Blackouts, Funny Turns and Dizziness Flashcards

1
Q

What are blackouts?

A

-Episodes of loss of consciousness
=Consciousness is not a simply localised brain function
=Blackouts results from something that affects the brain GLOBALLY

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2
Q

What are the general causes of blackouts?

A

-Syncope
-Epileptic seizure
=Then dissociative attack

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3
Q

What is syncope?

A

-Loss of consciousness due to hypoperfusion of the brain

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4
Q

Types of syncope

A
  • Vaso-vagal (reflex)
  • Postural hypotension (failure of autonomic NS)
  • Cardiac (dysthymic/ structural)
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5
Q

What are epileptic seizure blackouts?

A

-Consciousness lost due to abnormal electrical discharges in the brain

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6
Q

Types of epileptic seizure

A
  • Generalised epileptic seizures

- Complex partial seizures

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7
Q

Clinical information needed to diagnose blackouts

A
  • Clinical background
  • Immediate context
  • Onset of attack
  • During attack (eye witness)
  • Recovery from attack
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8
Q

Clinical background for diagnosing blackouts

A
  • Age
  • PMH (brain injury)
  • FH (inherited condition for epilepsy)
  • Drug history
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9
Q

Differentiating between epilepsy and syncope

A

-Favours epilepsy:
=long duration, post-event confusion (long recovery), cyanosis, sever biting of sides of tongue
-Both:
=Pallor, jerking, warning and stiffness

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10
Q

What are the other causes of blackouts?

A
  • Functional blackouts
  • Non-epileptic attack disorder
  • Dissociative attacks
  • Hypoglycaemia (diabetes on treatment)
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11
Q

Investigations for blackouts

A
  • Depending on context, none
  • ECG (conduction abnormality)

-In certain syncope cases
=Ambulatory ECG
=Tilt table testing

-In selected epilepsy cases:
=Routine EEG (limited utility confirming epilepsy, useful for classification type)
=Video-telemetry ECG (difficult cases)
=Cerebral imaging (cause of epilepsy)

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12
Q

What are funny turns?

A
  • Discrete episode(s)
  • Due to some sort of brain event
  • Deliberately vague/ inclusive definition unusual to patient
  • Tend to reflect localised brain dysfunction
  • Affecting a particularly focal brain area
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13
Q

General causes of funny turns

A
  • TIA
  • Epileptic seizure
  • Migraine
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14
Q

What questions are used to differentiate funny turns?

A

-Onset/course
=Sudden onset/ simultaneous (multiple features)
=Sudden (occur at once) or spreading (develop)
-Sequential (multiple events)

-Nature
=Negative (loss of function)
=Positive
=Mixed

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15
Q

TIA as funny turn

A

-Dysphagia and loss of limb power

=Sudden and simultaneous, negative

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16
Q

Migraine as funny turn

A
  • Tingling in arm, spreading over 15 minutes, followed by dysphagia (positive, spreading, sequential)
  • Spreading, coloured pattern in vision with visual loss (spreading mixed)
17
Q

Epilepsy as funny turn

A

-Twitching fingers, spreading up arm, to whole arm twitching (positive, spreading and sequential)

18
Q

Other causes of funny turns

A
  • Dissociation/ depersonalisation/ derealisation
  • Anxiety/ panic attacks
  • Hypoglycaemia
  • Parasomnias
19
Q

Investigations of funny turns

A
  • Clearly migraine= none

- TIA/ epilepsy= appropriate investigation

20
Q

What could a patient mean when describing dizziness?

A
  • Imbalance (legs not head)
  • Pre-syncope
  • ‘Funny feeling’
  • Vertigo
21
Q

What is vertigo?

A

-On or more:
=A distortion of static gravitational orientation
=An erroneous perception of movement of the sufferer
=An erroneous perception of movement of the environment
(not all vertigo is rotational vertigo)

22
Q

Main causes of vertigo

A
  • Unusual stimulation of intact systems (motion sickness)

- Pathological dysfunction of those systems

23
Q

Three structural items dealing with static gravitational orientation and perception of motion

A

-Visual
-Vestibular (acceleration)
-Somatosensory
-Feed information to the vestibular nucleus in the brainstem
=motion perception and spatial orientation
=eye movements
=posture

=Mismatch= vertigo

24
Q

Components of vertigo syndrome

A
  • Oculomotor: nystagmus, ocular deviation
  • Autonomic: nausea, vomiting, sweating
  • Postural: ataxia, falls
  • Perceptual: vertigo, disorientation
  • Secondary: anxiety, avoidance behaviour

-Peripheral (inner ear) vs central (brainstem)

25
Q

Classifying vertigo (time)

A
  • Acute vertigo
  • Recurrent, episodic
  • Chronic
26
Q

Types of acute vertigo

A
  • Isolated
  • +deafness (peripheral/ ENT)
  • +other neurological features (central/ neurology)
27
Q

What is vestibular neuronitis?

A

-Acute Unliteral Idiopathic Peripheral Vertigo (AUIPV)
=Vestibular nerve affected before joins cochlear nerve
=Inflammatory/ viral/ vascular
=NOT labyrinthitis (cochlear also damaged)

28
Q

Clinical presentation of AUIPV

A
  • May come on over a few hours/ may awake with it
  • Severe rotatory vertigo, accompanying vertigo disturbances (nausea), exacerbated by head movement (injured vestibular side stimulated)
  • Often prostrate for around a week, gradual recovery with good prognosis
  • Vertigo on sudden head movements may persist

-Recovery: resolution and central compensation

29
Q

What is Benign Paroxysm Positional Vertigo?

A
  • BPPV
  • Attacks of rotational vertigo
  • Generally last 10-20 seconds, may be brief nausea
  • Provoked by positional change
30
Q

What is BPPV caused by?

A

-Fragments of otoliths in semi-circular canals
=Preceding history of head trauma in some cases
=Good prognosis
=Positional exercises can help