Cardio Meds Flashcards

(19 cards)

1
Q

What are calcium channel blockers suffixes?

A

-pine
-zem
-pril

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2
Q

ACE inhibitor suffixes?

A

-pril

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3
Q

Angiotensin receptor blocker suffixes?

A

-sartan

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4
Q

Beta blocker suffixes?

A

-olol

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5
Q

Antiarrythmic class 1a drugs: what do they do and names?

A

Sodium channels - moderate phase 0 and conduction slowing

Quinidine
Procainamide
Disopyramide

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6
Q

Class 1b antiarrythmics; what are they and what do they do?

A

Sodium channel blocker - little effect on phase 0 and no effect on APD

Lidocaine, mexilentine, totcaide

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7
Q

Class 1c antiarrythmics; what are they and what do they do?

A

Sodium channel blocker - large effect on phase 0 depression and conduction slowing, little effect on depolarisation

Flecainide, propafenone, moricizine

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8
Q

Class II antiarrythmics; what are they and what do they do?

A

Beta blockers - reduce heart rate

Examples include - atenolol, bisoprolol, metoprolol, propranolol,

Propranolol - non selective

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9
Q

Class III antiarrythmics; what are they and what do they do?

A

Potassium channel blockers - increase electrical refractory period

E.g. amiodarone, sotalol, ibutalide

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10
Q

Class IV antiarrythmics; what are they and what do they do?

A

Calcium channel blockers - veraprimil, diltiazem

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11
Q

Other antiarrythmic drugs?

A

Adenosine - adenosine receptor agonists

Digoxin - sodium, postassium ATPase inhibitors

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12
Q

What drugs can you not give in pregnancy?

A

ACE inhibitiors

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13
Q

Digotoxin used for?

A

Heart failure / NO EFFECT ON BP

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14
Q

Vasodilators do what?

A

Relaxes smooth muscle

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15
Q

Loop diuretics?

A

Remove excess fluid and salt - heart failure

Furosemide

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16
Q

Thazilde diuretics?

A

High BP

hydrochlorothiazide and indapamide.

17
Q

Ticagrelor?

A

Antiplatelet med
Aspirin another example of this

18
Q

Warfarin?

A

Anticoagulant

Other examples; herparin, DOACs (-ban)

19
Q

Action potentials of cardiac muscles?

A

Sure! Here’s a brief summary:

Cardiac action potentials vary by cell type:
• Pacemaker cells (e.g., SA node): Have a spontaneous, slow depolarization due to Na⁺ and Ca²⁺ influx. They control heart rhythm and lack a true resting potential.
• Contractile cells (e.g., ventricular muscle): Have a fast depolarization (Na⁺ influx), a plateau phase (Ca²⁺ influx balances K⁺ efflux), and repolarization (K⁺ efflux). This allows strong, coordinated contractions and prevents arrhythmias