Cardiovascular Flashcards

(51 cards)

1
Q

pulmonary circulation

A

right side of the heart, which carries blood from the heart to the lungs and back to the heart

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2
Q

systemic circulation

A

left side of the heart, which carries blood from the heart to the rest of the body and then back to the heart

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3
Q

atherosclerosis

A

cells and lipids accumulate within the tunica intima not on the artery walls

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4
Q

hypertension can lead to

A

hypertiensive heart disease

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5
Q

two basic types of coronary heart disease

A

chronic ischemic heart disease
acute coronary syndromes

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6
Q

chronic ischemic heart disease

A

stable angina
silent myocardial ischemia

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7
Q

acute coronary syndromes

A

unstable angina
acute myocardial infarction
sudden death
ventricular fibrillation

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8
Q

coronary heart disease medication side effects

A

xerostomia
calcium channel blockers (gingival hyperplasia)
antithrombotic treatment regimen: anticoagulant/antiplatelet therapy (excessive bleeding)

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9
Q

peripheral artery disease

A

atherosclerosis of the arteries in the lower extremities

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10
Q

dental implications peripheral artery disease

A

if taking anticoagulants, know the coagulation status
side effects of other meds
patient education
evaluate for perio disorders
possible referral

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11
Q

abdominal aortic aneurysm

A

weakness of the aorta causing an enlargement or bulging of the artery
aneurysm located in abdominal aorta

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12
Q

abdominal aortic aneurysm dental implications

A

side effects of medications
stress importance of maintaining healthy oral tissue
identification of individual at risk may prompt a medical referral

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13
Q

raynaud disease or phenomenon pathogenesis

A

ischemia caused by vasospasm in the vessels of the extremities and sometimes ears and nose
brought on by cold or emotional stress
associated with smoking

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14
Q

raynaud disease or phenomenon dental implications

A

refer for medical evaluation
side effect of medications

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15
Q

thromboembolic venous disorders

A

formation of blood clots
can break away and lodge anywhere
deep vein thrombosis (DVT)

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16
Q

dental implications of thromboembolic venous disorders

A

low dose anticoagulant therapy (no risk of blood loss)
stronger anticoagulants (increase the risk of uncontrolled bleeding)
medical consultation to determine coagulation status
local hemostatic measures

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17
Q

common blood thinners

A

warfarin
eliquis
xarelto
pradaxa
heparin

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18
Q

2 forms of stroke

A

ischemic - thrombus/embolus
intracranial hemorrhage - trauma, hypertension, aneurysm

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19
Q

pathogenesis of stroke

A

blood obstructed by growing thrombus, or by an embolus
tissues distal to obstruction become damage and necrotic
resulting pressure in cranium due to blood flow causes an interruption of blood flow to tissues

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20
Q

perioral and intraoral characteristics after stroke

A

unilateral weakness may affect: chewing, swallowing, ability to clear the mouth of residual food
uncontrollable drooling
favor chewing on unaffected side of mouth
food pocketing
motor impairment

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21
Q

dental implications of stroke fall into four categories

A

management of side effects of meds
management of post stroke physical effects
home care modifications
emergency management of stroke

22
Q

dental implications of stroke - management of side effects of meds

A

determine coagulation status
provide local hemostatic measures
avoid tissue trauma
xerostomia management
calcium channel blockers may cause gingival overgrowth

23
Q

knowledge of heart disorders will enable the dental professional to

A

recognize potential problems
modify the treatment to protect the patient and provide the safest possible therapy

24
Q

shunts cause

A

mixing of oxygenated and deoxygenated blood

25
ventricular septal defect
hole in septal wall between left and right ventricles causing mixing of blood higher risk for infective endocarditis, aortic valve prolapse and blood clots eventual congestive heart failure clubbing of fingertips fatigue, shortness of breath and tachycardia
26
finger clubbing is an important feature with
chronic hypoxia cyanosis
27
atrial septal defect
hole in the wall between the right and left atria cyanosis and hypoxia abnormal heart beats (dysrhythmias) clubbing of fingers higher risk for infective endocarditis and emboli
28
patent ductus arteriosus
ductus arteriosus does not close after birth premature babies heart murmur development of pulmonary hypertension higher risk for infective endocarditis and arteritis
29
pulmonary stenosis
narrowing of pulmonary valve caused by a fusion of the valve cusps forming a funnel when the valve opens instead of a wide opening if severe- will be difficult for the heart to pump blood into the lungs CHF may result
30
tetralogy of fallot (TOF)
4 defects: VSD, PD, overriding aorta, and right ventricular hypertrophy right to left shunting of blood, cyanosis and hypoxia shortness of breath polycythemia - abnormal increase in the number of red blood cells increased risk of infective endocarditis, emboli, brain abscesses, acute episodes of cyanosis during which seizures, loss of consciousness and sudden death are possible
31
coarctation of the aorta (COA)
constriction of the aorta BP and pulse in the upper body differs compared to the lower body lower volume of blood is able to get past constriction left ventricular hypertrophy, dizziness, headache, epistaxis weakness, pain, pallor and coldness in lower extremities arteritis at constriction site untreated - increase risk for stroke, ruptured aortic and cerebral aneurysms, infective endocarditis at the aortic valve
32
transposition of the great arteries (TGA)
aorta exits from right ventricle and pulmonary artery exits from left ventricle most times also septal defect if no septal defect to allow for mixing of blood then is fatal cyanosis and hypoxia
33
dental implications of congenital heart defects
presence of cyanosis should have medical investigation prior treatment modifications - prophylactic antibiotics supplemental oxygen stress reduction techniques caution administering local anesthetics patients ed - link of oral infection and bacterial endocarditis how adequate OH can decrease the risk of an autogenous infection
34
dental antibiotic prophylaxis
antibiotic given to a patient for prevention of harmful consequences of bacteremia that may be caused by oral flora
35
heart valve defects
narrowing of heart valves muscles of heart have to compensate insufficient heart valves do not close all the way, causing regurgitation muscles will compensate to try to push more blood through blood flow through valve is more turbulent causing more damage heart eventually weakens and congestive heart failure will result
36
two kinds of heart valve defects
benign/physiologic murmurs pathologic murmurs
37
pathologic murmurs
caused by valve defect stenotic valves - symptoms manifest when the heart is under stress shortness of breath, possible chest pains
38
benign/physiologic murmurs
mostly heard in small children grow out of, no treatment needed no risk for endocarditis
39
heart valve defects dental implication
daily home care could cause bacteremia bacteria within the blood to attach to endocardium no prophylactic antibiotics for physiologic heart murmurs anticoagulant therapy (xerostomia) individuals with valve replacement are at a high risk for bacterial endocarditis and thrombus formation (may be on prophylactic antibiotics, augmented antibiotic prior to, consultation with patient's physician)
40
mitral valve prolapse dental implications
the ADA no longer requires prophylactic antibiotic coverage with or without regurgitation education should be given to patients on the importance of decreasing the risk of creating a bacteremia that might result in infective endocarditis
41
rheumatic heart disease dental implications
patients may be on anticoagulants patients may be on daily antibiotics should be suspected for patients reporting a history of rheumatic fever (at risk for recurrence, which could cause more heart damage)
42
infective endocarditis etiology
bacteria (staphylococcus and streptococcus) gram negative bacillus, yeast, fungi bacteremia must be present can form in a healthy heart, but develops most often in a heart compromised
43
infective endocarditis peri/intraoral characteristics
petechiae may be found on any of the oral mucosal surfaces, especially soft palate and buccal mucosa
44
infective endocarditis extraoral characteristics
fever, chills, shortness of breath, could have low grade fever and flu like symptoms
45
dental implications of infective endocarditis
prophylactic antibiotics prior to dental treatment gingival and periodontal infections are the most common source of transient bacteremia identify persons at risk
46
congestive heart failure perioral and intraoral characteristics
cyanosis of the lips and oral tissues severe: right side jugular vein may become distended and can be observed when patient is in an upright position
47
dental implications of congestive heart failure
xerostomia because of medications for CHF modifying the chair to a semi-upright or upright might be indicated as well as supplemental oxygen to aid in breathing
48
dysrhythmias
abnormal heart rhythms or beats
49
tachycardia
fast heartbeat
50
bradycardia
slow heartbeat
51
dental implications of dysrhythmias
often able to identify abnormal heart rhythms refer to physician implanted pacemakers or defibrillators do not indicate the need for prophylactic antibiotic coverage careful using magnetostrictive devices around unipolar pacemakers bipolar and ICD