Burns Flashcards

(27 cards)

1
Q

Epidermis

A

Protective outer layer
- hair

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2
Q

Dermis

A

2nd layer
- capillaries
- nerves
- sebaceous gland
- hair follicle

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3
Q

Hypodermis

A

3rd layer
- fat
- smooth muscle
- nutrient deposit
- heat insulator
- shock absorber

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4
Q

Skin’s Function

A
  • Maintain body temp
  • Barrier to evaporative water loss
  • Metabolic ativity
    > vit D production
  • 1st line against microbes
  • Protect against envirn’t
  • Sensations
    > touch, pressure, pain
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5
Q

Pathophysiology of a Burn

A
  • Intravascular fluid vol deficit due to loss of fluids from the intravascular space to the extravascular space results in
    > incrs blood viscosity; blood more like syrup, hard on heart = more resistance = incrd afterload
    > incrd afterload
    > dcrd peripheral & capillary flow; blood too thick to get into these smaller spaces, oxygen exchange & ventilation affected
    > multiple organ systems are affected
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6
Q

Patho of a Burn - Multi Organ Systems Affected

A
  • CV
    > heart goes into over-drive
  • Renal
    > hypoperfused; AKI
  • Pulmonary
  • GI
  • Immunological
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7
Q

Zones of Injury

A
  • Immediately after injury, the burn wound can be divided into 3 zones
    > what areas can be saved vs what areas are going to die
  • Zone of Coagulation
  • Zone of Stasis
  • Zone of Hyperemia
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8
Q

Zones of Injury - Zone of Coagulation

A

Is the central portion with the most damage, bc its usually the site of greatest heat transfer, leading to irreversible skin death
surrounded by zone of stasis

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9
Q

Zones of Injury - Zone of Stasis

A

or Zone of Ischemia: characterized by dcrd perfusion tht is potentially salvageable if edema is controlled & perfusion is incrd
yellow skin

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10
Q

Zones of Injury - Zone of Hyperemia

A

The outermost region of the wound characterized by incrd inflammatroy vasodilation
pinkish/red skin

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11
Q

All burns cause

A

Tissue destruction due to energy transfer

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12
Q

Causes of Burns

A
  • Thermal
  • Chemical
  • Radiation
  • Electrical
    > direct current (DC)
    > alternating current (AC)
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13
Q

Causes of Burns - Thermal

A

Steam, scalds, contact w/ heat, fire

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14
Q

Causes of Burns - Chemical

A
  • Acidic agents: cause coagulation necrosis, tissue looks dry
  • Alkali agents: cause colliquative necrosis, tissue looks wet
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15
Q

Causes of Burns - Radiation

A
  • Exposure to industrial equipment, equipment used for medical treatment
    > don’t appear immediately
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16
Q

Causes of Burns - Electrical

A
  • Direct Current (DC)
    > everything tht runs off a battery, plugs in to the wall w/ an AC adapter, or uses a USB cable for power relies on DC (dry cell battery, car battery, DC generator, radio, television, defibrillator)
    > DC causes a single convulsion or contraction, usually propelling the person away from the electrical source
    > exposure is not very long
  • Alternating Current (AC) (maintain contract until source is cut off)
    > AC is used to deliver power to houses, office buildings (electrical outlets, power lines)
    > maintain contract w/ the source
    > may present w/ only superficial burns, many devasting injuries if there is prolonged contract or muscle tetany
    > low-voltage AC injuries may potentially result in cardiac or resp arrest, arrhythmias (v-fib), or seizures tht are unwitnessed
    > high-voltage AC injuries are more likely to result in highly destructive thermal burns
17
Q

Causes of Burns - Electrical: Lightning

A
  • Has both the properties of an AC and DC signal
    > it has polarity and always travels in a single direction (DC)
    > voltage of lightning is not constant, it has variable amplitude
18
Q

1st Degree Burns

A
  • Burns tht affect the uppermost layer of skin (epidermis only) are classed as superficial (1st degree) burns; the skin becomes red/pink and the pain is limited in duration
    > sunburns & minor steam burns
19
Q

2nd Degree Burns - Superficial Partial Thickness

A
  • Burns tht involve all the epidermis and part of the underlying dermis
  • Appearance: light to bright red or mottled appearance may appear wet and weeping, may contain bullae (blister), are extremely painful and sensitive to air currents, blanch painfully
  • microvessels are injured = leaving plasma into interstitial space = blisters
  • brief contact w/ flames, hot liquid, or exposure to dilute chemicals
  • heal in 7-21 days, expect minimal scarring, generally don’t need surgery
  • may turn into full-thickness injuries if they become infected,
20
Q

2nd Degree Burns - Deep Partial Thickness

A
  • Involves the entire epidermal layer & deeper layers of the dermis
  • Appearance: red w/ patchy white areas tht blanch w/ pressure, appearance of deep-dermal wound changes over time, dermal necrosis & surface coagulated protein turn wound from white to yellow
    > don’t usually see blisters
    > long exposure to heat source
  • May require a skin substitute or surgial excision and grafting for wound closure
  • Treatment: surgical excision & skin grafting
    > maintain adequate perfusion
    > avoid infection
21
Q

Full-Thickness (3rd Degree) Burn

A
  • Involves the destruction of all the layers of the skin down to and including the subcutaneous fat and is poorly vascularized
    > full-thickness burns usually are painless & insensitive to palpation
    > bc all the epithelial elements are destroyed, the wound does not heal by re-epithelialization
    > appears pale white or charred, red or brown, and leathery. The surface of the burn may be dry, and if the skin is broken, fat may be exposed
  • Treatment: require sin grafting for closure
22
Q

3rd Degree pts are high risk for

A
  • Infections
  • Fluid & electrolyte imbalances
  • Alts in thermoregulation
  • Metabolic disturbances
23
Q

4th Degree Burn

A

Involves injury to deeper tissues, such as muscle or bone is often blackened, and frequently leads to loss of the burned part

24
Q

Inhalation Injury - Carbon Monoxide Poisoning

A
  • CMs
    > early: headache, dizziness, nausea, vomitting, dyspnea, tachycardia, tachypnea, confusion, & lightheadedness
    > late: dcrd end-organ perfusion
    > heart: myocardial ischemia & cardiac dysfunction
    > CNS: dcrd LOC, unconsciousness, coma, unresponsiveness
    > resp: resp failure
  • Treatment: high-flow oxygen admin at 100% through a tight-fitting nonrebreathing mask or endotracheal intubation
    > esp. if airway is compromised
25
Inhalation Injury - Upper Airway Injury ## Footnote assessment
- **Burns (from heat or chemicals) of the upper resp tract include burns involving the pharynx, larynx, glottis, trachea, and larger bronchi** - **Assessment**: extent of injury to face & neck, pressure of blisters on or redness of posterior pharynx, signs of singed nose hairs, incrd HbCO lvls, incrd RR, dcrd depth of breathing, hoarseness, stridor, incrd amnt of sputum > frequent airway assessment during hosp stay is essential in this pop as supraglottic edema may be delayed until fluid resus begins & 3rd spacing occurs
26
Inhalation Injury - Upper Airway Injury ## Footnote CMs treatment
- **CMs** > hoarseness > stridor > audible airflow turbulence > production of carbonaceous sputum - **Treatment** > early intubation > after airway is secure: HOB elevated, deep breath & cough, suctioning, ambulation, pulm hygiene, meds for bronchospasm
27
Inhalation Injury - Lower Airway Injury
**Chemical injuries to the mucosal surfaces**