Cardiology Flashcards

(71 cards)

1
Q

murmur of mitral stenosis

A

low-pitched, rumbling, diastolic with loud S1 and opening snap

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2
Q

causes of mitral stenosis

A

rheumatic fever, atrial myxoma

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3
Q

murmur of mitral valve regurgitation

A

loud holosystolic murmur best heard at the apex that radiates to the base

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4
Q

murmur of mitral valve prolapse

A

early to midsystolic click with with late systolic murmur best heard at left lateral heart border

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5
Q

causes of acute mitral regurgitation

A

endocarditis, AMI, trauma

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6
Q

murmur of aortic stenosis

A

crescendo-decrescendo systolic murmur radiating to the neck

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7
Q

causes of aortic stenosis

A

calcific valve degeneration, bicuspid aortic valve

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8
Q

murmur of aortic regurgitation

A

high-pitched, blowing diastolic murmur best heard at left sternal border, decreases with valvsalva

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9
Q

causes of acute aortic regurgitation

A

endocarditis, aortic dissection

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10
Q

hypertrophic cardiomyopathy murmur

A

harsh systolic ejection murmur that increases with standing/Valsalva and decreases with squatting and handgrip

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11
Q

signs associated with aortic regurgitation

A

water hammer pulse, de Musset sign (head bobbing with pulse), Quincke sign (pulsating nailbed), wide pulse pressure, Muller sign (visible pulsations of uvula)

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12
Q

symptoms of mitral valve prolapse

A

asymptomatic or may have dyspnea, palpitations, nonexertional chest pain, fatigue

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13
Q

most common cause of aortic regurgitation

A

endocarditis

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14
Q

symptoms of aortic regurgitation

A

exertional dyspnea, angina, symptoms of heart failure

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15
Q

what is austin flint murmur

A

late diastolic murmur best heard at apex.

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16
Q

high intensity statins

A

atorvastatin 40-80 mg
rosuvastatin 20-40 mg

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17
Q

moderate intensity statins

A

lovastatin 40-80 mg, pravastatin 40-80 mg, simvastatin 20-40 mg, atorvastatin 10-20 mg, rosuvastatin 5-10 mg

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18
Q

low-intensity statins

A

pravastatin 10-20 mg, lovastatin 20 mg, fluvostatin 20-40 mg, simvastatin 10 mg

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19
Q

most common type of cardiomyopathy

A

dilated

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20
Q

most common cause of dilated cardiomyopathy (NOT in the setting of CAD)

A

idiopathic

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21
Q

most common infectious causes of dilated cardiomyopathy

A

Viruses (especially enteroviruses, coxsackie virus)

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22
Q

toxic causes of dilated cardiomyopathy

A

ETOH abuse, cocaine, anthracyclines (doxorubicin), radiation

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23
Q

metabolic causes of dilated cardiomyopathy

A

thyroid disorders, thiamine deficiency

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24
Q

less common infectious causes of dilated cardiomyopathy

A

postviral myocarditis, HIV, Lyme disease, Parvovirus B19, Chagas disease

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25
echocardiogram findings of dilated cardiomyopathy
left ventricular dilation, thin ventricular walls, decreased ejection fraction, ventricular hypokinesis
26
hallmark PE finding for dilated cardiomyopathy
S3 gallop
27
Cause of S3 gallop
filling of a dilated ventricle
28
Agents for mortality reduction in dilated cardiomyopathy
ACE inhibitors/ARBs, beta blockers
29
when to use AICD in dilated cardiomyopathy
EF<30-35%
30
Normal axis
predominantly positive QRS in lead I and aVF (unless it is also predominantly negative in lead II, in which case it is left axis deviation)
31
Left axis deviation
predominantly positive QRS in lead I and negative in aVF OR predominantly positive QRS positive in lead I and in lead aVF and predominately negative in lead II
32
right axis deviation
QRS is mostly negative in lead I and positive in lead aVF
33
normal PR interval
0.12-0.2 seconds (3-5 boxes)
34
left atrial enlargement ECG
m-shaped P-wave in lead II >0.12 seconds, biphasic P in V1 with larger terminal component
35
right atrial enlargement ECG
tall P-wave in lead II >3mm, biphasic P in V1 with larger initial component
36
normal QRS width
<0.12 seconds
37
left BBB ECG
Wide QRS, broad/slurred R in V5/V6, deep S wave in V1, ST elevation in V1-V3
38
right BBB ECG
Wide QRS, RsR' in V1/V2, wide S wave in V6
39
right ventricular hypertrophy ECG
R>S in V1 or R>7 mm in height in V1
40
Left ventricular hypertrophy ECG
S in V1 + R in V5 (or V6) > 35 mm in men or 30 mm in women
41
definition of pathological Q waves
greater than 1 box in width or in depth
42
ECG findings for LAD occlusion
ST elevations/Q waves in leads V1-V4
43
ECG findings for proximal LAD occlusion only
ST elevations/Q waves in V1, V2
44
ECG findings for circumflex artery occlusion
ST elevations/Q waves in I, aVL, V5, V6
45
ECG findings for RCA occlusion
ST elevations/Q waves in II, III, aVF
46
ECG findings for mid LAD +/- circumflex occlusion
ST elevations/Q waves I, aVL, V4, V5, V6
47
portion of heart perfused by LAD
anterior wall
48
portion of heart perfused by proximal LAD
septum
49
portion of heart perfused by circumflex
lateral wall
50
portion of heart perfused by RCA (in most people)
inferior wall
51
ECG findings of posterior wall MI
ST depressions in V1-V2
52
What arteries perfuse posterior wall
RCA, circumflex
53
Normal QRS axis numbers
-30 to +90
54
causes of left axis deviation
LBBB, LVH, inferior MI, elevated diaphragm, L anterior hemiblock, WPW
55
causes of right axis deviation
right ventricular hypertrophy, lateral MI, COPD, left posterior hemiblock
56
preferred antiarrhythmic for WPW
Procainamide
57
1st line antiarrhythmic for atrial flutter or atrial fibrillation
Beta blocker or CCB
58
what type of CCBs are antiarrhythmics
non-dihydropyridines
59
hallmark of sinus arrhythmia
heart rate increases with inspiration and decreases with expiration
60
what is sick sinus syndrome
dysfunction of sinus node that leads to combination of sinus arrest with alternating paroxysms of atrial tachyarrhythmias and bradyarrhythmias
61
causes of sick sinus syndrome
sinus node fibrosis, older age, corrective cardiac surgery, medications, systemic diseases affecting the heart
62
definitive treatment of sick sinus syndrome
permanent pacemaker +/- AICD
63
1st degree AV block criteria
PR interval >0.2 seconds with all P waves followed by QRS Complexes
64
1st degree AV block treatment
often none needed, but can try atropine if symptomatic
65
what is a 2nd degree AV block, type I
AKA Wenkebach or Mobitz I, progressive PR interval lengthening followed by a dropped QRS
66
2nd degree AV block, type I causes
high vagal tone, inferior wall MI, AV node blocking agents (BBs, CCBs), hyperkalemia
67
what is 2nd degree AV block, type II
interruption of electrical impulses resulting in occasional non-conducted impulses (constant PR interval except for the dropped QRS complexes)
68
block location in Mobitz I
commonly above the bundle of His
69
block location in Mobitz II
commonly at the bundle of His
70
causes of Mobitz II
rarely seen in Pts without structural heart disease (AMI, myocardial fibrosis, etc.)
71
what is 3rd degree AV block
total AV dissociation (no atrial impulses reach the ventricles)