sounds found in diastole
S3 and S4
39
atrioventricular valves (AV) Because they separate the atrium and ventricles
The Tricuspid valve and Mitral valve
40
3 leaflets (Mercedes Benz)
aortic and pulmonic valves
40
❖ Venous Side ❖ Responsible for pulmonary Circulation: ❖ circulation to the lungs and pulmonary vasculature ❖ LOW PRESSURE SYSTEM
right side of heart
41
❖ Arterial Side ❖ Responsible for Systemic Circulation ❖ circulation to the most distal portions of the body ❖ HIGH PRESSURE SYSTEM
left side of heart
41
(EDV-ESV)
stroke volume
41
volume of blood ejected with a single beat from the ventricle.
stroke volume
42
CO/HR
stroke volume
43
stroke volume normal
60-100mL/beat
44
the volume of blood ejected from each ventricle over 1 minute ❖ HR x SV ❖ Dependent on Preload, Contractility, and Afterload
cardiac output
44
the ability of the cardiac muscle (and cell/myocyte), when given a load (volume of blood that stretches the muscle), to shorten. = THE SQUEEZE POWER. Increased by: sympathetic stimulation Decreased by: ischemia (hypoperfusion) to the myocardium
myocardial contractility
44
The volume of blood (the load that puts stretch pressure) that is present in the Ventricle at the end of diastole = HOW MUCH IS THE VENTRICLE BEING STRETCHED? Increased by: increased venous return, inspiration, increased intravascular volume Decreased by: exhalation, decreased LV output, pooling of blood in the capillary bed or venous system
preload
45
There is an optimal length between sarcomeres (stretch) at which the tension in the muscle fiber is greatest resulting is the greatest force of contraction.
frank starling law
46
the degree of vascular resistance to ventricular contraction. Or... the force of resistance that the ventricle must overcome to empty its contents at the beginning of systole. Increased by: increased arterial vascular tone, increased aortic volume, Aortic/Pulmonic valve stenosis, increased PVR
afterload
46
The ability of the aorta/large arteries to distend (stretchiness)
distensibility
47
esistance in the circulatory system to the flow of blood. Some resistance is required to create a blood pressure Particularly at the arteriole level Poiseuille’s Law:
peripheral vascular resistance