Cardiology Flashcards
Pulse
โ
Character of the pulse Determined by
1. Stroke volume
2. Arterial compliance
โ
Pulse is best assessed in
Major arteries such as radial and Carotid
โ
Bounding pulse
+AR
+Anaemina
+Sepsis
โ
Slow rising pulse
+AS
JVP
โ
Height of the JVP is determined by right atrial pressure
+ elevated in rt atrial pressure
+ reduced in hypovolemia
โ
a wave - atrial systole
+ Absent: AF
+ Giant:
โTS
โPS
โ Pulmonary hypertension
โ Right heart failure
+ Irregular canon โaโ wave: 3ยฐ heart block
+ regular canon โaโ wave:
โ VT
โ
v waves- ventricular systole
+ Giant v wave : TR
โ x descent - atrial relaxation & apical displacement of tricuspid valve ring
โ
y descent - atrial emptying early in diastole
+Prominent & deep y descent: constrictive pericarditis
+Absent or slow Y descent: Cardiac tamponade
โ
Paradoxical JVP
+ Constructive pericarditis
+ Cardiac tamponade
+ Pericardial effusion
Annulus Fibrosus
โ
Separate atria and ventricles
โ
Forms the skeleton for AV valves
โ
Electrically insulate atria and ventricle / Forms a conduction barrier between atria and ventricle
โ
Prevent conduction of transmission except av node
Cardiac Silhouette
On left is formed by
โ
aortic arch
โ
pulmonary trunk
โ
left atrial appendage
โ
LV
On right
โ
RA
โ
RV
โ
Superior and inferior vena cava
๐๐ผ๐ฟ๐ผ๐ป๐ฎ๐ฟ๐ ๐ฐ๐ถ๐ฟ๐ฐ๐๐น๐ฎ๐๐ถ๐ผ๐ป
๐๐ผ๐ฟ๐ผ๐ป๐ฎ๐ฟ๐ ๐ฐ๐ถ๐ฟ๐ฐ๐๐น๐ฎ๐๐ถ๐ผ๐ป
Lt main coronary artery
+ LAD :
โ ๐๐ป๐๐ฒ๐ฟ๐ถ๐ผ๐ฟ ๐ถ๐ป๐๐ฒ๐ฟ๐๐ฒ๐ป๐๐ฟ๐ถ๐ฐ๐๐น๐ฎ๐ฟ ๐ด๐ฟ๐ผ๐ผ๐๐ฒ
โ ๐ฆ๐๐ฝ๐ฝ๐น๐ถ๐ฒ๐ ๐ฎ๐ป๐๐ฒ๐ฟ๐ถ๐ผ๐ฟ ๐ฝ๐ฎ๐ฟ๐ ๐ผ๐ณ ๐๐ฒ๐ฝ๐๐๐บ & ๐ฎ๐ป๐๐ฒ๐ฟ๐ถ๐ผ๐ฟ, ๐น๐ฎ๐๐ฒ๐ฟ๐ฎ๐น & ๐ฎ๐ฝ๐ถ๐ฐ๐ฎ๐น ๐๐ฎ๐น๐น๐ ๐ผ๐ณ ๐๐ฉ
+ Lt circumflex artery :
โ ๐ฃ๐ผ๐๐๐ฒ๐ฟ๐ถ๐ผ๐ฟ๐น๐ ๐ถ๐ป ๐๐ฉ ๐ด๐ฟ๐ผ๐ผ๐๐ฒ
โ ๐ฆ๐๐ฝ๐ฝ๐น๐ถ๐ฒ๐ ๐น๐ฎ๐๐ฒ๐ฟ๐ฎ๐น, ๐ฝ๐ผ๐๐๐ฒ๐ฟ๐ถ๐ผ๐ฟ & ๐ถ๐ป๐ณ๐ฒ๐ฟ๐ถ๐ผ๐ฟ ๐๐ฒ๐ด๐บ๐ฒ๐ป๐๐ ๐ผ๐ณ ๐๐ฉ
*** ๐๐๐๐ก๐ช๐จ๐๐ค๐ฃ ๐ค๐ ๐ก๐๐๐ฉ ๐ข๐๐๐ฃ ๐๐ค๐ง๐ค๐ฃ๐๐ง๐ฎ ๐๐ง๐ฉ๐๐ง๐ฎ ๐๐จ ๐ช๐จ๐ช๐๐ก๐ก๐ฎ ๐๐๐ฉ๐๐ก
Right coronary artery
+ ๐ฅ๐๐ป๐ ๐ถ๐ป ๐ฟ๐ถ๐ด๐ต๐ ๐๐ฉ ๐ด๐ฟ๐ผ๐ผ๐๐ฒ
+ ๐ฆ๐๐ฝ๐ฝ๐น๐ถ๐ฒ๐ ๐ฅ๐, ๐ฅ๐ฉ & ๐ถ๐ป๐ณ๐ฒ๐ฟ๐ผ๐ฝ๐ผ๐๐๐ฒ๐ฟ๐ถ๐ผ๐ฟ ๐ฎ๐๐ฝ๐ฒ๐ฐ๐๐ ๐ผ๐ณ ๐๐ฉ, ๐ฆ๐ ๐ป๐ผ๐ฑ๐ฒ (๐ฒ๐ฌ%) & ๐๐ฉ ( ๐ต๐ฌ%)
+ ๐๐ง๐ค๐ญ๐๐ข๐๐ก ๐ค๐๐๐ก๐ช๐จ๐๐ค๐ฃ ๐ค๐ ๐๐พ๐ผ ๐ฉ๐๐๐ง๐๐๐ค๐ง๐ ๐ค๐๐ฉ๐๐ฃ ๐ง๐๐จ๐ช๐ก๐ฉ๐จ ๐๐ฃ ๐จ๐๐ฃ๐ช๐จ ๐๐ง๐๐๐ฎ๐๐๐ง๐๐๐ & ๐ผ๐ ๐ฃ๐ค๐๐๐ก ๐๐ก๐ค๐๐ .
*** Posterior descending artery
+ ๐ฅ๐๐ป๐ ๐ถ๐ป ๐ฝ๐ผ๐๐๐ฒ๐ฟ๐ถ๐ผ๐ฟ ๐ถ๐ป๐๐ฒ๐ฟ๐๐ฒ๐ป๐๐ฟ๐ถ๐ฐ๐๐น๐ฎ๐ฟ ๐ด๐ฟ๐ผ๐ผ๐๐ฒ
+ ๐ฆ๐๐ฝ๐ฝ๐น๐ถ๐ฒ๐ ๐ถ๐ป๐ณ๐ฒ๐ฟ๐ถ๐ผ๐ฟ ๐ฝ๐ฎ๐ฟ๐ ๐ผ๐ณ ๐ถ๐ป๐๐ฒ๐ฟ๐๐ฒ๐ป๐๐ฟ๐ถ๐ฐ๐๐น๐ฎ๐ฟ ๐๐ฒ๐ฝ๐๐๐บ
+ ๐ง๐ต๐ถ๐ ๐ถ๐ ๐ฎ ๐ฏ๐ฟ๐ฎ๐ป๐ฐ๐ต ๐ผ๐ณ ๐ฅ๐๐ ๐ถ๐ป ๐ฎ๐ฝ๐ฝ๐ฟ๐ผ๐
๐ถ๐บ๐ฎ๐๐ฒ๐น๐ ๐ต๐ฌ% ๐ผ๐ณ ๐ฝ๐ฒ๐ผ๐ฝ๐น๐ฒ (dominant right system) & is
supplied by CX in the remainder (dominant left system).
Depolarization starts in?
SA node
- Situated at the junction of SVC and RA
- Rate is Influenced by ANS
Nerve Supply of heart
- Effects of sympathetic activity
+ ฮฒ๐ญ-๐ฎ๐ฑ๐ฟ๐ฒ๐ป๐ผ๐ฐ๐ฒ๐ฝ๐๐ผ๐ฟ๐ ๐ถ๐ป ๐ต๐ฒ๐ฎ๐ฟ๐ ๐ฟ๐ฒ๐๐๐น๐๐: ๐ฃ๐ผ๐๐ถ๐๐ถ๐๐ฒ ๐ถ๐ป๐ผ๐๐ฟ๐ผ๐ฝ๐ถ๐ฐ & ๐ฐ๐ต๐ฟ๐ผ๐ป๐ผ๐๐ฟ๐ผ๐ฝ๐ถ๐ฐ ๐ฒ๐ณ๐ณ๐ฒ๐ฐ๐๐,
+ ฮฒ๐ฎ-๐ฎ๐ฑ๐ฟ๐ฒ๐ป๐ผ๐ฐ๐ฒ๐ฝ๐๐ผ๐ฟ๐ ๐ถ๐ป ๐๐ฎ๐๐ฐ๐๐น๐ฎ๐ฟ ๐๐บ๐ผ๐ผ๐๐ต ๐บ๐๐๐ฐ๐น๐ฒ : ๐ฉ๐ฎ๐๐ผ๐ฑ๐ถ๐น๐ฎ๐๐ฎ๐๐ถ๐ผ๐ป - Parasympathetic
+ Pre- Preganglionic & sensory fibers reach the heart through vagus nerves
+ Cholinergic nerves supply AV & SA nodes via muscarinic (M2) receptors
+ Under resting conditions, vagal inhibitory activity predominates & heart rate is slow
The basic unit of contraction
Sarcomere
Cardiac peptide
***ANP
+ ๐ฉ๐ฎ๐๐ผ๐ฑ๐ถ๐น๐ฎ๐๐ผ๐ฟ๐: ๐ฅ๐ฒ๐ฑ๐๐ฐ๐ฒ ๐ฏ๐น๐ผ๐ผ๐ฑ ๐ฝ๐ฟ๐ฒ๐๐๐๐ฟ๐ฒ (๐๐ฃ)
+ ๐๐ถ๐๐ฟ๐ฒ๐๐ถ๐ฐ: ๐ฅ๐ฒ๐ป๐ฎ๐น ๐ฒ๐
๐ฐ๐ฟ๐ฒ๐๐ถ๐ผ๐ป ๐ผ๐ณ ๐๐ฎ๐๐ฒ๐ฟ & ๐ก๐ฎ
+ ๐ฅ๐ฒ๐น๐ฒ๐ฎ๐๐ฒ๐ฑ ๐ฏ๐ ๐๐๐ฟ๐ถ๐ฎ๐น ๐บ๐๐ผ๐ฐ๐๐๐ฒ๐ in response to stretch
*** BNP
+ Produced by ๐๐ฒ๐ป๐๐ฟ๐ถ๐ฐ๐๐น๐ฎ๐ฟ ๐ฐ๐ฎ๐ฟ๐ฑ๐ถ๐ผ๐บ๐๐ผ๐ฐ๐๐๐ฒ๐ in response to stretch (Ex - heart failure)
+ ๐๐ฎ๐ ๐ฑ๐ถ๐๐ฟ๐ฒ๐๐ถ๐ฐ ๐ฝ๐ฟ๐ผ๐ฝ๐ฒ๐ฟ๐๐ถ๐ฒ๐.
*** Neprilysin
+ ๐๐ป๐๐๐บ๐ฒ ๐ฝ๐ฟ๐ผ๐ฑ๐๐ฐ๐ฒ๐ฑ ๐ฏ๐ ๐ธ๐ถ๐ฑ๐ป๐ฒ๐ & ๐ผ๐๐ต๐ฒ๐ฟ ๐๐ถ๐๐๐๐ฒ๐
+ ๐๐ฟ๐ฒ๐ฎ๐ธ๐ ๐ฑ๐ผ๐๐ป ๐๐ก๐ฃ, ๐๐ก๐ฃ & ๐ผ๐๐ต๐ฒ๐ฟ ๐ฝ๐ฟ๐ผ๐๐ฒ๐ถ๐ป๐
+ ๐๐ฐ๐๐ ๐ฎ๐ ๐ฎ ๐๐ฎ๐๐ผ๐ฐ๐ผ๐ป๐๐๐ฟ๐ถ๐ฐ๐๐ผ๐ฟ
+ ๐ง๐ต๐ฒ๐ฟ๐ฎ๐ฝ๐ฒ๐๐๐ถ๐ฐ ๐๐ฎ๐ฟ๐ด๐ฒ๐ ๐ถ๐ป ๐ฝ๐ฎ๐๐ถ๐ฒ๐ป๐๐ ๐๐ถ๐๐ต ๐๐
importance of Windkessel effect of the central artery
๐ฃ๐ฟ๐ฒ๐๐ฒ๐ป๐๐ ๐ฒ๐ ๐ฐ๐ฒ๐๐๐ถ๐๐ฒ ๐ฟ๐ถ๐๐ฒ๐ ๐ถ๐ป ๐๐๐๐๐ผ๐น๐ถ๐ฐ ๐๐ฃ ๐๐ต๐ถ๐น๐ฒ ๐๐๐๐๐ฎ๐ถ๐ป๐ถ๐ป๐ด ๐ฑ๐ถ๐ฎ๐๐๐ผ๐น๐ถ๐ฐ ๐๐ฃ there by ๐ฅ๐ฒ๐ฑ๐๐ฐ๐ฒ๐ ๐ฐ๐ฎ๐ฟ๐ฑ๐ถ๐ฎ๐ฐ ๐ฎ๐ณ๐๐ฒ๐ฟ๐น๐ผ๐ฎ๐ฑ & ๐บ๐ฎ๐ถ๐ป๐๐ฎ๐ถ๐ป๐ ๐ฐ๐ผ๐ฟ๐ผ๐ป๐ฎ๐ฟ๐ ๐ฝ๐ฒ๐ฟ๐ณ๐๐๐ถ๐ผ๐ป
Substances released from endothelium
** Substances released from endothelium
Vasodilators
+ Nitric Oxide
+ Prostacyclin
+ Endothelium-derived hyperpolarising factor
Vasoconstrictors
+ Endothelin-1
+ Angiotensin II
Von Willebrand factor (glycoprotein) : Promotes thrombus formation
Tissue plasminogen activator : Induce fibrinolysis & thrombus dissolution
Pulsus paradoxus
*** Pulsus paradoxus (exaggerated / > 10 mmHg โ BP during inspiration)
+ ๐ฆ๐ฒ๐๐ฒ๐ฟ๐ฒ ๐ฎ๐๐๐ต๐บ๐ฎ ๐ผ๐ฟ ๐๐ข๐ฃ๐
+ ๐๐ต๐ฎ๐ฟ๐ฎ๐ฐ๐๐ฒ๐ฟ๐ถ๐๐๐ถ๐ฐ ๐ผ๐ณ ๐ฐ๐ฎ๐ฟ๐ฑ๐ถ๐ฎ๐ฐ ๐๐ฎ๐บ๐ฝ๐ผ๐ป๐ฎ๐ฑ๐ฒ
P wave
P wave
+ ๐๐๐ฟ๐ถ๐ฎ๐น ๐ฑ๐ฒ๐ฝ๐ผ๐น๐ฎ๐ฟ๐ถ๐๐ฎ๐๐ถ๐ผ๐ป
+ ๐๐ฏ๐๐ฒ๐ป๐ : ๐๐๐ฟ๐ถ๐ฎ๐น ๐ณ๐ถ๐ฏ๐ฟ๐ถ๐น๐น๐ฎ๐๐ถ๐ผ๐ป (๐ฆ๐๐)
+ ๐ง๐ฎ๐น๐น ๐ฃ : ๐ฅ๐ ๐ฎ๐๐ฟ๐ถ๐ฎ๐น ๐ฒ๐ป๐น๐ฎ๐ฟ๐ด๐ฒ๐บ๐ฒ๐ป๐ (๐ฃ ๐ฝ๐๐น๐บ๐ผ๐ป๐ฎ๐น๐ฒ)
+ ๐ก๐ผ๐๐ฐ๐ต๐ฒ๐ฑ ๐ฃ : ๐๐ ๐ฎ๐๐ฟ๐ถ๐ฎ๐น ๐ฒ๐ป๐น๐ฎ๐ฟ๐ด๐ฒ๐บ๐ฒ๐ป๐
(๐ฃ ๐บ๐ถ๐๐ฟ๐ฎ๐น๐ฒ)
PR interval
PR interval
+ ๐ฅ๐ฒ๐ณ๐น๐ฒ๐ฐ๐๐ ๐ฑ๐๐ฟ๐ฎ๐๐ถ๐ผ๐ป ๐ผ๐ณ ๐๐ฉ ๐ป๐ผ๐ฑ๐ฎ๐น ๐ฐ๐ผ๐ป๐ฑ๐๐ฐ๐๐ถ๐ผ๐ป
+ ๐ฃ๐ฟ๐ผ๐น๐ผ๐ป๐ด๐ฒ๐ฑ : ๐๐บ๐ฝ๐ฎ๐ถ๐ฟ๐ฒ๐ฑ ๐๐ฉ ๐ป๐ผ๐ฑ๐ฎ๐น ๐ฐ๐ผ๐ป๐ฑ๐๐ฐ๐๐ถ๐ผ๐ป
+ ๐ฆ๐ต๐ผ๐ฟ๐ : ๐ช๐ฃ๐ช ๐๐๐ป๐ฑ๐ฟ๐ผ๐บ๐ฒ (๐ฆ๐๐)
+ ๐๐ฒ๐ฝ๐ฟ๐ฒ๐๐๐ถ๐ผ๐ป : ๐ฆ๐ฝ๐ฒ๐ฐ๐ถ๐ณ๐ถ๐ฐ ๐ผ๐ณ ๐ฎ๐ฐ๐๐๐ฒ ๐ฝ๐ฒ๐ฟ๐ถ๐ฐ๐ฎ๐ฟ๐ฑ๐ถ๐๐ถ๐ (๐ฆ๐๐)
QRS complex
duration Increased : RBBB / LBBB
Increased QRS amplitude: Left ventricular hypertrophy
T wave
T wave
+ ๐ฉ๐ฒ๐ป๐๐ฟ๐ถ๐ฐ๐๐น๐ฎ๐ฟ ๐ฟ๐ฒ๐ฝ๐ผ๐น๐ฎ๐ฟ๐ถ๐๐ฎ๐๐ถ๐ผ๐ป
+ ๐ง๐ฎ๐น๐น, ๐ฝ๐ฒ๐ฎ๐ธ๐ฒ๐ฑ ๐ง : ๐๐๐ฝ๐ฒ๐ฟ๐ธ๐ฎ๐น๐ฒ๐บ๐ถ๐ฎ
+ T inversion: Ishchemia, Hypokalemia
ST segment
ST segment
+ ๐ช๐ถ๐ฑ๐ฒ๐๐ฝ๐ฟ๐ฒ๐ฎ๐ฑ ๐๐ฎ๐ฑ๐ฑ๐น๐ฒ ๐๐ฎ๐ต๐ฝ๐ฒ๐ฑ ๐ฒ๐น๐ฒ๐๐ฎ๐๐ถ๐ผ๐ป : ๐๐ฐ๐๐๐ฒ ๐ฝ๐ฒ๐ฟ๐ถ๐ฐ๐ฎ๐ฟ๐ฑ๐ถ๐๐ถ๐
+ ๐ฃ๐ฒ๐ฟ๐๐ถ๐๐๐ฒ๐ป๐ ๐ฆ๐ง ๐ฒ๐น๐ฒ๐๐ฎ๐๐ถ๐ผ๐ป : ๐๐ ๐๐ฒ๐ป๐๐ฟ๐ถ๐ฐ๐๐น๐ฎ๐ฟ ๐ฎ๐ป๐ฒ๐๐ฟ๐๐๐บ (๐ฆ๐๐)
+ Elevation: Ischemia
+ Depression: Ischemia or Infarction
๐๐๐๐๐จ of ECG
๐๐๐๐๐จ
๐1 & ๐2»_space;> ๐๐
๐3 & ๐4»_space;> ๐๐ฃ๐ฉ๐๐ง๐ซ๐๐ฃ๐ฉ๐ง๐๐๐ช๐ก๐๐ง ๐จ๐๐ฅ๐ฉ๐ช๐ข
๐5 & ๐6 ๐ค๐ซ๐๐ง »_space;> ๐๐
Normal Cardiac axis
Between -30 and +90 Degree
Exercise ECG (positive, Contraindication)
considered positive if
+ Angina occurs
+ BP falls or fails to increase or
+ ST segment shifts > 1 mm
๐๐ฉ๐ง๐๐จ๐จ ๐ฉ๐๐จ๐ฉ๐๐ฃ๐ ๐๐จ ๐๐ค๐ฃ๐ฉ๐ง๐๐๐ฃ๐๐๐๐๐ฉ๐๐ ๐๐ฃ
+ ๐๐๐ช๐ฉ๐ ๐๐ค๐ง๐ค๐ฃ๐๐ง๐ฎ ๐จ๐ฎ๐ฃ๐๐ง๐ค๐ข๐
+ ๐๐๐๐ค๐ข๐ฅ๐๐ฃ๐จ๐๐ฉ๐๐ ๐๐๐๐ง๐ฉ ๐๐๐๐ก๐ช๐ง๐
+ ๐จ๐๐ซ๐๐ง๐ ๐๐ฎ๐ฅ๐๐ง๐ฉ๐๐ฃ๐จ๐๐ค๐ฃ
NT-proBNP
NT-proBNP
+ Measured in preference to BNP since it is more stable
+ Indications
โ ๐๐ถ๐ฎ๐ด๐ป๐ผ๐๐ถ๐ ๐ผ๐ณ ๐๐ฉ ๐ฑ๐๐๐ณ๐๐ป๐ฐ๐๐ถ๐ผ๐ป
โ ๐๐๐๐ฒ๐๐ ๐ฝ๐ฟ๐ผ๐ด๐ป๐ผ๐๐ถ๐ & ๐ฟ๐ฒ๐๐ฝ๐ผ๐ป๐๐ฒ ๐๐ผ ๐๐ต๐ฒ๐ฟ๐ฎ๐ฝ๐ ๐ถ๐ป ๐ฝ๐ฎ๐๐ถ๐ฒ๐ป๐๐ ๐๐ถ๐๐ต ๐ต๐ฒ๐ฎ๐ฟ๐ ๐ณ๐ฎ๐ถ๐น๐๐ฟ๐ฒ
What is the cornerstone of Dx of MI?
TROPONIN I
increased in
CARDIAC
* MI, Myocarditis
* Pulmonary embolism
* Pulmonary edema
* Cardiac surgery, trauma
* Tachyarrhythmia
* Aortic dissection
NON CARDIAC
* Septic shock, Stroke,SAH
* Prolonged hypotension
* ESRD
* Burn
What are the baseline investigation for a patient with chest pain on exertion
- CBC / FBC
- FBS
- Lipid profile
- TFT
- ECG
What is the first line test of choice to diagnose angina due to coronary artery disease
CT Coronary angiography
Causes of sudden arrhythmic death
Coronary artery disease
- Myocardial Ischaemia
- AMI
- Prior myocardial infarction with myocardial scarring
Structural heart disease
- AS
- CHD
- Cardiomyopathy (hypertrophic, dilated, arrhythmogenic right ventricular)
Non structural heart disease
- Long QT Syndrome
- Brugada syndrome
- Wolff- Parkinson- White syndrome
- ADR (torsades de pointes)
- Severe electrolyte abnormality
Features of benign innocent heart murmur
Soft
Mid diastolic
Heard at left sternal border
No radiation
No other cardiac abnormality
First Heart Sound
Timing: Onset of systole
Cause: Due to the closure of Mitral and tricuspid valve
Nature: Usually single or narrowly splitting
Loud HS: Hyperdynamic circulation (anemia, Thyrotoxicosis, Pregnancy), MS
Soft HS: HF, MR
Second Heart Sound
Timing: End of the systole
Cause: The closure of Aortic and Pulmonary valve
Nature: Split on inspiration, Single on expiration
Features:
Fixed wide splitting- ASD
Wide but variable splitting- RBBB
Reversed Splitting- LBBB
Loud HS: HTN, ASD without P. HTN, Hyperdynamic state
Soft HS: AS
Third Heart Sound
Timing: Early in diastole Just after S2
Nature: Low pitched often heard as gallop (an early sign of LVF)
Origin: From ventricular wall
Causes: Physiological: Young People, Pregnancy
Pathological Cause: HF, MR
Fourth Heart Sound
End of diastole Just before S1
Ventricular Origin
Low Pitch
Absent In AF
Feature of severe LVF
Systolic Click
Early or Mid Systole
Brief High-intensity Sound
AS, PS, Floppy mitral valve
Prosthetic Heart
Opening Snap
Early in Diastole
Severe MS
Causes of Ejection Systolic Murmur
Aortic Stenosis
Pulmonary Stenosis
ASD
Aortic Or Pulmonary Flow Murmur
Benign Murmur
Pan Systolic Murmur
Mitral Regurgitation
Tricuspid Regurgitation
VSD
Late Systolic Murmur
Mitral Valve Prolapse
Early Diastolic Murmur
Aortic regurgitation
Pulmonary Regurgitation
Mid Diastolic murmur
MS
TS
Austin flint Murmur
Mitral Or tricuspid Flow murmur
Continuous murmur
PDA
Most common causes of right heart failure
Chronic lung disease
Pulmonary embolism
Pulmonary valvular stenosis
Biventricular heart failure occurs in
Dilated cardiomyopathy
Coronary heart disease affecting both ventricle
Cardiac Output is determined by
Preload
afterload
Myocardial contractility
Obstructive Cause of HF
Ventricular Outflow obstruction:
HTN, AS - LHF
P. HTN , PS - RHF
Ventricular Inflow Obstruction:
MS, TS
HF Due to Volume Overload
- Left ventricular volume overload: Aortic and Mitral regurgitation
- VSD
- Right ventricular volume Overload: ASD
- Increased Metabolic demand
Arrhythmitic Cause of HF
AF
Tachycardia
CHB
HF Due to Diastolic dysfunction
Constrictive Pericarditis
Restrictive Cardiomyopathy
Cardiac Tamponade
LVH and Fibrosis
HF Due Reduced Ventricular Contractility
MI
Myocarditis/ Cardiomyopathy
Causes of High Output failure
Large AV shunt
Beri-beri
Anemia
Thyrotoxicosis
Effect of prolonged sympathetic stimulation
Cardiac myocyte apoptosis
Cardiac Hypertrophy
Focal cardiac necrosis