Burns Flashcards

(76 cards)

1
Q

Management of Patients with Burn Injuries

Most burns occur in _____

A

the home.
Prevention is key!

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2
Q

Functions of the skin

A

Protection from infection and injury
Prevents loss of body fluids
Regulates body temp
Provides tactile sense

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3
Q

The strongest predictors for mortality

A

Increased % TBSA
Presence of inhalation injury
Increased age

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4
Q

Types of burns

A

Thermal
Chemical
Electrical
Radiation
Inhalation

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5
Q

Thermal burns:

A

: exposure to heat- flame, flash, scald, or contact with hot objects

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6
Q

Chemical burns

A

acids, alkaline agents, or organic compounds

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7
Q

Electrical burns:

A

severity based on voltage and length of exposure. Risk for potential cervical spine injury

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8
Q

Radiation exposure:

A

thermal effect; damage to the cellular DNA

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9
Q

Inhalation injuries:

A

inhalation of thermal and/or chemical irritants (upper vs lower airway injury)

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10
Q

_____ is the most common type of burn.

Especially:

A

Thermal
85% of all burns are thermal.

scalding in children (very curious) and elderly.

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11
Q

Chemical – tissue continues to burn until:

-\_\_\_\_\_\_ must begin immediately \_\_\_\_\_\_\_ ***outcome improved for victim
A

chemical is completely removed. Dust off dry chemical. Remove clothing articles touching the skin.

Continuous irrigation
at the scene

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12
Q

Electrical- make sure:

Severity difficult to assess, may have:

A

scene is safe and victim no longer in contact with source.

exit and entrance wound, organ damage.

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13
Q

Zones of burn injury

At the center is the zone of ______, zone of most damage, tissue is ______

Zone of _____, injured cells, potentially salvageable, but with ______, necrosis can occur

Zone of _____: minimal injury, full recovery

A

Zone of coagulation
not viable

Zone of stasis
persistent ischemia

Zone of hyperemia

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14
Q

burn can evolve and worsen over time

A
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15
Q

At the center is the zone of coagulation, zone of most damage, tissue is _____

A

not viable

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16
Q

Zone of stasis, injured cells, potentially salvageable, but with persistent ________________

A

ischemia, necrosis can occur

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17
Q

Severity of Burn Injury

A

Depth of burn
Extent of burn
Location of burn
Age
Risk factors

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18
Q

Burn Depth Classifications

A

Superficial thickness (1st degree)

Partial thickness (2nd degree)

Full thickness (3rd & 4th degree)

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19
Q

Layers of skin affected :

1st- 4th

A

1st- epidermis

2nd- dermis

3rd- subcutaneous

4th- muscle

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20
Q

First Degree - Superficial

Involves only _____

Causes:

S&S:

Treatment:

A

epidermis.

Causes: radiation burn or brief exposure to heat source.

S&S: Redness, pain, moderate to severe tenderness; minimal edema, peeling, itching

Treatment: Mild analgesics, cool compresses, skin lubricants; heals within a few days

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21
Q

Second Degree - Partial Thickness

Involves:

Causes:

S&S:

Treatment:

A

epidermis & dermis; may extend into hair follicles.

scalds, flash flame, contact

Moist blebs, blisters, edema, mottled white, pink to cherry-red, moderate to severe pain

Usually heal 2-3 weeks, depending on depth and area; may require grafting

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22
Q

Third Degree -

Includes:

Causes:

S&S:

Treatment:

A

Full Thickness

epidermis, dermis, and sometimes subcutaneous tissue; may involve connective tissue and muscle

Causes: flame, prolonged exposure, electrical, chemical, contact

S&S: Dry, leathery, eschar, waxy white, dark brown, or charred appearance, strong burn odor
No pain at burn sites due to loss of nerve endings; severe pain in surrounding areas.

Surgical intervention required.

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23
Q

Fourth Degree –

Includes:

______ appearance

Causes:

Treatment:

A

Full Thickness

deep tissue, muscle, and bone

Charred

Causes: prolonged exposure or high voltage, electrical injury

Amputations likely; grafting of no benefit

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24
Q

Inhalation injury

Caused by inhalation of _____ and/or _____ irritants

Upper vs lower airway injury:

History of injury important
Hx of injury: suspect inhalation with _________ and burns of ____, ____, and ____

Burns of the face, mouth, anterior neck

Clinical signs:

_______ for definitive diagnosis

A

thermal and/or chemical

Upper vs lower airway injury: Upper airway above the glottis, lower airway below the glottis

enclosed spaces

face, mouth, anterior neck

singed facial hair, carbonaceous sputum, hoarse voice, stridor

Bronchoscopy

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25
Extent of Burn – Rule of Nines Expressed as a percent of total body surface area (TBSA). Divide body surfaces into multiples of nine.
Head- 9% Left arm- 9% Right arm- 9% Anterior chest 18% Posterior chest 18% Left leg- 18% Right leg- 18% Genital region- 1% Note: anterior and posterior of head 9%. Anterior chest 9 + abd 9 =18% If approximately half of arm were burned, the TBSA burned = 4.5%; anterior thigh + anterior lower leg = 4.5 % (total 9%)
26
_______ is another method and is more accurate for children With all methods, estimate at initial evaluation and again _________
Lund-Browder chart 72 hours later.
27
Children and burns Prevention! Splash and spill burns from hot food off stove is common Hallmark signs of child abuse:
Definite line of demarcation Frequent or repetitive hospital visits Symmetrical burn wounds Cigarette burns
28
Other vulnerable populations
The elderly patient Clients with reduced mental capacity People with reduced mobility and/or sensory impairments
29
Electrical- make sure scene is safe and victim no longer in contact with source. Severity difficult to assess, may have exit and entrance wound, organ damage. Monitor:
ECG- arrhythmias possible
30
_____ of burn determines treatment- surgical grafting?
Depth
31
Do not put anything on a burn!!!!! Any burn that is circumferential- all around the arm- worried about:
compartment syndrome- check pulses, cap. Refill.
32
Burn Centers Most minor burn injuries can be managed in community hospitals Statewide: Jaycee Burn Center - Chapel Hill, NC Wake Forest Baptist Medical Center Burn Center - Winston Salem, NC Burn Center Referral Criteria -
Partial thickness burns greater than 10% total body surface area (TBSA).
33
On the Scene Care
Prevent injury to rescuer Stop injury: extinguish flames or remove from the source Cool the burn Remove restrictive objects Cover the wound Irrigate chemical burns Primary survey: ABCDE
34
On the Scene Care Treat patient with falls and electrical injuries as for potential ______ injury. Don’t touch person if still in contact with the _______ Do:
cervical spine electrical current assessment surveying all body systems and obtain a history of the incident and pertinent patient history
35
Airway management Administer _______ if carbon monoxide poisoning suspected Consider_____ & ______ (esp. burns to the face & neck) Place in ______ position (unless spinal cord injury) _______ if needed to relieve resp. distress Turn, cough, deep, breath Provide suctioning & chest physiotherapy Bronchoscopy _______ to treat severe bronchospasm
100% humidified O2 early intubation and ventilator support high Fowlers Escharotomy Bronchodilators
36
Why consider early intubation and ventilator support for burns to face and neck? Edema? What is escharotomy? – Dead tissue- cutting in to dead tissue to allow for expansion Signs and symptoms of carbon monoxide poisoning?
Dull headache Weakness Dizziness Nausea or vomiting Shortness of breath Confusion Blurred vision Loss of consciousness
37
carbon monoxide Odorless, tasteless, colorless, gas, binds to O2 molecule..?
38
Phases of Burn Injury
I. Emergent: onset of injury to completion of fluid resuscitation II. Acute: from beginning of diuresis to near completion of wound closure III. Rehabilitative: from wound closure to return to optimal physical and psychosocial adjustment
39
Emergent Phase: (____ days) Onset of ______ and _____
0-3 days Onset of hypovolemic shock and edema
40
Emergent Phase Fluid & Electrolyte Shifts Increased ____________ loss of intravascular proteins & fluids into the _______ _____ and _____ Increased insensible water loss by ______ Hemolysis of RBCs (elevated Hct from ______) Major electrolyte shifts:
CAPILLARY PERMEABILITY interstitial compartment edema and decreased blood volume evaporation HEMOCONCENTRATION HYPERkalemia, HYPOnatremia
41
What does Increased capillary permeability mean? Fluid loss from burn skin 5-10 times greater from undamaged skin Peak fluid leak at ___, but continues up to ___
Water, electrolytes, proteins leak out of vasculature=EDEMA 6-8 hr 36 hr
42
Massive fluid shift ***Electrolyte shift– sodium and potassium switch leading to hyperkalemia and hyponatremia Low sodium- <135 Hyperkalemia- >5
43
Burn Shock Fluid shifts + fluid losses = What do you think vital signs will look like in burn shock?
Intravascular volume depletion Low bp, high hr
44
Emergent Phase Pathophysiology continued Inflammation & healing ______ occurs-repair begins within ______ Immunologic changes ____ immune response Burns < ___ TBSA produce primarily a local response; burns > ___ = local and systemic response (includes release of cytokines and other mediators) Burns cause the proteins inside the cells to denature and coagulate, resulting in a form of cell death known as ________
Coagulation necrosis - 6-12 hrs after injury Reduced 25% coagulative necrosis.
45
CV: Pulmonary: Kidney: Thermoregulatory: GI:
CV: decreased cardiac output, decreased BP, increased HR, edema Pulmonary: inhalation injury, upper and lower airway injury Kidney: function may be affected with hypovolemia Thermoregulatory: inability to regulate body temp (low at first, then elevated) GI: organ ischemia and dysfunction
46
Management of Emergent Phase
Fluid resuscitation (next slide) Pain management Foley catheter NPO or NGT to low intermittent suction Continuously monitored ECG with electrical burns Emotional support NGT with burns > 25% TBSA
47
Therapeutic Management:Fluid resuscitation ___ of injury _____ or ____ preferred for major burns ___ during first 24 hours Fluid calculation based on BSA ex. ABA Indicator of adequate fluid resuscitation: Baseline ____
Time Large bore IV or central line LR urine output: 0.5-1 mL/kg/hr* *more for electrical burns Rely on urine output instead of BP (bc BP is not an accurate estimate) Want at least 30 ml/hr weight
48
Fluid resuscitation formula Don’t have to memorize. Just know a massive amount of fluids are given ~1400 ml in 24 hrs
49
Acute phase (_______) Begins with: Concludes when:
Acute phase- 48-72 hrs to weeks to months Begins with mobilization of extracellular fluid and diuresis Concludes when partial thickness wounds are healed, or full thickness burns are covered by skin grafts lymphatics reabsorb fluid=diuresis
50
Acute Phase Fluid and Electrolyte Shifts Fluid reenters the vascular space from the interstitial space (______) Increased _____ Sodium is lost with diuresis and due to dilution as fluid enter vascular space: hyponatremia, varies Potassium shifts from extracellular fluid into cells: potential _____ Metabolic ____
HEMODILUTION urinary output HYPOkalemia acidosis
50
Acute Phase Fluid and Electrolyte Shifts Fluid reenters the vascular space from the interstitial space (______) Increased _____ Sodium is lost with diuresis and due to dilution as fluid enter vascular space: hyponatremia, varies Potassium shifts from extracellular fluid into cells: potential _____ Metabolic ____
HEMODILUTION urinary output HYPOkalemia acidosis
51
What happens if you were overaggressive with fluid resuscitation during emergent phase?
SOB, crackles, …listen to lungs
52
Acute Phase Healing Partial thickness wounds: healing begins, after: Full thickness wounds: require _______________ to heal Early excision (surgical removal) reduces effects of _______
eschar is removed, re-epithelization begins, heals within 10-21 days surgical debridement and skin grafting inflammatory mediators
53
Rehabilitation Phase Begins once: May happen within 2 weeks or even months later. Depends on ____ of burns Rehab goals: Common Complications:
the client's wounds have healed and client is prepared to engage in self care extent Resume functional role in society Rehabilitate from reconstructive surgery (cosmetic or functional) contractures and scarring
54
Therapeutic Management:Pain management: Poor _______, IV in small repeated doses May give _____ prior to dressing changes
Opioids NSAIDs Anxiolytics Sedatives Anesthetic agents Antidepressants tissue perfusion PO meds
55
What’s the difference between background pain, procedural pain, and breakthrough pain?
56
Therapeutic Management:Wound care
Wound cleaning Topical antibacterial therapy Wound dressing Wound debridement (4 types)
56
Therapeutic Management:Wound care
Wound cleaning Topical antibacterial therapy Wound dressing Wound debridement (4 types)
57
Wound debridement (4 types)
Natural- from dressing removal Mechanical- done in bath (keep room warm) Chemical- topical Surgical- go to OR
58
Therapeutic Management:Wound grafting Types (autograft, homograft, xenograft) Care of donor site Care of graft site
59
Therapeutic Management:Prevention of infection ____ is the #1 issue.
Infection controlled environment Monitor temperature (hyperthermia common after BI) Tetanus vaccination Antibiotic or antifungal per wound culture results Controlling hypergylcemia. A lot of patients will develop insulin resistance Sepsis
60
Therapeutic Management:Restoration of function
Proper positioning Specialty beds Passive and active ROM Splints of functional devices Compression garments OT and PT Functional and cosmetic reconstruction
61
Compression garments: wear __________, to minimize __________
most of the day, up to a year contractures and scarring
62
Complications Resp.: Cardio: GI: Kidney :
Sepsis Respiratory: acute resp failure, ARDS Cardiovascular: heart failure, pulmonary edema GI: paralytic ileus, Curling’s ulcer, translocation of bacteria, abdominal compartment syndrome Kidney: myoglobinuria-> ATN (ATN- acute tubular necrosis )
63
Nutrition ________ state – feed as soon as able to eat When oral route used: Intubated patients – Three-fold increase in basal metabolic rate
Hypermetabolic high-protein, high calorie meals and supplements enteral feedings
64
Emotional/psychological needs Remember, a burn injury can be a crisis for a family. Elicit patient wishes as early as possible.
65
Start enteral nutrition therapy early:
blunts the metabolic response, maintain gut viability, decrease bacterial translocation
66
Burn patients with >20% TBSA injury suffer long and severe response to injury:
hyperdynamic and hypermetabolic response (catabolic state, muscle wasting)
67
Inflammatory response can remain elevated for ____ after injury
months
68
Electrical injuries require:
baseline ECG and heart monitoring, larger fluid volume resuscitation (4ml/kg/TBSA) and want greater urine output, potential for cervical spine injury
69
Skin reproducing cells are located along shafts of hair follicles and sweat glands. Burn depth determines if spontaneous re-epithelization will occur
70
Smoke inhalation is major predictor of mortality: inhalation injury disrupts the supply of oxygen to the body by immense swelling of the upper respiratory tract, chemical irritation of the lower respiratory tract, and injuries resulting from noxious gases, such as carbon monoxide and cyanide
71
_________ is best indicator of adequate fluid resuscitation
Urine output
72
Early _____ reduces effects of inflammatory mediators
excision
73
Need ________ pain assessment and management
around the clock
74
Goal of ______ with insulin resistance
normoglycemic