Cardiovascular Flashcards

(50 cards)

1
Q

side effects of procainamide

A

Hypotension. Other side effects include: myocardial depression, QRS/QT prolongation, V-fib, and torsade de
pointes.

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2
Q

side effects of lidocaine

A

Drowsiness, nausea, vertigo, confusion, ataxia, tinnitus, muscle twitching, respiratory depression, and psychosis.
ECG changes may be seen also.

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3
Q

dobutamine for CHF

A

Potent inotrope with some vasodilation activity, used when heart failure is not accompanied by severe hypotension.
Dobutamine decreases afterload and increases contractility.

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4
Q

dopamine for CHF

A

Vasoconstrictor and positive inotrope, is used to increase cardiac output, especially if shock is present.

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5
Q

tx for A flutter

A
Initiate A-V nodal blockade with -adrenergic or calcium channel blockers or with digoxin. If necessary, in a stable
patient, attempt chemical cardioversion with a class IA agent such as procainamide or quinidine after digitalization.
If such treatment fails, or if patient is unstable and requires immediate electrocardioversion, do so with 25–50 J.
Sedation should be considered prior to electrical cardioversion.
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6
Q

tx for A fib

A

If patient is stable then control a fast ventricular rate with diltiazem bolus and/or IV infusion; consider digitalis, and if indicated, convert with procainamide, quinidine, or verapamil. Synchronized cardioversion at 100–200 J in an unstable patient requiring cardioversion. In a stable patient with a-fib of unclear duration, anticoagulation for 2–3 weeks should be considered prior to chemical or electrical cardioversion in order to decrease the chance of an
embolic stroke or other embolic problem.

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7
Q

causes of A fib

A

Hypertension, rheumatic heart disease, pneumonia, thyrotoxicosis, and ischemic heart disease are common causes.
Pericarditis, ETOH intoxication, PE, CHF, and COPD are other causes.

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8
Q

causes of SVT

A
digitalis toxicity (25% of digitalis induced arrhythmias), pericarditis, MI, COPD, preexcitation
syndromes, mitral valve prolapse, rheumatic heart disease, pneumonia, drug and alcohol abuse.
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9
Q

most common cause of acute mesenteric icschemia

A

Arterial embolism 40–50%. Source is usually the heart, most often from a mural thrombus (recent MI often). Most
common point of obstruction is the superior mesenteric artery.

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10
Q

Buerger’s disease

A

thromboangiitis obliterans, an inflammatory, nonatheromatous occlusive condition
that causes segmental lesions and thrombus formation in medium and small arteries with less blood flow to the feet
and legs, usually in heavy smokers, males in their 20s and 30s; symptoms are usually claudication, pain, cold feet,
eventual redness or cyanosis of legs, may lead to gangrene and amputation

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11
Q

contraindications to beta blockers

A
CHF, variant angina, AV block, COPD, asthma (relative), bradycardia, hypotension, and insulin dependent
diabetes mellitus (IDDM). Also, patients with recent cocaine use should not receive -blockers.
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12
Q

most common symptom of acute pericarditis

A

Sharp or stabbing retrosternal or precordial chest pain, and the pain increases when supine and decreases when
sitting-up and leaning forward. Pain may be increased with movement and deep breaths. Other symptoms include
fever, dyspnea described as pain with inspiration, and dysphagia.

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13
Q

physical findings of acute pericarditis

A

Pericardial friction rub is the most common. Rub is best heard at the left sternal border or apex in a sitting leaning
forward position. Other findings include fever and tachycardia.

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14
Q

s/s PE

A
Tachypnea (92%)
Chest Pain (88%)
Dyspnea (84%)
Anxiety (59%)
Tachycardia (44%)
Fever (43%)
DVT (32%)
Hypotension (25%).
Syncope (13%)
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15
Q

mitral stenosis: cause and symptom

A

The most common cause is rheumatic heart disease. The most common initial symptom is dyspnea.

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16
Q

acute mitral regurgitation

A

Most common cause is rupture of the chordae tendineae, rupture of the papillary muscles, or perforation of the valve leaflets. Common causes include AMI and infectious endocarditis.

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17
Q

aortic stenosis

A

Most common causes are rheumatic heart disease and congenital bicuspid valve. S/s
are syncope, angina, and left heart failure. As the disease progresses, systolic BP decreases and pulse pressure narrows.

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18
Q

s/s acute aortic regurgitation

A

Dyspnea, tachycardia, tachypnea, and chest pain. Causes include: infectious endocarditis, acute rheumatic fever,
trauma, spontaneous rupture of valve leaflets, or aortic dissection.

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19
Q

s/s chronic aortic regurgitation

A

Bobbing of the head with systole, bounding carotid pulse (water-hammer), pistol shot sound, the to-and-fro
murmur of Duroziez’s sign over the femoral arteries, and capillary pulsation of the nailbeds (Quincke’s sign).

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20
Q

most common cause of tricuspid stenosis

A

Rheumatic heart disease.

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21
Q

classic physical sign of endocarditis

A

A loud regurgitant heart murmur or a murmur that has changed in intensity or type.

22
Q

what increases the risk for endocarditis

A

damaged or prosthetic heart valve

23
Q

Janeway lesions

A

Purple-colored circular flat rashes (macules) on the palms or soles, due to embolic pieces of clot and infected
thrombi that break free of endocarditis and float to the distal circulation in the feet and hands. Other signs include
petechiae of the skin and mucous membranes and splinter hemorrhages under the nails.

24
Q

meds used to lower BP in thoracic dissection

A

Sodium nitroprusside, propranolol, or labetalol. An arterial line should be considered to closely monitor the blood
pressure.

25
most common med to treat hypertensive emergencies
Sodium nitroprusside (not DOC for eclampsia). Sodium nitroprusside works through production of cGMP which relaxes smooth muscle. This results in decreased preload and afterload, decreased oxygen demand, slight increased heart rate with no change in myocardial blood flow, cardiac output, or renal blood flow. Duration of action is 1–2 minutes. Sometimes, ß-blockade is required to treat rebound tachycardia.
26
nitroprusside ADRs
Hypotension. Thiocyanate toxicity with blurred vision, tinnitus, change in mental status, muscle weakness, and seizures is seen more often in patients with renal failure and after prolonged infusions. Cyanide toxicity is uncommon, it may occur with hepatic dysfunction, after prolonged infusions, and in rates greater than 10 μg/kg per minute.
27
s/s acute aortic dissesction
BP differences between arms, cardiac tamponade, and aortic insufficiency murmur. An abnormal ECG may also be present.
28
peripheral cyanosis
BP differences between arms, cardiac tamponade, and aortic insufficiency murmur. An abnormal ECG may also be present.
29
causes of peripheral cyanosis
Cyanosis with a normal SaO2 can be due to: decreased cardiac output. redistribution—may be 2◦ to shock, DIC, hypothermia, vascular obstruction.
30
causes of central cyanosis
The causes of cyanosis with a decreased SaO2 are: decreased PaO2, or decreased O2 diffusion. hypoventilation. V-Q mismatch, pulmonary shunting. dysfunctional hemoglobin (includes sickle cell crisis, drug-induced hemoglobinopathies).
31
moderate doses of dobutamine
Decreased peripheral vascular resistance and pulmonary occlusive pressure, and positive inotropic stimulation of the heart.
32
best tx for verapamil induced bradycardia
Calcium chloride 10%, give 10–20 ml IV.
33
s/s pericardial tamponade
Triad of hypotension, elevated CVP, and tachycardia is usually indicative of either acute pericardial tamponade or a tension pneumothorax in a traumatized patient. Muffled heart tones may be auscultated. Echocardiography is urgently needed to differentiate and diagnosis.
34
most common site of thrombophlebitis
The deep muscles of the calves, particularly the soleus muscle.
35
2 most common causes of pulsus paradoxus
COPD and asthma
36
Beck's triad
Hypotension, elevated CVP (distended neck veins), and distant muffled heart sounds.
37
what does Beck's triad indicate
pericardial tamponade
38
s/s aortic stenosis
Exertional dyspnea, angina, and syncope. Narrowed pulse pressure with decreased SBP. Slow carotid upstroke. Prominent S4.
39
tx for torsade de pointes
Pacemaker cranked to 90–120 bpm to “overdrive” pace. Isoproterenol. Magnesium sulfate 2 g IV. The goal is to accelerate the heart rate and shorten ventricular repolarization.
40
meds contraindicated for torsade de pointes
A drug which prolongs repolarization (QT interval). For example, class Ia antiarrhythmics (quinidine, procainamide). Other drugs that share this effect include TCAs, disopyramide, and phenothiazines.
41
meds commonly associated w/ torsade de pointes
Type I-A antiarrhythmics—quinidine and procainamide. | These drugs lengthen the Q-T interval.
42
most common cause of multifocal atrial tach
COPD
43
tx for multifocal atrial tach
Treat underlying disorder. | Magnesium sulfate 2 g over 60 seconds (with supplemental potassium to maintain serum K+ above 4 mEq/l).
44
tx for verapamil-induced hypotension
Calcium gluconate 1 g IV over several minutes.
45
3 types of cardiomyopathy
Dilated or congestive (most common), hypertrophic (hypertrophied left ventricle is small, unable to relax and fill properly), and restrictive cardiomyopathy (rare; stiff ventricles).
46
causes of dilated cardiomyopathy
Infection, metabolic and immunologic disorders, chronic alcohol abuse, pregnancy and postpartum disorders, and coronary artery disease.
47
causes of R sided HF
Left-sided heart failure (from AMI, VSD, cardiomyopathy, constrictive pericarditis, increased circulating blood volume, aortic and mitral valve stenosis or insufficiency, and other causes).
48
assessment of CV system
* family hx * edema * CP * dyspnea * vertigo/syncope * weight gain * leg cramps/pain
49
life changes for pt w/ HTN
* weight loss * eat more fruits/veg * low Na diet * exercise * limit alcohol * stop smoking
50
risk factors for CAD
* HTN * smoking * high lipids * DM * family hx * lack of exercise * obesity * oral contraceptives * men > women when under 70 y.o.