Cardio Mod 6 Flashcards

1
Q

which side of the heart is valve dysfunction more common

A

left side

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2
Q

2 types of valve dysfunction

A
  1. stenotic

2. insufficiency

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3
Q

3 etiologies of aortic valve stenosis

A
  1. congenital - abnormal bicuspid formation
  2. degenerative changes - more common in older population - deposits on valves that make it hard to open
  3. inflammatory - secondary to rheumatic heart dz
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4
Q

what two inflammatory conditions can cause aortic stenosis?

A
  1. rheumatic fever - post-infectious systemic inflammatory disease/reponse due to step - effects joints, skin, CNS and heart
  2. rheumatic heart dz - the inflammation associated with rheumatic fever can scar/deform the valves
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5
Q

primary effects of aortic valve stenosis

A
  1. reduced outflow of left ventricle
  2. increased end systolic LV volume/pressure (incomplete emptying of LV)
  3. output changes: decr SV/decr CO - (heart tries to compensate)
  4. decr systolic BP, potential decr pulse pressure (decr amplitude of pulse = pulse parvus)
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6
Q

secondary effects of aortic valve stenosis

A
  • hypertrophy of left ventricle as compensation to increased workload
  • –may cause coronary circulation insufficiency
  • –coronary capillary density insufficient for myocardial hypertrophy
  • –may lead to ischemia and potential dysrhythmias
  • cardiac pressure/congestion changes - incr left atrial pressure, incr pulm pressures, pulm HTN = pulm edema
  • all of this may progress to MI/heart failure
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7
Q

classic symptoms of aortic valve stenosis

A

syncope with exertion
angina
DOE as it progresses to heart failure

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8
Q

where is the heart murmur heard in aortic valve stenosis - systolic or diastolic?

A

systolic

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9
Q

where do you auscultate the aortic valve

A

right parasternal 2nd ICS

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10
Q

etiologies of mitral valve stenosis

-what population does it affect more?

A
  1. rheumatic heart dz most common cause
  2. degenerative not as common

females>males

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11
Q

primary effects of mitral valve stenosis

A
  1. reduced outflow of left atria
  2. increased LA volume/pressures (incomplete emptying of left atria)
  3. output changes: decr SV/decr CO - heart compensates to try and maintain normal SV and CO; exertional insufficiency of decre SV/CO
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12
Q

secondary effects of mitral valve stenosis

A
  1. left atria dilation due to incomplete emptying of left atria/hypertrophy as compensation to increased workload
  2. cardiac pressure/congestion changes - incr left atrial pressure, incr pulm pressure, pum HTN, leads to edema can eventually lead to RVF
  3. increased A wave due to pulmonary congestion
  4. atrial wall changes may lead to ischemia and potential atrial dysrhythmias - blood flow stasis of Aflutter/fib will add risk of thrombi formation
  5. all can lead to MI/heart failure
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13
Q

s/s of mitral valve stenosis

A
  1. progressive dyspnea due to pulmonary HTN/congestion
    (also orthopnea, PND)
  2. non angina chest discomfort
  3. right sided heart failure due to pulmonary HTN/congestion
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14
Q

where is the heart murmur heard in mitral valve stenosis - systolic or diastolic

A

diastolic

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15
Q

aortic valve insufficiency etiologies

A
  1. congenital
  2. secondary to disease pathology (rheumatic heart dz, bacterial endocarditis, CAD)
  3. CT disorders and other systemic dz
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16
Q

primary effects of aortic valve insufficiency

A
  1. during ventricular relaxation blood from aorta back flows into left ventricle
  2. increased end diastolic filling volumes of left ventricles
  3. output changes: incr SV/CO to accomodate for back flow loss; incr systolic BP, decr diastolic BP, incr pulse pressure (wide pulse pressure)
17
Q

secondary effects of aortic valve insufficiency

A
  1. dilation and hypertrophy of left ventricle to compensate for SV/CO volumes and workload
  2. cardiac pressure/congestion changes - potential for incr pulm pressure and edema
  3. potential for dysrhythmias and associated complications due to wall changes
  4. heart failure occurs as compensation mechanisms can’t maintain CO
18
Q

s/s of aortic valve insufficiency

A
  1. bounding peripheral pulses from increased pulse pressure changes
  2. DOE, orthopnea, PND, angina chest pain
  3. progressive symptoms that may develop over decades
19
Q

when is the heart murmur heart in aortic valve insufficiency - systolic or diastolic

A

diastolic

20
Q

mitral valve insufficiency etiologies

A
  1. rheumatic heart dz, endocarditis
  2. mitral valve prolapse progressing to regurgitation
  3. other: CAD, congestive cardiomyopathy, systemic CT disorders
21
Q

effects on heart with mitral valve insufficiency

A
  1. increased left atrial volumes/pressures: back flow of blood into left atria during ventricular contraction which leads to left atrial dilation/hypertrophy; viscous cycle as atrial dilation may enlarge mitral valve opening more
  2. increased ventricular fililng columes/pressures due to increased filling volumes from atria - hypertrophy and dilation of left ventricle follows
  3. increased atrial pressures also may lead to congestive back up into pulmonary circulation - incr pulm pressure, pulm HTN/edema
  4. eventually compensation fails to maintain CO and heart failure occurs
22
Q

s/s of mitral valve insufficiency

A

DOE - progresses to heart failure

23
Q

where is the heart murmur hear in mitral valve insufficiency

A

systolic

24
Q

where do you auscultate the mitral valve?

A

left 5th ICS space along the midclavicular line

25
Q

what is the most common valve disorder in the US

A

mitral valve prolapse syndrome

26
Q

what is mitral valve prolapse syndrome

A

enlarged cusps of mitral valve prolaps back into left atrium during systole

27
Q

pathogenesis of mitral valve prolapse syndrome

A
  1. abnormal connective tissue accumulation/degeneration in the valves cause enlargements
  2. the cusps will billow back into the atria
  3. chordae tendineae become stretched/elongated as cusps billow back
  4. eventually cusps may not be able to fully close and mitral valve regurgitation develops
28
Q

s/s of mitral valve prolapse syndrome

A

may or may not be symptomatic

29
Q

what is the heart sound heard in mitral valve prolapse syndrome

A

mid systolic click or systolic murmur

30
Q

prognosis of mitral valve prolapse syndrome

A

good without complications

31
Q

severe consequences of mitral valve prolapse syndrome

A
  1. rupture of chordae tendinae, emboli formation, ventricular failure
  2. infective endocarditis, stroke, death
32
Q

etiologies of tricuspid valve insufficiency

A
  1. congenital

2. secondary results of pulm HTN pathologies

33
Q

mechanical effects on cardiac pump with tricuspid valve insufficiency

A
  1. pulm HTN/congenital regurgitation causes back flow into right atria
    - increased right atrial pressures creates increased venous pressures
    - distended jugular veins and lower extremity/generalized edema
    - increased atrial volumes/pressures progress to increased right ventricular volumes/pressures
    - right ventricular hypertorphy/dilation compensate to maintain cardiac output
  2. compensation eventually fails and right sided heart failure occurs
34
Q

where is the heart murmur heart in tricuspid valve insufficiency - systolic or diastolic

A

systolic

35
Q

where is the tricuspid valve heard

A

left 5th intercostal space along parasternal line