Cardiac Arrest Flashcards

1
Q

What are the 2 different algorithm paths used to treat Adult Cardiac Arrest?

A

-VF/pVT
-Asystole/PEA

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2
Q

Because many pts will sudden cardiac arrest demonastrate VF at some point in their arrests, most ACLS providers will often follow the VF/pVT algorithm—Why is this the best approach?

A

Rapidly treating VT according to this sequence is the best approach to restoring spontaneous circulation.

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3
Q

VF/pVT will require CPR until a defibrillator is available to deliver high-energy Unsynchronized shocks.

A
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4
Q

What are atonal gasps a S/O cardiac arrest?

A

-Agonal gasps are not normal breathing
-A pt who gasps usually appears to be drawing air in very quickly
-The mouth may be open and the jaw, head or neck may move w/ gasps
-Gasps may appear to be forceful or weak, usually a slow rate
-The gasp may sound like a snort, snore or groan
-Gasping is a S/O cardiac arrest.

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5
Q

Describe the shock energy for VF/pFVT for a biphasic defibrillator:

A

-Manufacturer recommendation (Zoll—120/150/200)
-If unknown, recommended shock energy use maximum available; second and subsequent shock doses should be equivalent & higher doses may be considered.

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6
Q

What is the shock energy dose for VF/pVT for a Monophysite defibrillator?

A

-360 J

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7
Q

What is the drub therapy for VF/pVT?

A

-Epinephrine 1 mg Q3-5 minutes IV/IO
-Amiodarone 300 mg IV/IO first dose; 150 mg IV/IO second dose
OR
-Lidocaine 1-1.5 mg/kg IV/IO first dose; 0.5-0.75 mg/kg IV/IO second dose

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8
Q

Coronary perfusion pressure (CPP) is Aortic Relaxation (diastolic) pressure minus Right Atrial relaxation (diastolic) pressure. During CPR, PCC correlates w/ both may cardiac blood flow & ROSC. In 1 human study, ROSC did not occur unless a CPP 15 mg Hg or higher was achieved during CPR. What can providers do to achieve the best CPP possible?

A

-Provide good chest compressions w/ minimal interruptions.

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9
Q

In adult cardiac arrest but do VF/pVT, the heart is quivering & not effectively pumping blood to vital organs. What can be done so that these pts have a much higher survival rate?

A

-Immediate chest compressions and CPR

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10
Q

The first 4-6 minutes after cardiac arrest, referred to as ‘Clinical Death’, no damage occurs to the brain. In 6-10 minute period, ‘biological death’ after cardiac arrest, damage is likely to occur to the brain. Brain damage is usually irreversible, except in special circumstances such as accidental hypothermia or cold water drowning.

A
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11
Q

Why is the interval from collapse (cardiac arrest) to defibrillation one of the most important determinants of survival from cardiac arrest?

A

-A common initial rhythm in out-of-hospital witnessed cardiac arrest is VF.
-pVT rapidly deteriorates to VF and then the heart is quivering & does not pump blood
-Electrical defibrillation is the most effective way to treat VF (delivery of shock to stop the VF) & pVT.
-The probability of successful defibrillation decreases over time.
-VF deteriorates to Asystole if not treated.

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12
Q

When VF is present, CPR can provider a small amount of blood flow to the heart & brain but cannot restore an organized rhythm. Why is early defibrillation important?

A

-The earlier defibrillation occurs, the higher the survival rate.
-Restoring a perfusing rhythm is more likely w/ immediate CPR & defibrillation within a few minutes after the initial arrest.

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13
Q

What should be done after defibrillation?

A

-Immediately resume CPR beginning w/ compressions. Do not perform a rhythm check at this point unless the pt is showing S/O life, such as ROSC.

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14
Q

What is the effect of Epinephrine of the body and how does it help w/ cardiac arrest?

A

-Epinephrine causes vasoconstriction which will increase cerebral & coronary blood flow during CPR by increasing mean arterial pressure and aortic diastolic pressure.

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15
Q

When is Magnesium Sulfate given?

A

-for Torsades de Pointes
-Loading Dose 1-2 g IV/IO over 20 minutes.

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16
Q

For the intubated pt, what is the best way to monitor CPR quality, optimize chest compressions & detect ROSC.

A

-Quantitative waveform caphography

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17
Q

How is ROSC determined by observing PETCO2?

A

-ROSC is evident from the abrupt increase in PETCO2 to >50 mm Hg
-PTCO2 >50 mm Hg is consistent w/ a substantial improvement in blood flow.

18
Q

What is the main determinant of ERCO2 during CPR?

A

-Blood delivery to the lungs

19
Q

How can ERCO2 reading be improved?

A

-Improve chest compressions
-Vasopressor therapy

20
Q

When drugs are given via peripheral IV route during CPR how long does it take to reach the central circulation?

A

-1-2 minutes—> follow drug w/ a 20 ml bolus of IVF and elevate extremity for about 10-20 seconds to help deliver drug to central circulation

21
Q

What is the preferred route of administration for drugs during CPR?

A

-IV or IO route
-ET is not preferred if IV/IO are available.

22
Q

Studies demonstrate that the circulation system absorbs epinephrine, vasopressin & Lidocaine after administration via ER route. When giving via ET route, dilute drug in 10-15 ml SWI or NSS & inject directly into the ETT.

A
23
Q

A post cardiac arrest reasonable B/P is a MAP of 65 or greater. Adjust fluid administration or vasoactive or IO tropic agents as needed to aptitude cardiac output and systemic perfusion.

A
24
Q

Ultrasound may be applied to pts receiving CPR to help assess myocardial contractility & identify potentially treatable causes of cardiac arrest. What are some causes of cardiac arrest that can be determined by U/S?

A

-Hypovolemia
-Pneumothorax
-Pulmonary Thromboembolism
-Pericardial Tamponade

25
Q

What are the rhythms for PEA?

A

-Rate—>too fast or too slow
-Width of QRS—>wide vs narrow

26
Q

What drugs are used for PEA & Asystole?

A

-Epinephrine
-Other medications, depending on the cause of PEA or Asystole arrest.

27
Q

What is PEA?

A

-Any organized rhythm w/o a pulse

28
Q

What is the definition of an organized rhythm?

A

-QRS complexes that are similar in appearance from beat-to-beat
-QRS may b e narrow or wide
-QRS complexes may occur at rapid or slow rate, regular or irregular

29
Q

What are examples of rhythm for PEA

A

-Sinus Rhythm
-A Fib/Flutter
-BBB
-Idioventrilcular & ventricular escape rhythm

30
Q

What rhythm is the most common present after successful defibrillation?

A

-PEA due to cardiac function being too weak to produce a pulse or cardiac output

31
Q

If good PCR produces a strong pulse, relatively high ETCO2 or B/P it is more likely that the L ventricle is full and the cause of PEA is a poorly contractile L ventricle.

A
32
Q

If good CPR does not produce evidence of good cardiac output, what is a likely cause that the L ventricle is empty.

A

-Consider H’s and T’s
-Hypovolemia

33
Q

When is Asystole most often seen?

A

-As a terminal rhythm ini a resuscitation attempt that started w/ another rhythm.
-As the first rhythm identified in a pt w/ unwitnessed or prolonged.
-Persistent Asystole represents extensive myocardial ischemia & damage from prolonged periods of inadequate coronary perfusion.

34
Q

News the 5 H’s

A

-Hypovolemia
-Hypoxia
-Hydrogen Ion (Acidosis)
-Hyper/Hypokalemia
-Hypothermia

35
Q

Name the 5 T’s

A

-Tension Pneumothorax
-Tamponade (cardiac)
-Toxins
-Thrombosis (pulmonary)
-Thrombosis (coronary)

36
Q

Special resuscitation interventions (such as Extracorporeal CPR or ECMO (Extracorporeal Membrane Oxygenation)) & prolonged resuscitative efforts may be indicated for [pts w/ hypothermia, drug OD, or other potentially reversible causes of arrest.

A
37
Q

What temp is considered “severe hypothermia”?

A

<30 C or 86 F

38
Q

Is it appropriate to defibrillate a pt w/ severe hypothermia?

A

Yes

39
Q

Is it reasonable to attempt additional defibrillation attempts (after Initial)?

A

Yes—while active rewarding is taking place

40
Q

When hypothermic, pts have a reduced drug metabolism & drugs may accumulate to toxic levels. What drug is considered safe to give according to standard ACLS protocol?

A

Vasopressin

41
Q

What is considered “mild” hypothermia?

A

30-34 C (86-93.2 F)

42
Q

What should be done for pts w/ mild hypothermia?

A

Start CPR, attempt defibrillation, give meds according to local protocols & (if in hospital) provide active core rewarding.