Cardiac Diseases Flashcards

(49 cards)

1
Q

This is a progressive condition in which plaque builds up in the tunic intima of arteries.

A

Atherosclerosis

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2
Q

Another name for the deposit of lipids and calcified cells found during atherosclerosis

A

Atheroma

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3
Q

True or False: atherosclerosis can occur in any artery and is categorized by location

A

True

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4
Q

List 3 modifiable risk factors

A

cigarette smoking, dyslipidemia, hypertension, DM, obesity, sedentary lifestyle

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5
Q

True or False: atherosclerosis is more common in males than females

A

True

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6
Q

What are the 5 risk factors for cardiovascular disease?

A

atherosclerosis, dyslipidemia, diabetes, htn, lifestyle factors

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7
Q

Difference between type 1 and type 2 diabtes

A

type 1: early onset, beta cells of pancreas are destroyed and can’t make insulin
type 2: gradual onset, NOT autoimmune, person becomes insulin resistance

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8
Q

3 P’s of Diabetes

A
  1. polyuria - excessive urination
  2. polydipsia - excessive thirst
  3. polyphagia - excessive appetite
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9
Q

Lab findings for Diabetes Patients

A

fasting BG greater than 126
glucose intolerance test greater than 200
elevated HgbA1C

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10
Q

HgbA1C value for it to be considered diabetes?

A

greater than 6.5%

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11
Q

Onset, Peak and Duration of Rapid Acting Insulin (humalog/novolog)

A

Onset: 15-30 min
Peak: 1-2 hours
Duration: 3-6 hours

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12
Q

Onset, Peak and Duration of Short Acting Insulin (regular)

A

Onset: 30-60 min
Peak: 2-4 hours
Duration: 3-6 hours

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13
Q

Onset, Peak and Duration of intermediate insulin (NPH)

A

Onset: 2-4 hours
Peak: 8-10 hours
Duration: 10-18 hours

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14
Q

Onset, Peak and Duration of long acting insulin (LANTUS)

A

Onset: 1-2 hours
Peak: none
Duration: 19-24 hours

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15
Q

LDL versus HDL

A

LDL - low density lipoproteins or “bad cholesterol”
HDL - high density lipoproteins or “good cholesterol”

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16
Q

High levels of HDL inversely correlate with cardiovascular risk - T or F

A

True

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17
Q

Normal LDL

A

Less than 100

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18
Q

Normal HDL

A

greater than 60

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19
Q

normal cholesterol

A

less than 200

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20
Q

3 meds for lowering cholesterol

A

statins, nonstatins, and naicin

21
Q

Statin-associated muscle symptoms

A

weakness, aches and cramps

22
Q

Naicin is prescribed to do what for dyslipidemia?

A

ordered to increase HDL alongside other anti-lipid agents

23
Q

Examples of statins

A

Lipitor or Zocor

24
Q

Example of nonstatins

A

Zetia or Fenofibrate

25
Primary (essential) HTN versus Secondary HTN
primary: unknown cause - coorelates with lifestyle factors secondary: known primary cause - medication, endocrine disorder
26
Hypertensive Urgency Definition
BP > 180-110 without evidence of organ dysfunction
27
Hypertensive Crisis
Systolic BP>180 and/or diastolic BP>120, with impending or progressive organ dysfunction
28
DASH diet
high in fruits, veggies and lean meats, low in sugar and red meat suggested for those with HTN
29
Classes of medications for HTN
ACE inhibitors ARBs Calcium Channel Blockers Thiazide diuretics
30
True or False: HTN is more common in men less than 50 and woman over 50.
True
31
Angina Pectoris
chest pain caused by narrowed coronary arteries and presents with negative trop, ST depression and T-wave changes
32
Stable angina versus unstable
stable: resolves quickly, with rest or medications, can be triggered by exertion, large meals and hot/cold temperatures unstable: occurs any time, lasts longer than 20 minutes, more severe and not easily relieved
32
Stable angina versus unstable
stable: resolves quickly, with rest or medications, can be triggered by exertion, large meals and hot/cold temperatures unstable: occurs any time, lasts longer than 20 minutes, more severe and not easily relieved
33
Prinzmetal Angina/Variant Angina/vasoplastic
episodes of angina and TEMPORARY ST elevations caused by spasms of coronary artery - easily relieved by nitrates
34
STEMI versus NSTEMI
STEMI: elevated ST (>1) = COMPLETE occlusion NSTEMI: ST depression or T-wave inversion
35
Anterior Wall MI ST change location and artery affected
V1-V4; LAD
36
LAD supplies what part of the heart
blood to anterior left atrium and ventricle
37
Inferior Wall MI ST change location and artery affected
II, III, aVF; RCA
38
Right ventricular MI ST change location and area affected
V4-V6; interior wall MI
39
Lateral wall MI ST change location and artery affected
I, aVL, V5, V6; Left Circumflex
40
posterior wall MI ST change location and artery affected
V7-V9 elevation and ST depression in V1-V4 RCA or left circumflex artery
41
Issues after right ventricular MI
tachycardia, hypotension and JVD positive inotropes avoid pre-load reducing meds (beta blockers; diuretics; morphine, nitrates)
42
Issues after inferior wall MI
bradycardia/hypotension increase in AV blocks increase risk for papillary muscle rupture avoid reducing preload
43
Papillary muscle rupture
occurs 2-8 days post MI hemodynamic compromise, pulmonary edema and new LOUD systolic murmur requires EMERGENT repair
44
issues after anterior wall MI
increased risk of left heart failure increased risk of 2nd degree blocks increased ventricular rupture
45
Diagnostic tests to look for MI
elevated troponin (>0.1 ng/ml) elevated CK-MB (>2.5%)
46
STEMI Process _______ min door to balloon _______ min door to fibrinolytic therapy
90 min 30 min
47
NSTEMI meds
nitro - vasodilation beta blocker/calcium channel blockers - reduce myocardial oxygen demand heparin: improve blood flow morphine: pain not relieved with nitro
48
If patient cannot take aspirin for managing MI, what is the other option?
Clopidogrel