Cardiac Flashcards
(130 cards)
function of the SA node
pacemaker
60-100 beats/min
function of the AV node
receives impulses from SA node
if SA node fails, AV node starts
40-60 beats/min
function of purkinje fibers
acts as pacemaker when SA and AV nodes fail
20-40 beats/min
what happens when the coronary arteries are blocked?
increased risk of MI
heart sounds
1st (S1): heard at apex of heart
2nd (S2): heard at base of heart
3rd (S3): occurs with heart failure and regurg
4th (S4): atrial asystole, abnormal, causes cardiac hypertrophy disease or injury
sinus tach
> 100 beats/min
faster the heart rate the less cardiac output
sinus brady
<60 beats/min
treat if patient is symptomatic (decreased cardiac output)
low HR is normal for athletes
what does renin do?
vasoconstriction
increases BP
what does vasopressin (ADH) do to BP
ADH influences regulation of vascular volume.
when there is an increase blood volume less ADH will be released, increase urination, decreasing blood voulme and BP
when there is a decrease in blood volume more ADH will be released, which promotes blood volume to increase and increase BP
arteries
take oxygenated blood AWAY from heart
veins
transport deoxygenated blood to the heart
troponin
protein that increases with an MI
rises within 3 hours and last 7-10 days
normal level < 0.3
myoglobin
oxygen binding protein that rises within 2 hrs of cell death
RBC
4.2-6.1
decreases in rheumatic heart disease and infective endocarditis
increases in conditions where there is an inadequate tissue oxygenation
WBC
5-10
increases with infection and inflammation and after MI
H&H
hgb 12-18
hct 32-57
elevated hct: vascular volume depletion
decreased H&H: anemia
effects of potassium on the heart
hypokalemia: ventricular dysrythmias and increase risk of dig toxicity. flat and inversed T wave, U wave, and depression of ST
hyperkalemia: asystole and ventricular dysrhythmia. tall peaked T wave, wide QRS complex, prolonged PR intervals, flat P waves
effects of sodium on heart
decreases with diuretics
decreases with heart failure
effects of calcium on heart
hypocalcemia: ventricular dysrhythmias, prolonged ST and QT interval, and cardiac arrest
hypercalcemia: short ST segment and wide T wave, AV block, tachycardia or bradycardia, dig hypersensitivity and cardiac arrest
effect of phosphorus on heart
interpreted with calcium levels because kidney retain or excrete one or the other
when calcium is high phosphorus is low or vice versa
effects of mag on heart
low mag: vtach or vfib
tall T wave
depressed ST segment
high mag: muscle weakness hypotension and bradycardia.
long PR interval
wide QRS
BUN and heart
elevated BUN with heart failure and cardiogenic shock due to effects on renal circulation
BNP
anything >100 is heart failure
meanings of the ECG components
reflects electrical activity of cardiac cells and record electrical activity
P wave: atrial depolarization
PR interval: time it take impulse to get from atria to AV nose bundle of his to purkinje fibers (0.12-0.2 sec)
QRS complex: ventricular depolarization (0.04-0.1 sec)
ST segment: early ventricular repolarization
T wave: ventricular repolarization
U wave: follows T wave and indicates electrolyte issues
QT interval: total time required for ventricular depolarization and repolarization (0.32-0.4)