Cardio Flashcards

(54 cards)

1
Q

Infective Endocarditis: Patho

A

Vegetations growing on valves break off and embolism to various organs -> life threatening complications

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2
Q

Infective Endocarditis: Life threatening complications

A

Stroke, Spinal cord ischemia, Ischemia to extremities, Intestinal infarction, Splenic infarction

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3
Q

Ischemia to extremities meaning:

A

Pain, pallor, and cold foot or arm

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4
Q

Which is reported immediately?

A

Pain and Pallor in one foot

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5
Q

Chronic venous insufficiency: Patho

A

Valves in veins of lower extremities consistently fail to keep venous blood moving

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6
Q

What does CVI cause?

A

Chronic increased venous pressure; pushing fluid out of vascular space to surrounding tissues, tissue enzymes then break RBC down

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7
Q

Clinical finding for Chronic venous insufficiency

A

Brownish, hardened skin on lower extremities

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8
Q

Chronic venous insufficiency clinical manifestations

A

Brownish skin discoloration: chronic edema and inflammation; Tissue then harden appearing leathery, this skin prone to breakdown and ulcerations inside of the ankle

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9
Q

Emphasis of Discharge teaching for DVT

A

Minimize risk factors and promote blood flow and venous return and prevent reoccurrence

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10
Q

Teaching points for DVT education

A

Drink plenty of fluids: avoid caffeine and alcohol
Elevate legs when sitting and dorsiflex
Begin exercise program
Change position frequently
Stop Smoking
Avoid wearing restrictive clothes

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11
Q

Irregular waveforms of varying shapes/amplitudes

A

VFIB

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12
Q

EO: Potentially lethal dysrhythmia, irregular waveforms of varying shapes/amplitudes

A

VF

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13
Q

Dysrhythmia: Ventricle quivering with no effective contraction or cardiac output

A

VF

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14
Q

What does VF result in?

A

Unresponsive pulse less, apneic state

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15
Q

Treatment for VF

A

Rapid:
CPR
Defib
Drug Therapy

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16
Q

What drug therapies are used for VF?

A

Epinephrine
Vasopressin
Amiodarone

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17
Q

Discharge instructions for CABG

A

Hygiene
Medications
Activity Level
Sexual activity
Symptoms to report

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18
Q

CABG: Hygiene teaching

A

Shower instead of bath
No soaking or applying lotions to incisions

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19
Q

CABG: Activity level

A

No lifting objects >5lb
No driving for 4-6 weeks

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20
Q

CABG: Sexual activity

A

Resume able to walk 1 block or climb 2 flights of stairs w/o symptoms

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21
Q

CABG: Symptoms to report

A

Chest pain or SOB during rest
Signs of infection

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22
Q

Modify cardiac risk factors for CABG

A

No smoking, lose weight, maintain healthy diet, and increasing activity levels through exercises

23
Q

Heart failure: Full Patho

A

Cardiac output reduced: unable to pump blood right
Leads to: Reduce perfusion to vital organs
Reduced perfusion leads to: Decreased renal blood flow
Decreased renal leads to: Renin-angiotensin system to compensate by increasing blood volume via increased water resorption
Final result of compensation: Fluid volume excess and dilutional hyponatremia

24
Q

Heart Failure: Dilutional hyponatremia

A

Treated with:
Fluid restrict
Loop diuretics
ACE inhibitors

25
Heart Failure: Furosemide
Resolve hyponatremia by: Promote free water excretion Allow hemoconcentration and increased sodium
26
Pt with heart failure 8-hour urine output of 200 mL. What action should be first?
Auscultation of pts breath sounds
27
What does Urine output of <30 mL/hr indicate?
Low vascular volume: Dehydration or blood loss Decreased Renal Perfusion: Low cardiac output Intrinsic kidney injury Urinary obstruction: Prostate or foley
28
Why would auscultation be necessary for client’s with heart failure, low urine output?
Decreased cardiac function and buildup of fluid: Leads to need to assess lung sounds for crackles
29
S/S Failing pacemaker
Failure to capture with: Bradycardia Hypotension
30
Failure to capture in pacemaker
Pacer spikes without associated QRS
31
PRIORITY for Failed Pacemaker
Initiate transcutaneous pacing
32
Acute blood loss: Interventions
Improve perfusion to brain and vital organs
33
Acute blood loss: Priority
Lowering the head of the bed prior to other interventions
34
PAD: Description
Chronic atherosclerotic disease Caused by buildup of plaque in arteries
35
What does PAD commonly affect?
Lower extremities: Leading to tissue necrosis
36
PAD: Management
Lower extremities below heart when sitting/lying (improves arterial blood flow) Engage moderate exercise Daily skin care Stop smoking Avoid tight clothes Take meds as prescribed
37
Focuses of management
Improving blood flow and circulation to extremities via lifestyle changes and medications
38
Halter Monitor: Pt education
Diary of activities and symptoms Do not bathe or shower Engage in normal activities
39
Clinical manifestations of DVT:
Calf pain Lower leg warmth, and redness Unilateral leg edema
40
DVT occurs:
Commonly from decreased activity or mobility
41
DVT S/S
Unilateral edema Calf Pain or TTP Warmth Erythema Low-grade fever
42
Clients with … are at risk for life-threatening dysrhythmias (Heart block, V-tach, V-fib)
Myocardial Infarction
43
Myocardial Ischemia damages what? Causing what?
Cardiac muscle cells causing electrical irritability (PVC)
44
What increase the risk for dysrhythmias?
Electrolyte imbalances/ Hypokalemia
45
How does hypokalemia relate to Dysrhythmias?
Hyperpolarizes cardiac conduction pathways increasing risk for dysrhythmias
46
Medications administered for pts w/ NSR w/ PVC and lab values of Potassium 3.3 mEq/L
Potassium chloride IVPB once
47
A-fib discharge pt stable w/ meds (Digoxin). What education is needed before discharging?
Visit doctor to check drug levels Report to HCP if nausea and vomiting Tell HCP if HR below 60 BPM NO need for increasing potassium intake
48
Troponin:
High specific cardiac marker for detection of MI
49
Difference from Troponin and CK MB
Troponin:Greater sensitivity and specificity for myocardial injury
50
Troponin serum levels increase…
4-6 hours after onset of MI
51
Troponin value indicative of cardiac muscle damage
0.7 ng/mL Immediate focus
52
Highest priority for intervention with patients having chest pain
Elevated Troponin value: indicative of myocardial injury
53
Pt education for Mitral valve prolapse
Stay hydrated Avoid caffeine and EOTH Exercise regularly Reduce stress Take beta blockers (for palp and chest Px)
54
S/S of Mitral Valve Prolapse
Experiencing occasional palpitations, lightheadedness, and dizziness