cardiac/cardiac monitoring and support Flashcards

(35 cards)

1
Q

Conditions associated with bradycardia

A

Autonomic
-Raised ICP
-Visceral pain
-Drugs (beta blockers)
-Epidural

Non autonomic
-MI
-Hypoxia
-Hypothermia
-Hypothyroidism

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2
Q

Heart territories ecg

A

Anterior: V1-V4
Inferior: 2, 3, aVf
Posterior infarct: Isolated ST depression V1, V2

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3
Q

Treatment of STEMI

A

-Aspirin
-PCI followed by anticoagulation with heparin/LMWH/thrombolysis
-glycoprotein 2b/3a inhibitors
-Glycaemic control
-Beta blockers
-Thrombolysis if pci not availabe if not contraindicated

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4
Q

Contraindications to thrombolysis:

A

<2 weeks post op
-Active peptic ulcer
-Previous haemorrhagic stroke
-Recent head injury
-Prolonged traumatic CPR

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5
Q

What main parameters affect cardiac function?

A

Preload
Intrinsic cardiac function
Afterload

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6
Q

Describe the causes of cardiac failure

A

Factors affecting preload:
-Hypovolaemia
-Fluid overload
-Pneumothorax/cardiac tamponade

Factors affecting intrinsic myocardial function
-ACS
-Arrythmia
-Chronic heart failure + ‘operative stress’
-Pneumothorax/cardiac tamponade
-Electrolyte disturbance (e.g. cardiac tamponade)

Conditions affecting afterload
-Aortic stenosis
-PE
-Pneumothorax/cardiac tamponade
-Aortic dissection

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7
Q

How would you manage someone in acute heart failure?

A

-CCRISP principles
-Oxygen, sit pt up, CPAP if practicable
-Stop IVI
-IV diuretics (80mg furosemide IV)
-IV morphine (2.5-5mg diamorphine) to aid vasodilatation (reduce afterload)
-Nitrates (patch, sublingual/buccal/IV)
-ECG
-Tx underlying cause (Arrythmia/PE/tamponade)

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8
Q

What is the definition of cardiogenic shock?

A

-Severe impairment of cardiac function with hypotension <90mmhg or 30mmhg less than patients ‘normal’systolic

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9
Q

What is the most common cause of cardiogenic shoci?

A

Ischaemia/infarction

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10
Q

What are the important points in managing a patient with a pacemaker?

A

-Any pt undergoing surgery should have had recent pacemaker check
-Diathermy should be as far from pt as possible (e.g. on thigh or under buttocks). Never put it behind the pacemaker
-Short bursts of diathermy rather than long bursts
-Bipolar is safer
-Avoid diathermy near pacemaker

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11
Q

What is the definition of shock?

A

-Acute circulatory failure, with inadequate tissue perfusion causing cellular hypoxia

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12
Q

What are the most common mechanisms of shock?

A

-Hypovolaemic
-Cardiogenic
-Obstructive
-Vasodilatory

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13
Q

What are the causes of hypovolaemic shock? (preload)

A

-Haemorrhage
-Dehydration
-Fluid loss

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14
Q

Causes of cardiogenic shock (intrinsic cardiac function)

A

-MI
-Arrythmia
-Heart failure
-Cardiac contusions due to trauma

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15
Q

Causes of obstructive shock (intrinsic myocardial function)

A

-PE (obstruction to right ventricular outflow)
-Cardiac tamponade (construction on heart)
-Pneumothorax (pressure on heart)

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16
Q

Vasodilatory (afterload)

A

-Sepsis
-Neurogenic shock
-Anaphylaxis
-Adrenal insufficiency

17
Q

Neurogenic shock

A

-Follows spinal transection (above level of T6) or brainstem injury with loss of sympathetic outflow beneath the level of the injury and consequent vasodilation

18
Q

What is the definition of cardiogenic shock?

A

-Inadequate tissue perfusion resulting directly from myocardial dysfunction

19
Q

CVP interpretation

A

High CVP (>15mmhg)–> right ventricular/biventricular failure

Low CVP (<5mmhg) –> hypovolaemia

Normal CVP: >8mmhg

Static measurement can be misleading; young pt may have normal CVP due to vasoconstriction but may be underfilled

FLuid challenge can resolve doubt: small fluid challenge can be given (100-200ml) and then CVP measured, if significant rise and remains high this suggests myocardial failure/dysfiunction

20
Q

What are the indications for invasive cardiac monitoring?

A

-Failure to maintain cardiovascular homeostasis with simple measures
-Procedures that give rise to rapid/profound changes in preload or afterload, e.g. AAA repair
-Treatement with vasoactive drugs that influence preload/afterload/myocardial function, to monitor response to treatment
-Pts at risk of developing low perfusion states e.g. high risk pt with poor cardiac function

21
Q

What is the CVP a measurement of?

A

-Preload
-Pressure within SCV as it enters right atrium, reflects ability of right heart to accept and deliver circulating volume

22
Q

What influences the CVP?

A

-Venous return
-Right heart coimpliance
-Intrathoracic pressure

23
Q

What are the indications for a central line?

A

-Administration of fluid replacement therapy for hypovolaemia when conventional access not possible, e.g. when concern exists about overtransfusion when there is uncertainty about fluid volume status
-Measure effect of vasoactive drugs on venous capacitance, particularly vasodilators
-To aid diagnosis of right heart failure: high pressure with low cardiac output
-Administration of potent drugs e.g. inotropes
-TPN (needs dedicated clean lumen)

24
Q

Complications of central line

A

Related to insertion
-Haemotoma/haemothorax
-Tension pneumothorax
-Air embolus
-Extravascular catheter placement
-Neuropraxia
-Lymphatic puncture
-Tracheobronchial puncture
-Sepsis

Related to catheter
-Knotting of catheter
-Catheter breakage

25
Descrbibe noradrenaline receptor/effect/clinical use
Receptor -Alpha adrenoreceptor agonist Effect -Arteriolar vasoconstriciton Clinical use -Septic shock with low SVR
26
Describe Adrenaline receptor/effect/clinical use
Receptor -Alpha and beta adrenoreceptor agonist, predominantly beta 1 adrenoreceptor agonist at low doses Effect -Positive inotropic and chronotropic. Vasoconstricts at high doses Clinical use -Widespread in conidtions of low cardiac output: usful in emergency situations
27
Dopamine
Receptor: -Alpha and beta adrenoreceptors. Dopamine (DA) 1 and 2 receptors Effect: -Low dose: splanchnic vasodilatation, increased renal and hepatic blood flow (DA1). High dose: vasoconstriction Clinical use -Used less frequently
28
Dobutamine
Receptor: -DA1, DA2 and beta adrenoreceptor agonist Effect -Reduses SVR and increases cardiac output CLinical use -Cardiogenic shock
29
What are the functions of the kidney?
-Elimination of water soluble waste products of metabolism other than CO2 -Elimination of water soluble drugs -Fluid and electrolyte homeostasis -Acid-base balance -BP control: RAAS system -Endocrine function: EPO and vitamin D production
30
What is normal urine output?
Normal: 1.5-2ml/kg/hr Oliguria: <0/5 Anuria: <100ml/day
31
What is the definition of AKI
-Abrupt (within 48 hrs) reduction in kidney function defined as absolute increase in creatinine of >26 micromol/L OR -Percentage increase in serum creatinine level of >50% from baseline OR -Reduction in UO (oliguria <0.5ml/kg/hr for >6 hrs)
32
What are common causes of AKI?
Prerenal: -Hypovolaemia -Sepsis -Low cardiac output Intrinsic renal -ATN -Nephortoxics: drugs, contrast -Abdominal compartment syndrome -Hepatorenal syndrome -Glomerulonephritis Post renal -Bladder outflow obstruction -Bilateral ureteric obstruction
33
What are nephrotoxic drungs?
-ACE inhibitors -NSAIDS -Diuretics -Aminoglycosides
34
Indiciations for renal replacement therapy
Absolute: -Refractory hyperkalaemia (>6mmol/L) -Refractory pulmonary oedema and fluid overload -Uraemic encephalopathy Relative -Acidosis (pH <7.2) -Uraemia -Pericarditis -Toxin removal
35
What is the emergency treatment for hyperkalaemia?
-Continuous cardiac monitoring -Insulin (10-20 units actrapid) in 100ml 20% dextrose intravenously over 30 minutes -Sodium bicarb 50mmol intravenously over 5-10 mins -10% calcium gluconate IV (10-30 ml) -B2 agonist: nebulised salbutamol