Cardiovascular Flashcards

(64 cards)

1
Q

usual position for assessment JVP

A

Position the patient at 45 degrees.

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2
Q

Cachectic.
Causes

A

Definition:(severe loss of weight and muscle wasting.
Cause:
1- severe malignancy
2- Severe cardiac failure (cardiac cachectic).
- This resuit from:
1- Anorexia : due to congestive enlargement of the liver.
2- Impaired intestinal absorption : due to congestive intestinal
veins.

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3
Q

Most common causes of clubbing

A

Common:

CVS:
1- Cyanotic congenital heart disease.
2- Infective endocarditis.
Respiratory:
1 Lung carcinoma.
2 Chronic pulmonary suppuration: infecti..
- Bronchiectasis.
- Lung abscess.
- Empyema. T B
- Idiopathic pulmonary fibrosis.
2) Uncommon:
Respiratory:
1 Cystic fibrosis.
2 Asbestosis.
3 Pleural mesothelioma.
GIL:
1 Cirrhosis
Inflammatory bowel disease. 3- Celiac disease.
3) Rare:
1- Neurogenic diaphragmatic tumours. 2- Pregnancy.

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4
Q

Unilateral clubbing :

A

1- Bronchial arteriovenous aneurysm.
2- Axillary artery aneurysm.

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5
Q

Grades of clubbing:

A

1- Bogginess at the nail base.
2- Increased transverse diameter than the AP diameter.
3- Parrot beak appearance.
4- HPOA - Hypertrophic Pulmonary Osteo Arthropathy.

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6
Q

Splinter Hemorrhage:
• Causes:

A

• Definition: linear hemorrhages lying parellel to the long axis of the nail.
1- Trauma (mostly).
2- infective endocarditis due to vasculitis in the nail bed.
3- Other rare causes:
(A) Vasculitis.
B) Rheumatoid arthritis.
C) Polyarteritis nodosa.
D) Sepsis.
E) Hematological malignancy.
F) Profound anemia.

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7
Q

Osler’s nodes:
• Definition:
• They are rare manifestation of

A

These are red raised tender nodules on the pulps of the
fingers or toes or in the thenar or hypothenar eminence.

infective endocarditis.

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8
Q

4) Janeway lesions :
• Definition:

A

Non tender erythromatous maculopopular lesions containing bacteria occur rarely in palms or pulps of the fingers in patient with infective endocarditis.

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9
Q

Tendon xanthomata :
• Definition:

Palmar xanthomata and tuboeruptive xanthomata.
• Occur in the

A

1-Yellow or orange deposits of lipid in tendons of the hand or arms which occur in type II hyperlipidemia.
2-elbow and knee and it’s character of type III
hyperlipidemia.

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10
Q

• Normal resting heart rate:

A

about 60 - 100 beats per min.
Bradycardia → HR< 60 beats/min.
Tachycardia → HR ≥ 100 beats/min.

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11
Q

Arterial- pulse

A

rate
rhytum (regular.or Not only
Volum (good Valum or low Valum) ( low in HF with low cardiac output, elderly patients, dehydration)
character
Symchronictry

Method: palpate the radial artery of the wrist. The pulse usually felt medial to the radius using forefinger and middle finger pulps of the
examining hand:
* For Brachial and corotid arteries use the thumbs.
* Brachial artery → medial to biceps muscle.
* Carotid artery → between the larynx and the anterior border of sternocleidomastoid muscle. NEVER palpates arotid arteries
simultaneously.

femoral pulse (below inguinal ligament 1/3 of the way up from pubic tubercle).

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12
Q
  • Radial-radial delay occurs in :
A

large arterial occlusion by an
atherosclerosis plaque or aneurysm. Subelavian/ brachial Artery

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13
Q

• Radiofemoral delay occurs in:

A

when there is coarctation of the
aorta.

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14
Q

Pulsus alternans : (rare)
Definition:
Occur in:

A

A regular alternate of strong and weak beats (in volume).
Mechanism: unknown.
Occur in: Severe myocardial disease and heart failure.

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15
Q

Pulsus paradoxus :
Definition:

A

A pulse that increases in volume on expiration and decreases in volume in inspiration. Note: This phenomenon is physiological and not detectable in normal individuals. It is considered abnormal if the pulse pressure decrease, on inspiration, below 10 mmHg.
Mechanism: During inspiration , the systolic and diastolic blood
pressure normally decrease (because intrathoracic pressure becomes more negative, blood pools in the pulmonary vessels, so left heart filling is reduced).
Occur in: constrictive pericarditis, pericardial effusion and severe asthma.

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16
Q

D) Character and volume :
• Method: Use the

A

brachial and carotid arteries’
‘They are more better in determining the character and volume than radial artery. But for collapsing pulse and
pulsus alternans, used the radial artery.

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17
Q

Collapsing (bounding) pulse :
Definition:
Occur in:

A

High volume but ill sustained pressure wave form.

Aortic regurgitation.

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18
Q

Bisferiens pulse: MODEST
Definition:
Occur in:

A

Slow rise, with normal or high volume, with sudden collapse.
Occur in: combined aortic stenosis and regurgitation.

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19
Q

Postural blood pressure :

A

• Take BP of the patient on lying and standing positions.
• Postural hypotension is considered when :
1 - Systolic fall more than 15 mmHg or
2- Diastolic fall more than 10 mmHg.

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20
Q

Corneal arcus:

A

Cause: Precipitation of cholesterol crystals at the periphery of the
cornea.
hyperlipidemn thypercakem

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21
Q

1) Jaundice

A

CHF, hepatic congestion or prosthetic heart valve’).

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22
Q

4) Mitral faces (Malar flush):
Occur in:

A

Appearance: Rosy cheeks with bluish tinge.
Cause: dilatation of malar capillaries.
Occur in:
1) pulmonary HTN.
2) low cardiac output as in mitral stenosis.

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23
Q

5) The mouth:
A) for any high arched palate. This occurs in

A

Marfan’s syndrome
which is associated with congestive heart disease.

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24
Q

The abdomen..
Physical signs to be noticed:
1) Enlarged tender liver :
2) Pulsatile liver ‹
3) Ascites :
4) Splenomegaly:
5) Implanted cardioverter-defibrillator.

A

1-in the presence of Right heart failure.
2) tricuspid regurgitation (because the right ventricular systolic pressure wave is transmitted to the hepatic veins).
3-may occur in severe right heart failure.
4-may indicates infective endocarditis.

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25
Carotid arteries : • Importance:
tell about the aorta and left ventricular function.
26
Jugular venous pressure (JVP) Meaning:Pulsation (which is seen in the internal jugular vein)
Meaning:Pulsation (which is seen in the internal jugular vein) sternal angle is taken as the Zero point and the maximum height of pulsation in the vein indicates the JVP - The center of the right atrium lies 5 cm below the zero point. • Importance: tell about right arterial and right ventricular function,
27
Positive Hepatojugular reflux ( sustain increase JVP)
Right heart failure
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Difference between carotid and jugular pulsation
Carotid 1) Rapid outward movement 2) One peak per heartbeat 3) Palpable 4) independent of respiration, position or abdominal pressure (no hepatojugular reflux). Jugular Rapid inward movement 2 peaks per heartbeat Impalpable Dependent of respiration (decreased during inspiration), position and abdominal pressure.
29
Causes of raised JVP
Right ventricular failure LVF Tricuspid stenosis or regurgitation Pericardial effusion or constrictive pericarditis Superior vena caval obstruction Fluid overload Hyperdynamic circulation Cor pulmonal Restrictive cardiomyopathy
30
JVP wave forms Normal
1- a wave: Coincides : with right atrial systole. Also coincides with Ist heart sound and precedes the carotid pulsation. -Cause: atrial contraction. 2- V wave: Coincides: with ventricular systole. Cause: atrial Filling in the period when the tricuspid valve remains closed during ventricular systole. There are 2 decents in normal JVP: 1-X decent: * Caused by atrial relaxation, 2- Y decent: * Caused by rapid ventricular filling when the tricuspid valve open •C wave: (not usually visible) Cause: a transmitted pulsation from the carotid artery.
31
JVP wave forms abnormal
Large v wave: Occur in tricuspid regurgitation. Cannon a wave: "occurs when the right atrium contract against the closed tricuspid valve (in complete heart block ) Gianta wave: These are large but not explosive a wave with each beat. It occurs when the right atrial pressure raised because of the: 1 Obstruction in the outflow (tricuspid stenosis). 2 Elevated pressure in pulmonary circulation.
32
an increased JVP during inspiration.)
Kussmaul's sign: Examination: This sign is best examined with the patient sitting up at 90° and breathing quietly through his mouth.
33
1-Increase of JVP 2-Kussmaul's sign 3-Absent a wave 4-Giant a wave 5-Giant v wave
1- Heart failure - SVC obstruction 2- Constrictive pericarditis * Cardiac tamponade * Right ventricular infarction 3-Atrial fibrillation 4-Tricuspid stenosis 5-Tricuspid regurgitation
34
The precordium.. Inspection :-
1) Scars. - These are previous cardiac operations. -The position of the scar can be a clue to the valve lesion that has been operated, e.g. A) Valve surgery (cardiopulmonary bypass) :(median sternotomy and usually hidden under the chest hair: B)Previous closed mitral valvotomy : left or even right sided lateral thoractomy scars. 2) Some skeletal abnormalities. A) Pectus excavatum (funnel chest) when the Broast deep mside; B)Kyphoscoliosis (curvature of the vertebral column) which may be part of Marfan's syndrome. These can cause distortion of position of the heart and great vessels in the chest and this alter the position of the apex beat. GEERTVEBI
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1) Apex beat must be palpable. • The position of apex beat is defined as
the most lateral and inferior point at which the palpating fingers are raised with each systole. NOTE: The normal position of apex beat is in the 5 left intercostal space 1 cm medial to the midclavicular line.
36
Types of abnormal apex beats: -
1) The pressure loaded (hyperdynamic or systolic overload) forcefull apex beat. Occurs with aortic stenosis or hypertension. 2) The volume loaded (hyperkinetic or diastolic overloaded) uncoordinated impulse. Occurs with left ventricular dysfunction. 3) The double impulse apex beat, where 2 distinct impulses are felt with each systole. Occurs with hypertrophic cardiomyopathy. 4) The tapping apex beat will be felt when the I st heart sound is actually palpable (heart sounds are NOT palpable in healthy people). Occur with mitral or rarely tricuspid stenosis.
37
Thrills :
• Definition: These are palpable murmurs. Cause: Turbulent blood flow of cardiac murmuls.
38
• Note: In some cases the apex beat may not be palpable due to:
1- Thick chest wal! 3- Pericardial effusion. 4- Shock (or death). 2- Emphysema. 5- Rarely to dextrocardia (where there is inversion of heart and great vessels) The apex beat will be palpable to the right of the sternum in many cases of dextrocardia.
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4) Heaves : • Definition: These are palpable impulses from either the right or left ventricle. • Note: A pulsation over the left parasternal area (right ventricular heave) is usually due to
right ventricular hypertropby (as of pulmonary hynertension)
40
The stethoscope 1 The bell: Designed for amplifying low pitched sounds. Examples : 2 The diaphragm: Designed for higher pitched sounds. Examples: Examination Sequence :
The stethoscope 1 The bell: Designed for amplifying low pitched sounds. Examples : 1- diastolic murmur of mitral stenosis. 2- 3rd heart sound. 2 The diaphragm: Designed for higher pitched sounds. Examples: 1 Systolic murmur of mitral regurgitation. 2 4th heart sound. Examination Sequence : 1) Mitral area with the bell and then by the diaphragm. 2) Tricuspid area with the diaphragm. 3) Pulmonary area with the diaphragm. 4) Aortic area with the diaphragm.
41
Physiological splitting
(the closure of the aortic valve earlier than the pulmonary valve) occurs because the pressure of systemic cigulation is higher than the pressure of pulmonary circulation. - This splitting is increased by inspiration (because increased venous return to the right ventricle which further delay pulmonary valve closure). - It disappears on expiration. - Heard as: lub d/dub (inspiration), lub dub (expiration).
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This aids in distinguishing b/w the Is and 2nd heart sounds.
Ist heart sound → precedes the pulse. 2nd heart sound → follows the pulse.
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Abnormalities of the heart sounds The cause: Forcefull closure of the valves. *Stenosis + Tachycardia loud sound. * Regurgitation + bradycardia → soft sound. Special consideration: 1st heart sound : Loud: Soft: 2nd heart sound : Loud: Soft:
1st heart sound : Loud: Any case of short AV conduction time (short P-R interval) , mitral stenosis and tachycardia . Soft: Any case of long AV conduction time (long P-R interval). _first degree heart block ( prorlonged diastolic filling time ) . _ left bundle branch block ( delayed onset of left ventricular systole ) _ mitral regurgitation .. 2nd heart sound : Loud: Systemic HTN (loud Az) or pulmonary HTN (loud Pz). Soft: Calcific aortic stenosis (the leaflet movement is reduced).
44
2nd heart sound Splitting.. 1) Increased normal splitting (wider on inspiration) : When there is any delay in right ventricular emptying. Examples:
A)Right bundle branch block (delayed right ventricular depolarization). B) Pulmonary stenosis (delayed right ventricular ejection). C) Ventricular septal defect (increased right ventricular volume). D)Mitral regurgitation (because of earlier aortic valve closure due to more rapid left ventricular emptying)
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2) Fixed splitting : i.e. There is no respiratory correlation. Example
Atrial septal defect where equalization of volume loads b/w the 2 atria occurs through the defect (atria acting as a common chamber).
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3) Reversed splitting : ile. P2 occurs first and splitting occurs in expiration. Examples:
A)Left bundle branch block (delayed left ventricular depolarization). B) Aortic stenosis (delayed left ventricular ejection). C)Large patent ductus arteriosus (increased left ventricular volume).
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_ 3d heart sound It is a low pitched mid-diastolic sound. -It leads to the impression of gallop rhythm resembling a galloping horse) which is a triple rhythm consists of 3 heart sounds. Cause: rapid filling of the ventricles after opening of AV valves. Usually pathological after the age of 40. -Cause: -Occurs in (physiologically):
Usually pathological after the age of 40. Cause: poor contracting ventricle (although diastolic filling is not especially rapid). Left ventricular S3: - Louder at the apex and on expiration. -Occurs in: 1- Left ventricular failure (an important sign). 2- Aortic or mitral regurgitation. 3 VSD. 4 Patent ductus arteriosus. Right ventricular S3: - Louder at the left sternaVedge. -Occurs in: 1- Right ventricular failure. 2- Constrictive pericarditis. Occurs in (physiologically): 1- Children 2- Young adults 3- pregnancy 4- Athletes. 5- Fever: Heard as: lub dub dum.
48
4th heart sound (never occur physiologically) -It is a low pitched presystolic sound. -It also leads to the impression of triple (gallop) rhythm. Cause: high pressure atrial wave reflected back from a poor compliant? ventricle. Heard as: da lub dub.
Left ventricular S4: occurs whenever left ventricular compliance is reduced as in: 1 Aortic stenosis 2Acute mitral regurgitation. 3- Systemic hypertension. 4 IHD (the sound may be the only physical sign in angina or MI). 5 Advanced age. Right ventricular S4: occurs whenever right ventricular compliance is reduced as in: 1- Pulmonary stenosis. 2- Pulmonary hypertension.
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Summation gallop -The combining of S3 and S4 to one sound. -It may occur if the….. Quadruple rhythm -When both S3 and S4 are present. -It indicates …….
heart rate is greater than 120/min. severe ventricular dysfunction.
50
A) Systolic murmurs (Occurs during ventricular systole) 1) Pansystolic: Occurs in 2) Mid systolic (ejection) murmur = crescendo decrescendo murmur Occurs in 3- Late systolic murmur :
Occur in:Mitral regurgitation,tricuspid regurgitation and VSD. 2-Aortic stenosis,pulmonary stenosis, Hypertrophic cardiomyopathy. 3- Character: It could be distinguished from midsystolic murmur by the presence of a gap between the Is heart sound and the murmur which then continues right up to the 2nd heart sound. Occur in: Mitral valve prolapse or papillary muscle dysfunction where mitral regurgitation begins in mid-systole.
51
Diastolic murmurs (occurs during ventricular diastole) 1) Early diastolic murmur: Character: A high pitched sound begin immediately with the 2nd heart sound and has a decrescendo quality. Cause: 2) Mid-diastolic murmur: Character: Begin later in diastole and may be short or extend right up to the Is heart sound.
1-Regurgitation through leaking aortic or pulmonary valves. Occur in: Aortic or pulmonary regurgitation. 2-Cause: Impaired flow during ventricular filling. Occur in: mitral or tricuspid stenosis and atrial myxoma (rarely occur, where the tumour obstructs the valve orifice). 3) Presystolic murmur: Character. These are extension of the mid-diastolic murmurs of the mitral or tricuspid stenosis and usually don't occur when atrial systole is lost in atrial fibrillation. Cause and Occurence: the same of mid-diastolic murmur:
52
5) Continuous murmuns: Character: Extend throughout systole and diastole.
Cause: the presence of communication between 2 parts of the circulation with a permenant pressure gradient so that blood flow occurs continuously. Occur in: Patent ductus arteriosus, arteriovenous fistula (coronary, pulmonary or systemic)
53
Pitting edema of the sacrum →
for signs of severe right cardiac ailure.
54
Pitting unilateral lower limb edema:
A) DVT: B) Tumour or lymph nods compressing a large vein. 3-cellulite 4-rupture of the cyst behind the knee
55
1- Pitting lower limb edema: (the skin is indented and only slowly refills).
A) Cardiac : CHF, constrictive pericarditis. B) Drugs : Calcium antagonists. C) Hepatic : cirrhosis. D) Renal : nephrotic syndrome E) GIT : Malabsorption, starvation.
56
3- Non-pitting lower limb edema:
A) Hypothyroidism. B) Lymphedema due to: i- Infection. ii- Malignancy. iii- Allergy iv- Milroy's disease (unexplained lymphedema which appears at puberty and is more common in females).
57
Peripheral vascular disease : Signs of this disease are:
1- Absent pulses. 2- Femoral systolic bruits. 3- Marked leg pallor. 4- Absence of hairs. 5- Cool skin. 6- Reduced capillary return (compress the toe nails - the return of the normal red colour is slow). Buerger's Test : (to confirm the presence of the disease) Elevate the leg to 45 degrees → a rapid appearance of pallor (indicates poor arterial supply). -Then place them dependent over the edge of the bed at 90 degrees → cyanosis occurs (indicates impaired arterial supply).
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Deep Venous Thrombosis (DVT) Physical signs to be examined
1) Swelling of the calf and the leg. 2)Dilated superficial veins. 3) Feel for increased warmth. 4)Squeeze the calf gently to determine if the area is tender.
59
Varicose veins malleolus). 3) Palpate the veins. A) Hard veins : suggest …….. B) Tenderness: suggest …… 4) Perform Cough impulse test Technique: Put the fingers over the long saphenous vein opening in the groin'. Ask the patient to cough, a fluid thrill is felt if the
A) Hard veins : suggest thrombosis. B) Tenderness: suggest thrombophlebitis saphenofemoral valve is incompetent.
60
Pericardial rub Definition• , Occurs in: Best heard in:
Definition: It is a characteristic physical sign or pericarditis which is described as superficial scratching sound with systolic and diastolic components. Occurs in: pericarditis. Best heard in: lower left sternal edge when the patient is sitting up and breathing out using the diaphragm.
61
Prosthetic (mechanical) heart sound
It is a high pitched "metallic" sound, commonly palpable and often audible without a stethoscope. It occurs during both the opening and closure of the valve.
62
Opening snaps Definition: Cause: Occurs in: Best heard in:
Definition: A high pitched sound occurs after S2. Cause: Sudden opening of the valve. Occurs in: Mitral (rarely tricuspid) stenosis. Best heard in: lower left sternal edges.
63
Systolic ejection click' Definition: Cause: Ocurrs in : Best heard in: Non-ejection systolic click. Definition: Cause: Occurs in: Best heard in:
Definition: A high pitched early systole sound. Cause: sudden opening of the valve. Ocurrs in: Congenital aortic or pulmonary stenosis!® Best heard in: aortic, pulmonary or left sternal edge area. Non-ejection systolic click. Definition: A high pitched systolic sound. Cause: prolapse of one or more redundant mitral valve leaflets. Occurs in: mitral valve prolapse and may be heard in ASD. Best heard in: mitral area (apex).
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1)Respiration: -Murmurs that arise o the right side of the heart tend to be louder during ،،،،،،،،. -Cause: increased venous return and theretore blood flow to the right side of the heart. -Expiration has the opposite effect. 2) Leaning forward : -The patient should lean forward in full expiration. - This is done to make the of the hear L closer to the chest wall. -It aids in determining the،،،،،،،،،،،،، nurmurs (which may not be detected only by wis manocavrey
inspiration mitral reguroitation