Burns Flashcards
(27 cards)
Epidermis
Protective outer layer
- hair
Dermis
2nd layer
- capillaries
- nerves
- sebaceous gland
- hair follicle
Hypodermis
3rd layer
- fat
- smooth muscle
- nutrient deposit
- heat insulator
- shock absorber
Skin’s Function
- Maintain body temp
- Barrier to evaporative water loss
-
Metabolic ativity
> vit D production - 1st line against microbes
- Protect against envirn’t
-
Sensations
> touch, pressure, pain
Pathophysiology of a Burn
-
Intravascular fluid vol deficit due to loss of fluids from the intravascular space to the extravascular space results in
> incrs blood viscosity; blood more like syrup, hard on heart = more resistance = incrd afterload
> incrd afterload
> dcrd peripheral & capillary flow; blood too thick to get into these smaller spaces, oxygen exchange & ventilation affected
> multiple organ systems are affected
Patho of a Burn - Multi Organ Systems Affected
-
CV
> heart goes into over-drive -
Renal
> hypoperfused; AKI - Pulmonary
- GI
- Immunological
Zones of Injury
-
Immediately after injury, the burn wound can be divided into 3 zones
> what areas can be saved vs what areas are going to die - Zone of Coagulation
- Zone of Stasis
- Zone of Hyperemia
Zones of Injury - Zone of Coagulation
Is the central portion with the most damage, bc its usually the site of greatest heat transfer, leading to irreversible skin death
surrounded by zone of stasis
Zones of Injury - Zone of Stasis
or Zone of Ischemia: characterized by dcrd perfusion tht is potentially salvageable if edema is controlled & perfusion is incrd
yellow skin
Zones of Injury - Zone of Hyperemia
The outermost region of the wound characterized by incrd inflammatroy vasodilation
pinkish/red skin
All burns cause
Tissue destruction due to energy transfer
Causes of Burns
- Thermal
- Chemical
- Radiation
-
Electrical
> direct current (DC)
> alternating current (AC)
Causes of Burns - Thermal
Steam, scalds, contact w/ heat, fire
Causes of Burns - Chemical
- Acidic agents: cause coagulation necrosis, tissue looks dry
- Alkali agents: cause colliquative necrosis, tissue looks wet
Causes of Burns - Radiation
-
Exposure to industrial equipment, equipment used for medical treatment
> don’t appear immediately
Causes of Burns - Electrical
-
Direct Current (DC)
> everything tht runs off a battery, plugs in to the wall w/ an AC adapter, or uses a USB cable for power relies on DC (dry cell battery, car battery, DC generator, radio, television, defibrillator)
> DC causes a single convulsion or contraction, usually propelling the person away from the electrical source
> exposure is not very long -
Alternating Current (AC) (maintain contract until source is cut off)
> AC is used to deliver power to houses, office buildings (electrical outlets, power lines)
> maintain contract w/ the source
> may present w/ only superficial burns, many devasting injuries if there is prolonged contract or muscle tetany
> low-voltage AC injuries may potentially result in cardiac or resp arrest, arrhythmias (v-fib), or seizures tht are unwitnessed
> high-voltage AC injuries are more likely to result in highly destructive thermal burns
Causes of Burns - Electrical: Lightning
-
Has both the properties of an AC and DC signal
> it has polarity and always travels in a single direction (DC)
> voltage of lightning is not constant, it has variable amplitude
1st Degree Burns
-
Burns tht affect the uppermost layer of skin (epidermis only) are classed as superficial (1st degree) burns; the skin becomes red/pink and the pain is limited in duration
> sunburns & minor steam burns
2nd Degree Burns - Superficial Partial Thickness
- Burns tht involve all the epidermis and part of the underlying dermis
- Appearance: light to bright red or mottled appearance may appear wet and weeping, may contain bullae (blister), are extremely painful and sensitive to air currents, blanch painfully
- microvessels are injured = leaving plasma into interstitial space = blisters
- brief contact w/ flames, hot liquid, or exposure to dilute chemicals
- heal in 7-21 days, expect minimal scarring, generally don’t need surgery
- may turn into full-thickness injuries if they become infected,
2nd Degree Burns - Deep Partial Thickness
- Involves the entire epidermal layer & deeper layers of the dermis
-
Appearance: red w/ patchy white areas tht blanch w/ pressure, appearance of deep-dermal wound changes over time, dermal necrosis & surface coagulated protein turn wound from white to yellow
> don’t usually see blisters
> long exposure to heat source - May require a skin substitute or surgial excision and grafting for wound closure
-
Treatment: surgical excision & skin grafting
> maintain adequate perfusion
> avoid infection
Full-Thickness (3rd Degree) Burn
-
Involves the destruction of all the layers of the skin down to and including the subcutaneous fat and is poorly vascularized
> full-thickness burns usually are painless & insensitive to palpation
> bc all the epithelial elements are destroyed, the wound does not heal by re-epithelialization
> appears pale white or charred, red or brown, and leathery. The surface of the burn may be dry, and if the skin is broken, fat may be exposed - Treatment: require sin grafting for closure
3rd Degree pts are high risk for
- Infections
- Fluid & electrolyte imbalances
- Alts in thermoregulation
- Metabolic disturbances
4th Degree Burn
Involves injury to deeper tissues, such as muscle or bone is often blackened, and frequently leads to loss of the burned part
Inhalation Injury - Carbon Monoxide Poisoning
-
CMs
> early: headache, dizziness, nausea, vomitting, dyspnea, tachycardia, tachypnea, confusion, & lightheadedness
> late: dcrd end-organ perfusion
> heart: myocardial ischemia & cardiac dysfunction
> CNS: dcrd LOC, unconsciousness, coma, unresponsiveness
> resp: resp failure -
Treatment: high-flow oxygen admin at 100% through a tight-fitting nonrebreathing mask or endotracheal intubation
> esp. if airway is compromised