Cardio Flashcards
(40 cards)
Thoracic duct ascending enters where and empties into ?
enters the aortic hiatus and empties into the left subclavian vein.
IVC is from from which veins?
Right and left common iliac veins at the level of L4 and L5. The IVC returns venous blood to the heart from the
lower extremities, portal system, and abdominal and pelvic viscera.
At the level of L1, the renal veins and arteries originate where
aorta
the IVC is anterior to right renal artery and right to the aorta
SVC is formed from
Left and right brachiocephalic veins
Right-sided endocarditis involving the tricuspid valve commonly occurs in IV drug users and is most often due
S.aureus
infective endocarditis can cause tricuspid regurgitation, which is
identified as
an early systolic murmur best heard over the left lower sternal border that is accentuated by
inspiration.
The right and left main coronary arteries arise directly from the root of the aorta and provide the blood supply
to the heart. The left main coronary artery divides
Left anterior descending (LAD) and circumflex
coronary arteries, which supply most of the anterior and left lateral surfaces of the heart.
In 85-90% of
individuals, the right coronary artery gives rise to
the posterior descending artery and right marginal branch of right coronary artery.
These patients are said to
have right dominant coronary circulation. In approximately 10% of patients, the posterior descending artery
arises from the circumflex branch of the left main coronary artery; these patients have left dominant
circulation.
The posterior descending artery of right supplies
most of the inferior wall of the left ventricle, which forms
the diaphragmatic surface of the heart. The right coronary artery also gives rise to the SA and AV nodal
arteries in most patients.
The LAD of left coronary artery normally supplies
anterior 2/3 of the interventricular septum (septal
branches}, the anterior wall of the left ventricle (diagonal branches}, and part of the anterior papillary muscle.
The most common cause of coronary sinus dilatation
is elevated right-sided heart pressure secondary to pulmonary artery
hypertension.
The coronary sinus serves as the endpoint of venous drainage from the coronary blood supply_ Since it
contains deoxygenated blood it drains into
the right atrium
Since the coronary sinus communicates freely with
the right atrium, it will become dilated by
any factor that causes dilatation of the right atrium.
Aortic dissection often occurs in patients with hypertension. Such patients might have
elevated
left heart pressures
This patient presents with progressive dyspnea and orthopnea (cough when lying down). His chest x-ray
shows cardiomegaly (heart> 1 hemithorax in size), increased vascular shadowing (alveolar edema) in a
“batwing” peri-hilum pattern, and blunting of the costophrenic angles (pleural effusions). These findings are
characteristic of
Left acute ventricular heart failure
acute left ventricular failure,
result from increased left atrial and ventricular filling pressure
(increased preload).
This increased pressure is transmitted to the pulmonary capillaries, causing fluid
transudation into the pulmonary interstitial and alveolar spaces (cardiogenic pulmonary edema).
Common triggers for acute heart failure include
myocardial infarction, severe hypertension, arrhythmias (eg,
atrial fibrillation), and drug use (eg, cocaine). Patients with acute heart failure complain of cough, dyspnea,
and fatigue, which can rapidly become more severe
Chest examination from acute heart failure usually reveals
crackles and sometimes wheezing
(cardiac asthma) due to edema of the bronchial airways
The chest x-ray can also show from acute heart failure show
Kerley B lines,
short horizontal lines situated perpendicularly to the pleural surface that represent edema of the interlobular
septa.
Acute respiratory distress syndrome (ARDS) results from
endothelial injury and leakage of
fluid from capillaries.
ARDS has what onset? and looks like what on chest X ray?
has an acute onset and is characterized by bilateral patchy airspace disease on chest
x-ray. It is caused by an inciting factor such as sepsis, aspiration, pneumonia, or trauma.
ARDS can be
distinguished from cardiogenic pulmonary edema by t
he absence of jugular venous distension and
cardiomegaly.
chest x-ray in idiopathic interstitial fibrosis (referred to histologically as usual interstitial
pneumonia) shows?
reticular, net-like, opacities involving the lung bases. Most patients present with slowly
progressive dyspnea and a nonproductive cough.
Obstructive pulmonary diseases such as asthma and chronic obstructive pulmonary disease
are associated with
hyperinflated lungs and a flattened diaphragm.