Cardio-respiratory Flashcards

(491 cards)

1
Q

What is included in physical examination in cardio-respiratory?

A

Inspection
Palpation
Percussion
Auscultation

(IPPA)

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2
Q

What is included in inspection?

A

Vital signs
Thoracic shape
Mechanism of ventilation
Head, neck, extremities
Speech, cough, sputum
Position
Alertness

(VTMHMHSPA)

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3
Q

What are the vital signs that we check?

A

HR, BP, SpO2, RR

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4
Q

How to take HR?

A

Palpate pts radial pulse using index and middle finger.
Pts should remain quiet during procedure.
Count # of beats in 10 sec x 6 or 15 x 4 or 30 x 2, ….
Take note of the rate, rhythm and strength of pulse.

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5
Q

What is the Rate?

A

Bradycardia: lower than 60 bpm
Normal: 60-100 bpm
Tachycardia: higher than 100

Brady = Low
Tachy = High

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6
Q

Normal HR for neonates (1 to 28 days)

A

120-160 bpm

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7
Q

Normal HR for infants (1 to 12 months)

A

100-120 bpm

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8
Q

Normal HR for children (1 to 8 y.o)

A

80-100 bpm

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9
Q

Normal HR for adults

A

60-100 bpm

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10
Q

What is a rhythm?

A

Can be normal, irregular or irregularly irregular

If irregular/irregularly irregular = Referral

Normal - Regular Consistent pattern
Irregular - Irregular but Consistent pattern
Irregularly Irregular - Irregular but Inconsistent pattern

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11
Q

How to take RR?

A

Inspect covertly - don’t tell pt what you’re doing.

Take the number of breath in 10 sec x 6 or 15 sec x 4 ….

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12
Q

Different RR

A

Bradypnea - lower than 12 breaths/min
Eupnea - 12-20 breaths/min
Tachypnea - higher than 20 breaths/min

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13
Q

Normal RR for Neonates (1-28 days)

A

40-60

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14
Q

Normal RR for infants (1-12 months)

A

25-50

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15
Q

Normal RR for children (1-8 y.o)

A

15-30

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16
Q

Normal RR for adults

A

12-20

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17
Q

How to take BP?

A
  • Pt is sitting with L arm exposed + supported (level of the heart).
  • Palpate brachial artery pulse (medial to bicep tendon) and place cuff 1 inch above.
  • Place stethoscope onto pulse.
  • Instruct pt not to talk.
    Inflate the cuff until brachial pulse disappears then add 20 mmHg.
  • Deflate at rate of 2 mmHg/beat.

** Ensure the cuff encircle approx. 80% of the arm.

Too large = underestimate BP
Too small = overestimate BP

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18
Q

How to tell systolic from diastolic?

A

Systolic = 1st sound heard
Diastolic = when sound disappears

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19
Q

Values of BP - Hypotension

A

90/60 mmHg or less

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20
Q

Values of BP - Normal

A

120/80 mmHg

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21
Q

Values of BP - Hypertension

A

140/90 mmHg or more

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22
Q

What is orthostatic hypotension?

A

When the systolic measure drops by 20 mmHg when going from lying to upright.

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23
Q

What is SpO2?

A

Measurement of what % of hemoglobin are saturated with O2.

Hemoglobin - protein in our red blood cell that carries O2 from our lung to tissues.

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24
Q

Where do we take SpO2?

A

1) Finger probe
2) Ear probe
3) Toes

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25
What is a normal SpO2 value?
95-100% ** Below 90% we want to have further investigation + supplement of O2
26
What are we looking at when we are looking at mechanism of ventilation?
- Breathing pattern - Ratio of inspiration:expiration - Depth
27
Mechanism of ventilation - Breathing pattern
Diaphragmatic - 70% of time Lateral costal - 30% of time Apical - 0% of time
28
Mechanism of ventilation - Ratio of inspiration:expiration
Obstructive - 1:3 or more Normal - 1:2 Restrictive - 1:1
29
Mechanism of ventilation - Depth
Shallow or normal
30
What are some abnormal thoracic shapes?
- Funnel chest - Pigeon chest - Kyphoscoliosis - Barrel chest
31
What is a funnel chest?
Sternum goes inward - Congenital
32
What is a pigeon chest?
Sternum pokes forward - Congenital
33
What is a kyphoscoliosis?
Combination of kyphosis and scoliosis - Congenital or functional
34
What is a barrel chest?
Result of chronic hyperinflation
35
What can happen with an abnormal thoracic shape?
It can affect mechanics of ventilation and ventilation/perfusion matching.
36
What do we check for the head?
- Color (pallor) - Cyanosis (blue lips) - Nasal flaring
37
What do we check for the neck?
- Acc muscle use (often with apical breather or over use of SCM muscle) - Jugular venous distension (R side HF indicator)
38
What do we check for the extremities?
- Capillary refill (2 to 4 sec) - Clubbing (nails together check for hole) - Color - Edema - Muscle wasting
39
What are we looking with cough?
- Effective (strength) - Productive (if sputum come out) - Persistent (freq) - Wet (productive) or dry (unproductive)
40
What are we looking with Sputum?
- Quantity - Color - Consistency - Thick = fibrosis - Odor - Foul smell = bronchiectasis or lung abscess
41
What are we looking with speech?
- Any trouble speaking or breathing
42
What does a pink - frothy colored sputum means?
Pulmonary edema
43
What are we looking during palpation?
1) Chest wall expansion 2) Diaphragmatic excursion 3) Edema 4) Pain and crepitus 5) Tracheal positioning 6) Tractile fremitus
44
what are the chest wall expansion methods?
1) Manual method 2) Circumferential method
45
Chest wall expansion - Manual method
More of a Dx tool. Looking if mvt is symmetric Place thumb together and ask pt to take deep breath. Ax 3 regions; - Upper lobe (sterno-costal) - Middle lobe (vertebro-costal) - Nipple line - Lower lobe (lateral costal) - Around T10
46
Chest wall expansion - Circumferential method
Measurement. Get baseline. Track progress. Common locations of measurement: - Axilla - 10th rib Take 3 measurement and record the best of 3.
47
What are the diaphragmatic excursion methods?
- Manual method - Circumferential method - Diaphragmatic percussion
48
Diaphragmatic excursion - Manual method
Hand placed on apex of belly during inspiration.
49
Diaphragmatic excursion - Circumferential method
Place tape at level of apex of belly and instruct pt to first exhale and then max inhale. Measure/see the difference.
50
What are we looking with edema?
- Pitting vs Non-pitting - Level (how far it as extend)
51
What medical condition can cause edema?
- R side HF - Pregnancy - Lymphedema - Systemic diseases
52
What is pitting?
When we palpate, there's an indentation, but when we remove the pressure the indentation persist = Fluids retention
53
What is non-pitting?
Indentation doesn't occur (or very rare)
54
What does it says if when palpating there's inc pain?
The problem is more likely to be musculoskeletal than angina.
55
What is a crepitus?
Is a crunchy sound.
56
What is the normal position of the tracheal?
Between the sterno-costal joints. Take 2 fingers and run it down between the trachea to see if there's any deviation. If there's an Inc volume = pushes away If there's a Dec volume = ipsilat.
57
What is tactile fremitus?
With palm of hand or ulnar side we palpate and feel the vibration of the number 99. If there's an Inc of sound = more dense tissues If there's an Dec of sound = hyperinflation or inc distance between lungs/chest wall
58
Why use percussion?
1) Dx percussion 2) Diaphragmatic excursion
59
Why use dx percussion?
- Determine the density of the underlying tissue. - Identify the normal or abnormal ventilation. - Identify the changes in lung density. ** Abnormality can only be detected 5 cm in depth. Dx percussion differs from Tx percussion
60
How to do dx percussion?
Strike the DIP Fast snap of the wrist Perform 2-3 strikes Better to perform on exposed skin
61
What are the sounds we can possibly hear with dx percussion?
- Resonant - Dull - Hyper-resonant
62
Resonant...
Normal aerated lung tissue (air:tissue ratio normal)
63
Dull...
Non-aerated lung tissue - more dense/consolidation (air:tissue ratio below normal)
64
Hyper-resonant...
Hyperinflated lung - more air (Hallow sound) (air:tissue ratio above normal)
65
How to measure diaphragmatic excursion?
- Pt sitting, access to the back - Make sure pt know how to properly use diaphragm - Pt max exhale and hold as the therapist percuss - Therapist marks when the dull sound is heard (diaphragm) - Then pt max inhale and hold as the therapist percuss down from the point of marking - Therapist makes a mark at the dull sound. Normal = 3-5 cm difference Diaphragm sits higher on R than L because the liver is on R.
66
What are we hearing when ausculting?
1) Breath sounds 2) Voice sounds 3) Heart sounds
67
How to properly auscultate?
- Quiet environment - Stethoscope directly on skin Pt should take slow, deep breaths in and out through the mouth - Ear tips facing forwards inside ears - Bell for low frequency sounds (BP) - Diaphragm for high frequency sounds (bigger surface, pulmonary ax) - Sanitize stethoscope - Auscultation should be performed over entire lung space - At least one entire breath per region *** See manual for Pattern
68
Definition of ausculting
The art of listening to sounds produced by the body.
69
What is special about the R lung?
3 parts (Upper lobe, middle lobe and lower lobe) Compared to 2 parts on L (Upper lobe and lower lobe)
70
What separate lung lobes?
Fissures
71
What do we hear with normal breath sounds?
- Vesicular - Bronchovesicular - Bronchial
72
What do we hear with abnormal breath sounds?
- Crackles (rales) - Wheezes (rhonchi) - Pleural friction rub - Stridor
73
What is a vesicular breath sound?
- Indicates normal lung - Soft and low-pitched - I:E = 3:1 - Heard over peripheral lung tissue Normal breath sound
74
What is a bronchovesicular breath sound?
- Mixture of bronchial and vestibular - I:E = 1:1 - I is soft and low-pitched - E is loud and high-pitched - Heard over main stem bronchi in 1st and 2nd intercostal spaces and posteriorly between the scapulae Normal breath sound
75
What is a bronchial breath sound?
- Loud, high-pitched, hollow quality - Louder on E - I:E = 1:1 or 1:2 - Distinct pause between I and E - Heard over the trachea and manubrium Normal breath sound
76
What is a crackle breath sound?
- Short and explosive - Abnormal breath sound
77
What are the classification for crackle breath sound?
Timing: Inspiratory vs expiratory Quality: Coarse vs fine
78
Course crackle...
- Air moving retained secretion causing intermittent opening and closing of airway. - Wet (from fluids) - Inspiration and/or expiration - Any area of the lung
79
Fine crackle...
- Sudden opening of collapsed alveoli - Dry - Inspiration - Basal lung
80
What is a wheeze breath sound?
- Musical - Can be affected by coughing - Abnormal breath sound
81
82
What is a pleural friction rub breath sound?
- Long, low-pitched, leathery creaking sound - Produced by frictional resistance between layers - Pain may be associated to pleural friction rub - May be confused with pericardial rub from heart - To differentiate ask pt to hold their breath, if rub persist then it is a pericardial rub, if rub sound disappears, it is a pleural friction rub. - Abnormal breath sound - Extrapulmonary sound
83
What is a stridor breath sound?
- Loud, musical, high-constant pitch - Audible from a distance without stethoscope - Most prominent during inspiration - Due to turbulent air flow (upper airway obstruction or narrow airway) - Abnormal breath sound
84
What is a voice sounds - heard with auscultation?
Are produced as a result of loss of selective filtering of high frequency sounds. 3 technique to Ax. Only one is necessary as all 3 test for the same thing.
85
What are the technique to Ax voice sounds?
- Egophony - Whispered pectoriloquy - Bronchophony
86
How to egophony?
Pt repeat the letter E as we auscultate. If we hear A = consolidation (can be water, blood, pus...).
87
How to whispered pectoriloquy?
Whisper word change from muffled (normal tissue) to clearer over consolidation.
88
Bronchophony
Instead of whispering we ask pt to talk normally. Inc intensity and clarity = consolidation
89
Why is pulmonary function tests used for? De quoi prend t'il en compte?
- To evaluate the mechanical function of the lungs - Prend en contre l'age, sex, race, grandeur et poids - Value are compared to predicted values to see if pt falls within normal or as an obstructive/restrictive disease
90
What are the different categories of PFT's?
- Volume - Capacity - Flow - Diffusion studies - Respiratory muscle strength
91
What is the clinical utility of PFT's?
- Determine if pt present with respiratory condition (obstructive/restrictive/combination) - Determine severity - Determine response to bronchodilator tx - Outcome measure to determine progression of disease, effectiveness of tx and medications
92
How do we perform an arterial blood gas test?
Sample of blood (needle or catheter)
93
Why do we use an arterial blood gas test?
Measure the amount of arterial gases and acidity (pH) of arterial blood.
94
Normal values of pH
7.35-7.45
95
Normal values of PaCO2
35-45 mmHg
96
Normal values of HCO3
22-26 mEq/L
97
Normal values of SPO2
95-100%
98
Normal values of PaO2
80-100 mmHg
99
pH is...
Acidity Below 7.35 is acidic Above 7.45 is base/alkalosis A measure of hydrogen ion concertation in the blood which indicates the alkalinity or acidity of the blood.
100
What is PaCO2?
Refers to partial pressure of carbon dioxide. Pt hypoventilating leads to inc PaCO2 (higher than 45 mmHg) = Respiratory acidosis Pt hyperventilating leads to dec PaCO2 (lower than 35 mmHg) = Respiratory alkalosis
101
HCO3...
Bicarbonate Higher than 26 = Metabolic alkalosis Lower than 22 = Metabolic acidosis Higher than 26 = Base Lower than 22 = Acidic
102
PaO2...
Mild hypoxemia = 60-80 mmHg Moderate hypoxemia = 40-60 mmHg Severe hypoxemia = lower than 40 mmHg TRUC ----- Severe - Moderate - Mild - Normal 40-60-80-100
103
What are the ABG compensation?
- Uncompensated - Partially compensated - Compensated
104
Uncompensated....
pH level is abnormal with 1 component (PaCO2 or HCO3) abnormal and the other normal.
105
Partially compensated...
pH is abnormal with the other components (PaCO2 or HCO3) also being abnormal. (BOTH)
106
Compensated....
pH is normal but the other components (PaCO2 or HCO3) are abnormal. (BOTH)
107
What is an obstructive lung disease?
Trouble getting air out. Characterized by inc airway resistance and obstruction affecting expiratory airflow.
108
All-inclusive term for obstructive lung disease... Obstruction diseases...
- Chronic bronchitis - Emphysema - Asthma - Bronchiectasis
109
What can cause an obstructive lung diseases?
- Smocking - Air pollution - Genetic (alpha-1 antitrypsin deficiency) - Infection - Aging - Allergy
110
How does smocking affect our lungs?
Affect cillias Inhibate cillias motarory - causing obstructive disease + asthma
111
Why is cillias used?
Air like projecting Help clear the lungs Help move secretion
112
Airway obstruction will lead to...
- Dec forced expiratory flow - Inc air trapping
113
How does aging affect our lungs?
Dec elastine = lungs doesn't recoil as much
114
Terme parapluis - COPD...
Chronic bronchitis, edema and asthma
115
What is a chronic bronchitis?
Productive cough on most days for 3 months/year for 2 consecutive year. = Inc + retaining mucus.
116
Pathophysiology - Chronic bronchitis
- Hypertrophy + hyperplasia of mucous glands and goblet cells (Inc mucus) - Dec # of cillia (secretion retention) - Chronic inflammatory changes in brochial walls - Dec gas exchange
117
Causes - Chronic bronchitis
Long term irritation of tracheobronchial tree 1. Smocking 2. Pollution
118
Clinical presentation - inspection - chronic bronchitis
- Blue bloater - obese and cyanotic - Mucus white, yellow, green - Inc JVP and ankle edema
119
Clinical presentation - palpation - chronic bronchitis
Tactile fremitus - Dec air trapping, Inc secretion retention - Ankle edema
120
Clinical presentation - percussion - chronic bronchitis
- Hyper-resonant - air trapping - Dull secretion retention
121
Clinical presentation - auscultation - chronic bronchitis
- Dec BS - Early inspiratory wet crackles - Wheezing
122
Clinical presentation - ABG's - chronic bronchitis
Large dec PaO2 and Inc PaCO2
123
Clinical presentation - CXR - chronic bronchitis
Cardiomegaly and white haziness
124
What is emphysema?
Enlargement of the airway distal to the terminal bronchioles accompanied by destruction of their walls. Floppy lung - Alot of air trap inside
125
What are the types of emphysema?
- Centrilobar - Panlobar
126
Centrilobar...
- More common - Affects respiratory bronchioles - M more than F - Rare among non-smockers - Common with pt with chronic bronchitis
127
Panlobar...
- Affect terminal and respiratory bronchioles - M more than F - Due to alpha-antitrypsin deficiency
128
Pathophysiology - Emphysema
- Bullae may be found in these pt - Dev from obstruction of ai flow during expiration - Leads to hyperinflation - destruction of alveolar walls - dec elastic recoil, inc dead space, dec gas exchange
129
Causes - Emphysema
- Smocking - Pollution - Alpha-antitrypsin - panlobar
130
Clinical presentation - inspection - emphysema
- Pink puffer - Thin and wasted (muscle atrophy) - Barrel chest - I:E prolonged - 1:3 - Pursed lip breathing - Inc acc respiratory muscle use (30% diaphragm, 70% acc muscles) - Signs of respiratory distress (leaning over)
131
Clinical presentation - palpation - emphysema
- Tactile fremitus - Dec (less vibration) - Chest wall expansion - Dec
132
Clinical presentation - percussion - emphysema
- Hyper-resonant (vide)
133
Clinical presentation - auscultation - emphysema
- Dec BS - May have dry crackles
134
Clinical presentation - ABG's - emphysema
- Dec PaO2 (moderate hypoxemia) - Normal or Inc PaCO2
135
Clinical presentation - CXR - emphysema
- Inc black area (hyperinflated + dec lung tissue) - Flattened diaphragm (hyperinflated) - Flattened ribs (no angles) - Narrow mediastinum (thin elongated heart)
136
What is asthma?
Chronic inflammatory condition of the airways characterized by hyper-responsiveness of the airways to various stimuli results in narrowing the airways.
137
Pathophysiology - asthma
- Acute attack - Dec threshold of airway smooth muscle reactivity - Leads to bronchospasm, bronchial wall edema and inflammation, and inc secretions within the lumen of the airways - Narrow airways inc airway resistance (both in and out)
138
Etiology - asthma
Unknown
139
What are some factors that can trigger an asthma attack?
- Intrinsic - Extrinsic
140
What are intrinsic factors that trigger asthma attack?
- Drugs - Exercise - Inhaled irritants - Respiratory infection - Stress - Weather
141
What are extrinsic factors that trigger asthma attack?
- Animals - Dust - Feather - Food - Mold - Pollen
142
Clinical presentation - asthma - Inspection
- Chest tightness - Dyspnea - Inc acc respiratory muscle use - Other signs of respiratory distress
143
Clinical presentation - asthma - Palpation
- Tactile fremitus - Dec (air trapping) - Chest wall excursion - Dec
144
Clinical presentation - asthma - Percussion
- Hyper-resonant (air trapping)
145
Clinical presentation - asthma - Auscultation
- Dec BS (breath sound) - wheezing - Possible crackles
146
Clinical presentation - asthma - ABG's
- Dec PaO2 - Inc PaCO2 in sever cases - Dec pH
147
Air trapping = Tactile fremitus
Dec
148
Air trapping = percussion
Hyper-resonant
149
Secretion/consolidation = Tactile fremitus
Inc
150
Secretion/consolidation = percussion
Dull
151
What is a bronchiectasis?
- Irreversible, abnormal dilation of bronchi and bronchioles resulting in airflow obstruction and secretion retention - Commonly associated with chronic inflammation and infection airways - Considered an extreme form of chronic bronchitis - Can be obstructive or restrictive
152
Pathophysiology - bronchiectasis
- Destruction of bronchial wall causing permanent dilation of airways - Ciliated walls replaced by non-ciliated, mucus-secretion cells - Pooling of infected secretions leading to recurrent infections - May cause atelectasis distal to obstruction
153
Etiology - bronchiectasis
- Post infection - Congenital disorders - Bronchial obstruction
154
Clinical presentation - bronchiectasis - inspection
- Thin and fatigued - Clubbing - Inc acc respiratory distress - Severe cough - Other sings of respiratory distress - Inc mucus
155
Clinical presentation - bronchiectasis - palpation
- Tactile fremitus - Depends on lungs changes - Chest wall excursion - Dec (air trapping)
156
Clinical presentation - bronchiectasis - percussion
Depends on lungs changes
157
Clinical presentation - bronchiectasis - auscultation
- Dec BS - Wheezing - possible coarse crackles
158
Clinical presentation - bronchiectasis - CXR
- Dilated airways - Dark lung fields in areas of trapping - Flattened diaphragm - May or may not see areas of consolidation or atelectasis - High resolution CT is more commonly used to help dx bronchiectasis
159
What are some restrictive diseases?
- Parenchymal diseases - Pleural diseases - Chest wall deformities - Neuromuscular diseases
160
What is a restrictive lung diseases?
- Disease that restrict the lung from expending fully - Trouble getting the air in - Dec compliance - Dec neg pressure - Dec air entry RESULTING in; - inspiratory problem - dec volume of air in the lungs (dec ventilation) - With an restrictive diseases - Inc work of breathing RESULTING in; - inc RR - inc acc muscle use - inc pressure required to maintain lung expansion and ventilation - inc fatigue
161
What are some parenchymal diseases?
- Interstitial pulmonary fibrosis (IPF) - Sarcoidosis - Atelectasis - Acute respiratory distress syndrome (ARDS)
162
What is IPF?
- Thickening of the interstitium of the alveolar walls which progress to fibrosis or scaring of the lungs - Bilat. disease
163
Pathophysiology - IPF
- Dec lung compliance (inc airway resistance on inhalation) - Inc elastic recoil - Inc fibroblasts results in inc collagen leading to fibrosis or scaring - Dec diffusion capacity
164
Etiology - IPF
- Idiopathic (most common) - Environmental exposure to inorganic dust, toxic gases, certain drugs - May be genetic factors - Some connective tissue disorders are associated with IPF (RA) - Abnormal healing of the wounds
165
Clinical presentation - IPF - inspection
- Dyspnea - Inc RR + shallow breathing - Dry unproductive cough - Clubbing - Cyanosis - Dec chest expansion
166
Clinical presentation - IPF - palpation
- Tactile fremitus - Inc (feel more vibration, thicker)
167
Clinical presentation - IPF - percussion
- Dull (because thicker)
168
Clinical presentation - IPF - auscultation
- Late fine dry inspiratory crackles (sounds like pulling a Velcro)
169
Clinical presentation - IPF - ABG's
- Dec PaO2 - Dec PaCO2
170
Clinical presentation - IPF - CXR
- Small contracted lungs - Raised diaphragm - Diffuse reticular marking (mainly in lower lobe) - High resolution CT is more commonly used to help assess the severity of IPF. Ground glass opacities is a key feature seen in IPF using HRCT.
171
What is sarcoidosis?
Disease involving a lot of granuloma (collection of inflammatory cells all over that forms a lump) dev in the lungs, skin, lymph nodes and other organs Etiology - unknow Not super common Most of the time it resolve by itself
172
What is atelectasis?
- Unilat disease - Collapse of alveoli or lung tissue (may have sub-segmental, segmental or lobar distribution)
173
Pathophysiology/etiology - atelectasis
- Obstruction (mucus plug, tumor, foreign body - it blocks the air from getting to any area distal) - Dec nitrogen (Nitrogen help to maintain alveoli open) - Dec surfactant (Inc surface tension) - Surfactant is a substance that usually dec surface tension so the alveoli stays open - Compression - from the substance - Hyperventilation - Hypoventilation - often post-opp because pt don't take slow deep breath
174
Clinical presentation - atelectasis - inspection
- Dyspnea - Cyanosis - Inc RR + shallow breathing - Ipsilat tracheal deviation
175
Clinical presentation - atelectasis - palpation
Tactile Fremitus - Dec Chest wall expansion - Dec on affected side
176
Clinical presentation - atelectasis - percussion
Dull directly over compressed tissue
177
Clinical presentation - atelectasis - Auscultation
- Dec BS or absent - Fine dry inspiratory crackles
178
Clinical presentation - atelectasis - ABG's
- Dec PaO2 - good blood flow, poor ventilation
179
Clinical presentation - atelectasis - CXR
- Ipsilat shift of mediastinum - Inc density in area of atelectasis - elevated hemi-diaphragm
180
What is an ARDS?
- Not an specific disease, it can be trigger by a lot of pathologies - An acute lung injury characterized by respiratory distress, sever hypoxemia and inc permeability of the alveolar-capillary membrane
181
Pathophysiology - ARDS
- Inc permeability of capillaries due to injury - leads to edema in 1- interstitial space and then into 2 - alveoli -3 - diff breathing - Dec surfactant production leading to inc alveolar surface tension causing dec lung compliance - V/Q mismatch - R to L shunt - arterial hypoxemia (blood passing threw without picking any O2) - Rapid fibrosis in later disease progression
182
Etiology - ARDS
- Shock - Severe pneumonia - Severe trauma - Sepsis (infection) - Aspiration
183
Clinical presentation - ARDS - inspection
- Severe dyspnea (often require ventilation) - Cyanosis - Inc RR + shallow breathing
184
Clinical presentation - ARDS - palpation
Tactile fremitus - Inc
185
Clinical presentation - ARDS - percussion
Dull
186
Clinical presentation - ARDS - auscultation
- Inspiratory crackles - Diffuse wheezes
187
Clinical presentation - ARDS - ABG's
- Severe dec PaO2 - Dec PaCO2
188
Clinical presentation - ARDS - CXR
Patchy infiltrated in periphery of lungs
189
What are some pleural diseases?
- Pneumothorax - Pleural effusion
190
What is a pneumothorax?
An abnormal collection of air in the pleural space (outside of the lungs/alveoli)
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Pathophysiology - pneumothorax
Loss of neg pressure in pleural cavity causes expanded rib-cage and/or collapsed lung Pressure outside of the lungs make it hard to expand, then collapse
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Etiology - pneumothorax
- Trauma to chest wall - Complication of invasive procedure (can cause a hole) - Idiopathic - Rupture of respiratory structure (bullea rupture) - Complication from mechanical ventilation (+ve pressure)
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Types of pneumothorax...
- Spontaneous pneumothorax - Traumatic pneumothorax - Tension pneumothorax
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Spontaneous pneumothorax
- Dev suddenly due to rupture in air containing structure - Most common young men - Primary (bleb/bullea) vs secondary (sec to another disease)
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Traumatic pneumothorax
- Due to penetrating (stabbing, gunshot wound) or non-penetrating (contusion) injury to the chest wall
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Tension pneumothorax
- Tear in pleura that acts as a one-way valve (air out but not in) - Medical emergency - acute life-threatening situation
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Clinical presentation - pneumothorax - inspection
- Sings of respiratory distress (especially with tension type) - Dyspnea - Inc RR - Chest pain (most common sign) - Dry cough
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Clinical presentation - pneumothorax - palpation
Tactile fremitus - dec (a lot of air trapping)
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Clinical presentation - pneumothorax - percussion
Hyper-resonant
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Clinical presentation - pneumothorax - Auscultation
Dec or absent BS
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Clinical presentation - pneumothorax - ABG's
Dec PaO2
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Clinical presentation - pneumothorax - CXR
- Blackened area around lungs - flattened hemi-diaphragm
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What is a pleural effusion?
Unilat disease An abnormal collection of fluid in pleural space ** Don't mistake for pulmonary edema = fluids inside the lungs **
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Pathophysiology - pleural effusion
Inc production or dec clearance of fluids
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Types of pleural effusion
Exudative and Transudative
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Etiology - pleural effusion
Secondary to infection, cancer, or disease
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Clinical presentation - pleural effusion - inspection
- May have dyspnea - Inc RR - May have chest pain (especially with deep breathing and coughing) - Dry cough
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Clinical presentation - pleural effusion - palpation
- Tactile fremitus - Dec - Chest wall excursion - Dec on side of effusion
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Clinical presentation - pleural effusion - percussion
Dull
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Clinical presentation - pleural effusion - auscultation
Dec, absent directly over effusion, may hear pleural friction rub
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Clinical presentation - pleural effusion - ABG's
- Dec PaO2 - Dec PaCO2
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Clinical presentation - pleural effusion - CXR
- White in the aera of inc fluid in pleural space - Contralateral tracheal deviation with large effusion - Blunting or loss of distinction of the costophrenic angle (angle between the diaphragm and chest wall)
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What are some chest wall deformities?
- Chest wall diseases - Bony deformities
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What is a bony deformities?
Various deformities of the bony structures of the body. Affecting the bone - affecting the lung expansion.
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What is included (Patho) in bony deformities?
- Ankylosing spondylitis - Congenital deformities - Kyphosis - Pectus carinatum - pigeon chest - Pectus excavatum - funnel chest - Scoliosis
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Etiology - Bony deformities
- Idiopathic - Neuromuscular disease - Congenital
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Clinical presentation - bony deformities - inspection
- Dyspnea - SOBOE (on excursion) - Abnormal thorax shape
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Clinical presentation - bony deformities - palpation
- Tactile fremitus - Depends on the chest deformity (DEC) - Chest wall expansion - Depends on the chest deformity (DEC)
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Clinical presentation - bony deformities - percussion
Depends on the chest deformity (Normal)
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Clinical presentation - bony deformities - auscultation
Depends on the chest deformity (Normal)
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Clinical presentation - bony deformities - ABG's
- Dec PaO2 if inc WOB
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Clinical presentation - bony deformities - CXR
Depends on bony deformity, possible atelectasis in lower lobes.
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What are some example of neuromuscular disorders affecting the mechanism of respiration? - Acting as a restrictive disease.
- ALS - Guillian-Barre syndrome - Multiple sclerosis - Muscular dystrophy - Myasthenia Gravis - Parkinson's disease - Poliomyelitis - SCI
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What muscles help with ventilation?
- Diaphragm - Intercostals - Abdominals - Acc muscles of ventilation
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Diaphragm...
- Primary muscle of ventilation - Innervation: phrenic nerve C3-4-5 - Dependent on the intercostal and abdominal muscles
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Intercostals...
- Innervation: T1-T12 - Acts to stabilize the rib cage - Internal intercostals = active exhalation - External intercostals = inhalation (quiet and forceful) - External intercostal muscles are primary muscles of ventilation
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Abdominals...
- Innervation T6-L1 - Stabilize inferior border of the rib cage - Inc intra-thoracic pressure for a strong effective cough
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What are other acc muscles of ventilation?
- Erector Spinae - Pectoralis Major and Minor - Serratus Anterior - Scalene (ant, middle, post) - SCM - Trapezius - Lats - QL
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What is cystic fibrosis (CF)?
- Present as a lung disease more than anything else - Affect a lot of organ in the body - Systemic hereditary disease of the exocrine glands (secrete something at the surface, EX - sweat glandes, salvatory glandes, mucus glandes) of the body - Results in copious amount of thick secretions - Dec lifespan
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Pathophysiology - CF
- Most commonly manifest in lungs, liver, kidney and intestine - Ion transport dysfunction: Inc electrolyte (Na & Cl) content in sweat - Inc obstruction of exocrine ducts by thick secretion - Inc secretion retention in lungs leads to recurrent lung infection, fibrosis/scarring, cystic dilations of the bronchi - Malabsorption of nutrients - pancreas deficiency - diff producing good amount of enzymes (enzyme use to absorb nutrients)
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Etiology - CF
- Autosomal-recessive gene that affects the exocrine glands - Dx in childhood (impaired growth and dev)
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Clinical presentation - CF - inspection
- Low weight (despite healthy appetite, possible osteoporosis) - Inc RR - Barrel chest - Clubbing - Chronic productive cough - because producing a lot of mucus - Copious amounts of mucopurulent or purulent mucus (often with blood in sputum)
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Clinical presentation - CF - palpation
Tactile fremitus: Can be obstructive, restrictive, mixed Inc if secretion Dec if trapping air
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Clinical presentation - CF - percussion
Can be obstructive, restrictive, mixed Hyper-resonant Dull
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Clinical presentation - CF - auscultation
- Dec BS - Inspiratory/expiratory crackles - Wheezing
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What are some infectious lung diseases?
- Pneumonia - Tuberculosis
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What is a pneumonia?
- Secretion retention - Acute inflammation of the lungs associated with alveolar filling by exudates (consolidation) - Common complication and cause of morbidity and mortality in hospitalized pt - Longer the stay in hospital - greater risk of pneumonia - Leading cause of death in Canada
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Pathophysiology - pneumonia
- Infectious agent (virus or bacteria) or irritant reaches the lungs triggering inflammatory reaction - May also be as a result of an auto-immune disease
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Etiology - pneumonia
May be viral, bacterial, fungal or other - Aspiration - Contact (trauma or chest tube) - Inhalation (droplet) - Hematogenous (circulation) - Inc risk: Infant, elderly, those with chronic cardiac or respiratory disease, immunosuppressed pt
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Clinical presentation - pneumonia - inspection
- Dyspnea - Cyanosis - Inc RR + shallow breathing - Cough (productive = bacterial, non-productive = viral) - Fever (high fever = bacterial, moderate fever = viral)
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Clinical presentation - pneumonia - palpation
Tactile fremitus - Inc (consolidation)
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Clinical presentation - pneumonia - percussion
Dull
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Clinical presentation - pneumonia - auscultation
- Wet inspiratory crackles (a lot of secretion) - Bronchial or bronchovesicular BS (normal BS heard somewhere it shouldn't be heard)
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Clinical presentation - pneumonia - ABG's
- Dec PaO2 - May have dec PaCO2
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Clinical presentation - pneumonia - CXR
- Air bronchograms - Opacities in surrounding alveoli
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What is tuberculosis?
- Airborne disease - Primary affects the lungs and other organ - An infectious, systemic, inflammatory disease
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Pathophysiology/etiology - tuberculosis
- Mycobacterium tuberculosis (bacteria) ** Most infection don't have sympts, may lay dormant then reactivate when immune system weakens
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Clinical presentation - tuberculosis - respiratory
Cough more than 2 weeks - Dry cough (early) - Productive cough (mucus/blood)
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Clinical presentation - tuberculosis - systemic
- Fever - Fatigue - Night sweats - Weight loss (poor appetite) - May have swollen lymph nodes (fighting infection)
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Diagnosis - tuberculosis
TB infection - TB skin test - TB blood test TB disease - CXR (infiltrates and cavitation typically in the upper lobes) (May have pleural involvement and/or parenchymal fibrosis) - Sputum sample - Medical hx - Physical examination
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Intervention - tuberculosis
- Meds 6-12 months - Prevent exposure to other - Placed in a -ve pressure room - Anyone entering the room must follow universal precautions and wear N-95 mask - Secretion clearance techniques - Deep breathing - Coughing
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What is different with a small pneumothorax and a large?
Small: Air collects between the lung and the chest wall. Large: A lot of air collects and pushes on the lungs and heart. + tracheal deviation. Tx - Trapped air is removed by using a chest tube.
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Exudative...
- Inc permeability of the pleural surface leading to inc fluid, proteins, white blood cells and immune cells into the pleural space - Fluid is cloudy (opaque) - Caused by inflammation, infection or cancer
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Transudative...
- Inc hydrostatic pressure in pleural capillaries - Fluid is clear and has very few proteins
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Pectus carinatum
Pigeon chest
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Pectus excavatum
Funnel chest
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What are some cardiovascular diseases?
- Acute coronary syndrome - Congestive heart failure - Pulmonary edema - Pulmonary embolism - Peripheral vascular disease
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What is an acute coronary syndrome?
= Impairments in the blood supply to the heart.
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What condition is included in acute coronary syndrome?
- Myocardial ischemia - Myocardial infarction
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What is a myocardial ischemia?
- Insufficient blood flow to myocardium - Typically presents with angina - No sympts = silent myocardial ischemia
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What are the different possible angina?
- Angina - Stable angina - Unstable angina
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Angina...
- Type of chest pain/pressure that you get as a result of coronary artery disease or insufficient blood flow to the heart - Diffuse retrosternal pressure, heaviness, tightness or constriction in the chest caused by reduced blood flow to myocardium - Pt may clench their fist over their sternum (Levine sign) - May radiate to the L jaw, L arm, and/or upper back between the scapula
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Stable angina...
- Precipitated by activities that inc myocardial oxygen demand (physical activities, sexual activities, emotional stress...) - Relived by rest or nitroglycerin
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Unstable angina...
- Occurs at rest without any precipitating factors or with minimal exertion - Not relieved by rest and may or not be relieved with nitro Requires immediate medical attention (high risk of myocardial infarction)
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What is a myocardial infarction?
- Heart attack - acute injured myocardial tissue - Death of cardiac muscle due to lack of blood flow - Caused by a sudden complete occlusion of 1 or more coronary arteries
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What are some ways that a myocardial infarction can be detected?
- By there symptoms - ECG changes - Cardiac biomarkers
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What are some myocardial infarction symptoms?
- Angina - Anxiety - Diaphoresis - Dyspnea - Dizziness - Fatigue - Nausea
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What is the next step to take if we have myocardial infarction symptoms?
- Check the cardiac biomarkers by blood work - ECG
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What are some ECG changes for myocardial infarction?
- If ischemia - ST-segment depression - Inverted T-wave - Small acute MI with small injury to myocardial tissue - No ST-segment change - Referred to as non-ST segment elevation myocardial infarction (NSTEMI) or non-Q-wave myocardial infarction (NQMI) - Large acute MI with injury to myocardial tissue - ST-segment elevation - Pathological Q-wave - Referred to as ST segment elevation myocardial infarction (STEMI) or Q-wave myocardial infarction (QMI)
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What cardiac biomarkers can be found in blood work of pt with MI?
- Troponin I - Troponin T - Myoglobin - Creatine kinase - Myocardial Band (CK-MB)
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What are the medical management for MI?
- Surgery - Medication
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What are the surgery option for MI?
- Percutaneous Transluminal Coronary Angioplasty (PTCA) - Stent - Coronary Artery Bypass Graft (CABG) - Donor vessels (saphenous vein, internal thoracic artery, radial artery of nondominant)
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What are the medications for MI?
- Beta blockers - Dec HR - Calcium channel blockers - Nitrates - Angiotensin-converting enzyme (ACE) inhibitors - Angiotensin receptor blockers (ARB) - Supplemental oxygen ** They all dec the BP **
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What is the goal of physiotherapy for MI?
- Improve exercise capacity - Improve exercise efficiency - Improve exercise tolerance - Improve self management - Improve QOL
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What consist of a cardiac rehabilitation?
Exercise, education, lifestyle modification program designed to optimize physical, psychological, social and vocational function.
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What are the phase of a cardiac rehabilitation?
- Phase 1: Acute/Inpatient Phase - Phase 2: Subacute/Conditioning Phase - Phase 3: Intensive Rehabilitation Phase - Phase 4: Maintenance Phase
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What is Phase 1: Acute/Inpatient Phase?
- Traditionally begun in acute hospital setting - Role of PT: prepare for D/C, monitor activity tolerance, support risk factor modification techniques, provide emotional support, build self-efficacy and educate the pt on how to recognize adverse signs/sympts with activity, and collaborate with other members of the team. Focus of interventions is on assessing hemodynamic responses to activity, and independence in functional mobility activities. - Vital signs should be monitored before, during and after. - Intensity should be low level.
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What is Phase 2: Subacute/Conditioning Phase?
- Typically begins after D/C in the outpatient setting - Conditioning exercises are done with close cardiac monitoring
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What is Phase 3: Intensive Rehabilitation Phase?
- Exercise in large groups - Resistance training typically initiated in this phase
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What is Phase 4: Maintenance Phase?
- Pt is encouraged to continue exercise training in a group setting or self-monitored program
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What are the phase 1 levels?
Level 1 (1 METs) - Pt in ICU medically stable for 24 hours - Bed rest but allow gentle AROM exercises of UE/LE and deep breathing exercises Level 2 (2METs) - Allow sitting up in a chair for meals, performing ADLS and walking to the bathroom/inside the room (up to 50 ft) a few times a day Level 3 (3 METs) - Ambulate up to 250 ft a few times a day Level 4 (4 METs) - Performing ADLs independently and ambulate up to 1000 ft a few times a day - Allow climbing of 1 flight of stairs
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Education for pt with heart disease...
- Activity guidelines - Self-monitoring - Symptom recognition and response - Nutrition - Medication - Sexual activity - Psychological/social issues
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Activity guidelines...
Pt and family members should understand specific activity guidelines including planned exercised session, leisure time and rest.
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Self-monitoring...
- Teach pt to monitor the intensity of their activities by; palpating pulse, HR monitor, RPE scale - Includes awareness of other signs/sympts that may suggest exercise intolerance (dyspnea, confusion, light-headed)
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Symptom recognition and response...
- Pt should be able to recognize specific cardiac sympts and know how to respond - Pt should know when to call their physician or go to the hospital - Info on action that should be taken should be provided
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Nutrition...
- Pt meet with nutritionist to discuss their dietary habits Recommendation: Reduce fat intake, monitor salt and fluid (CHF)
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Medication...
- Pt should receive info on effects, side effects, dosage and timing - Pt should disclose all supplements and herbal remedies being taken - Pt should understand which non-prescribed drugs may have -ve interactions with prescribed and should be avoided
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Sexual activity...
- Encourage to verbalize their concern to each others - Likely ready once their energy level is satisfying for them, they can walk outdoors and climb stairs comfortably - Is like any other physical activity, planning, pacing and warm-up contribute to more comfort/better outcome
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Psychological/social issues...
- Physio should recognize psychosocial - PT plays an IMP role in reassuring pt that the sympts are normal du to cardiac event - Pt should seek guidance and counseling - Referral to mental health professional - PT work with pt for a pt centered approach to dev goals, outcome and plan of care that matches the client needs, values and level of function
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What is CHF?
Syndrome characterized by impairment in the hearts pump function - Problem pumping the blood out of the heart Collection of S+S that create this Dx (Not actually a disease) Now pt are living longer
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CHF - Epidemiology
- Leading cause of cardiac deaths in North America - Most frequent cardiac diagnosis for hospital admission - M more than F
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CHF types
2 types; based on structure and based on function Based on structure: - Left-sided heart failure - Right-sided heart failure - Biventricular heart failure
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LSHF
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RSHF
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Biventricular heart failure
Lt heart failure is so bad = backup in lungs = pulmonary artery is full (too much pressure) = R ventricule as too much pressure = Rt heart failure
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Pathophysiology - CHF
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Risk factors - CHF
- Cardiac muscle dysfunction - Cardiac dysrhythmias (altered electrical function) - Cardiomyopathies (myocardial cells damage) - Long term CAD - Hypertension - Valve abnormalities - Pericardial pathology ** If you have any of those --- Inc risk of dev CHF **
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S+S - CHF
LSHF: - Dyspnea - Fatigue - Weakness - Pulmonary edema (fluids in lungs) - Paroxysmal nocturnal dyspnea (sudden episode of SOB at night) - Orthopnea (SOB when lying down) RSHF: - Dyspnea - Fatigue - Weakness - Jugular vein distension - Peripheral edema - Pitting edema - Fluid weight gain - Ascites (excess fluids in belly area)
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Interventions - CHF
- Positioning to reduce orthopnea (semi-faller) - Relaxed breathing ex's (diaphragmatic breathing - Demande moins d'énergie) - Supplemental oxygen - Progressively inc ambulation - Progressively return to exercise - Cardiac rehabilitation program ** The rest is more medical - out of our scope **
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What is pulmonary edema?
Abnormal accumulation of fluid in the lungs. Fluid moves from the pulmonary capillaries --- interstitial space --- alveolar space
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Pathophysiology/etiology - Pulmonary edema
- Inc capillary membrane permeability (normally shouldn't allow easy exchanges) - Inc capillary hydrostatic pressure - Dec capillary osmotic pressure - Lymphatic insufficiency (normal will drain fluids)
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Types of pulmonary edema
- Cardiogenic pulmonary edema - Non-cardiogenic pulmonary edema
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Cardiogenic pulmonary edema
- High pressure (inc pulmonary capillary hydrostatic pressure) - Backflow of blood in the system due to kidney, heart muscle or valve damage leading to inc blood accumulation in pulmonary capillaries which inc pulmonary capillary hydrostatic pressure - LSHF
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Non-cardiogenic pulmonary edema
- Low pressure - Inc permeability of the pulmonary capillaries and alveolar endothelium due to trauma or toxins - ARDS
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Clinical presentation - pulmonary edema - inspection
- Dyspnea - Inc work of breathing - Cyanotic - Orthopnea - Cough with pink frothy sputum
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Clinical presentation - pulmonary edema - palpation
Tactile fremitus: Normal or inc
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Clinical presentation - pulmonary edema - percussion
Dull
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Clinical presentation - pulmonary edema - auscultation
- Dec BS - Fine inspiratory crackles - Coarse crackles
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Clinical presentation - pulmonary edema - ABG's
Dec PaO2
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Clinical presentation - pulmonary edema - CXR
- Cardiomegaly (Inc size) - Enlarged pulmonary vessels - White fluffy/hazy airspace - Kerley B lines (short hori. line near the pleural space)
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What is pulmonary embolism?
- A blood clot that has been lodged in a pulmonary artery - Commonly associated with DVT in the lower extremities (blood cloth in calf) - If DVT dislodge and makes it way to lungs = Pulmonary embolism - Life-threatening
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Pathophysiology - pulmonary embolism
- Pulmonary embolus obstructs blood flow to an area of the lungs - If blood flow to lung tissue completely blocked, can lead to infarction and necrosis of lung tissue - Very large embolism can lead to inc pulmonary artery resistance --- inc work load of R ventricle - RHF
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Risk factors - pulmonary embolism
- Immobilization (post-op) - Secondary inc coagulation - Other: CHF, Hx of DVT, obesity, pregnancy, trauma, varicose veins
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Clinical presentation - pulmonary embolism - inspection
- Acute onset of dyspnea - Inc RR - *** Chest pain *** - May have cough with bloody sputum (hemoptysis)
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Clinical presentation - pulmonary embolism - ABG's
- Dec PaO2 - Dec PaCO2 - Inc pH
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Clinical presentation - pulmonary embolism - CXR
Infarcted area of lung appears white (rare)
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Clinical presentation - pulmonary embolism - Dx
Using CT scan or V/Q scan
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Pulmonary embolism - interventions
- Prophylactic post-op - Anti-coagulant meds (not PT scope) - Bed exercises (such as ankle pumps) - Early mobilization (better than bed ex's) - Compression stocking (preventive) - If PE or DVT is suspected or confirmed: - Discontinue exercise and mobilization until further notice - Notify nurse or surgeon - Document - Inc anti-coagulant meds - Thrombolytic meds
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What is peripheral vascular disease (PVD)?
- Disorders of the blood vessels (arteries and veins) - Primarily due to atherosclerosis (hardening and narrowing of the arteries) - Significant narrowing of vessels must occur before there is enough occlusion of blood flow to produce sympts
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S+S of PVD
- Leg pain (intermittent claudication) - Coldness in affected leg - because of poor circulation - Dec pulses in lower limb - because of poor circulation - Dec mobility and function of limb due to pain - Possible numbness - nerve damage - Possible pain and paleness of leg with elevation (Buerger's test - like SLR) - Dec hair growth - because of poor circulation - Skin breakdown - Ulceration - Gangrene
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What is intermittent claudication?
- Pain or cramping in the buttock or legs (mainly calf) as a result of poor circulation to the affected area - Inc pain with inc activity due to inc energy demands on the muscle that has poor circulation - Dec pain at rest (even in standing) - Must differentiate between intermittent claudication vs neurogenic claudication
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Interventions - claudication
- Progressive inc of activity and aerobic exercise - to inc tolerance to walk - May stimulate the dev of collat. blood vessels - Education (risk factors and self management) - Self-ax of skin, educate skin-care
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Intermittent vs neurogenic claudication
Intermittent: Also known as vascular claudication Pain with WB - walking Static (stop), no pain (standing or sitting) Neurogenic: Also known as spinal stenosis - nerve roots are compressed as they exit the interverbal foreman Inc pain with lsp extension Dec pain with lsp flexion Pain with WB - walking Feels better in sitting LSP IMPACT
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What is CI with PVD - pulmonary vascular disease?
Compressive socks, because there's already poor circulation it will only make it worst.
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Categories of infectious agents
- Bacteria - Virus - Fungi - Protozoa
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What are some personal protective equipment? PPE
- Hand hygiene - Gloves - Gowns - Face protection (mask, goggles)
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Hand hygiene
- Should take at least 15 sec - Use alcohol-based hand rub preferred method when not visibly soiled - Wash hand with soap and water visibly soiled with blood or other fluids
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Gloves
- When in contact with blood or body fluids, non-intact skin or contaminated items - Change between pt and between sites - Be aware of touching self, cloths and environment - Wash hand after removal
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Gowns
- If your clothing may get soiled - Wash hand after removal
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Mask and eye protection
- For all coughing pt - Procedure where splashing, spray, sneeze may occur
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What are the 5 moments for hand hygiene? Key moments when health care workers should perform hand hygiene
- Before touching a pt - Before clean/aseptic procedures - After body fluid exposure/risk - After touching pt - After touching pt surrounding
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Donning vs Doffing PPE
Don 1. Sanitize 2. Gown 3. Mask 4. Eye protection 5. Gloves Doff 1. Gloves 2. Gown 3. Sanitize 4. Goggles 5. Mask 6. Sanitize
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What are some safety additional precautions?
- Contact - Droplet - Airborne
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Contact transmission
- Transmission of organisms via skin-to-skin contact or indirect physical contact with infected environmental surfaces
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Droplet transmission
- Transmission of organisms through droplets generated from the respiratory tract (coughing, sneezing, talking) - Droplets travel short distances and settle on surfaces - Deposit on the nasal or oral mucosa of the new host
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Airborne transmission
- Transmission of organisms which have become aerosolized and remain suspended in the air - Inhaled by susceptible host - Special ventilation systems are required to control of airborne transmission of microorganisms
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Isolation type for contact transmission
Room: Private room or group pts with the same infection Pt transport: Minimize transport of pt
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Isolation type for droplet transmission
Room: Private Pt transport: Mask pt when transport is necessary
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Isolation type for airborne transmission
Room: Private room with -ve pressure system and ensure door is closed Transport: Mask pt when transport is necessary
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What are some infectious agents that are considered contact precautions?
- Methicillin-Resistant Staphylococcus Aureus (MRSA) - Vancomycin-Resistant Enterococcus (VRE) - Extended Spectrum Beta Lactamases (ESBL) - Clostridium Difficile (C-Diff) - Norovirus - Uncontained diarrhea/drainage
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What are the contact precaution procedure?
- Hand hygiene prior to pt encounter - Gloves required for all pt and pt environment contact - Long sleeved gown required if skin or clothing will come into contact with pt or pt environment - Use dedicated equipment or disinfect prior to use with other pts - Hand hygiene after pt encounter
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What are some infectious agents that are considered droplet precautions?
- Mumps - Rubella - Pertussis (whooping cough) - Influenza (droplet and contact) - Pneumonia (droplet and contact) - Meningitis (droplet and contact) - Acute Respiratory Illness (droplet and contact)
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What are the droplet precaution procedure?
- Hand hygiene prior to pt encounter - Mask on pt when outside room or for duration of clinic visit - Mask and eye protection for anyone within 2 meters of pt - Hand hygiene after pt encounter - Clean and disinfect equipment and surface
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What are some infectious agents that are considered airborne precautions?
- TB if evidence of respiratory disease - Disseminated Shingles (airborne and contact) - Measles - SARS - Varicella (airborne or contact)
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What are the airborne precaution procedure?
- Airborne infection isolation room (-ve pressure room) - Mask on pt when outside room - Fit tested N95 respirator and eye protection for all health care workers - Only immune staff to have contact with pts with measles or varicella
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Why is thoracic surgery used?
Remove an irreversibly damaged area of the lung.
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What are the thoracic surgery types?
- Pneumonectomy - Lobectomy - Segmental resection - Wedge resection - Lung volume reduction surgery or bullectomy
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What are the thoracic surgery incisions?
- Median Sternotomy (Cut through the sternum) - Thoracotomy (Cut in intercostal space of ribs)
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Pneumonectomy
Removal of a lung Avoid lying with surgical side up until further notice from surgeon
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Lobectomy
Removal of a lobe of a lung
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Segmental resection
Removal of a segment of a lobe
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Wedge resection
Removal of a portion of a lung
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Lung volume reduction surgery or bullectomy
Removal of large emphysematous tissue
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What is a thorocotomy?
- Commonly performed for lung resections or to remove an irreversibly damaged area of the lung - Posterolateral thoracotomy is most common procedure - Incision follows the path of the 4th intercostal space - Chest tubes are placed to evacuate air and fluid from pleural space
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What are the muscles incised during thorocotomy?
- Latissimus dorsi - SA - External intercostals - Internal intercostals - Trapezius - Rhomboids
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What are some positioning with thorocotomy?
- Lying on side of chest tube is NOT CI, as long as the tube is not kinked or pulled (often avoided but pts) - IMP to change positions as with all surgeries in order to avoid pressure ulcers
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Education pre/post thoracotomy
- Deep breathing - Supportive cough - Lines - Scar management - infection prevention - Relaxation - Bed mobility - Positioning - Transfers - Early mobilization
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Potential complications of pulmonary surgery
- Aspiration - Inc pain - Phrenic nerve impairment - Atelectasis - Ulcers - DVT
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What is DVT?
- A thrombus (blood cloth) that forms in a deep vein of the body - Typically the calf/leg - Thrombus may partially or completely block blood flow - Thrombus may potentially dislodge and travel to the lungs (pulmonary embolism), heart or brain = fatal - Venous stasis due to immobility post-op can inc the risk of DVT. along with hypercoagulation and changes to blood vessel wall
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S+S - DVT
- Leg pain - Tenderness - Ankle edema - calf swelling - Dilated veins - Positive Homan's sign (DF of ankle)
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Prevention - DVT
- Early mobilization - Ankle pumps - Anti-coagulants - Graduated compression stockings
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What to do if we suspect a pt with DVT?
- Stop tx, which may be CI until further notice - Alert surgeon, doctor or nurse (they will do a droppler ultrasound) - Document findings
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What is a cardiovascular surgery?
- Specialized thoracic surgery involving the heart and great vessels - Pt is placed on an extracorpal membrane oxygenator (bypass machine) due to interrupted blood flow during procedure
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What are some common cardiovascular surgeries?
- Heart surgery - CABG (Coronary Artery Bypass Graft) - Saphenous vein graft (mobility and leg exercises restricted until further notice) - Internal thoracic artery graft - Radial artery graft - Valve replacements (aortic and mitral valve replacement) - Heart transplant - Surgery on great vessels - Aortic aneurysm repair - Abdominal aortic aneurysm (AAA) repair (laparotomy)
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Sternal (cut) precautions
Limit UE exercise while sternal incision are healing - No pushing (pec activation - insertion) - No pulling - No lifting one arm above 90 deg - No hand behind back - No driving for 4 weeks (hori. abd, activate pecs) - No lifting more than 10 lbs for 6 weeks
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Benefits of early mobilization after cardiovascular surgery
- Improves breathing - Chest mobility - Secretion clearance - Assists GI function/bowel mobility - Improves conditioning and activity tolerance - Allow increased independence - Improves mood - Prevents: - Aspiration - Thrombus formation - Muscle atrophy - Contractures - Pressure sores - Neuropathy
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What is a bypass machine?
Machine that takes over the function of the lungs and heart. Surgeons oxygenate the blood and pumps it out of the body so that the heart can be silent and they can do the surgery.
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Sternal timeline
Take 6-8 weeks to heal
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What are the different types of ventilation?
-ve pressure Vs +ve pressure -ve pressure: Normale - The way our bodies uses ventilation +ve pressure: Pushing/forcing air in the lungs
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What is mechanical ventilation?
The use of a machine or device to assist or replace spontaneous ventilation. Mechanical - Refers to a machine
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Where do we use mechanical ventilation?
Mostly used in hospital setting, especially in ICU
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Ventilation Vs Respiration
Ventilation = Mvt of air in and out of the lungs Respiration = Mvt of gaz across a membrane
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Administration of mechanical ventilation: Invasive vs Non-invasive
Invasive (intubation): Going IN the body - Endotracheal tube (ETT) (Passage of artificial airway to pt trachea) - Mouth or nose - Tracheostomy (Incision in neck) Non-invasive: Nothing is getting in - Nasal mask (goes over nose) - Complete face mask (both nose and mouth)
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What is a nasogastric tube?
Feeding tube that goes from the nose to the stomach
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Indication for mechanical ventilation
- Severe hypoventilation, hypoxia or hypoxemia - Apnea - Person is not breathing - Acute hypercarbia - Elevated CO2 - PaO2 lower than 50 mmHg with supplement O2 - RR higher than 30 breaths/min - Vital capacity less than 10L/min - Inspiratory force lower than -25 cm H2O - Central depression (nervous system as slow down) - Dec level of consciousness - Anesthesia or sedation - Head injury - Drug overdose - Dec work of breathing and respiratory muscle fatigue - Poor pulmonary hygiene (secretion clearance)
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What are some mechanical ventilation possible complications?
- Barotrauma - Volutrauma - Ventilator acquired pneumonia (VAP) - Diaphragm atrophy - Hemodynamic compromise
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Barotrauma
Is due to high pressure Can lead to an alveolar rupture (then to a pneumothorax)
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Volutrauma
Is from excessive volume Will cause an overdistension of alveolar
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Ventilator acquired pneumonia (VAP)
Result of intubation Micro aspiration (foreign object is being breath in leading to pneumonia) of gastric contents or oral secretions
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Diaphragm atrophy
As a result of disuse Mechanical ventilation is doing all the work After their off ventilation it can be hard to breath because the muscle are so weak
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Hemodynamic compromise
Hemodynamic - Refers to flow of the blood Overinflated lungs --- compression of great vessels --- dec venous return --- dec cardiac output
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What are the type of mechanical ventilation breaths?
- Mandatory - Assisted - Spontaneous
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Assist control ventilation (ACV)
Used when pt can't make adequate respiratory effort. Assisting their breaths. Disadvantage: - If RR is fast pt still get full tidal volume and it can lead to respiratory alkalosis and hyperinflated - Tidal volume and minimum # of mandatory breaths/min (RR) delivered by ventilator. This will deliver a minimum minute ventilation (amount of air getting in and out in a min). - Pt able to initiate inspiration but still receives preset tidal volume. - If pt does not initiate breath within a specific time period, the ventilator will deliver a breath to maintain the RR.
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Mandatory - mechanical ventilation breaths
Pt is completely dependent Ventilation is initiated, controlled and ended by the ventilator
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Non-invasive - modes of ventilation
- Continuous positive airway pressure (CPAP)
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Assisted - mechanical ventilation breaths
Ventilation initiated by pt but controlled and ended by the ventilator
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What are the different modes of ventilation?
- Invasive - Non-invasive - Continuous mandatory ventilation (CMV)
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Spontaneous - mechanical ventilation breaths
Entire by pt Ventilation is initiated, controlled and ended by the pt but the volume and pressure of the breath delivered by the ventilator is based on pt demand.
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Continuous mandatory ventilation (CMV) - modes of ventilation
- Tidal volume (Depth of breath) and preset respiratory rate delivered by ventilator - Ventilation provides total support (pt has no control) Mostly used for someone completely sedated and not breathing on its own.
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Invasive - modes of ventilation
- Continuous mandatory ventilation (CMV) - Assist control ventilation (ACV) - Synchronized intermittent mandatory ventilation (SIMV)
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Synchronized intermittent mandatory ventilation (SIMV)
- Preset mandatory tidal volume and RR (usually set low so pt can initiat breath) delivered by ventilator - Pt able to breath spontaneously between ventilator breaths. - Spontaneous pt-initiated breaths are synchronized with ventilator
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Ventilator adjuncts
- Positive End Expiratory Pressure (PEEP) - Pressure Support Ventilation (PSV)
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Continuous positive airway pressure (CPAP)
Always providing some sort of pressure Can be invasive or non-invasive - Pt spontaneous breaths are augmented with predetermined level of (+ve) pressure delivered throughout the entire respiratory cycle (inspiratory and expiratory phase) - Keeps airway open continuously in pts who can breathe spontaneously on their own, but require assistance keeping airway unobstructed - Commonly used as a weaning mode for those who are intubated or an attempt to postpone intubation - When used with mask is considered non-invasive ventilation - Helps prevent alveolar collapse, improve FRC, and enhance oxygenation - Used in pts with obstructive sleep apnea, adults with neuromuscular diseases, acute and chronic ventilatory failure, and children whit acute respiratory failure
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Positive End Expiratory Pressure (PEEP)
Preventing alveoli to collapse When we exhale alveoli begin to close as the air is escaping but PEEP prevent it from closing all the way - A +ve pressure is applied to the lung at the end of the expiratory phase of ventilation - Helps keep alveoli open during expiration and reduces pulmonary shunting
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Pressure Support Ventilation (PSV)
- Pt-initiated breaths are augmented by the ventilator to maintain a constant preset inspiratory pressure - Pt initiates all breaths and controls the respiratory rate, and inspiratory time - Helps dec work of breathing A step to normal Working on getting out of ventilator Disadvantage: If the pt cant breath it's not safe because it wont activate (related to muscle weakness non-activated)
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Complications of PEEP may include:
Inc physiological dead space Dec cardiac output Ventilator associated pneumonia Inc risk of barotrauma ** High level of PEEP can cause excessive alveolar distension (volutrauma) or pulmonary barotrauma ( Ex - Pneumothorax) **
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How to communicate when intubated?
Because the communication is altered when intubated or wearing a face mask we need to find ways to communicate - Writing - Hand signals - Communication board
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What is weaning?
- The process of dec mechanical ventilation - A spontaneous breathing trial while being closely monitored is typically performed to Ax readiness to begin weaning process We put them on low ventilation and see how they react
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What are we looking when we exercise testing?
- Pre-exercise test evaluation - Maximal exercise test - Submaximal exercise tests
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Why pre-exercise testing?
- Exercise stresses the body and may lead to adverse responses to the pt. Pre-screening pts prior exercises identifies whether a pt is at risk of an adverse response to exercises.
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Purpose of pre-exercise testing
- Purpose: to screen for pts at risk of adverse effects with exercise who require further ax prior to starting an exercise program
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Pre-exercises testing may include...
- Detailed hx - Physical examination - Laboratory tests - Risk stratification processing - Test selection
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What are some pre-exercises testing risk factors?
- Age - Family history - Physical activity level - Obesity - Pre-diabetes - Dyslipidemia - Hypertension - Stress/psychosocial - Smoker - Alcohol consumption - Diet
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Age - pre-exercises testing risk factors
Men older than 45 y.o Woman older than 55 y.o
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Family history - pre-exercises testing risk factors
Sudden death MI Coronary revascularization - Before 55 y.o in father/brother - Before 65 y.o in mother/sister
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Physical activity level - pre-exercises testing risk factors
Not engaging in at least 30 min of moderate physical activity at least 3x/week in the last 3 months (any exercises that cause SOB but still allows to talk)
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Obesity - pre-exercises testing risk factors
- BMI higher or equal to 30 kg/m2 - Waist circ (male higher 102 cm, woman higher than 88 cm)
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Pre-diabetes - pre-exercises testing risk factors
- Any hx of diabetes - Any blood tests looking into glucose levels - If pt is unaware count as a risk if (older or equal to 45 y.o, BMI higher or equal to 25 kg/m2)
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Dyslipidemia - pre-exercises testing risk factors
- LDL (Bad): higher or equal to 130 mg/dL - HDL (Good): lower than 40 mg/dL - Pt on statin meds - Total serum cholesterol: higher or equal to 200 mg/dL
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Hypertension - pre-exercises testing risk factors
- Systolic BP higher or equal to 140 mmHg and distolic BP higher or equal to 90 - Taking hypertension meds
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Stress/psychosocial - pre-exercises testing risk factors
- Depression/anxiety/stress levels - SF-36: QOL - Beck depression inventory: depression
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Smoker - pre-exercises testing risk factors
- Current smoker - Quit within last 6 months - Exposure to second hand smoke
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Alcohol consumption - pre-exercises testing risk factors
Male more than 14 drink/week Woman more than 9 drink/week
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Diet - pre-exercises testing risk factors
No specific guideline, just see if pt considering having good nutrition habits.
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What are some exercise testing indication for Dx?
- Maximal functional work capacity - Submaximal functional work capacity - Endurance - Mvt economy - Onset of exertional sympts - Subjective - Dyspnea, chest pain, leg pain, cerebral sympts - Objective - EKG changes, HR and BP response - Effect of meds - Evaluate progress
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What are some exercise testing indication for prescription?
- Exercise program - Meds
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What are some exercise testing indication for prognostic?
- Determine likelihood of adverse cardiovascular events - Determine surgical risk
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Absolute CI to exercises testing
- A recent significant change in resting ECG suggesting significant ischemia, recent MI (within 2 days) or other acute cardiac events - Unstable angina - Uncontrolled cardiac dysrhythmias causing sympts or hemodynamic compromise - Symptomatic severe aortic stenosis - Uncontrolled symptomatic HF - Acute pulmonary embolus or pulmonary infarction - Acute myocarditis or pericarditis - Suspected or know dissecting aneurysm - Acute systematic infection, accompanied by fever, body aches or swollen lymph glands
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Relative CI to exercises testing
- Left main coronary stenosis - Moderate stenotic valvular heart disease - Electrolyte abnormalities - Severe arterial hypertension at rest SBP higher 200 mmHg DBP higher 110 mmHg - Tachydysrhythmia or bradydysrhythmia - Hypertrophic cardiomyopathy and other forms of outflow tract obstruction - Neuromotor, musculoskeletal or rheumatoid disorders that are exacerbated by exercise - High-degree atrioventricular block - Ventricular aneurysm - Uncontrolled metabolic disease - Chronic infectious disease - Mental or physical impairment leading to inability to exercise adequately
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Maximal exercise testing
- Is administered by physician (Not in our scope) - Also known as stress test - Graded exercise tests (intensity progressively inc) - Used to predict VO2 max - Treadmill tests (Bruce protocol) Subject work at complete exhaustion
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Submaximal testing
- May be administer by PT - Evaluative submaximal exercise tests - Is used as a measure of endurance and exercise capacity - 6MWT - Distance covered by pt walking at a comfortable pace for 6 min - Subject is allowed to take brake as needed - 2 practice trial are required - Variant (2MWT, 3MWT) - Disadvantage - HR, BP not monitored and the level of motivation impact scoring - Advantage - Easy to addmister, no equipment needed - Predictive submaximal exercise tests - (Locking at HR and BP) - Used to predict VO2 max - Treadmill tests (modified Bruce protocol) - Cycle ergometer tests
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General signs/sympts to prematurely terminate an exercise test
- Fatigue (physical or verbal) - Signs of poor perfusion - Lightheadedness - Confusion - Ataxia - Pallor - Cyanosis - Nausea - Cold or clammy skin - SOB - Wheezing
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Cardiovascular signs/sympts to prematurely terminate an exercise test
- Onset of angina or angina like sympts - Leg cramps or claudication - Drop in SBP - Higher/equal to 10-15 mmHg with an inc in work rate - Drops below the value obtained in the same position prior testing - Excessive inc in BP - SBP higher 200-250 mmHg - DBP higher 110-115 mmHg - Failure of SBP to rise as exercise continues - Failure of HR to inc with inc exercise intensity - Noticeable change in heart rhythm by palpation or auscultation or ECG
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Other sings/sympts to prematurely terminate an exercise test
- Subject request to stop - Failure of testing equipment
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What are we locking at when exercise prescription?
- Exercise selection - Parameters - Monitoring - Safety - Indication to stop exercise
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On what is based the exercise selection?
- Goals - Ability - Pt preference - Safety
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On what are based the exercise parameters?
- Goals - Evaluation - Established guidelines - Safety
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Parameters for exercise includes...
FITT Frequency Intensity Time Type
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Intensity
- The level of difficulty of a given exercise or exercise session Intensity may be prescribed by... - RPE (rating perceived exertion) - HR max - VO2 max - Maximal oxygen uptake reserve VO2R - Max metabolic equivalents METS max - Standard intensity ranges based on the above methods (minus RPE) is 60-85% (some place 70%) - High risk intensity ranges based on the above methods (minus RPE) is 50-75% (some place 60%)
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Frequency
- # of days/week or time/day dedicated to an exercise or exercise program - Aerobic is recommended 3-5day/week at moderate to vigorous intensity
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Rating of perceived exertion (RPE and modified Borg)
RPE 6-20 Multiply by 10 = HR 6 - 7 - Very very light 8 - 9 - Very light 10 - 11 - Fairly light 12 - 13 - Somewhat hard 14 - 15 - Hard 16 - 17 - Very hard 18 - 19 - Very very hard 20 - MAX Borg 0 - Nothing 0.5 - Very very slight (just noticeable) 1 - Very slight 2 - Slight 3 - Moderate 4 - Somewhat severe 5 - Severe 6 - 7 - Very severe 8 - 9 - Very very severe (almost max) 10 - MAX
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Type
- The type or mode of activity or exercise chosen Types of exercise may include ... - Aerobic exercise (walking, running, swimming, biking) - Aerobic metabolism - Resistance training ( strength - Anaerobic, endurance, power - Anaerobic)
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Time
- A measure of the amount of time or the reps/sets physical activity of the exercise session is performed - It is recommended that most adults accumulate 30-60 min of moderate intensity exercise per day (equal/more than 150 min/week) or 20-60 min of vigorous exercise per day (more/equal to 75 min/week) - Less than 20 min of exercise per day may still be beneficial for sedentary individuals - Duration of physical activity may be performed continuously or intermittent (10 - 10 - 10 min) in multiple sessions
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Canadian physical activity guidelines
- Adults should accumulate at least 150 min of moderate-vigorous intensity aerobic per/week - Suggest to add muscle and bone strengthening using major muscle groups at least 2 days/week ** For anyone older than 18 y.o **
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Monitoring
- If pt is at any risk of adverse response to activity it is advise to be supervised - HR, BP, SpO2, dyspnea and other signs/sympts of respiratory distress should be monitored for pt at risk - Stable pt should be trained and educated on self-monitoring, maintaining intensity of exercise within appropriate limits and keeping record of details of exercise sessions prior to carrying out an exercise program indepently
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Safety
- Appropriate screening and testing should take place prior to beginning an exercise program - All CI for exercise testing also apply to exercise training - All pts should refrain from using a Valsalva maneuver - Upper body exercise may inc SBP, hypertensive individuals should be cautious when performing any upper body exercises and avoid high intensive upper body exercises
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What is the goal of intervention?
- Optimize gas exchange at all levels - Improve pt condition - Dec sympts - Optimize function ** Pt status should be ax prior every tx session and re-ax after tx **
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What is included in cardiovascular interventions?
- Positioning - Breathing exercises - Airway clearance techniques - Forced expiratory techniques - Exercise
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What is included in positioning?
- V/Q matching - Dyspnea in COPD
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What is the optimal positioning for V/Q matching?
- Unilat lung disease: Lie on unaffected side "Good lung down" - Gravity will bring all blood to good lung OPTIMIZE - Bilat. lung disease: Lie in prone - Pneumonectomy: Do not lie with affected side up "Bad lung down" - We don't want the saline to go in good lung - ARDS: Lie in prone
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Positions to dec dyspnea in COPD
- Standing, leaning back against a wall with hands bearing onto thighs to unload thorax - Standing, leaning against a table - Sitting, leaning forward with elbows resting on thighs - Sitting, leaning forward against table Helping because unloading thorax + requires less energy so less oxygen = less SOB
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Positioning for V/Q matching
V/Q = ventilation over perfusion ratio, optimal is 0.8 Cardiovascular pts may experience greater distress when placed in certain position. This distress can be explained by V/Q inequalities. Positioning is often the first step in every tx procedure.
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What are some breathing exercises?
- Deep diaphragmatic breathing - Pursed lip breathing for COPD - Inspiratory muscle training - Segmental breathing - Sustained maximal inspiration
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Deep diaphragmatic breathing
- Teaching pts deep diaphragmatic breathing helps promote dec use of acc muscles of breathing and promote inc use of diaphragm muscle for breathing - Also known as belly breathing - Diaphragmatic breathing is more efficient (dec energy cost) than using acc muscle for breathing - Pt performs long slow breaths - Promotes relaxation - May use hands on belly to guide diaphragmatic breathing or may cue diaphragmatic breathing by instructing pt to take a quick sniff through the nose ** Not encourage for pt with COPD **
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Pursed lip breathing for COPD
- Performed by taking a breath in through the nose and exhaling through tightly pressed pursed lips. Smell the roses, blow out the candles. - Exhalation phase should be 3x as long as inspiration (ex 2 sec in, 6 sec out) - Creates +ve back pressure that splints small airways open longer - Helps control and reduce RR - More efficient emptying of the lung (dec hyperinflation) - Improved gas exchange - Promotes relaxation ** Better for COPD pt **
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Inspiratory muscle training
- Resistive exercise training for the muscle of respiration aimed at inc the strength or endurance of respiratory muscles - Inspiratory muscle training is commonly performed using an inspiratory muscle trainer (IMT) Think of it like a weight training... Parameters: Strength - F: 2-4x/week, I: 60-85% PImax (max inspiration pressure), T: 8-12 reps 1-3 sets Endurance - F: 4-6x/week. I: 40-85% PImax, T: more than 15 mins (as tolerated)
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Segmental breathing
- Localized breathing towards a segment of a lung that requires greater expansion or ventilation - Uses tactile input to inc expansion of specific areas - Questionable whether a person can expand a localized area of lung specifically while not expanding others
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Sustained maximal inspiration
- Pt performs sustained maximal inspiration to TLC for 3-5 sec - Incentive spirometer (IS) is a method of performing a sustained maximal inspiration (SMI) using a device to measure flow or volume - Often used post-op to prevent atelectasis or airway closure - IS provides visual feedback and helps provide incentive/goal for pt which in turn helps with pt compliance 2 types: Flow meter: Instruct pt to keep the balls up and level as much as possible when inhaling Volume meter: Instruct pt to inhale deeply with constant flow keeping the flow indicator within the prescribed ranges
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What are the airway clearance techniques?
- Postural drainage - Percussion - Vibrations - PEP device - Independent breathing techniques - Suctioning
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Postural drainage
- Pt is placed in a position that allows drainage of secretion from bronchial airways via gravity - Often used in conjunction with percussions and vibrations - Pt maintains postural drainage position for 5-10 mins or longer (if tolerated) - The pt face and SpO2 should be monitored during tx - Signs of tx intolerance includes inc SOB, anxiety, nausea, dizziness, hypertension and bronchospasm ** Position may be modified when pt presents with a precaution or relative CI for traditional postural drainage positioning ** What ever lobe/segment is affected you want it up so that the gravity make it go down
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CI for postural drainage
- Intracranial pressure higher than 20 mmHg - Head and neck injury (spinal instability) until stabilized - Active hemorrhage whit hemodynamic instability - Recent spinal surgery or acute spinal injury - Active hemoptysis - Empyema - Bronchopleural fistula - Pulmonary edema associated with HF - Large pleural effusion - Elderly, confused or anxious pts - Rib fx - Surgical wound or healing tissue - Pulmonary embolism - Untreated pneumothorax
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What is Trendelenberg?
Head lower than torso and legs
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Tredelenberg position is CI in adults for...
- Pt in whom inc intracranial pressure is to be avoided - Uncontrolled hypertension - Distended abdomen - Esophageal surgery or GERD (acid reflux) - Uncontrolled airway at risk for aspiration ** We can do postural drainage but as to lie flat
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Percussions
- Traditional secretion mobilization technique which uses rhythmical force of a therapist or caregivers' hands in a cupped position against the thorax of a pt - Percussions are performed over specific lung segments believed to have inc secretion retention with the aim of loosening or dislodging the bronchial secretions from the airways so that they may be expelled through the central airways via coughing or suctioning - In preparation for percussion, a thin towel or hospital gown should cover the area of skin to be percussed for comfort and to avoid erythema - Sound should be HOLLOW, slapping sound indicate that your hand positioning may not be in the correct cupped position. This may cause erythema and discomfort for the pt - Percussion should be rhythmic with equal force applied by both hands - Avoid bony prominences (scapula, spine, clavicula, ribs), breast tissue and directly over the heart - The pt face and SpO2 should be monitored during tx - Pt may perform self-percussion on areas that can be reached comfortably - Mechanical percussion devices may be used to dec caregiver strain or pt strain when performing percussions
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CI for percussions
- Severe osteoporosis - Rib fx - Pulmonary embolism - Pneumothorax - Anticoagulation therapy - Malignancy - Burns/skin grafts - Open wounds - Inc intracranial pressure - Subcutaneous emphysema - GI bleeding
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Vibrations
- Traditional secretion mobilization technique in which a vibratory force is applied while applying pressure over a pt chest wall over the involved lung segment - Performed only on exhalation - It is proposed that vibrations enhance mucociliary transport from periphery to central airways - Mechanical vibration devices may be used to dec caregiver strain or pt strain when performing vibration For baby we can use a electric tooth brush
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CI for vibration
- Severe osteoporosis - Rib fx - Pulmonary embolism - Pneumothorax - Anticoagulation therapy - Malignancy - Burns/skin grafts - Open wounds - Inc intracranial pressure - Subcutaneous emphysema - GI bleeding
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PEP device
- +ve expiratory flow device are hand-held devices that create back pressure to splint airways while exhaling through the device - Back pressure created by PEP device allow for air to pass through inter-alveolar connections with pressure to dislodge or move mucous proximally - Can be used with aerosolized medication - Performed longer or equal to 15 min, 2-3x/day
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What are the types of vibrations?
- Coarse - Fine
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Coarse vibrations
Large amplitude, low frequency force (2Hz)
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Fine vibrations
Low amplitude, high frequency force (12-20Hz) Usually better tolerated
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What are the different PEP devices?
- Low pressure PEP - High pressure PEP - Non-Oscillating PEP devices - Oscillating PEP devices - Flutter
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Low pressure PEP
- 10-20 cm H2O - More commonly used - Provides equal effectiveness as high-pressure PEP, but with a lower presumed risk of pneumothorax
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High pressure PEP
- 50-120 cm H2O - PEP mask is used - Less commonly used due to inc risk of pneumothorax
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Non-Oscillating PEP devices
TheraPEP, Threshold PEP - Smooth flow. Creates a back pressure in a similar way as pursed lip breathing.
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Oscillating PEP devices
Acapella, Flutter, Cornet, Quake, Bubble PEP - Provides accelerated expiratory flow rates and interrupts airflow through oscillation of airways which loosens secretions and helps move them centrally
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Flutter
Plastic pipe with a steel ball and perforated cover - Moving ball end up: Inc pressure - Moving ball end down: Dec pressure * Only device which is position dependent
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What are the different independent breathing techniques?
- Active cycle of breathing technique (ACBT) - Autogenic drainage (AD)
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Active cycle of breathing technique (ACBT)
- Used to help clear bronchial sections using 3 ventilatory phases: Breathing control (BC), thoracic expansion exercises (TEE), and forced expiratory techniques (FET) Repeated cycles of 3 ventilatory phases: 1. Breathing control: Gentle tidal volume breathing (relaxed upper chest and shoulders) - Breathing normal 2. Thoracic expansion exercises: Deep inspiration (deep diaphragmatic breaths). This phase loosens the secretions. May be accompanied by percussion or vibration. 3. Forced expiration technique: 1 or 2 huffs or coughs.
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Autogenic drainage (AD)
- Is performed using varying expiratory flow at various lung volumes to help mobilize secretions from peripheral airways into central airways to be expelled through huffing or coughing - 3 phases: Unstick, Collect, Evacuate - Breathing is always done through the nose using diaphragmatic breathing - At the end of one cycle perform 2-3 huffs/coughs followed by deep breathing to prevent collapse of airways - Tx time 30-45 min/session once a day
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Advantage of PEP devices
- Easy to use - Dec risk of bronchospasm - Easy to learn - Accessible - Promote independent - Doesn't require a professional
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Disadvantage of PEP device
- Need to carry device - Social stigma Afraid to draw attention or to everybody now
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Advantage of independent breathing techniques
- No dec in O2/no desaturation - Don't need equipment - Can do anywhere at anytime
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Disadvantage of independent breathing techniques active cycle of breathing technique
- Take time (10-30 min) - Some pt prefer PT Tx
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Unstick
Exhaling to low volumes (ERV) will mobilize mucus (5-6 reps, 3 sec hold)
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Collect
Mucus will collect when breathing in mid lung volumes (5-6 reps, 3 sec hold)
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Evacuate
Evacuation of mucus occurs when breathing into larger lung volumes (5-6 reps, 3 sec hold). May perform expectoration via huff or cough at end
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Disadvantage of independent breathing techniques for autogenic drainage
- A lot of concentration - Cognitive demand high - Not efficacy for children - Some pt prefer passive tx - Require good proprioception, tactile skills
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Suctioning
A procedure to remove secretions through insertion of catheter or device via the nasopharynx, oropharynx or an artificial airway General indications: - To remove secretions or stimulate cough when pt is unable to do so independently - Suctioning should be performed when clinically indicated, not routinely (visible secretion in airways, chest auscultation findings - crackles, pt describe feeling some secretion, +ve tactile fremitus, SOB, ABG's - hypoxemia or hypercarbia, chest rx)
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Port of entry for suctioning
- Nose/mouth - Nose/mouth with artificial airway - Endotracheal tube or tracheostomy tube
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CI for suctioning
- Severe dec O2 saturation less 92% - Inc ICP - Hemoptysis - Malignant arrhythmia - Hyperinflation post-CABG and head injury
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Potential complication for suctioning
- Infection - Mucosal (tracheal and bronchial) trauma - Hypoxia/hypoxemia - Hemodynamic instability - Laryngospasm/bronchospasm - Atelectasis - Pneumothorax - Inc ICP - Pain - Anxiety
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Methods to minimize complication of suctioning
- Infection control measures (Sterile environment/equipment, handwashing, PPE) - Hyperoxygenation - Hyperinflation - Limit suctions time (no greater than 10-15 sec per pass) and allow recovery time between each pass (30 sec between each pass) - Meds and sedation prior procedure
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Ax of outcome - suctioning
- Improved breath sounds - Removal of secretions - Improved blood gas (ABG's) data or pulse oximetry (SpO2) - Dec work of breathing (dec RR or dyspnea)
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What are the forced expiratory techniques?
1) Cough 2) Huff 3) Assisted cough 4) Cough assisted machine
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Forced expiratory techniques - Cough
A cough is a forced expiratory technique with a closed glottis that may be used to help expel secretions 4 stages of coughing: 1. Inspiration - Adequate inspiratory volumes for a cough are approx. 60% of predicted VC 2. Glottal closure 3. Compression - Active contraction of abdominal and intercostal muscles causing an inc intrathoracic pressure distal to the glottis 4. Expulsion - Opening of the glottis and forceful expulsion of air Cueing = Strong double barrel cough (2 cough) Closed glottis = Cough Open glottis = Huff
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Forced expiratory techniques - Huff
- Is a forced expiratory technique with an open glottis that may assist to mobilize and clear secretions from airways - A deep breath is followed by a forced exhalation with mouth and glottis open - Huffing is preferred to coughing in pt with obstructive lung diseases due to the risk of small airway collapse from the high intra-thoracic pressure created with coughing (Regression of a cough) Huff from mid to low lung volumes = Clearing peripheral airways Huff from mid to high lung volume = Clearing proximal airways Cue = Imagine you are trying to fog up a pair of glasses
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What are the assisted cough techniques?
- Costophrenic assist - Heimlich - Type assist (abdominal thrust)
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Costophrenic assist
- Therapists hands are placed on the costophrenic angles of the rib cage - Therapist times pt's next exhalation and applies a quick manual stretch down and towards pt's navel. This quick stretch facilitates a stronger contraction of the diaphragm and intercostals for the inhalation which follow - The pt holds their breath for 3 sec and then coughs maximally with the therapist provide a sustained pull pressure on the costophrenic angle
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Heimlich - Type assist (abdominal thrust)
- Therapist places the heel of their hand over the epigastric area (underneath xiphoid process). Avoid the xiphoid process. - The pt is instructed to take a deep breath in and hold for 3 sec. - The pt is instructed to cough on the count of 3 and the therapist provides a quick pressure up and in (J stroke) as in a heimlich maneuver. ** CI for those with GERD or recent abdominal surgery **
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Cough assist machine
- Stimulates a cough and pulling of sections from the airways through alternating =ve and -ve pressures. - Is used with a mask, mouthpiece or tracheostomy adaptor. - +ve pressure created by the machine delivers a large volume of air on inspiration - The machine creates a rapid reversal to -ve pressure, which stimulates a cough