Cardiac Rhythm Analysis Flashcards by Megan Pirtle

automaticity

ability to initiate impulse spontaneously and continuously

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excitability

ability to be electrically stomulated

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conductivity

ability to transmit impulses along a membrane in an orderly manner

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contractility

the mechanical response to an impulse

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SA node

where the beat starts (P wave)

pacemaker of the heart

60-100 bpm normally

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AV node

fires 40-60 bpm normally

escape beat

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parasympathetic nervous system effect on SA node

decreases rate

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sympathetic nervous system effect on SA node

increases rate

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dysrhythmias

disorder of impulse formation, conduction of impulses or both

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P wave

depolarization of the atria

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QRS complex

depolarization of the ventricles

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T wave

repolarization of the whole heart

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artifact

electrodes are not secre, muscle interference or electrical interference

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How fast is normal sinus

60-100 BPM

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where does normal sinus start

SA node and follows normal conduction pathways

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when can sinus brady be normal

aerobically trained athletes and during sleep

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what nervous system contributes to sinus brady

parasympathetic

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S/s sinus brady X7

hypotension
pale, cool skin
weakness
angina
dizziness or syncope
confusion or disorientation
SOB

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sinus brady tx X3

atropine
pacemaker
stopping offending drugs

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atropine class

anticholinergic

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atropine MOA

raises HR

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how much atropine can you give without an order

max of 3 mg titrating up from 0.5

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what class of drugs can cause sinus brady

beta blockers

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how fast is sinus tachy

101-200 bpm

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what causes sinus tachy X3

vagal inhibition sympathetic stimulation drugs

sinus tachy s/s X6

``` dizziness dyspnea hypotension angina r/t CAD diaphoresis SOB ```

sinus tachy treatment X3

treat by cause vagal maneuver beta blockers

SVT HR

150-220 bpm

what is SVT assosicated with X7

``` overexertion stress deep inspiration stimulants disease dig toxicity ```

what does SVT over 180 lead to

decreased cardiac output and stroke volume

SVT s/s 4

hypotension dyspnea angina chest pain WILL BE SYMPTOMATIC

SVT Tx X6

``` vagal stimulation Adenosine beta blockers ca channel blockers amiodarone cardioversion ```

IV adenosine half life

10 seconds

adenosine admin rate

1-2 second fast push followed immediately by fast 20 mL flush

adenosine dosage

6 mg first dose 12 mg second dose

a fib treatment X6

``` amiodarone ibutilide cardioversion anticoag ablation maxe procedure ```

what does a flutter increase the risk of

stroke

a flutter tx X3

drugs cardioversion ablation

what are PVC's associated with X4

stimulants electrolyte imbalances hypoxia heart disease

are PVC's harmful

not in normal heart

PVC tx X2

correct cause | antidysrhythmics

V tach rate

150-200 BPM

why is v tach bad

life threatening d/t decreased CO and possibility of moving to v fib

torsades de points

v tach with different sized waves

what is v tach associated with X4

heart disease electrolyte imbalances drugs CNS disorder

V tach symptoms X4

hypotension pulmonary edema decreased cerebral blood flow cardiopulmonary arrest

stable v tach tx X2

antidysrhythmics | cardioversion

unstable v tach tx X2

CPR | rapid defibrillation

what is v fib associated with X5

``` MI ischemia disease procedures electrolyte imbalances ```

v fib s/sx X3

unresponsive pulseless apneic

v fib tx X3

CPR defibrillation ACLS drug protocol

asystole s/sx X3

unresponsive pulseless apneic

what is asystole the result of X3

cardiac disease severe conduction disturbance end-stage HF

asystole tx X3

CPR ACLS drug protocol Intubation

pulseless electrical activity

electrical activity is observed on the EKG

PEA causes (H's and T's

``` Hypovolemia Hypoxia Hydrogen ion (acidotic) Hyper/hypo kalemia Hypoglycemia Hypothermia ``` ``` Toxins Tamponade Thrombosis Tension pneumothorax Trauma ```

PEA tx X4

CPR intubation ACLS fix underlying cause

1st degree Heart block s/sx

not symptomatic

1st degree heart block tx

no tx

third degree heart block tx X2

pacemaker | drugs to increase HR

when is ST elevation significant

if 1 mm over the isoelectric line

initial biphasic defibrillation

120-200 Joules

initial monophasic defibrillation

360 joules

when does CPR begin with defibrillation

immediately after

where shoul dyou never put defibrillator pads

over a pacemaker or implantable cardioverter/defibrillator

what is cardioversion ideal for X2

stable v tach | SVT

what is cardioversion

shock is given on the R wave

initial biphasic cardioversion

70-75 joules

initial monophasic cardioversion

100 joules

what do you do if patient goes pulseless during cardioversoin

turn sync button off and defibrillate

Cardiac Rhythm Analysis Flashcards

(70 cards)